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Chapter 14

Cardiovascular

Emergencies

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Anatomy and Physiology (1 of 21)

• Heart’s job is to pump blood to supply

oxygen-enriched red blood cells to tissues.

• Divided into left and right sides

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Anatomy and Physiology (2 of 21)

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Anatomy and Physiology (4 of 21)

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Anatomy and Physiology (5 of 21)

• Heart’s electrical system controls heart rate

and coordinates atria and ventricles.

• The cardiac muscle is the myocardium.

•  Automaticity allows cardiac muscles tospontaneously contract. 

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Anatomy and Physiology (6 of 21)

Electrical conduction system of the heart

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Anatomy and Physiology (9 of 21)

• The myocardium must have a continuous

supply of oxygen and nutrients to pump

blood.

• Increased oxygen demand by myocardiumis supplied by dilation (widening) of

coronary arteries.

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Anatomy and Physiology (10 of 21)

• Stroke volume is the volume of blood

ejected with each ventricular contraction.

• Coronary arteries are blood vessels that

supply blood to heart muscle.

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• Coronary arteries start at the first part of the

aorta:

 – Right coronary artery

 – Left coronary artery

Anatomy and Physiology (11 of 21)

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Anatomy and Physiology (12 of 21)

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Anatomy and Physiology (19 of 21)

Carotid Femoral

Brachial Radial Posterior tibial Dorsalis pedis

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Anatomy and Physiology (20 of 21)

• Cardiac output is the volume of blood that

passes through the heart in 1 min.

 – Heart rate× volume of blood ejected with each

contraction (stroke volume)

• Perfusion is the constant flow of oxygenated

blood to tissues

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Anatomy and Physiology (21 of 21)

• Good perfusion requires:

 –  A well-functioning heart

 –  An adequate volume of blood

 –  Appropriately constricted blood vessels

• If perfusion fails, cellular and eventually

patient death occur.

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Pathophysiology (1 of 26)

• Chest pain usually stems from ischemia,

which is decreased blood flow.

 – Ischemic heart disease involves a decreased

blood flow to one or more portions of the heart. – If blood flow is not restored, the tissue dies.

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Pathophysiology (2 of 26)

•  Atherosclerosis is

the buildup of

calcium and

cholesterol in the

arteries.

 – Can cause

occlusion of

arteries

 – Fatty material

accumulates with

age.

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Pathophysiology (4 of 26)

•  A thrombo-embolism is a blood clot

floating through blood vessels.

• If clot lodges in coronary artery, acute

myocardial infarction (AMI) results.

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Pathophysiology (5 of 26)

• Coronary artery disease is the leading

cause of death in the US.

• Controllable AMI risk factors:

 – Cigarette smoking, high blood pressure, high

cholesterol, high blood glucose level (diabetes),

lack of exercise, and stress

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Pathophysiology (6 of 26)

• Uncontrollable AMI risk factors:

 – Older age, family history, and being a male

•  Acute coronary syndrome (ACS) is caused

by myocardial ischemia.

 –  Angina pectoris

 –  Acute myocardial infarction

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Pathophysiology (7 of 26)

•  Angina pectoris occurs when the heart’s

need for oxygen exceeds supply.

 – Crushing or squeezing pain

 – Does not usually lead to death or permanentheart damage

 – Should be taken as a serious warning sign

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Pathophysiology (8 of 26)

• Unstable angina

 – In response to fewer stimuli than normal

• Stable angina

 – Is relieved by rest or nitroglycerin

• Treat angina patients like AMI patients.

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Pathophysiology (9 of 26)

•  AMI pain signals actual death of cells in

heart muscle.

 – Once dead, cells cannot be revived.

 –   ―Clot-busting‖ (thrombolytic) drugs orangioplasty within 1 hour prevent damage.

 – Immediate transport is essential.

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Pathophysiology (10 of 26)

• Signs and symptoms of AMI

 – Weakness, nausea, sweating

 – Chest pain that does not change

 – Lower jaw, arm, back, abdomen, neck pain

 – Irregular heartbeat and syncope (fainting)

 – Shortness of breath (dyspnea)

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Pathophysiology (11 of 26)

• Signs and symptoms of AMI (cont’d)

 – Pink, frothy sputum

 – Sudden death

•  AMI pain differs from angina pain

 – Not always due to exertion

 – Lasts 30 minutes to several hours

 – Not always relieved by rest or nitroglycerin

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Pathophysiology (12 of 26)

•  AMI patients may not realize they are

experiencing a heart attack.

•  AMI and cardiac compromise physical

findings: – Fear, nausea, poor circulation

 – Faster, irregular, or bradycardic pulse

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Pathophysiology (13 of 26)

•  AMI and cardiac compromise physical

findings (cont’d):

 – Decreased, normal, or elevated BP

 – Normal or rapid and labored respirations

 – Patients express feelings of impending doom.

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Pathophysiology (14 of 26)

• Three serious consequences of AMI:

 – Sudden death

• Resulting from cardiac arrest

 – Cardiogenic shock

 – CHF

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Pathophysiology (15 of 26)

•  Arrhythmias:

heart rhythm

abnormalities

 – Prematureventricular

contractions

 – Tachycardia

 – Bradycardia 

Source: From Arrhythmia Recognition: The Art of Interpretation, courtesy of Tomas B. Garcia, MD.

Source: From Arrhythmia Recognition: The Art of Interpretation, courtesy of Tomas B. Garcia, MD.

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Pathophysiology (16 of 26)

•  Arrhythmias

(cont’d)

 – Ventricular

tachycardia – Ventricular

fibrillationSource: From Arrhythmia Recognition: The Art of Interpretation, courtesy of Tomas B. Garcia, MD.

Source: From Arrhythmia Recognition: The Art of Interpretation, courtesy of Tomas B. Garcia, MD.

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Pathophysiology (17 of 26)

• Defibrillation restores cardiac rhythms.

 – Can save lives

 – Initiate CPR until a defibrillator is available.

•  Asystole: no heart electrical activity – Reflects a long period of ischemia

 – Nearly all patients will die.

Source: From Arrhythmia Recognition: The Art of Interpretation, courtesy of Tomas B. Garcia, MD.

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Pathophysiology (18 of 26)

• Cardiogenic shock

 – Often caused by heart attack

 – Heart lacks power to force enough blood

through circulatory system – Inadequate oxygen to body tissues causes

organs to malfunction.

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Pathophysiology (19 of 26)

• Cardiogenic shock (cont’d)

 – Often caused by a heart attack

 – Heart lacks the power to pump

 – Recognize shock in its early stages.

• Congestive heart failure

 – Often occurs a few days following heart attack

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Pathophysiology (20 of 26)

• Congestive heart failure

 – Increased heart rate and enlargement of left

ventricle no longer make up for decreased heart

function – Lungs become congested with fluid

 – May cause dependent edema

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Pathophysiology (21 of 26)

• Hypertensive emergencies

 – Systolic pressure greater than 160 mm Hg

 – Common symptoms

• Sudden, severe headache

• Strong, bounding pulse

• Ringing in the ears

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Pathophysiology (22 of 26)

• Hypertensive emergencies (cont’d)

 – Common symptoms

• Nausea and vomiting

• Dizziness• Warm skin (dry or moist)

• Nosebleed

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Pathophysiology (23 of 26)

• Hypertensive emergencies (cont’d)

 – Common symptoms include altered mental

status and pulmonary edema.

 –If untreated, can lead to stroke or dissectingaortic aneurysm.

 – Transport patients quickly and safely.

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Pathophysiology (24 of 26)

•  Aortic aneurysm is weakness in the wall of

the aorta.

 – Susceptible to rupture

 – Dissecting aneurysm occurs when inner layersof aorta become separated (Table 14-1).

 – Primary cause: uncontrolled hypertension

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Pathophysiology (25 of 26)

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Pathophysiology (26 of 26)

•  Aortic aneurysm (cont’d)

 – Signs and symptoms

• Very sudden chest pain

• Comes on full force• Different blood pressures

 – Transport patients quickly and safely.

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Scene Size-up 

• Scene safety

 – Ensure the scene is safe.

 – Follow standard precautions.

• Mechanism of injury (MOI)/nature of illness

(NOI)

 – Clues often include report of chest pain,

difficulty breathing, loss of consciousness. – Obtain clues from dispatch, the scene,

patient, bystanders

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Primary Assessment (1 of 2)

• Form a general impression.

 – If unresponsive, pulseless, or apneic, assess

for use of AED.

•  Airway and breathing

 – If difficulty breathing: apply oxygen via

nonrebreathing mask; if not breathing: give

100% oxygen via bag-mask device.

 – CHF patients: use CPAP.

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Primary Assessment (2 of 2)

• Circulation

 – Check skin color, temperature, condition.

• Transport decision

 – Transport in a stress-relieving manner.

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History Taking (1 of 2)

• Investigate the chief complaint (eg, chest

pain, difficulty breathing).

 –  Ask about recent trauma.

• Obtain a SAMPLE history from a consciouspatient.

 – Use OPQRST (Table 14-2).

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History Taking (2 of 2)

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Secondary Assessment 

• Physical examination

 – Focus on cardiac and respiratory systems.

• Circulation

• Respirations

• Vital signs

 – Obtain a complete set of vital signs

 –If available, use pulse oximetry

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Reassessment (1 of 4)

• Reassess vital signs every 5 min or when

patient’s condition changes significantly.

• Sudden cardiac arrest is always a risk.

• Interventions

 – Give oxygen.

 –  Assist unconscious patients with breathing.

 – Follow local protocol for administration of low-dose aspirin or prescribed nitroglycerin (see

Skill Drill 14-1).

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Reassessment (3 of 4)

• Interventions (cont’d)

 – If cardiac arrest occurs, perform CPR

immediately until an AED is available.

• Reassess your interventions.

• Provide rapid patient transport.

Heart Surgeries and

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Heart Surgeries andPacemakers (1 of 6)

• Many open-heart operations have been

performed in the last 20 years.

• Coronary artery bypass graft (CABG)

 – Chest or leg blood vessel is sewn from the aortato a coronary artery beyond the point of

obstruction.

• Others have implanted cardiac pacemakers.

Heart Surgeries and

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Heart Surgeries andPacemakers (2 of 6)

• Percutanous transluminal coronary

angioplasty (PTCA)

 –  A tiny balloon is inflated inside a narrowed

coronary artery.• Patients who have had open-heart

procedures may have long chest scar.

Heart Surgeries and

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Heart Surgeries andPacemakers (3 of 6)

• Cardiac pacemakers

 – Maintain regular cardiac rhythm and rate

 – Deliver electrical impulse through wires in direct

contact with the myocardium – Implanted under a heavy muscle or fold of skin

in the upper left portion of the chest

Heart Surgeries and

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Heart Surgeries andPacemakers (4 of 6)

• Cardiac pacemakers

(cont’d)

 – This technology is very

reliable. – Pacemaker malfunction

can cause syncope,

dizziness, or weakness

due to an excessivelyslow heart rate.

• Transport patients

promptly and safely.

Source: © Carolina K. Smith, MD/ShutterStock, Inc.

Heart Surgeries and

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Heart Surgeries andPacemakers (5 of 6)

•  Automatic implantable cardiac defibrillators

(AICDs)

 – Used by some patients who have survived

cardiac arrest due to ventricular fibrillation – Monitor heart rhythm and shock as needed.

 – Treat chest pain patients with AICDs like they

are experiencing a heart attack.

Heart Surgeries and

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Heart Surgeries andPacemakers (6 of 6)

•  AICDs (cont’d)

 – Electricity is low; it

will not affect

responders.

 – When an AED is

used, do not place

patches directly

over pacemaker.

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Cardiac Arrest

• The complete cessation of cardiac activity

•  Absence of a carotid pulse

• Was terminal before CPR and external

defibrillation were developed in the 1960s

Automated External

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Automated ExternalDefibrillation (1 of 14)

•  Analyzes electrical

signals from heart

 – Identifies

ventricularfibrillation

 –  Administers shock

to heart when

neededSource: The LIFEPAK® 1000 Defibrillator (AED) courtesy of Physio-Control.

Used with Permission of Physio-Control, Inc, and according to the

Material Release Form provided by Physio-Control.

Automated External

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Automated ExternalDefibrillation (3 of 14)

•  AED models

(cont’d):

 – Most are

semiautomated – Monophasic vs.

biphasic shocks

•Source: The LIFEPAK® 20e Defibrillator monitor courtesy of Physio-Control.

Used with permission of Physio-Control, Inc, and according to

the Material Release Form provided by Physio-Control

Automated External

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Automated ExternalDefibrillation (4 of 14)

• Potential problems:

 – Check daily and exercise the battery as

recommended by manufacturer.

 – Apply to pulseless, unresponsive patients.

 – Most computers identify rhythms faster than 150

to 180 beats/min as ventricular tachycardia,

which should be shocked.

Automated External

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Automated ExternalDefibrillation (5 of 14)

•  Advantages of AED use:

 – Quick delivery of shock

 – Easier than performing CPR

 –  ALS providers do not need to be on scene

 – Remote, adhesive pads safer than paddles

 – Larger pad area = more efficient shocks

Automated External

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Automated ExternalDefibrillation (6 of 14)

• Other considerations

 – Though all cardiac arrest patients should be

analyzed, not all require shock.

 – Asystole (flatline) = no electrical activity

 – Pulseless electrical activity usually refers to a

state of cardiac arrest.

Automated External

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Automated ExternalDefibrillation (7 of 14)

• Early defibrillation

 – Few cardiac arrest patients survive outside a

hospital without a rapid sequence of events.

 –Chain of survival:• Early recognition and activation of EMS

• Immediate bystander CPR 

Automated External

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Automated ExternalDefibrillation (9 of 14)

• Early defibrillation (cont’d)

 – CPR prolongs period during which defibrillation

can be effective.

 –Has resuscitated patients with cardiac arrestfrom ventricular fibrillation

 – Nontraditional responders are being trained in

 AED use.

Automated External

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Automated ExternalDefibrillation (10 of 14)

• Integrating the AED and CPR

 – Work the AED and CPR in sequence.

 – Do not touch the patient during analysis and

defibrillation. – CPR must stop while AED performs its job.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (1 of 7)

• Preparation

 – Make sure the electricity injures no one.

 – Do not defibrillate patients in pooled water.

 – Do not defibrillate patients touching metal.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (2 of 7)

• Preparation (cont’d)

 – Carefully remove nitroglycerin patch and wipe

with dry towel before shocking.

 –Shave hairy chest to increase conductivity.

 – Determine the NOI and/or MOI.

• Perform spinal stabilization.

• Performing defibrillation

 – See Skill Drill 14-2.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (3 of 7)

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (4 of 7)

• Follow local protocol for patient care after AED shocks.

 –  After AED protocol is completed, one of the

following is likely:• Pulse regained

• No pulse regained and no shock advised

• No pulse regained and shock advised

• Wait for ALS, and continue shocks and

CPR on scene.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (5 of 7)

• If ALS is not responding and protocolsagree, begin transport when:

 – The patient regains a pulse.

 –6 to 9 shocks are delivered.

 –  AED gives three consecutive messages

(every 2 min of CPR) advising no shock.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (6 of 7)

• Transport considerations:

 – 2 EMTs in patient compartment, 1 driving

 – Deliver additional shocks on scene or, if medical

control approves, en route. –  AEDs cannot analyze if vehicle is in motion.

Emergency Medical Care for

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Emergency Medical Care forCardiac Arrest (7 of 7)

• Coordination with ALS personnel

 – If AED available, do not wait for ALS.

 – Notify ALS of cardiac arrest.

 – Do not delay defibrillation.

 – Follow local protocols for coordination.

Review

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Review

1.  All of the following are common signs andsymptoms of cardiac ischemia, EXCEPT:

 A. headache.

B. chest pressure.C. shortness of breath.

D. anxiety or restlessness.

Review

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Review

Answer: A 

Rationale: Cardiac ischemia occurs when the

heart’s demand for oxygen exceeds the

available supply. Common signs andsymptoms of cardiac ischemia include chest

pain or discomfort, shortness of breath

(dyspnea), and anxiety or restlessness.

Headache is not a common symptom ofcardiac ischemia.

Review (1 f 2)

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Review (1 of 2)

1.  All of the following are common signs andsymptoms of cardiac ischemia, EXCEPT:

 A. headache.

Rationale: Correct answer

B. chest pressure.

Rationale: Chest pressure is a definite

symptom of a cardiac event.

Review (2 f 2)

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Review (2 of 2)

1.  All of the following are common signs andsymptoms of cardiac ischemia, EXCEPT:

C. shortness of breath.

Rationale: This is a response to decreased

cardiac output and hypoxia.

D. anxiety or restlessness.

Rationale: This is caused by a fear of death

and is a result of hypoxia, decreased cardiac

output, and ischemia.

Review

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Review

2. While palpating the radial pulse of a 56-year-old man with chest pain, you note that

the pulse rate is 86 beats/min and irregular.

This indicates:

 A. pain.

B. fear.

C. anxiety.

D. an arrhythmia.

Review

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Answer: D 

Rationale: An irregular pulse in a patient with

a cardiac problem suggests an arrhythmia—

an abnormality in the heart’s electrical

conduction system. Patients with signs of

cardiac compromise, who have an irregular

pulse, must be monitored closely for cardiac

arrest.

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Review (1 of 2)

2. While palpating the radial pulse of a 56-year-old man with chest pain, you note that

the pulse rate is 86 beats/min and irregular.

This indicates:

 A. pain.

Rationale: Pain could be a result or symptom

of cardiac ischemia.

B. fear.Rationale: Fear of impending death is usually

the psychological result of having chest pain.

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Review (2 of 2)

2. While palpating the radial pulse of a 56-year-old man with chest pain, you note that

the pulse rate is 86 beats/min and irregular.

This indicates:

C. anxiety.

Rationale: Anxiety is also a symptom of a

cardiac event.

D. an arrhythmia.Rationale: Correct answer

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3.  A 56-year-old man has an acutemyocardial infarction. Which of the

following blood vessels became blocked

and led to his condition?

 A. Coronary veins

B. Coronary arteries

C. Pulmonary veins

D. Pulmonary arteries

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Answer: B 

Rationale: The coronary arteries, which

branch off the aorta, supply the myocardium

(heart muscle) with oxygen-rich blood.Occlusion of one or more of these arteries

results in a cessation of oxygenated blood

beyond the area of occlusion and results in

acute myocardial infarction (AMI).

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Review (1 of 2)

3.  A 56-year-old man has an acutemyocardial infarction. Which of the

following blood vessels became blocked

and led to his condition?

 A. Coronary veins

Rationale: Coronary veins do not carry

oxygen and would not be the direct cause of

an acute MI.

B. Coronary arteries

Rationale: Correct answer

Review (2 of 2)

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Review (2 of 2)

3.  A 56-year-old man has an acutemyocardial infarction. Which of the

following blood vessels became blocked

and led to his condition?

C. Pulmonary veins

Rationale: Pulmonary veins are the primary

blood supply to the lungs and not the heart.

D. Pulmonary arteriesRationale: Pulmonary arteries are the route of

blood return to the left atrium from the lungs.

 An obstruction would not cause an acute MI.

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4. Major controllable risk factors for an AMIinclude:

 A. older age.

B. family history.C. cigarette smoking.

D. male sex.

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Answer: C 

Rationale: Smoking is a major controllable

risk factor for any cardiovascular disease.

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Review (1 of 2)

4.  A major controllable risk factor for an AMIincludes:

 A. Older age.

Rationale: This is an uncontrollable risk

factor.

B. Family history.

Rationale: This is an uncontrollable risk

factor.

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Review (2 of 2) 

4.  A major controllable risk factor for an AMIincludes:

C. Cigarette smoking.

Rationale: Correct answer.

D. Male sex.

Rationale: This is an uncontrollable risk

factor.

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5.  A patient with cardiac arrest secondary toventricular fibrillation has the greatest

chance for survival if:

 A. CPR is initiated within 10 minutes.B. oxygen and rapid transport are provided.

C. defibrillation is provided within 2 minutes.

D. paramedics arrive at the scene within

5 minutes.

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Answer: C 

Rationale: Survival from cardiac arrest

secondary to ventricular fibrillation is highest if

CPR is provided immediately and defibrillationis provided within 1 minute of the patient’s

cardiac arrest. Early high-quality CPR and

defibrillation are the two most important

factors that influence survival from cardiacarrest.

Review (1 of 2)

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Review (1 of 2)

5.  A patient with cardiac arrest secondary toventricular fibrillation has the greatest

chance for survival if:

 A. CPR is initiated within 10 minutes.Rationale: CPR needs to be initiated

immediately.

B. oxygen and rapid transport are provided.

Rationale: Oxygen therapy and transport areimportant parts of resuscitation, but CPR and

early defibrillation afford the greatest chance

of survivability.

Review (2 of 2)

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Review (2 of 2)

5.  A patient with cardiac arrest secondary toventricular fibrillation has the greatest

chance for survival if:

C. defibrillation is provided within 2 minutes.Rationale: Correct answer

D. paramedics arrive at the scene within

5 minutes.

Rationale: Not necessarily; CPR and earlydefibrillation will provide the greatest chance

of survivability.

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6.  A 59-year-old woman presents with chestpressure. She is conscious and alert, but

her skin is cool, pale, and clammy. Your

first step in providing care (treatment)

should be:

 A. apply the AED.

B. administer high-flow oxygen.

C. ask her if she takes nitroglycerin.

D. take a complete set of vital signs.

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Answer: B 

Rationale: Any patient with suspected cardiac

compromise should be given high-flow oxygen

as soon as possible. Obtaining vital signs andinquiring about the use of nitroglycerin are

appropriate; however, you should administer

oxygen first. The AED is only applied to

patients in cardiac arrest.

Review (1 of 2)

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Review (1 of 2)

6.  A 59-year-old woman presents with chestpressure. She is conscious and alert, but

her skin is cool, pale, and clammy. Your

first step in providing care (treatment)

should be:

 A. apply the AED.

Rationale: The AED is only applied to

patients in cardiac arrest.

B. administer high-flow oxygen.

Rationale: Correct answer

Review (2 of 2)

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Review (2 of 2)

6.  A 59-year-old woman presents with chestpressure. She is conscious and alert, but

her skin is cool, pale, and clammy. Your

first step in providing care (treatment)

should be:

C. ask her if she takes nitroglycerin.

Rationale: Inquiring about the use of

nitroglycerin is appropriate, but not the first step.

D. take a complete set of vital signs.

Rationale: Obtaining vital signs is important, but

not the first step.

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7. If a patient with an implanted pacemaker isin cardiac arrest, the EMT should:

 A. avoid defibrillation with the AED and transport

at once.

B. not apply the AED until he or she contacts

medical control.

C. place the AED pads at least 1″ away from the

pacemaker.D. apply the AED pads directly over the

implanted pacemaker.

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Answer: C 

Rationale: The only modification required for

cardiac arrest patients with an implanted

pacemaker is to ensure that the AED pads areat least 1″ away from the pacemaker. Placing

the AED pads directly over the pacemaker will

result in a less effective defibrillation and may

damage the pacemaker.

Review (1 of 2)

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Review (1 of 2)

7. If a patient with an implanted pacemaker isin cardiac arrest, the EMT should:

 A. avoid defibrillation with the AED and transport

at once.

Rationale: AEDs can be used with

pacemakers, but avoid placing pads over

pacemakers.

B. not apply the AED until he or she contacts

medical control.

Rationale: EMTs are trained in the use of

 AEDs.

Review (2 of 2)

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Review (2 of 2)

7. If a patient with an implanted pacemaker isin cardiac arrest, the EMT should:

C. place the AED 1″ away from the pacemaker.

Rationale: Correct answer

D. apply the AED pads directly over the

implanted pacemaker.

Rationale: AEDs should not be placed over

pacemakers.

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8. The main advantage(s) of the AED is:

 A. it provides quick delivery of a shock.

B. it is easier than performing CPR.

C. there is no need for ALS providers to be onscene.

D.  All of the above.

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Answer: D 

Rationale: The AED provides quick delivery

of a shock, is easier to perform than CPR, and

does not require ALS providers to operate it.

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e e ( )

8. The main advantage(s) of the AED is:

 A. it provides quick delivery of a shock.

Rationale: The AED provides quick delivery

of a shock.

B. it is easier than performing CPR.

Rationale: The AED is easier to perform than

CPR.

Review (2 of 2)

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( )

8. The main advantage(s) of the AED is:

C. there is no need for ALS providers to be on

scene.

Rationale: ALS providers do not need to be

on scene to put the AED to use.

D.  All of the above.

Rationale: Correct answer.

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9.  After administering a nitroglycerin tablet toa patient, the EMT should:

 A. check the expiration date of the nitroglycerin.

B. reassess the patient’s blood pressure within 5minutes.

C. instruct the patient to chew the tablet until it is

dissolved.

D. ensure that the nitroglycerin is prescribed tothe patient.

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Answer: B 

Rationale: Nitroglycerin is a vasodilator and

can lower the patient’s BP; therefore, you

should reassess the patient’s BP within5 minutes after giving nitroglycerin. Instruct the

patient to allow the nitroglycerin to dissolve

under his or her tongue; it should not be

chewed. You should check the drug’s expirationdate and ensure that it is prescribed to the

patient before administering it.

Review (1 of 2)

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( )

9.  After administering a nitroglycerin tablet toa patient, the EMT should:

 A. check the expiration date of the nitroglycerin.

Rationale: This is checked before medication

administration.

B. reassess the patient’s blood pressure within

5 minutes.

Rationale: Correct answer

Review (2 of 2)

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( )

9.  After administering a nitroglycerin tablet toa patient, the EMT should:

C. instruct the patient to chew the tablet until it is

dissolved.

Rationale: The tablet is placed under the

patient’s tongue and allowed to dissolve.

D. ensure that the nitroglycerin is prescribed to

the patient.

Rationale: This is checked before medicationadministration.

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10. Nitroglycerin is contraindicated inpatients:

 A. with a systolic blood pressure less than

100 mm Hg.

B. with chest pain of greater than 30 minutes

duration.

C. who are currently taking antibiotics for an

infection.D. who are less than 40 years of age and have

diabetes.

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Answer: A 

Rationale: Nitroglycerin is a vasodilator and

may cause a drop in BP; therefore, it is

contraindicated in patients with a systolic BPof less than 100 mm Hg and in patients who

have taken erectile dysfunction (ED) drugs

(eg, Viagra, Cialis, Levitra) within the past 24

to 36 hours. ED drugs are also vasodilators; ifgiven in combination with nitroglycerin, severe

hypotension may occur.

Review (1 of 2)

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( )

10. Nitroglycerin is contraindicated inpatients:

 A. with a systolic blood pressure less than

100 mm Hg.

Rationale: Correct answer

B. with chest pain of greater than 30 minutes

duration.

Rationale: Cardiac chest pain is the primary

indication for the use of nitroglycerin.

Review (2 of 2)

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10. Nitroglycerin is contraindicated inpatients:C. who are currently taking antibiotics for an

infection.

Rationale: Antibiotics are not a contra-indication for nitroglycerin use. Prior adverse

reactions to the drug and the use of other

nitrates would be a contraindication.

D. who are less than 40 years of age and havediabetes.

Rationale: While the patient’s age must be

considered, it is not a contraindication.

Credits

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• Background slide image (ambulance):Galina Barskaya/ShutterStock, Inc.

• Background slide images (non-ambulance):

© Jones & Bartlett Learning. Courtesy ofMIEMSS.