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Reporter:Ms. Reyna Marie P.
Labadan
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Epidemiology ofBurns
2 million people requiremedical attention eachyear
51,000 requireadmission
4,500 people dieannually
Most burns cover lessthan 5-10% of bodysurface area
Most injuries occur at
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Mortality fromBurns
Most deathsoccur at home
Causes of death
Smokeinhalation, sepsis,pneumonia,shock
the risk of deathincreasessignificantly if thepatient has
sutained bothcutaneous burn
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What areburns???
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PREDISPOSING
FACTORS all age groups at risk
Mortality increases withage
Males predominate
Young children and elderlypeople are at particularlyhigh risk for major burns
Occupation
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PRECIPITATINGFACTORS
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burns
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em c
al BB
UURR
NNss
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Electrical burns
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Radiation burns
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bb
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INTEGUMENTARY SYSTEM and
BURNS
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DEGREES OF BURNS
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First Degree
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Second Degree
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Third Degree
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PATHOPHYSIOLOGY
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Be familiar
Hyperkalemia Hyponatremia
Anemia
Acute tubularnecrosis
Laryngeal edema
Hypoxemia
ARDS
Respiratoryacidosis
Hemoconcentrati
Edema
Pain
Hypovolemicshock
Oliguria Ileus
Curlings ulcer
Altered LOC
Metabolic acidosis
Major organdamage
Renal failure
Respiratory
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DETERMINING EXTENT OFINJURY
E ti ti
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EstimatingBURNS in
AdULts
RULE OF9
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Estimating BURNS inChildren
LUND andBROWDER
METHOD
-
recognizes thatTBSA
especiall
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PALM METHOD
- Used inpatients with
scatteredburns
- Size of ptspalm is
approx. 1 %
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D
I
A
G
NO
S
T
I
S
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LABORATORYTESTS
SERUMELECTROLYTESa.Emergentphase:1st48hourshyperkalemia >5meq/Lhyponatrem
ia
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Bloodtest..
Elevated hematocrit (emergentphase)
Decreased RBC count (electricalburns)
Elevated glucose level
elevated BUNelevated creatinine (acute
tubular necrosis,
electrical burns)
UR
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Elevated specific gravity
Color: Dark amber
concentratedBurgundy-colored
presence of
myoglobin
Glucosuria
URINALYSIS
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Indicated if:
positive history of cardiacdisease
Electrical injury
Pulse is dysrhythmic orrate is abnormally slow or
rapid
ECGmonitoring
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ABG
analysisRespiratory acidosis
Metabolic acidosis
Not valid in carbon monoxidepoisoning
Usually normal
Hemoglobin is well saturated
Pulseoximetry
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Carbon monoxide level..
5-10%
11-20%
21-30%
31-40%
41-50%
>50%
Impaired visualacuity
Flushing, headache Nausea, impaired
dexterity
Vomiting, dizziness,syncope
Tachypnea,
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Management of the patient
with Burn injury
mergen
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mergenResuscitative
phase On-the-sceneCareExtinguish theflame
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Cool the burn
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remove restrictive objects
cover the wound
Chemical burns
irrigate
I iti l i iti i
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Initial priorities inthe E.R.
AirwayBreathing
Circulation
If edemadevelops,
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Large bore needle inserted
NGT if nauseated
Indwelling urinary catheter
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Acute or intermediatephase
Continued assessment
Maintenance of respiratory andcirculatory status
Fluid and electrolyte balance
infection prevention
Pain managementNutritional support
Burn wound care
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WOUNDCLEANING
Hydrotherap
y
Topical antibacterial
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Topical antibacterialtherapySilver sulfadiazine(silvadene)
Silver nitrate
Mafenide acetate(sulfamylon)
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Wounddebridement
to remove tissuecontaminated
by bacteria andforeign bodies
To removedevitalized tissue
or
burn eschar inpreparation for
raftin and
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i h i d h i
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Biosynthetic and syntheticdressings
Biobran
e
Dermal
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Dermalsubstitutes
Integr
a
Alloderm
A t ft
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Autografts
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Disorders
ofwound healing
Scars
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Keloids
Contractures
REHABILITAtION
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REHABILITAtIONPHASE
Wound healing, psychologicalsupport, and restoring maximal
functional activity remain priorities
Reconstructive surgery maybe needed
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urs ng
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urs ng
Management
EMERGENT PHASE
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EMERGENT PHASE
INTERVENTIONS RATIONALE
1. Provide humidified oxygen
2. Monitor for signs of hypoxia
and report to the physician
immediately
3. Place patient in semi fowlers
position4. Prepare to assist with
intubation and escharotomies
5. Monitor mechanically
ventilated patient closely
1. Humidified oxygen provides
moisture to injured tissues;
supplemental oxygen
increases alveolar oxygenation
2. To detect worsening of
condition
3. To promote lung expansion4. Intubation allows mechanical
ventilation. Escharotomy
enables chest excursion in
circumferential chest burns
5. It allows for early detection ofdecreasin res irator status
PROBLEM 1
Assessment: dyspnea, shortness of breath, irritability, tachypnea,
tachycardia, restlessness
Nursing diagnosis: Impaired gas exchange related to upper airway
obstruction secondary to smoke inhalation injury
Goal: Maintenance of adequate tissue oxygenation
Assessment: SOB, dyspnea, crackles,
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Assessment: SOB, dyspnea, crackles,tachycardia, tachypneaNursing diagnosis: Ineffective airway clearancerelated to edema and effects of
smoke inhalationGoal: Maintain patent airway and adequateairway clearanceINTERVENTIONS RATIONALE
1. Maintain patent airway
through proper patient
positioning
2. Suction secretions as
needed
3. Provide humidified oxygen
4. Encourage patient to turn,cough, and deep breathe
5. Encourage patient to use
incentive spirometry
1. A patent airway is crucial
to respiration
2. To remove accumulated
secretions if client is
unable to cough
effectively
3. Humidity liquefiessecretions and facilitates
expectoration
4. To mobilize secretions
5. To promote lung
expansion, mobilize
Assessment: tachycardia, weight loss, irritability,
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hypotension, tachypneaNursing Diagnosis: Fluid volume deficit related toincreased capillary permeability andevaporative losses from the burn wound
Goal: Restoration of optimal fluid and electrolytebalance and perfusion of vital organs
INTERVENTIONS RATIONALE
1. Observe vital signs and urine
output, and be alert for signs of
hypovolemia or fluid overload
2. Monitor urine output at least
hourly and weigh patient daily
3. Maintain IV lines and regulate
fluids at appropriate rates, as
prescribed
4. Elevate head of patients bed and
elevate burned extremities
5. Notify physician immediately of
decreased urine output, blood
pressure, or increased pulse rate
1. Hypovolemia is a major risk
immediately after the burn
injury. Overresuscitation might
cause fluid overload
2. Output and weight provide
information about renal
perfusion and fluid status
3. Adequate fluids are necessary to
maintain fluid and electrolyte
balance and perfusion of vital
organs
4. Elevation promotes venous
return
5. Because of the rapid fluid shifts
in burn shock, fluid deficit must
be detected early so that
ACUTE PHASE
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ACUTEPHASE
INTERVENTIONS RATIONALE
1. Monitor vital signs, intake
and output, weight. Assess
for edema, jugular veindistention (JVD), crackles,
increased arterial
pressures
2. Notify physician if urine
output
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Assessment: altered skin integrity, weakness, fullthickness burn injuryNursing diagnosis: Risk of infection related to loss ofskin barrier and impaired immune response
Goal: Absence of localized or systemic infectionINTERVENTIONS RATIONALE
1. Use asepsis in all aspects of
patient care:
a. Meticulous hand hygiene before
and after patient care
b. Use clean or sterile gloves forwound care
c. Wear isolation gown or protective
plastic apron for patient care
d. Wear mask and hair cover when
wounds are exposed and during
sterile procedurese. Change invasive lines and tubings
as recommended
2. Screen visitors for possible
infection
3. Exclude plants and flowers in water
from patients room
1. Aseptic techniques minimize risk
of cross contamination and spread
of bacterial contamination
2. Avoiding known infecting agents
prevents introduction of additionalmicroorganisms
3. Stagnant water is a potential
source of bacterial growth
4. Antibiotics reduce and kill bacteria
5. Increased WBC count indicates
infection. Culture and sensitivityindicate microorganisms present
and what appropriate antibiotics
to be used.
Health teachings
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Health teachings
instruct to wash burned areas
daily with mild soap and waterand to apply the prescribedtopical agent or dressing
Written and verbal instructionsshould be given about
prevention of complications,
pain management, and nutritionGive information about specificexercises to preventcomplications like DVT and
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TheEnd
Thank