Mekanisme Gangguan Kesadaran Power Point

Embed Size (px)

Citation preview

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    1/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    2/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    3/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    4/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    5/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    6/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    7/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    8/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    9/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    10/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    11/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    12/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    13/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    14/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    15/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    16/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    17/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    18/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    19/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    20/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    21/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    22/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    23/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    24/103

    Tanda Klinik Fase Diensefalik.

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    25/103

    Tanda Klinik Fase Diensefalik.

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    26/103

    Tanda Klinik Fase Midbrain Pons Atas.

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    27/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    28/103

    Tanda Klinik Herniasi Unkus Fase Dini N. III

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    29/103

    Tanda Klinik Herniasi Unkus Fase Lanjut N.III

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    30/103

    Tanda Klinik Fase Midbrain Pons Bawa

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    31/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    32/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    33/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    34/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    35/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    36/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    37/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    38/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    39/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    40/103

      Cardiac death: Heartbeat and breathing stop

      Brain death:

    Irreversible cessation of all functions of theentire brain, including the brain stem

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    41/103

      First introduced in a 1968 report authored b aspecial committee of the Harvard !edical "chool

      #dopted in 198$, %ith modi&cations, b the'resident(s Commission for the "tud of )thical'resident(s Commission for the "tud of )thical

    'roblems in !edicine and Biomedical *esearch'roblems in !edicine and Biomedical *esearch,as a recommendation for state legislatures andcourts

       +he brain death standard %as also emploed in

    the model legislation -no%n as the .niform.niform/etermination of /eath #ct/etermination of /eath #ct, %hich has beenenacted b a large number of 0urisdictions andthe standard has been endorsed b theinuential #merican Bar #ssociation2

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    42/103

    12 Clinical or radiographic evidence of anacute catastrophic cerebral eventconsistent %3 d4 of brain death

    52 )4clusion of conditions that confoundclinical evidence i2e27metabolic

    2 Con&rmation of absence of druginto4ication or poisoning

    #lso barbiturates, !B;s

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    43/103

      Cerebral motor response to pain "upra7orbital ridge, the nail beds, trape>ius

    !otor responses ma occur spontaneouslduring apnea testing spinal ree4es

    "pinal ree4 responses occur more often inoung

    If pt had !B, then test %3 train7of7four

     

    "pinal arcs are intact?

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    44/103

     

    *ound, oval, or irregularl shaped  !idsi>e e, the

    light ree4 remains intact onl in the absence ofbrain death I@ atropine does not mar-edl aAect response 'araltics do not aAect pupillar si>e +opical administration of drugs and ee trauma

    ma inuence pupillar si>e and reactivit 're7e4isting ocular anatomic abnormalities ma

    also confound pupillar assessment in brain death

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    45/103

      culocephalic ree4 doll;s ees

      @estibulo7ocular cold caloric test

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    46/103

      culocephalic ree4 *apidl turn the head 9$D on both sides

    ormal response deviation of the ees tothe opposite side of head turning

    Brain death oculocephalic ree4es areabsent no /oll;s ees no ee movement inresponse to head movement

     

    ot Barbie, but old fashioned tpe dolls 'ainted vs2 %ooden ees in porcelain heads

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    47/103

    http://images.google.com/imgres?imgurl=http://telemedicine.orbis.org/data/1/rec_imgs/57_9.jpg&imgrefurl=http://telemedicine.orbis.org/bins/volume_page.asp?cid=1-3-4-14&h=115&w=181&sz=8&tbnid=MQ3goRdz9I4J:&tbnh=60&tbnw=94&start=4&prev=/images?q=%22oculocephalic%22&hl=en&lr=&sa=G

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    48/103

      )levate the HB $D

      Irrigate both tmpanic membranes %ithiced %ater

    bserve pt for 1 minute after each earirrigation, %ith a E minute %ait bet%eentesting of each ear

    Facial trauma involving the auditor canal and

    petrous bone can also inhibit these ree4es

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    49/103

      stagmus both ees slo% to%ard cold,fast to midline ot comatose

      Both ees tonicall deviate to%ard cold%ater Coma %ith intact brainstem

      !ovement onl of ee on side of stimulus

    Internuclear ophthalmoplegia "uggests brainstem structural lesion

      o ee movement Brainstem in0ur 3 death

    http://www.fpnotebook.com/NEU90.htmhttp://www.fpnotebook.com/NEU160.htmhttp://www.fpnotebook.com/NEU160.htmhttp://www.fpnotebook.com/NEU90.htm

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    50/103

      Corneal ree4es are absent in brain death Corneal ree4es 7 tested b using a cotton7

    tipped s%ab

    rimacing in response to pain can be testedb appling deep pressure to the nail beds,supra7orbital ridge, +!G, or s%ab in nose

    "evere facial trauma can inhibit interpretationof facial brain stem ree4es

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    51/103

      Both gag and cough ree4es are absentin patients %ith brain death ag ree4 can be evaluated b stimulating

    the posterior pharn4 %ith a tongue blade, butthe results can be dicult to evaluate in orallintubated patients

    Cough ree4 can be tested b using )++suctioning, past end of )++

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    52/103

      'aC5 levels greater than 6$ mmHg, 5$mmHg over baseline

       +echniJue: 're7o4genate %ith 1$$K o4gen several min

    #llo% baseline 'aC5 to be L

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    53/103

      )) $ minutes

      < vessel angiograph

      Cerebral blood o% perfusion scan

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    54/103

    http://images.google.com/imgres?imgurl=http://pedsccm.wustl.edu/All-Net/media/gif/neurogif/trauma/cpscanbd.jpg&imgrefurl=http://pedsccm.wustl.edu/All-Net/english/neurpage/trauma/head-5.htm&h=249&w=538&sz=16&tbnid=oBGKQkhMfkYJ:&tbnh=60&tbnw=129&start=3&prev=/images?q=%22cerebral+perfusion+scan%22&hl=en&lr=&sa=N

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    55/103

      ecessar to repeat the clinical e4amination afteran Mappropriate; observation period has passed

      Con&rmator )) unless it is determined thatthere is no blood o% to the brain

    Age 7 days to 2 monthsAge 7 days to 2 months  +%o e4aminations

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    56/103

    12 Clinical or radiographic evidence of anacute catastrophic cerebral eventconsistent %3 d4 of brain death

    52 )4clusion of conditions that confoundclinical evidence i2e27metabolic

    2 Con&rmation of absence of druginto4ication or poisoning

    #lso barbiturates, !B;s

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    57/103

      Cerebral motor response to pain "upra7orbital ridge, the nail beds, trape>ius

    !otor responses ma occur spontaneouslduring apnea testing spinal ree4es

    "pinal ree4 responses occur more often inoung

    If pt had !B, then test %3 train7of7four

     

    "pinal arcs are intact?

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    58/103

     

    *ound, oval, or irregularl shaped  !idsi>e e, the

    light ree4 remains intact onl in the absence ofbrain death I@ atropine does not mar-edl aAect response 'araltics do not aAect pupillar si>e +opical administration of drugs and ee trauma

    ma inuence pupillar si>e and reactivit 're7e4isting ocular anatomic abnormalities ma

    also confound pupillar assessment in brain death

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    59/103

      culocephalic ree4 doll;s ees

      @estibulo7ocular cold caloric test

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    60/103

      culocephalic ree4 *apidl turn the head 9$D on both sides

    ormal response deviation of the ees tothe opposite side of head turning

    Brain death oculocephalic ree4es areabsent no /oll;s ees no ee movement inresponse to head movement

     

    ot Barbie, but old fashioned tpe dolls 'ainted vs2 %ooden ees in porcelain heads

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    61/103

    http://images.google.com/imgres?imgurl=http://telemedicine.orbis.org/data/1/rec_imgs/57_9.jpg&imgrefurl=http://telemedicine.orbis.org/bins/volume_page.asp?cid=1-3-4-14&h=115&w=181&sz=8&tbnid=MQ3goRdz9I4J:&tbnh=60&tbnw=94&start=4&prev=/images?q=%22oculocephalic%22&hl=en&lr=&sa=G

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    62/103

      )levate the HB $D

      Irrigate both tmpanic membranes %ithiced %ater

    bserve pt for 1 minute after each earirrigation, %ith a E minute %ait bet%eentesting of each ear

    Facial trauma involving the auditor canal andpetrous bone can also inhibit these ree4es

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    63/103

      stagmus both ees slo% to%ard cold,fast to midline ot comatose

      Both ees tonicall deviate to%ard cold%ater Coma %ith intact brainstem

      !ovement onl of ee on side of stimulus Internuclear ophthalmoplegia "uggests brainstem structural lesion

      o ee movement Brainstem in0ur 3 death

    http://www.fpnotebook.com/NEU90.htmhttp://www.fpnotebook.com/NEU160.htmhttp://www.fpnotebook.com/NEU160.htmhttp://www.fpnotebook.com/NEU90.htm

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    64/103

      Corneal ree4es are absent in brain death Corneal ree4es 7 tested b using a cotton7

    tipped s%ab

    rimacing in response to pain can be testedb appling deep pressure to the nail beds,supra7orbital ridge, +!G, or s%ab in nose

    "evere facial trauma can inhibit interpretationof facial brain stem ree4es

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    65/103

      Both gag and cough ree4es are absentin patients %ith brain death ag ree4 can be evaluated b stimulating

    the posterior pharn4 %ith a tongue blade, butthe results can be dicult to evaluate in orallintubated patients

    Cough ree4 can be tested b using )++suctioning, past end of )++

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    66/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    67/103

      )) $ minutes

      < vessel angiograph

      Cerebral blood o% perfusion scan

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    68/103

      #bsent Cerebral Function

      #bsent Brainstem Function

     

    #pnea

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    69/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    70/103

    Cerebral Corte4

    Brain "tem

    *eticular#ctivating"stem

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    71/103

      Cognition

      @oluntar

    !ovement  "ensation

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    72/103

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    73/103

     Midbrain

    Cranial Nerve III

     pupillary function

     eye movement

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    74/103

    Pons

      Cranial Nerves IV, V, VI

     conjugate eye movement

     corneal reflex

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    75/103

     Medulla

      Cranial Nerves IX, X

     Pharyngeal (Gag) eflex

     !racheal (Cough) eflex

      espiration

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    76/103

      *eceives multiple

    sensor inputs

      !ediates

    %a-efulness

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    77/103

    NormalCere"ral #noxia

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    78/103

    Normal Cere"ral $emorrhage

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    79/103

    Normal%u"arachnoi& $emorrhage

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    80/103

    Normal !rauma

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    81/103

    Normal'eningitis

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    82/103

     Neuronal Injury

    ecrease& Intracranial

    loo& *lo+

    Neuronal %+elling

    Increase& Intracranial

    Pressure

    ICP'#P is

    incompati"le

    +ith life

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    83/103

      'ersistent @egetative "tate

      Noc-ed7in "ndrome

     

    !inimall *esponsive "tate

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    84/103

      ormal "leep7Oa-e Ccles

      o *esponse to )nvironmental "timuli

      /iAuse Brain In0ur %ith 'reservation

    of Brain "tem Function

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    85/103

    !entral Pontine Infar"t

     Complete Paralysis

     Preserve& Consciousness

     Preserve& -ye 'ovement

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    86/103

      /iAuse or !ulti7Focal Brain In0ur

      'reserved Brain "tem Function

      @ariable Interaction %ith

    )nvironmental "timuli

    %tatic -ncephalopathy

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    87/103

    Clinical 'rereJuisites:

    Pno%n Irreversible Cause

    )4clusion of 'otentiall *eversible Conditions /rug Into4ication or 'oisoning

    )lectrolte or #cid7Base Imbalance )ndocrine /isturbances

    Core Bod temperature = 5D C

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    88/103

      Coma

      #bsent Brain "tem *ee4es

      #pnea

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    89/103

    o *esponse to o4ious "timuli

    ail Bed 'ressure

    "ternal *ub

    "upra7rbital *idge 'ressure

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    90/103

      'upillar *ee4

      )e !ovements

      Facial "ensation and !otor *esponse

      'harngeal ag *ee4

       +racheal Cough *ee4

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    91/103

    Pupils &ilate& +ith no constriction to "right light

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    92/103

    .cculo/Cephalic esponse

    0oll1s -yes 'aneuver2

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    93/103

    .culo/Vesti"ular esponse

    0Col& Caloric !esting2

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    94/103

      Corneal *ee4

       Ga% *ee4

      rimace to "upraorbital or

      +emporo7!andibular 'ressure

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    95/103

    'rereJuisites

    Core Bod +emperature = 5D C

    "stolic Blood 'ressure 9$ mmHg

    ormal )lectroltes

    ormal 'C5

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    96/103

    12 're74genation 1$$K 4gen via +racheal Cannula

    '5 5$$ mm Hg

    52 !onitor 'C5 and '5 %ith pulse o4imetr

    2 /isconnect @entilator

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    97/103

      Facial +rauma

      'upillar #bnormalities

      C" "edatives or euromuscularBloc-ers

      Hepatic Failure

      'ulmonar /isease

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    98/103

    ##

    $

    Normal -lectrocere"ral %ilence

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    99/103

    Cerebral#ngiograph

    Normal No Intracranial *lo+

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    100/103

     +echnetium799 Isotope Brain "can 

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    101/103

    !*7 #ngiograph

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    102/103

    !ranscranial

    3ltrasonography

  • 8/18/2019 Mekanisme Gangguan Kesadaran Power Point

    103/103

    "omatosensor )vo-ed 'otentials