162 AMERICAN HEART JOURNAL LEG ULCERS,THEIR CAUSESANDTIUMTMENT. By S. T. Arming, Boston, 19.54, Little, Brown & Company. At first the title of this book is misleading, because it was not until this reviewer began to read it that he realized the extent to which this subject concerns the cardiovascular specialist. The leg ulcers are the last event in a chain of developments most of which fall definitely within the scope of “Cardiology.” The author fully discusses these factors: arterial disease, venous disease, including varicosities, causes of disturbed circulation in the leg, venous thrombosis arising either from changes in the wail of the veins or in the clotting tendencies of the blood. The book is really a comprehensive discussion of circulatory disturbances of the leg in relation to the venous circulation. As such it can warmly be recommended as part of the physician’s library. The book is beautifully written and well arranged, and the publishers are to be congratulated. L'.~PPARATO CARDIOVASCOI~ARE NELI,A BRUCELLOSI (THE CARDIOVASCULAR SYSTEM IN BRUCEL- LOSE). By M. Poli, Pisa, 1953, Edizione Omnia Medica, li9 pages and 6 figures. This monograph analyzes the various cardiovascular lesions caused by Bmcellu melitmsis and the resulting clinical pictures, and tries to establish their relative frequency. Endocardial, myocardial, and pericardial lesions during brucellosis have been repeatedly reported. Out of 166 cases of brucellosis observed by the author, 80 presented evidence of cardiovascular lesions. However, only in 10 could other etiologic causes be definitely excluded. The other patients may have had cardiac lesions due to brucellosis, but previous rheumatic lesions in patients with brucellosis could not be excluded. Endocarditis is usually severe and malignant. It can be diagnosed if the valvular lesions are in evolution and blood cultures reveal only brucellae. However, in one case a mixed etiology, (Str. vKdans and brucella) was demonstrated. In general, the aortic valve is the most common localization. Atrial fibrillation, bundle branch or I.V. block, or S-T and T changes reveal, according to the author, the onset of brucelIar myocarditis. Fifteen autopsied cases revealed endocarditis in 33 per cent, pericarditis in 33 per cent, and myocarditis in 40 per cent. Detailed therapeutic considerations follow. This book is of interest to the internist and the cardiologist. A more clear preseirtation and a better final summarization, however, would have increased its utility. :l.il.L Note In the March, 1955, issue of the AMERICAN HEART JOURNAL (49:367-384) there appeared an article by Doctors A. R. Dawe and P. R. Morrison, entitled “Characteristics of the Hibernating Heart.” At the time this article was written, the authors were unaware of previous work that had been done in this field by Doctor H. S. Samuli Sarajas, of Nortulls Hospital, Stockholm, Sweden, reported in the Acta Physiologica Scandinavica, 1:43, 1954. Dr. Dawe has now asked that a note to this effect appear in the AMERICAN HEART JOURNAL. Editor.

L'apparato cardiovascolare nella brucellosi (The cardiovascular system in brucellosis): By M. Poli, Pisa, 1953, Edizione Omnia Medica, 179 pages and 6 figures

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Page 1: L'apparato cardiovascolare nella brucellosi (The cardiovascular system in brucellosis): By M. Poli, Pisa, 1953, Edizione Omnia Medica, 179 pages and 6 figures

162 AMERICAN HEART JOURNAL

LEG ULCERS,THEIR CAUSESANDTIUMTMENT. By S. T. Arming, Boston, 19.54, Little, Brown & Company.

At first the title of this book is misleading, because it was not until this reviewer began to read it that he realized the extent to which this subject concerns the cardiovascular specialist. The leg ulcers are the last event in a chain of developments most of which fall definitely within the scope of “Cardiology.” The author fully discusses these factors: arterial disease, venous disease, including varicosities, causes of disturbed circulation in the leg, venous thrombosis arising either from changes in the wail of the veins or in the clotting tendencies of the blood. The book is really a comprehensive discussion of circulatory disturbances of the leg in relation to the venous circulation. As such it can warmly be recommended as part of the physician’s library. The book is beautifully written and well arranged, and the publishers are to be congratulated.

L'.~PPARATO CARDIOVASCOI~ARE NELI,A BRUCELLOSI (THE CARDIOVASCULAR SYSTEM IN BRUCEL- LOSE). By M. Poli, Pisa, 1953, Edizione Omnia Medica, li9 pages and 6 figures.

This monograph analyzes the various cardiovascular lesions caused by Bmcellu melitmsis and the resulting clinical pictures, and tries to establish their relative frequency. Endocardial, myocardial, and pericardial lesions during brucellosis have been repeatedly reported. Out of 166 cases of brucellosis observed by the author, 80 presented evidence of cardiovascular lesions. However, only in 10 could other etiologic causes be definitely excluded. The other patients may have had cardiac lesions due to brucellosis, but previous rheumatic lesions in patients with brucellosis could not be excluded.

Endocarditis is usually severe and malignant. It can be diagnosed if the valvular lesions are in evolution and blood cultures reveal only brucellae. However, in one case a mixed etiology, (Str. vKdans and brucella) was demonstrated. In general, the aortic valve is the most common localization.

Atrial fibrillation, bundle branch or I.V. block, or S-T and T changes reveal, according to the author, the onset of brucelIar myocarditis. Fifteen autopsied cases revealed endocarditis in 33 per cent, pericarditis in 33 per cent, and myocarditis in 40 per cent.

Detailed therapeutic considerations follow. This book is of interest to the internist and the cardiologist. A more clear preseirtation

and a better final summarization, however, would have increased its utility. :l.il.L

Note

In the March, 1955, issue of the AMERICAN HEART JOURNAL (49:367-384) there appeared an article by Doctors A. R. Dawe and P. R. Morrison, entitled “Characteristics of the Hibernating Heart.” At the time this article was written, the authors were unaware of previous work that had been done in this field by Doctor H. S. Samuli Sarajas, of Nortulls Hospital, Stockholm, Sweden, reported in the Acta Physiologica Scandinavica, 1:43, 1954. Dr. Dawe has now asked that a note to this effect appear in the AMERICAN HEART JOURNAL.

Editor.