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Valvular Heart Disease/Myopathy/Aneur ysm

Valvular Heart Disease/Myopathy/Aneurysm. Valvular Heart Disease HeartPoint: HeartPoint Gallery mmon/articles.cfm?ARTID=187

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Page 1: Valvular Heart Disease/Myopathy/Aneurysm. Valvular Heart Disease HeartPoint: HeartPoint Gallery  mmon/articles.cfm?ARTID=187

Valvular Heart Disease/Myopathy/Aneury

sm

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Valvular Heart Disease

HeartPoint: HeartPoint Gallery

http://www.heartcenteronline.com/myheartdr/common/articles.cfm?ARTID=187

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Risk FactorsRheumatic Heart Disease MICongenital Heart DefectsAgingCHF

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PathophysiologyStenosis- narrowed valve, increases afterloadRegurgitation or insufficiency- increases preload. The heart has to pump same blood**Blood volume and pressures are reduced in front of the affected valve and increased behind the affected valve.This results in heart failureAll valvular diseases have a characteristic murmur murmurs

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Mitral Valve Stenosis

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Mitral StenosisDec. flow into LVLA hypertrophyPulmonary pressures increasePulmonary hypertensionDec. CO* early symptom is DOELater get symptoms of R heart failureA fib is common- anticoagulantsUsually secondary to rheumatic fever

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Mitral RegurgitationRegurg of blood into LA during systoleLA dilation and hypertrophyPulmonary congestionRV failureLV dilation and hypertrophy-to accommodate inc. preload and dec CO

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Mitral Valve ProlapseA type of mitral insufficiencyUsually asymptomaticMay get atypical chest pain related to fatigueTachydysrhythmias may developRisk for endocarditis is increased and need prophylactic antibiotics

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A&P 1 Heart part 1

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Aortic StenosisIncrease in afterloadReduced COLV hypertrophyIncomplete emptying of LAPulmonary congestionRV strain

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Symptoms

Syncope

Angina

Dyspnea

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Aortic StenosisMay be asymptomatic for many years due to compensationDOE, angina, and exertional syncope are classic symptomsLater get signs of R heart failureUntreated-poor prognosis- 10-20%sudden cardiac death

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Aortic RegurgitationGet increased preoad- 60% of SV can be regurgitatedCharacteristic water hammer pulseRegurgitation of blood into the LVLV dilation and hypertrophyDec. CO

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Water Hammer pulse

Pulse, water hammer: A jerky pulse that is full and then collapses because of aortic insufficiency (when blood ejected into the aorta regurgitates back through the aortic valve into the left ventricle ).

Also called a Corrigan pulse or a cannonball, collapsing, pistol-shot, or trip-hammer pulse.

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Tricuspid and Pulmonic Valve Disorders

Result in R side heart failure

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Diagnostic TestsEcho- assess valve motion and chamber sizeCXREKGCardiac cath- get pressures

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MedicationsLike Heart Failure

ACE, DigDiureticsVasodilatorsBeta blockersAnticoagulants*Prophylactic antibiotics

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Medical/ Surgical Treatment

Percutaneous balloon valvuloplastySurgery

Open commissurotomy- open stenotic valvesAnnuloplasty- can be used for bothValve Replacement

Mechanical-need anticoagulantBiologic-only last about 15 yearsRoss Procedure

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Ross Procedure

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Nursing DiagnosesDecreased Cardiac OutputActivity IntoleranceRisk for InfectionIneffective Protection

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Case study

http://edcenter.med.cornell.edu/Pathophysiology_Cases/96-97_Pathophysiology_Cases/96-97Case_04.html

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CardiomyopathyPrimary-idiopathicSecondary

Ischemiainfectious diseaseexposure to toxinsMetabolic disordersNutritional deficiencies

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PathophysiologyDilated

Most commonCocaine and alcohol abuseChemotherapy pregnancyHypertensionGenetic* Heart chamber dilate and contraction is impaired and get dec. EF%*Dysrhythmias are common- SVT Afib and VTPrognosis poor-need transplant

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Pathophysiology Hypertrophic

GeneticAlso known as IHSS or HOCMGet hypertrophy of the ventricular mass and impairs ventricular filling and COSymptoms develop during or after physical activitySudden cardiac death may be first symptomSymptoms are dyspnea, angina and syncope

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PathophysiologyRestrictive

Least commonRigid ventricular walls that impair fillingContraction and EF normalPrognosis-poor

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DiagnosticsEcho-wall motion and EFEKGCXRHemodynamicsPerfusion scanCardiac cathMyocardial biopsy

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MedicationsSame as for heart failure except for hypertrophic

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TreatmentSurgery

Vad-bridge to transplantHeart TransplantMyoloplastyICD- antiarrhythmics are negative inotropesDual chamber pacemakerHypertrophic- excision of ventricular septum

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Heart transplant

 

virtual transplant

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Nursing DiagnosesDecreased Cardiac OutputFatigueIneffective Breathing PatternFearIneffective Role PerformanceAnticipatory grieving

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Aortic Aneurysms

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DefinitionAbnormal dilation of a blood vessel at a site of weakness or a tear in the vessel wall. Usually secondary to atherosclerosis.

Most commonly affect the aorta

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Layers of an artery

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AAA

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Aneurysms location

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Saccular

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Fusiform

Most are fusiform and 98% are below the renal artery

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False or Pseudo

A pseudoaneurysm is actually a disruption in the intima and media of the

aorta, and only the adventitia/surrounding tissue retains

blood within the aortic cavity.

Ex. A hemotoma after a heart cath

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DissectingBlood invades or dissects the layers of the vessel wall

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Aortic dissection occurs when blood enters the wall of aorta, separating its layers, and creating a blood filled cavity.

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Thoracic Aortic AneurysmFrequently asymptomaticMay have substernal, neck or back painOther symptoms depend on location and structures they compress

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Abdominal AorticPulsating mass in mid and upper abdomen and bruit over the massPain intensity correlates to size and severityMay have thrombiIt can rupture causing shock and death in 50% of rupture cases

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Aortic dissectionLife threatening emergencyHappens when the intima tears and causes hemorrhage into the mediaHypertension is main cause*Primary symptom is excruciating painMay get syncope,dyspnea and weaknessPeripheral pulses are absent

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Diagnostic Tests for Aneuysms

Most are diagnosed on routine work-upCXRAbd. UltrasoundTEECT or MRIAngiography

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MedicationsAnti-hypertensives

Beta blockers, VasodilatorsCalcium channel blockersNipride

SedativesNiacin, mevocor, statinsPost-op anti-coagulants

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SurgeryUsually repaired if >5cmOpen procedure- abd incision, cross clamp aorta,aneuysm opened and plaque removed, then graft sutured in place

Pre-op assess all peripheral pulsesPost-op-check urine output and peripheral pulses hourly for 24 hours- (when to call Dr.)

Endovascular stents- placed through femoral artery

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Nursing AssessmentPain- chest, abd or back painHypertension(other vital signs)Peripheral pulsesPulsation in upper abdomenSOB

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Nursing DiagnosesRisk for Ineffective Tissue PerfusionRisk for InjuryAnxietyPainKnowledge Deficit

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PreventionPrevent atherosclerosisTreat and control hypertensionDiet- low cholesterol, low sodium and no stimulantsCareful follow-up if less than 5cm. It can grow .5cm /year

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ComplicationsRupture

Back painHypotensionPulsating mass

ThrombiRenal Failure

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Rupture Triad

Back pain

HypotensionPulsating hematoma

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http://www.cotc.edu/vstone/wk3.htm

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http://www.medi-smart.com/cardce5.htm