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KEHAMILAN KEMBARKEHAMILAN KEMBAR= MULTIPLE PREGNANCIES == MULTIPLE PREGNANCIES =
(GEMELLI)(GEMELLI)
Pendahuluan
• Two for the price of one” atau “instant family”• High Complication Risk→Morbiditas &
mortalitas ↑ 50% 32-38 minggu, 10% dibawahnya• Pe↑ Malpresentasi:
- kedua janin sungsang 41%- Janin kembar I sungsang 17%- Locked twins (jarang)
• Persalinan operatif & resiko persalinan preterm ↑
Definisi & Klasifikasi
Kehamilan 2 janin atau lebihKembar dizigotik (66%) Binovular-fraternal twins1. fertilisasi 2 ovum oleh 2 sperma2. Dikorionik: Amnion terpisahKembar monozigotik (33%) Mono ovular-identical twins - Pembelahan 1 ovum, fertilisasi oleh sperma sperma yang sama- Pembelahan <72 jam: Dikorionik diamnotik (96%)- Pembelahan 4-8 hari: Monokorionik diamniotik (4%)
Mono ovular-identical twins, diamniotik monokorionik
- Pembelahan 8-13 hari: Monokorionik, Monoamniotik
- Pembelahan >13 hari: Conjoined twins
Fetus Papyraceous
- Salah satu janin kembar tidak berkembang
- Tak berbentuk, mengkerut & rata
Perbandingan Mono/Dizigotik 1:2
Faktor resiko untuk kembar dizigotik:
- ♀ tua
- Multiparitas
- Riwayat keluarga kehamilan kembar dizigotik
Fetus Papyraceous, salah satu fetus yang tidak berkembang
Insiden
1% dari kehamilan, 2/3 dizigot & 1/3 monozigot
Etnik (1:50 Afrika, 1:80 Causasia, 1:50 Asia)
Usia (2% > 35 thn)
Paritas (2% setelah kehamilan ke-4)
Metode konsepsi (20% induksi ovulasi)
Riwayat keluarga
Insidensi menurut hukum Hellin adalah 1 dalam 80n-1 kehamilan
Etiologi
• Bangsa, hereditas, umur & paritas→ binovular fraternal-twins
• Obat klomid & gonadotropin hormon→ dizigotik
• Fertilisasi in vitro & transfer embrio (IVF&ET)
Patofisiologi
Fertilisasi ovum&sperma di tuba falopii
Ovum yang telah dibuahi turun uterus
nidasi dan Pertumbuhan fetus
Selama proses ini kembar dapat terbentuk
Kehamilan berasal dari satu telur terjadi :Akibat adanya kerja faktor penghambat (inhibiting factor) pada masa awal pertumbuhan embrio intrauterin, mempengaruhi segmentasi selanjutnya pada berbagai tingkatan.
Tipe Presentasi
• Janin kembar I presentasi vertex 75%
• Kedua janin presentasi vertex 45%
• Salah satu janin vertex, lainnya bokong 37%
• Kedua janin presentasi bokong 10%
tipe-tipe presentasi
Distribusi dari letak dan posisi janin kembar (dalam %) antara lain:
KEMBARDUA
KEMBAR PERTAMA
Kepala Sungsang Lintang
Kepala 39 13 0,6
Sungsang 26 9 0,6
Lintang 8 4 0,6
Early Diagnosis
Anamnesa Ultrasonografi
Gemelli
Pemeriksaan klinis Radiologi
Diagnosis Awal Twins
DIZYGOTICDIZYGOTIC MONOZYGOTICMONOZYGOTIC
Ultrasonografi kehamilan kembar pada usia kehamilan 38-40 hari
Diagnosa dini gagal → - P↑ PJT & persalinan prematur - P↑ mortalitas & morbiditas perintal - P↑ komplikasi
Berdasarkan observasi
36-37 mgg +++ Amnion <<<
P’tbhan janin 24-35 mgg plasenta matang++
Kematian intra uterin ↑ 37-38 mgg
• Differential Diagnosis
Kehamilan lewat waktu
Polihidramnion
Tumor fibroid uterus
Kista
Mola hidatiforma
Anemia Atonia uteri Hidramnion
PPH Abortus Komplikasi maternal
Retensio plasenta Partus prematur
Inersia uteri Pre-eklampsia
Solusioplasenta Malpresentasi Plasenta Previa
KPD
Komplikasi fetal
Prematuritas
BBLR
Insufisiensi plasenta
Kelainan kongenital
Prolapsus tali pusat
Komplikasi Intrapartum
Plasenta Insufisiensi plasenta
kebutuhan nutrisi>> Polihidramnion
Kondisi lain
Prolapsus tali pusat Malpresentasi
LockedPPH Komplikasi Peripartum Twins
Solusio Plasenta Transfusion Syndrom
Penatalaksanaan
A. Tindakan umum- Diet & Pola makan yang baik- Besi & Asam folat- Aktivitas << & aktivitas +++
B. Pem. Klinis setiap 2mgg setelah 24 mgg- keadaan servik setelah 24 mgg- pengetahuan kehamilan preterm- pergerakan bayi setelah 32 mgg
C. USG setiap 4-6 mgg setelah dignosis- kemungkinan plasenta previa- kemungkinan gangguan pertumbuhan janin- presentasi janin
D. Nonstress test setelah setelah 32mgg- keadaan janin- penekanan taki pusat
E. Konsultasi perinatologi
Kembar discordant: janin resepient lebih besar dari pada janin donor abnormalitas arteriovenous tampak pada permukaan plasenta, darah arteri kaya O2 donor bercampur dengan darah resepient
PENANGANAN PERSALINAN
• KALAU ANAK I SUNGSANG ATAU LINTANG SEBAIKNYA S.CESAR.
• KALAU ANAK I P.KEPALA DIUPAYAKAN DENGAN P/ VAGINAL ANAK KE DUA DENGAN V.EKSTRAKSI.
• SELAMA DJJ NORMAL TIDAK ADA ALASAN UNTUK MEMPERCAPAT KELAHIRAN ANAK KEDUA
• PENGAWASAN YANG KETAT MENENTUKAN OUTCOME PERSALINAN
anak pertama lintang atau sungsang dan anak kedua memanjang (terjadi posisi saling mengunci interlocking)
Panduan penanganan persalinan spontan pada kehamilan kembar
Janin pertamaSiapkan peralatan resusitasi & perawatan bayiPasang infus & cairan intravenaPantau keadaan janin, djjPeriksa presentasi janin- vertex → PSP, monitor persalinan- bokong → indikasi SC- lintang → SC
Tinggalkan klem pada ujung maternal tali pusat
• Janin kedua atau berikiutnya
Segera setelah bayi pertama lahir:
- Palpasi abdomen → letak janin
- lakukan versi luar
- Periksa djj
• Periksa dalam
- Presentasi janin kedua
- keutuhan selaput ketuban
- Prolapsus tali pusat
Monoamniotic twins mortality
• 2 to 5% loss every 2 weeks from 15 to 32 weeks
• 9% at 33 wks 29% at 36-38 wks
• 95% cord entanglement (prenatal diagnosis 28%)
Comparison of rates of complications in singleton and multiple gestations
Complications Rate for twins (increase)
Chorioamnionitis 4-foldPremature rupture of membranes 4-foldFetal asphyxia 5-foldTwin-twin transfusion 1 of 9 monoamniotic twinsCongenital malformations 3-foldHydramnios 1 of 12 twinsAbruptio placentae 2-foldPlacenta previa 2-foldCompression of cord 2-foldBirth injury 10-foldPrematurity 10-foldUmbilical cord knots 2-fold
Maternal morbidity and obstetric complications of quadruplet pregnancy (No. 22)
VARIABLE INCIDENCE (%)
Antepartum hospitalization 100Hyperemesis gravidarum 9.4Hyperemesis gravidarum, total parenteral nutrition required 3.1Gestational diabetes mellitus, A1 18.8Gestational diabetes mellitus, A2 3.1Anemia (Hct < 30%), no antepartum transfusion required 25.0Anemia (Hct < 30%), antepartum transfusion required 15.6Antepartum bleeding 3.1Placenta previa 0.0Preeclampsia 71.9HELLP syndrome 2.5PPROM 18.8PTL 100Twin-twin transfusion syndrome 3.1Chorioamnionitis 6.3
I. Psychological Support and Clinical Counseling
• All parents should be aware that pathologies such as fetal growth retardation, congenital anomalies, abnormal placentation, abruptio placentae, fetal malpresentation and preterm delivery, occur more commonly in multiple than in singleton pregnancy
• These aspects result in higher maternal and perinatal mortality and morbidity.
• Antenatal complications are three to five times higher in multiple pregnancy than in singleton pregnancy.
• From the first trimester onwards is required to help parents to cope with possible negative outcome and also with the socio-economic problems related to multiple birth.
The most important:The most important:
EARLY DIAGNOSISEARLY DIAGNOSIS WHY?WHY?
MULTIPLE MULTIPLE PREGNANCYPREGNANCY
HIGH-RISK HIGH-RISK PREGNANCYPREGNANCY
• COMPLICATIONS DURING PREGNANCYCOMPLICATIONS DURING PREGNANCY• SPECIFIC MALFORMATION SEQUENCESSPECIFIC MALFORMATION SEQUENCES• HIGHER PERINATAL MORBIDITIY AND MORTALITYHIGHER PERINATAL MORBIDITIY AND MORTALITY• INTRAPARTAL COMPLICATIONSINTRAPARTAL COMPLICATIONS
==
DIAGNOSIS OF MULTIFETAL PREGNANCY:SIMULTANEOUS VISUALIZATIONSIMULTANEOUS VISUALIZATION
• two or more embryostwo or more embryos
•or or corresponding bodycorresponding body partsparts of of twotwo or more fetusesor more fetuses
A firm diagnosis ofA firm diagnosis ofthe number of embryosthe number of embryos
after 7th weekafter 7th week ! !
EARLY DIAGNOSIS OF TWINSEARLY DIAGNOSIS OF TWINS
EMBRYOSEMBRYOS AND AMNIOTICAND AMNIOTICMEMBRANESMEMBRANES
MONOCHORIONICMONOCHORIONICMONOAMNIOTICMONOAMNIOTICTWINSTWINS
HIGH-ORDER MULTIPLE PREGNANCYHIGH-ORDER MULTIPLE PREGNANCYPregnancy with three or more fetuses
three amnioticthree amniotic
three chorionicthree chorionic
2D multiplanar imaging2D multiplanar imaging
3D3D reconstructionreconstruction
• volume scanning• volume rendering• spatial reconstruction • plastic imaging
TRIPLETSTRIPLETS
FRONTFRONT BACKBACK
QUADRUPLETS
HIGH ORDER PREHIGH ORDER PREGGNANCYNANCY
HIGH ORDER PREHIGH ORDER PREGGNANCYNANCY
HIGH ORDER PREHIGH ORDER PREGGNANCYNANCYSEPTUPLETSSEPTUPLETS
12 EMBRYOS12 EMBRYOS
HIGH ORDER PREHIGH ORDER PREGGNANCYNANCY
II. Correct Diagnosis andCharacterization of Chorionicity
• Multiple gestation should be suspected when the uterus is larger than predicted by menstrual history.
• Approximately one fifth of multiple gestations are monochorionic and four fifths are dichorionic.
• Type of placentation and chorionicity is helpful in the following three clinical situations: 1) The differentiation of twin to twin transfusion syndrome (TTS) from a twin gestation in which one fetus shows growth retardation; 2) the management of twins with congenital malformations, in which selective feticide may be considered as an option if the gestation is dichorionic and 3) the management of single fetal death in a multiple gestation.
• The thickness of dividing membrane is in 85% of monochorionic twins ~ 2 mm, in DC/DA the membrane is ~ 4 mm
• The “lambda” sign is an indicator of dichorionic pregnancy
II. Correct Diagnosis andCharacterization of Chorionicity
• The following criteria must be fulfilled to diagnose monoamniotic twins:
1. no dividing amniotic membrane is present2. only one placenta is seen3. both fetuses are of the same sex 4. the fetuses must have adequate amniotic fluid
surrounding them5. both fetuses must move freely within the
uterine cavity.
ACCURATE PRENATAL DIAGNOSISACCURATE PRENATAL DIAGNOSISOF CHORIONICITY IS OF PREDOMINANTOF CHORIONICITY IS OF PREDOMINANTIMPORTANCE FOR THE CLINICAL MANAGEMENT IMPORTANCE FOR THE CLINICAL MANAGEMENT OF OF MULTIPLE MULTIPLE PREGNANCIES PREGNANCIES
EARLY DIAGNOSIS OF CHORIONICITYEARLY DIAGNOSIS OF CHORIONICITY
NUMBER OF NUMBER OF GESTATIONAL GESTATIONAL SACSSACS
1st TRIMESTER1st TRIMESTER
ALARM !MONOCHORIONICMONOCHORIONIC
AND / ORAND / ORMONOAMNIOTIC TWINSMONOAMNIOTIC TWINS
FETAL FETAL COMPLICATIONS COMPLICATIONS
EARLY DIAGNOSIS OF AMNIONICITYEARLY DIAGNOSIS OF AMNIONICITYWhy is it important?
TWO SEPARATED PLACENTAS
PLACENTA 2
PLACENTA 1
DETERMINATION OF THE CHORIONICITYIN SECOND TRIMESTER
Sonographic counting of separated placentas is Sonographic counting of separated placentas is an accurate method of determining the an accurate method of determining the
chorionicity in the second trimester chorionicity in the second trimester
MONOCHORIONICBIAMNIOTIC TWINS
BICHORIONICBIAMNIOTIC TWINS
LAMBDA SIGN
BICHORIONIC BIAMNIOTIC TWINSBICHORIONIC BIAMNIOTIC TWINS
BIAMNIOTICBICHORIONIC
TWINS
MONOAMNIOTIC MONOCHORIONIC TWINSMONOAMNIOTIC MONOCHORIONIC TWINS
THE Y-SHAPEDY-SHAPED JUNCTION
Y-SIGNTRICHORIONICTRICHORIONICTRIAMNIOTICTRIAMNIOTIC
TRIPLETSTRIPLETS
““MERCEDES” SIGNMERCEDES” SIGN
III. Close Evaluation of Fetal Anatomy
Fetal Malformations and Prenatal Genetic Diagnosis
•The incidence of malformation in monozygotic twin pregnancies is twice that in dizygotics.
•Chromosomal anomalies are no more common in twins than singletons
•Anomalies not unique to twins but believed to be increased in frequency because of mechanical factors are positional defects (such as clubfoot and congenital dislocation of the hip) due to intrauterine crowding.
•Additional anomalies due to vascular consequences of fetal death are congenital skin defects, microcephaly, hydrancephaly, porencephaly, multicystic encephalomalacia, hydrocephalus, intestinal atresia and limb amputation.
III. Close Evaluation of Fetal Anatomy
Fetoplacental Markers in Twin Pregnancies Affected by Down Syndrome
• Around one-third of twin pregnancies are monozygous and their rate of Down syndrome is relatively independent of race and maternal age.
• Dizygous twins are more common in older mothers and as they arise from separate fertilisation of two simultaneously shed ova there is double the age-related risk than for a singleton pregnancy that either twin will have Down syndrome
CONJOINED (SIAMESE) TWINSCONJOINED (SIAMESE) TWINS INCIDENCE 1: 50 000 BIRTHSINCIDENCE 1: 50 000 BIRTHS
ULTRASOUND CRITERIA FOR DIAGNOSIS:ULTRASOUND CRITERIA FOR DIAGNOSIS:
1) LACK OF SEPARATE VISUALISATION OF FETUSES IN SPECIFIC ANATOMICAL REGIONS
2) FIXED POSITION OF THE TWIN TOWARD EACH OTHER
3) MISSING SEPARATING MEMBRANE
SYMMETRICAL SYMMETRICAL COMPLETE FORMCOMPLETE FORM Two fetuses shareTwo fetuses share a certain amount of tissuea certain amount of tissue
Surgical separation is Surgical separation is possible in general.possible in general.
PATTERNS OF PHYSICAL JOINING
PATTERNS OF PHYSICAL JOINING
SYMMETRICAL SYMMETRICAL INCOMPLETE FORMINCOMPLETE FORM
Surgical separation Surgical separation is usually impossibleis usually impossible
Conjoined twins: Conjoined twins:
subtotal fusionsubtotal fusionwith partial separation with partial separation of fetal headsof fetal heads
EARLY DIAGNOSIS OF CONJOINED TWINS
CONJOINED TWINS
THORACO-THORACO- OMPHALOPHAGUSOMPHALOPHAGUS
lack of separate visualisation of fetuses lack of separate visualisation of fetuses in thoraco-abdominal regionin thoraco-abdominal region
COLOR DOPPLERCOLOR DOPPLERSINGLE SHARED UMBILICAL SINGLE SHARED UMBILICAL CORDCORD
FIVE - VESSEL CORDFIVE - VESSEL CORD
THORACO-OMPHALOPHAGUS
VI. Avoidance of Most Frequent Complications
Complications of multiple pregnancies comprise:• Abortion,• Vanishing twin syndrome• Malformation• Vasa previa• Growth discrepancy• Intra uterine growth restriction (IUGR)• Polyhydramnios• Preeclampsia• Preterm-premature rupture of membranes (P-PROM)• Preterm delivery• Gestational diabetes• Intrauterine fetal death.
VANISHING TWINVANISHING TWIN• in in 20%20% of twin of twinss
• single fetal demisesingle fetal demise• high-risk surviving twinhigh-risk surviving twin• intintrarauterine hematomasuterine hematomas• better prognosis in dichorionicbetter prognosis in dichorionic• thromboplastine embolisationthromboplastine embolisation
VANISHING TWIN
SUBCHORIONIC HAEMATOMA
differentia
l diagnosis
differentia
l diagnosis
MONOCHORONIC / BIAMNIOTICMONOCHORONIC / BIAMNIOTIC::““TWIN TO TWIN” TWIN TO TWIN” TRANSFUSION SYNDROMETRANSFUSION SYNDROME
MONOAMNIOTIC:MONOAMNIOTIC:UMBILICAL CORD ENTAGLEMENTUMBILICAL CORD ENTAGLEMENT
ACARDIAC TWIN - ACARDIAC TWIN - TRAPTRAP SEQUENCE SEQUENCE CONJOINED TWINSCONJOINED TWINS
TTTSTTTS
•5% - 20% monochorionic twins5% - 20% monochorionic twins•arterioarterio venous venous anastomosesanastomoses•discordant growthdiscordant growth
DONOR DONOR RECIPIENTRECIPIENTOLIGOHYDRAMNIOS POLYHYDRAMNIOSOLIGOHYDRAMNIOS POLYHYDRAMNIOS
IUGR IUGR MACROSOMIA, HYDROPS MACROSOMIA, HYDROPS
MICROCARDIA CARDIOMEGALIAMICROCARDIA CARDIOMEGALIA
ANEMIA POLYCYTHAEMIAANEMIA POLYCYTHAEMIA
fetal loss 80%fetal loss 80%
TWIN TO TWIN TRANSFUSION SYNDROMETWIN TO TWIN TRANSFUSION SYNDROME
TWIN TO TWIN TRANSFUSION SYNDROMETWIN TO TWIN TRANSFUSION SYNDROME
RECIPIENT:RECIPIENT: Fetal hydrops
SCALP EDEMASCALP EDEMA
ASCITESASCITES
collapsed amniotic collapsed amniotic membranemembrane
DONOR:Stuck twin
TWIN TO TWIN TRANSFUSION SYNDROME
fixed twinfixed twinanhydramniosanhydramnios
POLYHYDRAMNIOS OF RECIPIENT TWIN
TWIN TO TWIN TRANSFUSION SYNDROME
TWIN TO TWIN TRANSFUSION SYNDROME
PULSATIONS WITHPULSATIONS WITHREVERSE- FLOW AT REVERSE- FLOW AT THE END OF DIASTOLETHE END OF DIASTOLE
UMBILICAL VEIN UMBILICAL VEIN SONOGRAM SONOGRAM IN RECIPIENT TWININ RECIPIENT TWIN
DUCTUS VENOSUSDUCTUS VENOSUSSONOGRAMSONOGRAM
IN RECIPIENT TWININ RECIPIENT TWIN
REVERSAL OF FLOWREVERSAL OF FLOWDURING ATRIALDURING ATRIALCONTRACTIONCONTRACTION
TWIN TO TWIN TRANSFUSION SYNDROME
Recipient : Recipient : venous return patternvenous return pattern
PlethoricPlethoric RECIPIENTRECIPIENT
AnaemicAnaemic DONORDONOR
TWIN TO TWIN TRANSFUSION SYNDROMETWIN TO TWIN TRANSFUSION SYNDROME
Weight difference > 25%Weight difference > 25%Haemoglobin difference >5%Haemoglobin difference >5%
VASCULAR ANASTOMOSES VASCULAR ANASTOMOSES IN A TWIN PLACENTA:IN A TWIN PLACENTA:
ARTERIOARTERIO VENOUSVENOUSARTERIO ARTERIO ARTERIOUSARTERIOUS VENO VENO VENOUSVENOUS
superficialsuperficial
deepdeep
VISUALIZATION WITHVISUALIZATION WITHPOWER ANGIO MODEPOWER ANGIO MODE
SURFACE ANASTOMOSESSURFACE ANASTOMOSES
TWIN REVERSED TWIN REVERSED ARTERIAL PERFUSIONARTERIAL PERFUSION
(TRAP)(TRAP)
IN MONOCHORIONIC TWINS ONE TWIN ( PUMP-TWIN ) ACTIVELY PERFUSES
THE SECOND TWIN ( PERFUSED TWIN ) VIA LARGE A -A AND/OR V - V ANASTOMOSES
1% of monozygotic1% of monozygotic twins are affected twins are affected
Incidence 1 : 35 000 birthsIncidence 1 : 35 000 births
ARTERIAL SUPPLY INTO PLACENTA BY THE PUMP TWIN IS ABLE TO OVERCOME THE BLOOD PRESSURE OF THECO-TWIN SO AS TO PERFUSE THAT TWINBY REVERSED FLOW (TOWARD CO-TWIN)IN THE UMBLICAL ARTERIES OF THE CO-TWIN
PATHOGENESIS
PERFUSED TWIN PERFUSED TWIN ACARDIUSACARDIUS
NORMALNORMAL( PUMP TWIN )( PUMP TWIN )
TRAP BLOOD FLOWS FROM AN BLOOD FLOWS FROM AN UMBILICAL ARTERY OF THE UMBILICAL ARTERY OF THE PUMP TWIN IN PUMP TWIN IN REVERSE DIRECTIONREVERSE DIRECTION VIA VIA ARTERIO - ARTERIAL ARTERIO - ARTERIAL ANASTOMOSES INTO ANASTOMOSES INTO UMBILICAL ARTERY OF THE UMBILICAL ARTERY OF THE PERFUSED TWIN.PERFUSED TWIN.
THE UMBILICAL VEIN OF THE PARASITIC FETUS RETURNS THE BLOOD INTO THE PLACENTA ANDBACK TO PUMP TWIN
REVERSE FLOW NORMAL FLOWNORMAL FLOW
EARLY REVERSE OF CIRCULATIONEARLY REVERSE OF CIRCULATION
REVERSE PASSIVE PERFUSION OF TWINREVERSE PASSIVE PERFUSION OF TWIN
PERFUSION IN OPPOSITE DIRECTION ANDPERFUSION IN OPPOSITE DIRECTION AND
PERFUSION WITH DEOXIGENATED BLOODPERFUSION WITH DEOXIGENATED BLOOD
INDUCTION OF DEVELOPMENTAL DISORDERSINDUCTION OF DEVELOPMENTAL DISORDERS
REDUCTION ANOMALIES ( EXTREMITIES )REDUCTION ANOMALIES ( EXTREMITIES )
DEVELOPMENTAL ATROPHIES ( HEART AND BRAIN ) DEVELOPMENTAL ATROPHIES ( HEART AND BRAIN )
PATHOGENESIS OF FETAL DYSMORPHIA:PATHOGENESIS OF FETAL DYSMORPHIA:
Ultrasound finding = early ultrasound detection the most bizzarre fetal malformations
PUMP - TWIN
normalmorphology
normaldirection ofblood flow
PERFUSED TWIN
acardius
reduction anomalies of head and extremities
reversed blood flowreversed blood flow
TWINS MC / MA, 15 TWINS MC / MA, 15 wkswks
REVERSEDREVERSEDPERFUSIONPERFUSION
COLORCOLORDOPPLERDOPPLER
ULTRASONIC CRITERIA FOR ACARDIUSULTRASONIC CRITERIA FOR ACARDIUS
An amorphous mass with An amorphous mass with its own umbilical its own umbilical cord in monochorionic- cord in monochorionic- monoamnioticmonoamniotictwin pregnancytwin pregnancy
ACARDIAC - ACEPHALIC
This acardiac twin consists mainly of lower extremities
No heart and brain
No trunkNo trunkand headand head
COMPLICATION SPECIFIC FORMONOAMNIOTIC MONOCHORIONICTWINS
CORD ENTAGLEMENTCORD ENTAGLEMENT
THE CLOSE INSERTION OF THE UMBILICAL THE CLOSE INSERTION OF THE UMBILICAL CORDS INTO PLACENTA IS ASSOCIATED WITH:CORDS INTO PLACENTA IS ASSOCIATED WITH:LARGE-CALIBER ANASTOMOSES LARGE-CALIBER ANASTOMOSES AND AND
HIGH PREDISPOSITION FOR ENTANGLEMENTHIGH PREDISPOSITION FOR ENTANGLEMENT
MONOAMNIOTIC MONOAMNIOTIC TWINNINGTWINNING
CORD ENTANGLEMENTCORD ENTANGLEMENT
POWER DOPPLERPOWER DOPPLERCOLOR DOPPLERCOLOR DOPPLER
CORD ENTANGLEMENTCORD ENTANGLEMENT
Multiple gestations present a significantMultiple gestations present a significantdedecrease in fetal growth crease in fetal growth which is which is
in direct relationship to the number in direct relationship to the number of fetusesof fetuses in in high order pregnancieshigh order pregnancies
TWIN-TO-TWIN TRANSFUSIONTWIN-TO-TWIN TRANSFUSION
should be should be considered when growth discordancy considered when growth discordancy is diagnosed in is diagnosed in monochorionic gestationsmonochorionic gestations
• COMPLEX BODY MOVEMENTS• HICCUPS• HAND-FACE CONTACTS• MOUTH OPENING• SWALLOWING• BREATHING MOVEMENTS• HEAD MOVEMENTS• EXTREMITY MOVEMENTS• JUMPING• TWISTING• STRETCHING• YAWNING
SPONTANEOUS MOTORIC ACTIVITYSPONTANEOUS MOTORIC ACTIVITY
The Ten Commandmentsin Multiple Pregnancies
I. Psychological Support and Clinical CounselingII. Correct Diagnosis and Characterization of
ChorionicityIII. Close Evaluation of Fetal AnatomyIV. Management at Referral CentersV. Individualization of CareVI. Avoidance of Most Frequent ComplicationsVII Consideration of Some Specific PathologiesVIII. Close Monitoring of FetusesIX. Planning of Time and Mode of DeliveryX. Monitoring of the Mother During Postpartum
Ultrasound assessment of multiple pregnancy:Ultrasound assessment of multiple pregnancy:
1. EARLY DIAGNOSIS OF MULTIPLE PREGNANCY1. EARLY DIAGNOSIS OF MULTIPLE PREGNANCY
2. DIAGNOSIS OF CHORIONICITY AND AMNIONICITY2. DIAGNOSIS OF CHORIONICITY AND AMNIONICITY
3. COMPLICATIONS IN MONOCHORIONIC TWINS3. COMPLICATIONS IN MONOCHORIONIC TWINS
4. FETAL CONGENITAL ANOMALIES 4. FETAL CONGENITAL ANOMALIES
5. APPROPRIATE VERSUS DISCORDANT GROWTH5. APPROPRIATE VERSUS DISCORDANT GROWTH
6. COLOR-DOPPLER OF MULTIFETAL PREGNANCY6. COLOR-DOPPLER OF MULTIFETAL PREGNANCY
7. PREDICTION OF PRETERM DELIVERY7. PREDICTION OF PRETERM DELIVERY
8. INTRAPARTUM ULTRASONOGRAPHY8. INTRAPARTUM ULTRASONOGRAPHY