19922021 Case Study Fistulectomy Ppt

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    Case Study:FistulectomyGroup 6:Bulatao, Lesley Charmaine C.Cabudoc, Maricar G.Comilang, Janielle Lyn M.Constante, Quolette M.Dela Cruz, Rhealyn N.

    Ebuenga, Allyssa O.Espanueva, Gaylen C.Fabon, Yvette Stephanie Nichol B.Franco, Ma. Eliza Joy L.Fuentes, Raquel F.

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    Introduction

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    An anal fistula is an abnormal connection

    between the epithelialised surface of the analcanal and (usually) the perianal skin. Analfistulae originate from the anal glands, whichare located between the two layers of the

    anal sphincters and which drain into the analcanal. If the outlet of these glands becomesblocked, an abscess can form which caneventually point to the skin surface. The tractformed by this process is the fistula.

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    Abscesses can recur if the fistula sealsover, allowing the accumulation of pus. It then

    points to the surface again, and the processrepeats. Anal fistulas do not generally harmand they often do not hurt, but they can beirritating because of the pus-drain (and, it isnot unknown for formed stools to be passedthrough the fistula); additionally, recurrentabscesses may lead to significant short termmorbidity from pain, and create nudes forsystemic spread of infection. A fistula is a

    tiny channel or tract that develops in thepresence of inflammation and infection. Itmay or may not be associated with an abscess,but like abscesses, certain illnesses such as

    Crohns disease can cause fistulas to develop.

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    The channel usually runs from the rectumto an opening in the skin around the anus.However, sometimes the fistula openingdevelops elsewhere. For example, in womenwith Crohns disease or obstetric injuries, thefistula could open into the vagina or bladder.

    Since fistulas are infected channels, there isusually some drainage. Often a draining fistulais not painful, but it can irritate the skinaround it. An abscess and fistula often occur

    together. If the opening of the fistula sealsover before the fistula is cured, an abscessmay develop behind it.

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    An anal fistula is almost always the result of aprevious abscess. Just inside the anus are smallglands. When these glands get clogged, they may

    become infected and an abscess can develop. Afistula is a small tunnel that forms under the skinand connects a previously infected anal gland tothe skin on the buttocks outside the anus. After

    an abscess has been drained; a tunnel may persistconnecting the anal gland from which the abscessarose to the skin. If this occurs, persistentdrainage from the outside opening may indicatethe persistence of this tunnel. If the outside

    opening of the tunnel heals, recurrent abscessmay develop. Symptoms related to the fistulainclude irritation of skin around the anus,drainage of pus (which often relieves the pain),

    fever, and feeling poorly in general.

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    Surgery is necessary to cure an analfistula. Although fistula surgery is usually

    relatively straightforward, the potential forcomplication exists, and is preferablyperformed by a specialist in colon and rectalsurgery. It may be performed at the same

    time as the abscess surgery, although fistulasoften develop four to six weeks after anabscess is drained, sometimes even months oryears later.

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    Fistula surgery usually involves opening upthe fistula tunnel. Often this will require

    cutting a small portion of the anal sphincter,the muscle that helps to control bowelmovements. Joining the external and internalopenings of the tunnel and con-verging it to a

    groove will then allow it to heal from theinside out. Most of the time, fistula surgerycan be performed on an outpatient basis.Treatment of a deep or extensive fistula mayrequire a short hospital stay. Discomfortafter fistula surgery can be mild to moderatefor the first week and can be controlled with

    pain pills.

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    CASEABSTRACT

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    This is the case of Patient X, a 34 year oldmale who was admitted last September 3,

    2009, under the service of Dr. R. Lopez ofValuecare. He came to the hospital with achief complaint of hematochezia and painfulbowel elimination.

    Three weeks prior to admission, Patient Xexperienced hematochezia. He noted painfuldefecation, however describes having

    constipation or diarrhea. He decided to seekconsult and was admitted to undergoproctosigmoidoscopy.

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    At the Recovery Room, Patient X wasinserted with a Foley Catheter for urine

    collection and was transferred to room. Hewas hooked to a liter of D5NM + Ketorolac 60mg to run for 6 hours and was placed on dietas tolerated. A few hours later, the foleycatheter was removed and he was able to voidfreely. The sack on the surgical site wasremoved later on.

    On September 5, 2009 at 5:40 a.m. theIVF was discontinued upon request and onSeptember 6, 2009, Patient X was dischargedambulatory.

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    Physical

    Assessment

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    GENERAL DATA

    1. General Information

    Name: D.Y. Age: 34 y/o

    Gender: Male

    Chief complaint: Hematochezia

    Admitting diagnosis: ProctosigmoidoscopyFistulectomy

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    2. Vital Signs Temperature: 36.4 Pulse Rate: 62

    Respiratory rate: 16 Blood Pressure: 100/70

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    3. General Survey

    3.1 Anthropometric Measurement Height: 55 Weight: 72kg

    3.2 General AppearanceThe patient shows sign of distress,

    conscious and coherent. He is oriented to the place,person, and time. He is well-developed, looksaccording to his age. Well- nourished and calm.

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    4. Skin The patients skin is brown, smooth and fair,

    without any abnormalities found. He has goodskin turgor and is warm to touch.

    5. Head The patients head size is proportion to the size

    of his body and with a normocephalic shape. Thehairs are evenly distributed. There is nopresence of dandruff or scar. The face issymmetrical and with negative facial musculture.The patients eyes are symmetrical. Eyebrows

    and eyelashes are evenly distributed. Paleconjunctiva. Anicteric sclera. The cornea and lensare clear. Pupil sizes are equal. The visual acquityis good (20/20).

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    6. Ears The ears of the patient are symmetrical, soft

    and pliable, and at the level of the outer cantus

    of the eye. There is no presence of discharges onthe ear canal. Able to hear sounds on both ears.

    7. Nose Patients nose is smooth, nasolobial fold is

    symmetrical, septum is located in the midline, nopresence of nasal discharge seen. Patent nostrils.

    8. Mouth and Pharynx The lips are pinkish in color and moist, no

    presence of cracks or lesions. Tongue is found at

    the midline and can move freely. Complete teethwithout presence of cavity. Gums and buccalmucosa are pinkish in color, smooth and moist.Uvula is on the midline. There is no presence ofinflammation of tonsils.

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    9. Neck Patients neck moves freely. Trachea is located in

    the midline. Cervical lymph nodes are non-palpable. There is no presence of masses.

    10. Chest and Lungs Patients chest is cylindrical with regular

    breathing pattern. Lung expansion is symmetricaland no retractions.

    11. Heart

    The precordium is flat. Apical pulse is located atthe fifth intercostal space left mid-clavicularline. Heart rhythm is regular.

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    12. Abdomen

    Patients abdomen appears globular and withoutpresence of scars/lesions, with a presence oftenderness upon palpation.

    13. Genitals

    Not assessed, the patient refused to.14. Anus and rectum Not assessed, the patient refused to.

    15. Back and Extremities Nail and nail beds are pinkish with no sign ofinflammation. Decreased ROM upon surgery.Spine is on the midline. Coordinated gait.

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    Anatomy & Physiology:Digestive Functions

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    TheDig

    estive

    Sys

    tem

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    Digestive functions

    Ingestionoccurs when foods enter thedigestive tract through mouth.

    Mechanical processingis the physicalmanipulation of solid foods, first by the

    tongue and the teeth and then by swirlingand mixing motions of the digestive tract. Digestionrefers to the chemical breakdown

    of food into small organic fragments that

    can be absorbed by the digestive epithelium. Secretionaids digestion through the releaseof water, acids, enzymes and buffers by thedigestive tract and accessory organs.

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    Absorptionis the movement of small organicmolecules, electrolytes, vitamins, and wateracross the digestive tract.

    Excretionis the elimination of waste

    products from the body. Within thedigestive tract, these waste products arecompacted and discharge through theprocess of defecation

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    Hist

    ological

    Features

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    1. Mucosa Inner lining.

    An example of a mucous membrane. Consist of an epithelial surface moistenedby glandular layer of loose connectivetissue, the lamina propria.

    Increase the surface area available forabsorption

    Four Major Layers of DigestiveTract

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    Permit expansion after a large meal. Forms fingerlike projections, called villi. Outer portion of the mucosa contains a

    narrow band of smooth muscle and

    elastic fibers Muscularis mucosae, move the mucosal

    folds and villi.

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    2.Submucosa

    Second layer of loose connective tissuethat surrounds the muscularis mucosae.

    Contains large blood vessels andlymphatics as well as network of nerve

    fibers, sensory neurons andparasympathetic motor neurons.

    This neural tissue submucosal plexushelps control and coordinate thecontractions of smooth muscle layer andalso helps regulate the secretion of thedigestive glands.

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    3. Muscularis externa Collection of smooth muscle cells arranged

    in an inner circular layer and an outerlongitudinal layer.

    Contractions of these layers in various

    combinations agitate or propel materialsalong the digestive tract. These are autonomic reflex movements

    controlled primarily by a network of nerve,

    the myenteric plexus, sandwiched betweenthe inner and outer smooth muscle layers.

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    4. Serosa A serous membrane

    Covers the muscularis externa along mostportions of the digestive tract inside theperitoneal cavity

    The parietal and visceral peritoneum that linesthe inner surfaces of the body wall. The parietal and visceral peritoneum are

    connected by double sheets of serous

    membrane called mesenteries, loose connectivetissue sandwiched between epithelia providesan access route for the passage of bloodvessels, nerves and lymphatics servicing the

    digestive tract.

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    TheMovemen

    tofDige

    stive

    Materia

    ls

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    Peristalsis and Segmentation

    Peristalsis Waves of muscular contractions that move along

    the length of the digestive tract.

    During a peristaltic movement, the circularmuscles first contract behind the digestivecontents. Then longitudinal muscles contract,shortening adjacent segments. A wave ofcontraction in the circular muscles then forcesthe materials in the desired direction.

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    Segmentation Movements that churn and fragment

    digestive materials. This action results in a thorough mixing

    of the contents with intestinalsecretions. Because they do not follow aset pattern, segmentation movements donot propel materials in a particulardirection.

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    The agitation of ingested materials withgastric juices secreted by the glands of the

    stomach produces a viscous, soupy mixturecalled chyme. The principal anatomical landmark of the

    stomach is a muscular organ with the shape

    of an expanded J. The esophagus connects to the stomach at

    the cardia. The bulge of the stomach superior to the

    cardia is the fundus of the stomach. The large area between the fundus and the

    curve of the J is the gastric body.

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    Pylorus the curve of the J connects thestomach with the small intestine.

    A muscular pyloric sphincter regulates theflow of the chyme between the stomach andthe small intestine.

    The stomach resembles a muscular tube withnarrow and constricted lumen. When full, itcan expand to contain 1-1.5 liters.

    This degree of expansion is possible because

    the stomach wall contains a number ofprominent ridges and folds called rugae.

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    The visceral peritoneum covering the outersurface of the stomach is continuous with apair of mesenteries. The greater omentumextends below that hangovers and protectsabdominal viscera. The much smaller lesser

    omentum extends from the lesser curvatureto the liver. Stomach is lined by an epithelium dominated

    by mucous cells. These secreted mucus

    produced helps protect the lining from theacids, enzymes, and abrasive materials itcontains.

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    Gastric pits shallow depressions andopen onto the gastric surface. Eachgastric pit communicates with thegastric glands that extends deep intothe underlying lamina propria. These

    glands are dominated by two types ofsecretory cells: parietal cells andchief cells. Together these cellssecrete about 1500 ml of gastric juice

    each day. Chief cells secrete pepsinogen, an

    inactive form of the enzyme pepsin.

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    Regulation of Gastric Activity

    The cephalic phase the sight, smell, taste orthought of food initiates at the cephalicphase of gastric secretion directed by theCNS, prepares the stomach to received food.Under the control of vagus nerve,parasympathetic fibers innervate parietalcells, chief cells, and mucous cells of thestomach.

    The gastric phase begins with the arrival of

    food in the stomach. Stimulation of stretchreceptors in the stomach wall andchemoreceptors in the mucosa triggers therelease of a hormone, gastrin, into the

    circulatory system.

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    3. The intestinal phase begins when chyme

    starts to enter the small intestine. Thepurpose of this phase is to control rate ofgastric emptying and ensure that thesecretory, digestive, and absorptivefunctions of the small intestine can proceedefficiently.

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    Digestion in the stomach

    The stomach performs preliminarydigestion of proteins by pepsin and, for avariable period, permits the digestion ofcarbohydrates by salivary amylase.

    This enzyme remains active until pHthroughout the material in the stomachfalls below 4.5, usually within 1-2 hoursafter a meal. As the stomach contents

    become more fluid and the pH approaches2.0, pepsin activity increases and proteindisassembly begins.

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    Protein digestion is not completed in thestomach, but there is usually enough timefor pepsin to breakdown complex proteinsinto smaller peptide and polypeptide

    chains before the chyme enters the smallintestine.

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    The Small Intestine

    About 6 meters (20 ft) long and has adiameter ranging from 4 cm at the stomachto about 2.5 cm at the junction with thelarge intestine. It has three subdivisions:

    the duodenum, the jejunum, and the ileum. The duodenum is the 25 cm (1 ft) closest tothe stomach. This portion receives chymefrom the stomach and exocrine secretionsfrom the pancreas and liver.

    Thejejunum, which is supported by a sheetof mesentery, is about 2.5 meters (8 ft) inlength. The bulk of chemical digestion andnutrient absorption occurs in the jejunum.

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    The jejunum leads us to the third segment,the ileum. The ileum ends at the sphincter,the ileocecal valve, which controls the flowof chyme from the ileum into the cecum ofthe large intestine.

    Plicae intestinal lining bears a series oftransverse folds.

    Villi lining of the intestine is also thrown intoseries of fingerlike projections. Small intestine were a simple tube with

    smooth walls, it have a total absorptive area

    around 3300 square centimeters, or roughly3.6 square feet. Lacteal refers to the pale, cloudy

    appearance of the lymph in these channels.

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    Intestinal Movements

    Two examples are thegastroenteric reflex and thegastroileal reflex.

    Gastroenteric reflex initiated by distention, whichimmediately accelerates glandular secretion and

    peristaltic activity in all segments. Gastroileal reflex is a response to circulating levels

    of hormone gastrin. Intestinal juice moistens the intestinal contents,

    assists in buffering acids, and dissolves bothdigestive enzymes provided by the pancreas and theproducts of digestion.

    I i l h

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    Intestinal hormones Secretin is released when the pH falls in the

    duodenum, occurs when acid chyme arrives fromthe stomach. The effect is to increase thesecretion of water and buffers by the pancreasand liver

    Cholecystokenin is secreted when chyme arrive

    in the duodenum, especially when it containslipids and partially digested proteins, targetsthe pancreas and liver, accelerates theproduction and secretion of all types of

    digestive enzymes. Gastric inhibitory peptide is released whenfats and glucose enter the small intestine. Thispeptide hormone inhibits gastric activity andcauses the release of insulin from pancreaticislets.

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    The Large Intestine

    The horseshoe-shaped large intestine beginsat the end of the ileum and ends at the anus. Lies below the stomach and liver. The principal functions of large intestine

    include reabsorption of water andcompaction of feces, the absorption ofimportant vitamins liberated by bilateralaction, and the storing of fecal materialprior to defecation.

    The large intestine often called the largebowel.

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    It has an average length of approximately 1.5meters (5 ft) and a width of 7.5 cm (3 in).

    It is divided into three major regions: the pouchlike cecum, the first portion of the large intestine;the colon, the largest portion of the large intestine;and the rectumthe last 15 cm (6 in) of the largeintestine and the end of the digestive tract.

    Large intestine absorbs a variety of othersubstances from the chyme Vitamins (2) bilirubinproducts bile salts toxins

    Movement from the transverse colon through therest of the large intestine results from thepowerful peristaltic contractions, called massmovement.

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    The Cecum

    Material arriving from the ileum first entersan expanded chamber called cecum.

    Ileocecal valve a muscular sphincter guardsthe connection between the ileum and thececum.

    It usually has the shape of a rounded sacand the slender veniform appendix attaches

    to the cecum along its posteromedialsurface. Appendix is almost 9 cm( 3.5 in)

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    Descending colon turns inferiorly.

    The descending colon continues along theleft side until it curves and recurves as thesigmoid colon.

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    The Rectum

    Forms the end of the digestive tract. Anorectal canal last portion of the rectum

    contains small longitudinal folds joined bythe transverse folds that mark the boundarybetween columnar epithelium of the rectumand a stratified squamous epithelium similarto that found in the oral cavity.

    Anus the opening of the anorectal canal, theepidermis becomes keratinized and identicalto that on the surface of the skin.

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    The circular muscle layer of themuscularis externa in this region formsthe internal anal sphincter.

    The external anal sphincter guardsthe exit of the anorectal canal. Consistof muscle fibers, is under voluntary

    control.

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    Pathology and

    Physiology

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    PRESSURE IN RECTUM

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    E E E

    PASSAGE OF HARDENED STOOL

    CAUSES FRICTION TO LINING

    PREDISPOSITION TO ANAL GLAND INFECTION IN THEINTERSPHINTERIC PLANE

    FORMS ABSCESS IN THE PERI-ANAL AREA

    FORM A CHRONIC TRACT ( FISTULA-IN-ANO)

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    WBC NEUTROPHILS

    INFLAMMATORY RESPONSE DUE TO INFECTION

    RELEASE OF CHEM. MEDIATORS

    HISTAMINE & PROSTAGLANDIN SEROTONIN

    PAIN FEVER

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    Past medical history

    Important points in the history that may suggesta complex fistula include the following:Inflammatory bowel disease

    DiverticulitisHistory of traumaPrevious radiation therapy for prostate or

    rectal cancer

    TuberculosisImmune suppression-Steroid therapy, HIV

    infection

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    Classification of fistula in-ano

    Parks classification system (all are inrelation to the sphincters)

    The Parks classification system defines4 types of fistula-in-ano that resultfrom cryptoglandular infections.

    1 I hi i 70%

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    1.Intersphincteric-commonest-70%

    Common course - Via internal sphincter to theintersphincteric space and then to the perineum.They result from perianal abscesses

    2. Transsphincteric -25%

    Common course - Low via internal and external

    sphincters into the ischiorectal fossa and then tothe perineum. Originate from ischiorectalabscesses

    3.Suprasphincteric -5%

    Common course - Via intersphincteric spacesuperiorly to above puborectalis muscle intoischiorectal fossa and then to perineum. Resultfrom supralevator abscesses

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    4. Extrasphincteric-1%

    Bypass the anal canal and sphincter

    mechanism, passing through the ischiorectalfossa and levator ani muscle, and open high inthe rectum

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    Laboratory

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    Drug Study

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    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

    Plasil Antacids,

    anti-emeticagents, anti-ulcerants

    1 amp x 1

    dose

    TIV Dopamine

    antagonistthat acts byincreasingreceptorsensitivity andresponse ofupper GITtissues to

    acerthylcoline.

    Disturbanc

    es of GImotility

    Give at least

    30 mins.. Beforemeals and atbedtime.Assess mentalstatus duringtreatment.Instruct pt. to

    avoid hazardousactivities for atleast 2 hours.Advice pt toavoid alcohol andotherdepressant thatenhancesedatingproperties ofthis drug.

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    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

    Ranitidin

    e

    Antacids,

    anti- emetic,anti-ulcerants

    1 amp x 1

    dose

    TIV Completely

    inhibits actionof histamineon the H2 atreceptor siteof parietalcells.Decreasinggastric acidsecretions.

    selected

    cases ofpersistentdyspepsia,stressulceration,& inpatientsat risk ofacidaspirationduringanesth

    Assess pt. for

    abdominal pain.Note forpresence ofblood in emesis,stool or gastricaspirate.May be addedto totalparenteralnutritionsolution.

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    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

    Coamoxiclav

    Antibiotic 625 mg/tab q 8hrs

    x 6 doses

    PO An antibiotic

    that is a

    combination

    of a penicillin

    (amoxicillin)

    and a

    substance

    called

    clavulanicacid. It kills

    bacteria, by

    interfering

    with their

    ability to

    form cell

    walls. The

    bacteria

    therefore

    break up and

    die.

    skin & softtissue

    infections,UTI, pre &post-surgicalprocedures, bone &joint

    Instruct

    patient to

    immediately

    report signs or

    symptoms of

    hypersensitivity

    reaction, such

    as rash, fever,

    or chills.Monitor

    patient

    carefully for

    signs and

    symptoms of

    hypersensitivityreaction.

    Monitor

    patients vital

    signs before,

    during and after

    medication.

    N f Cl f D / R M h f d N

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    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

    Flanaxforte

    non-steroidalanti-

    inflammatoryand antrheumaticproducts

    550 mg/tab q 8 hrs

    x 3 doses

    PO Reversiblyinhibits

    cyclooxygenase-1 and 2(COX-1 and 2)enzymes,which result indecreasedformation of

    prostaglandinprecursors;hasantipyretic,analgesic, andanti-inflammatoryproperties

    Relief of

    mild to

    moderatel

    y severe

    pain &

    fever w/

    or w/o

    accompany

    inginflammati

    on eg

    musculosk

    eletal

    trauma,

    post-op

    pain &

    post-

    dental

    extraction

    .

    Advice the pt.to take this

    medicationexactly asdirected; do notincrease dosewithoutconsultingphysician. Do

    not crushtabletsInstructclient to takewith food ormilk to reduceGI distress.

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    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

    Tell pt. thatshe may

    experiencedrowsiness,dizziness,lightheadedness, or headachealso, nausea,vomiting, orheartburnInstruct pt.to report DOB,chest pain, skinrash anditching.

    Name ofDrug

    Classification Dosage/frequency

    Route Mechanism ofaction

    Indication Nursingresponsibilities

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    Drug frequency action responsibilities

    Fibrosinesachets

    Laxatives andpurgatives

    1 sachet in 1glass ofwater TID x

    2 days

    PO Is a stimulantlaxative. Itacts directly

    on the bowels,stimulating thebowel musclesto cause abowelmovement.

    Fibersupplement to

    maintainregularityof bowelmovement.

    Instruct patientto report if she/he develop

    nausea, vomiting,or stomach pain.Instruct patientto stop laxativeand inform nurseor physician ifshe/ heexperienced

    Rectal bleedingor failure to havea bowel movementwithin 12 hoursafter use of alaxative, may be asign of a serious

    condition.

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    Nursing

    Care

    Plan

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    PRE-OP

    Assessment Diagnosis Planning Nursing

    Interventions

    Evaluation

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    Interventions

    Subjective:

    Masakit kapagako ay

    dumudumi, asverbalized by thepatient

    pain in rectalregion

    Objective:

    a pain rating of7 on a scale of 0-10

    Irritable

    Confused

    change in pulserate

    increase and

    decrease inrespiratory rate

    Acute pain related

    to actual tissue

    damage

    After 1 hour of

    nursing

    intervention, the

    client willexperience lesser

    pain.

    Performed a

    comprehensive

    determined

    whether the client isexperiencing pain

    the tine of initial

    interview.

    assessed pain in

    a client using a self

    report 0-10

    numerical painrating scale

    assessed and

    document the

    intensity of pain and

    discomfort.

    Goal has been

    partially met.

    The client

    experiencedlesser pain.

    Assessment Diagnosis Planning Nursing

    Interventions

    Evaluation

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    Interventions

    asked the client

    to describe

    appetite, bowel

    elimination andability to rest and

    sleep.

    obtained a

    prescription to

    administer by the

    doctor.

    Assessment Diagnosis Planning Nursing

    Interventions

    Evaluation

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    Interventions

    Subjective:

    Nahihirapan

    ako dumumi as

    verbalized bythe patient

    Objective:

    Bright red

    blood with stool

    (+) flatus

    Abdominal

    tenderness

    Constipation

    related to

    discomfort

    duringdefecation

    After 1 hour of

    nursing

    intervention the

    client will relieffrom discomfort

    of constipation

    Assessed

    usual pattern of

    defecation

    including timeof day, amount

    and frequency

    of stool

    Assessed

    history of bowel

    habitsPalpated for

    abdominal

    distention,

    percuss for

    dullness and

    auscultate for

    bowel sounds

    Goal has

    been partially

    met.

    The client hasexperienced

    lesser pain

    from the

    discomfort of

    constipation.

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    Assessment Diagnosis Planning Nursing

    Interventions

    Evaluation

    Encouraged

    the client toheed defecation

    warning signs

    Checked for

    impaction of

    feces in bowel

    Provide

    privacy for

    defecation

    Administer

    stool softeners

    ordered by the

    doctor

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    POSTOP

    Assessment Diagnosis Planning NursingInterventions

    Evaluation

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    Interventions

    Subjective:

    Objective:Open wound

    High risk for

    infection r/t

    inadequateprimary

    defense as

    manifested by

    broken skin

    After 2 hoursof

    nursing

    interventionthe

    patient willgain

    knowledge in

    infectioncontrol

    as evidencedby

    discussing the

    wound care.

    IndependentEstablishRapportTeach patient

    to wash hands

    often,

    especially

    before

    toileting,Before andafter meals.

    After 2 hours of

    Nursingintervention

    the patient will be

    able to gain

    knowledge in

    infection controlas

    evidenced by hisdiscussion inwound

    care. Therefore,the

    goal was met

    Assessment Diagnosis Planning NursingInterventions

    Evaluation

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    Interventions

    Discuss to

    patient the

    following signsof infection:

    redness,

    swelling,

    increased pain,

    or purulent

    drainage on thesite and feverDemonstrate

    and allow

    return

    demonstrationof wound care

    N

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    EVALUATIO

    N

    AND

    DISCH

    ARGE

    PLAN

    MEDICATION

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    MEDICATION

    Take Home Med. Fibrosine Sachet 1 sachet in 1 glass of water 3 times a day.

    EXERCISE

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    EXERCISE

    Avoid heavy lifting, straining and strenuousexercise for two weeks at a minimum (i.e.,weightlifting, jogging, swimming, etc.)

    TREATMENT

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    TREATMENT

    Fistulectomy - in a fistulectomy, the surgeon makesan incision in the fistula tract, opening it up andmerging it with the anal canal. This allows thetissues to heal from the inside out.For very smallfistulas, a fistulotomy may be performed in adoctors office, using only local anesthesia. Largerfistulas, however, require surgery under spinal orgeneral anesthesia, and are typically performed in ahospital or surgery center. Patients typicallyexperience mild or moderate discomfort or painfollowing this procedure, with a recovery time ofone to four weeks.

    HEALTH TEACHING

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    HEALTH TEACHING

    Maintain a liquid diet for two days after theprocedure (i.e., soup, Jell-O, etc.)

    Eat a high fiber diet after two days Use the bathroom once a day. A warm bath

    may help your symptoms. Take over-the-counter pain medicine as

    needed

    Shower standing up and bathe the area withwater to soothe and keep it clean. Do not sit in the bathtub

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    Do not use topical steroids or topical agentssuch as Preparation H

    Expect some drainage for two to four weeksafter the procedure as the Surgisis AFPplug is incorporated and the fistula tract isclosed.

    Using stool softeners and adhering to goodhygiene, such as sitz baths after everybowel movement, decreases discomfort andhelps for recovery.

    OPD

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    OPD For anal fissures, the WASH regimen is indicated.

    For anal fistulas, outpatient follow-up with asurgeon is indicated if consultation did not takeplace at the time of presentation.

    Botulinum toxin injection has been shown to be aneffective alternative to surgery for the treatment

    of uncomplicated idiopathic anal fissure. Topical application of clove oil cream has

    demonstrated significant benefit in patients withchronic anal fissure.

    The application of topical 0.5% nifedipine ointment

    has been used as a chemical sphincterotomy agent.It has been shown to offer a significant healingrate for acute anal fissure and may prevent it frombecoming a chronic fissure.

    DIET

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    DIET A high-fiber diet causes a large, soft, bulky stool

    that passes through the bowel easily and quickly.Because of this action, some digestive tractdisorders may be avoided, halted, or even reversedsimply by following a high-fiber diet. A softer,

    larger stool helps prevent constipation andstraining. This can help avoid or relievehemorrhoids. More bulk means less pressure in thecolon, which is important in the treatment ofirritable bowel syndrome and diverticulosis (defects

    in the weakened walls of the colon). In addition,fiber appears to be important in treating diabetes,elevated cholesterol, colon polyps, and cancer of thecolon.