Starchy Foods and Pancreas

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    test of comprehension, parapgrasing and critical reviee of one concept in the

    book, live to eat. Type of academic writting used depends on objective, but

    basics remain the same. Put in greek debt folder

    refined carbs and Low nutrition:

    Also, eating refined means, nutrients are not taken either even though they make up bulk of calories pg

    38 eat to live

    Also refined foods have little fiber and nutritents, two things the author cliams are necessary to surpress

    hunger and therefore you feel hungry

    again. pg 44 eat to live

    refined Carbs and Diabetes:

    Refined carbs have fiber and mineral removed and therefore get absored in blood easily and qucikly.

    Insulin is then punped out quickly to curb this sugar.

    Being fat also causes more insulin to be excreted, so if one is fat, and eating refined carbs, insulin is

    excessivley excreted by pancreas.

    Eating refined carbs increases sugar intake and so does being fat, leading to load on pancreas.

    When too much load on pancrease it can lead to diabetes (isnt this insulin sensitivyt which might lead to

    diabetes??) pg 38 eat to live

    HOWEVER i have not found this relation between load on pancreas and diabetes.

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    according to http://www.diabetes.org/diabetes-basics/diabetes-myths/?loc=DropDownDB-myths

    Type 1 diabetes is caused by genetics and unknown factors that trigger the onset of the disease; type 2

    diabetes is caused by genetics and lifestyle factors and NOT SUGAR itself

    Being overweight does increase your risk for developing type 2 diabetes, and a diet high in calories from

    any source contributes to weight gain.

    Research has shown that drinking sugary drinks is linked to type 2 diabetes.

    Well obesity surely plays a part..he is right, but is his reasoning right. But wht about about pancreatic

    overload?

    According to http://www.diabetes.org/diabetes-basics/prevention/pre-diabetes/?loc=DropDownDB-prediabete efore people develop type 2 diabetes, they almost always have "prediabetes" blood

    glucose levels that are higher than normal but not yet high enough to be diagnosed as diabetes.

    refined carbs and obesity:

    pg 44 eat to live, he says fat and carbs taken together are dangerous, However if you read, he is talking

    about BEING FAT as well as consuming fat.

    Fat and carbs, he says, together create an effect and promote a signal to accumulate fat in body.

    refined food scause sugar surge and then trigger incuslin release to dirve sugar out to be used.

    However INSULIN ALSO PROMOTES STORGAE OF FAT and swells fat cells.

    (i would not doubt, it uses sugar and probably stores extra sugar). As you get fatter

    http://www.diabetes.org/diabetes-basics/prevention/pre-diabetes/?loc=DropDownDB-prediabetehttp://www.diabetes.org/diabetes-basics/prevention/pre-diabetes/?loc=DropDownDB-prediabetehttp://www.diabetes.org/diabetes-basics/prevention/pre-diabetes/?loc=DropDownDB-prediabetehttp://www.diabetes.org/diabetes-basics/prevention/pre-diabetes/?loc=DropDownDB-prediabete
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    More fat on body means, more need for insulin, as it block muscles from using Insulin, therefore more

    insulin is required to get rid of sugar

    whihc the fat is blocking. E.g. fta arund tummy can cause 3-5 times more insulin release. ONce again

    higher insulin creates more fat.

    Eating refined foods it seems therefore casues set point of body, that the weight the body tries to

    maintain through hhormone, to increase.

    This is because refined carbs (and fats the author nows says) increase fat storing hormone, probably

    insulin, the set point is risen.

    refined carbs cause higher insulin, highe insuline makes you fat (temporarily, what makes it permanent

    is),

    and fat makes you release more insulin, eventually leading to a higher permanent

    steady weight.

    But is it relay causing fat storage as I was told by dietry asoiacoation book, is only when too high

    calories. So what causes fat storage, higher calories, or simply even intaking fat and being used by

    insulin)

    It is true Accordinhg to Modern Nutrition in Health and Diseases

    insulin stimulates fat storage through its contribution to adipocyte differentiation, its inhibition of

    lipolysis, and its stimulation of lipogenesis, although the latter is likely a minor effect in humans pg

    639

    https://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjA

    G&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRE

    SENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-

    XQ&sig2=lqgB2ySS2vtPXVl_rhZGygagrees that with dietry association that

    Insulin regulates the uptake of nutrients into the cells, the storage of nutrients not being used, and

    the conversion of one nutrient type to another.

    Excess glucose is preferentially stored as glycogen BUT

    https://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyghttps://www.google.com.pk/url?sa=t&rct=j&q=&esrc=s&source=web&cd=7&cad=rja&ved=0CGUQFjAG&url=http%3A%2F%2Fwww.iit.edu%2Fresearch%2Fservices%2Fdoc_ret_2004%2FPOWERPOINT_PRESENTATION.ppt&ei=dTmoUfbaEKuT0AWW14CAAQ&usg=AFQjCNGtMVWFvPC4rQFgJHKbelizQJr-XQ&sig2=lqgB2ySS2vtPXVl_rhZGyg
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    When cells are saturated with glycogen (livercells store 5 to 8% of their weight as glycogen,

    muscle cells 1 to 3%) additional glucose is converted

    to fat in the liver and stored as fat in adipose cells.

    Insulin promotes the conversion of all excess glucose in liver that cannot be stored as glycogen

    into fatty acids

    Fatty acids are packaged as triglycerides in low density lipoproteins transported by blood to

    adipose tissue

    Insulin activates lipoprotein lipase in the capillary walls of adipose tissue, which splits

    triglycerides into fatty acids. This enables them to be absorbed into adipose cells where they

    are converted again to triglycerides and stored.

    A deficiency of insulin will accelerate the breakdown of the bodys fat reserves for fuel.Insulin

    defeincy can cause metabolism of fat, leading to high chloestrol, heart attack etc

    However Also see

    http://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&

    sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=f

    alseInsulintakes extra glucose and stores it as fat. When insulin levels grow and spike, it can also lead to

    too much fat being stored. Therefore high incuslin levels means having more fat.

    Not all fat is same, for diabetes visceral obesity is worse, that is around the middle of the body. linked to

    extra fat storgae in lliven ,w hich can raise sugar level, and to extra fat in musc cells, making them even

    more reistant to insulin

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    Insulin

    When you eat a candy bar or a meal, the presence ofglucose, amino acids or fatty acidsin the intestine

    stimulates the pancreas to secrete a hormone called insulin. Insulin acts on many cells in your body,

    especially those in the liver, muscle and fat tissue. Insulin tells the cells to do the following:

    Absorb glucose, fatty acids and amino acids

    Stop breaking down glucose, fatty acids and amino acids; glycogen into glucose; fats into fatty acids and

    glycerol; and proteins into amino acids

    Start building glycogen from glucose; fats (triglycerides) from glycerol and fatty acids; and proteins from

    amino acids

    The activity of lipoprotein lipases depends upon the levels of insulin in the body. If insulin is high, then the

    lipases are highly active; if insulin is low, the lipases are inactive.

    The fatty acids are then absorbed from the blood into fat cells, muscle cells and liver cells. In these cells,

    under stimulation by insulin, fatty acids are made into fat molecules and stored as fat droplets.

    It is also possible for fat cells to take up glucose and amino acids, which have been absorbed into the

    bloodstream after a meal, and convert those into fat molecules. The conversion of carbohydrates or

    protein into fat is 10 times less efficient than simply storing fat in a fat cell, but the body can do it. If you

    have 100 extra calories in fat (about 11 grams) floating in your bloodstream, fat cells can store it using

    only 2.5 calories of energy. On the other hand, if you have 100 extra calories in glucose (about 25 grams)

    floating in your bloodstream, it takes 23 calories of energy to convert the glucose into fat and then store it.

    Given a choice, a fat cell will grab the fat and store it

    rather than the carbohydrates because fat is somuch easier to store.

    This hguy is somewhere in the middle and says

    http://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en

    &sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f

    =false

    how high insulin surges MAY influence whether the body uses or stores its glucose and fat

    insulin facilitates transport of glucose into cells, storage of fatty acids as fat and synthesis of cholesterol.

    insulin is one of many factors involved in body weight and metabolism.

    http://health.howstuffworks.com/food.htmhttp://science.howstuffworks.com/life/human-biology/diabetes.htmhttp://health.howstuffworks.com/http://science.howstuffworks.com/life/human-biology/blood.htmhttp://science.howstuffworks.com/life/cellular-microscopic/health.howstuffworks.com/calorie.htmhttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://books.google.com.pk/books?id=LhSLKVmauGoC&pg=PA128&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falsehttp://science.howstuffworks.com/life/cellular-microscopic/health.howstuffworks.com/calorie.htmhttp://science.howstuffworks.com/life/human-biology/blood.htmhttp://health.howstuffworks.com/http://science.howstuffworks.com/life/human-biology/diabetes.htmhttp://health.howstuffworks.com/food.htm
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    Insulin only causes problem when one develops insulin resistance, that is pancreas need to pump more.

    It doenst depend on carbs, fat or protein, but on being overweight. Reducing weight improves insulin

    response in any case.

    Relation between GI and weight? < the actual argument which needs to be studied further

    Studies show diets high in GI increase body weight.

    fructose doesnt lead to secretion of insulin however some research suggests that fructose favors the fat

    making pathways and impairs fat clearing pathways in the body. As the liver busily builds lipids, its

    handling of glucose become s unbalanced and insulin resistance develops. Reserach is suggesting links

    between high fructose intakes and prediabtes and metabolic syndrome.

    A study showed that insulin resiatnce and weight loss were impacted by low calories diet and not by the

    GI index. Low GI food may mean lower energy intake in next meal.

    Indidviausl also respond differently to Carbs. Some people have high insulin response. In one study,

    weight gain over 6 years was not dependent on the diet being igh or low in carbs, but now insulin

    response of indidvuals where people with high insulin response gained more weight (especially when on

    high carb diet) while for such people weight loss maybe greater on a low GI diet (he is talking about

    CARB and not GI)

    refined Carbs and heart disease:::::

    Because sugar enters body so quickly it can also increase triglycerides and therefore risk of heart disease

    in susceptible individuals i.e

    people who already have likelihood of developing such a problem. pg 38 eat to live

    According to The Ten Biggest Diet Myths And Greatest Health Secrets Revealed: A Summary ... By

    Warren Pear refined carbs do not cause blood sugar to rise alot

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    In Summary of above

    According to Dr something of eat to live

    Given that

    Blood Sugar requires insulin

    Fat on body decreases uptake of insulin

    Eating refined foods (eating fat makes it worse)

    More Sugar in Blood

    More insulin

    Being fat means less sugar intake requiring higher insulin

    More Sugar and Fat absorbed

    Leads to higher set point i.e. your hormone levels reach an excretion level where your weight is

    permanently increased to a new level. It is NOT a vicious cycle.

    Anything that increases insulin will cause the cycle?? Refined Carb, High GI etc

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    However question persist as it is not 100% clear.

    Does High GI food really cause fat storage, weight gain and a sighing of body set point of

    weight?

    Does high GI and blood sugar cause overload of pancreas?

    So insulin is storing extra glucose as fat.http://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=

    insulin%20and%20fat%20storage&f=falseInsulin

    And prefers to store fat as fat, rather than glucose as fat as it takes less

    energyhttp://science.howstuffworks.com/life/cellular-microscopic/fat-cell2.htmbut does it store when

    extra glucose/fat/calories are not present????

    need more articles in fat absorption and insulin to note if. See GI, insulin and fat storage for future

    |and these two questions have not been clearly answered.

    http://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://science.howstuffworks.com/life/cellular-microscopic/fat-cell2.htmhttp://science.howstuffworks.com/life/cellular-microscopic/fat-cell2.htmhttp://science.howstuffworks.com/life/cellular-microscopic/fat-cell2.htmhttp://science.howstuffworks.com/life/cellular-microscopic/fat-cell2.htmhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulinhttp://books.google.com.pk/books?id=XGaI1WDHo_0C&pg=PA35&dq=insulin+and+fat+storage&hl=en&sa=X&ei=d0eoUdaQA8iR7Abrl4GoBA&redir_esc=y#v=onepage&q=insulin%20and%20fat%20storage&f=falseInsulin
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    -----

    Implications:::

    (can be begiing for type of intro or proposal or at end [aswell] to show use of result): for low fat, lowcarb, high GI diets and incentive to maintain low fat structure. Wgat about women who have naturally

    more fat? Wgat abiut fat ppl, is it tougger because of this set point to become thin?

    Further research:::

    need better qlty sources, more reading on fat storage aswell as how and if pancreas are overloaded byextra work and does it cause sensitivity or sonething else eventually causing diabetes. Alsi see womanand if naturally hight fat means impact of insulin ie metabolism in them is different?