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Chapter 5 The Integumentary System
bull Protects Sensation Vit D Temp regulatn excretn
A Skin
1 Epidermis Roof
Stratified squamous
A Stratum basale
Bottom
Mitosis ~ every 19 days
1 surfaces ~ 40-56 days later
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
A Skin
1 Epidermis Roof
Stratified squamous
A Stratum basale
Bottom
Mitosis ~ every 19 days
1 surfaces ~ 40-56 days later
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
B Stratum Granulosum
Fill whard protein keratin (die)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
C Stratum Corneum
Upper
Protective cells coated wlipid
Top sloughs
Callus friction uarr cell layers
Corn thickened area over bone
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
2 Dermis
House
Collagen (strength)
Cleavage Lines (fiber orientation)
resist stretching
parallel incision heal best
striae stretch marks
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Skin Cont
Few fat cellsmacrophages
Dermal papillae (upper)
Blood vessels (move matlrsquos temp reg)
Shape fingerft print
Grip of handft
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
B Skin Color 1 Melanin
Brnblk pigment molecule
UV protection
Melanocytes Produce melanin vesicles melanosomes Vesicles phagocytized by epidermal cells
Frecklesmoles most
Palmssoles least
of melanocytes
Same for all races
- Skin cells
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Production based on
Genetics races
Albinism = recessive (no melanin)
Hormones pregnancy mask
UV light suntan
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
2 Tattoo bluish hue dermal fibers scatter light
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
3 Cyanosis bluish hue darr bld O2
Cyanosis of nail beds with extreme
pulmonary dysfunction
Irregular heart w Undersized lungs
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
4 Carotene yellowish lipid-soluble pigment (carrots)
5 Jaundice yellow wliver damage - bile in bld
Jaundice Yellow skin discoloration caused by hyperbilirubinemia Anatomy of the liver An obstruction in the bile duct may lead to jaundice
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
6 Birthmark congenital disorder of dermal BV
Capillary Malformation (Port-wine Stain)
Vascular birthmarks are caused by enlarged small blood vessels just beneath the skins surface The most common are known as angels kissesldquowhen located on the forehead or eyelids and as stork bites
when they appear on the back of the neck
Most birthmarks are no risk to health
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
C Hypodermis Foundation
Subcutaneous (under skin)
frac12 body fat (insulatepad) TBF count
Injection site
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
D Accessory Skin Structures 1 Hair
A Shaft (visible)
B Hair root (bulb) in follicle
Dermal papilla nourishes
Dividekeratinize
The cuticle or outer layer
of a healthy shaft of hair
A split end caused when
a hair shaft is pulled apart
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Phases
Growth lash-30 dysscalp-3 yrs
Rest lash 105 dysscalp-1-2 yrs
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
C Pattern baldness sex linked (hormone)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
D Color determined by bulb melanocytes
Age gray -darr activity white - no actvty
How does hair turn gray The melanocytes in hair stop producing melanins
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
2 Muscles arrector pili (smooth)
A Gooseflesh hair perpendicular
B Wfur insulatefierceness
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
3 Glands
A Sebaceous- sebum (oil) to follicle
B Eccrine- sweat (uarr H2O) to skin
C Apocrine- org-sweat to genitalaxillary follicle
Active wpuberty ndash bacterial decay=odor
An apocrine gland which
produces little sweat but is
responsible for the bodys
natural scent
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
E Physiology Temp reg (dermal BV) excretion (urea-sweat)
Vitamin D prodctn in skin wUV
Stimulates CaP uptake SI= musclebone health
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
F Diagnostic
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Burns degree of depth
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Partial-thickness part of basale viable
(regenerate from edges follicles)
1deg epidermis ndash edema no scar
2deg epidermis-blister may scar
The epidermis the outer layer is burned Reddening occurs and swelling is possible
The epidermis is burned through and the dermis is also injured An intense red discoloring is accompanied by severe pain swelling and blisters
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Full-thickness
3deg epidermis destroyed-skin graft
All layers of skin are burned through to the fat muscles and possibly to the bone There may be severe pain but sometimes extensive nerve damage results in little or no pain
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
First Degree Burn
Second Degree Burn
Third Degree Burn
Superficial (epidermal burn) Intermediate
(superficial dermal burn)
Deep (sub-dermal burn)
Fourth degree burn
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Skin Cancer
most common type of cancer UV exposure
Basal cell carcinoma common open ulcer treatable
Massive Ulcerating
Basal Cell Carcinoma
Histology
basal cell
carcinoma
high power
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Squamous cell carc keratinized tumordarr metastasis
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Malignant melanoma rare melanocytes-mole
metastasis common (fatal)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
Aging
darr skin activity amp functn
Age spots wuarr of melanocytes (some areas)
