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Brain stroke by Roel Tolentino Makati Philippines

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Page 1: Brain stroke   by Roel Tolentino Makati Philippines
Page 2: Brain stroke   by Roel Tolentino Makati Philippines
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com·pen·di·umkəmˈpendēəm/Noun

a collection of concise but detailed information about a particular subject, especially in a book or other publication

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Four PrinciplesSTROKE is a brain attack.

STROKE is an emergency.

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Four PrinciplesSTROKE is treatable.

STROKE is preventable.

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DefinitionsStroke

Clinical syndrome of rapid onset of brain deficits of lasting more than 24 hours or leading to death

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DefinitionsTransient Ischemic attack (TIA)

Clinical syndrome of rapid onset of brain deficits which resolves within 24 hours

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DefinitionsProgressive Stroke

A stroke in which the focal neurological deficits worsen with time

Completed StrokeA stroke in which the focal neurological deficits

persist and do not worsen with time

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BRAIN STROKE

Philippines' 2nd cause of death.

Prevalence of 1%

schemic stroke comprises 70%

hemorrhagic stroke comprises 30%

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BRAIN STROKE (PH)

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BRAIN STROKE (PH)

neurologist-to-patient ratio is 1:330,000

67% of neurologists in urban centers

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Anterior Circulation

Posterior Circulation

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Types of Stroke

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Ischemic Stroke80% of strokes

Arterial occlusion of an intracranial vessel

Two etiological typesThromboticEmbolic

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Etiology of ischemic stroke

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Etiology of ischemic stroke

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Etiology of ischemic stroke

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Thrombotic Stroke

Atherosclerosis is the most common pathology leading to thrombotic occlusion of blood vessels

Hypercoagulable disorders

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Etiology of ischemic stroke

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Blood flowIf zero leads to death of brain tissue within 4-

10min

Pathophysiology of Ischemic Stroke

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Intracerebral Hemorrhage• Result of chronic hypertension

• Small arteries are damaged due to hypertension

• In advanced stages vessel wall is disrupted and leads to leakage

• Headache

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CLINICAL FEATURES

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HistoryOnset and progression of neurological

symptoms?

History of previous TIAs

History of hypertension & diabetes mellitus

History of heart conditions like arrhythmias, RHD & prosthetic valves

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History of seizures & migraine

History of anticoagulant therapy

History of oral contraceptive use

History

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History of any hypercoagulable disorders like cancer

Substance abuse: cocaine, amphetamines

History

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General featureabrupt onset of a focal neurologic deficit

manifestations of stroke are highly variable

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DIFFERENTIAL DIAGNOSIS

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Hypoglycemia

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Space Occupying Lesions

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SEIZURES AND POST SEIZURE STATES

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MIGRAINE MIGRAINE

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INVESTIGATION MODALITIES: BRAIN

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ISCHEMIC LESION

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TREATMENT OBJECTIVES

1. MINIMIZE VOLUME OF BRAIN IRREVERSIBLY DAMAGED

2. PREVENT COMPLICATIONS

3. REHABILITATION

4. REDUCE RISK OF RECCURENCE

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REHABILITATIONPHYSIOTHERAPY - as early as

possible - prevents contractures,

spasticity and atrophy

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REHABILITATIONSPEECH THERAPY

IMPROVE QUALITY OF LIFE WITH MOTOR AIDS

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PREVENTIONBlood pressure control

Diabetes Management

Lipid Management

Smoking Cessation

Alcohol Moderation

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PREVENTIONWeight Reduction/Physical

Activity

Carotid Artery Interventions

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PREVENTIONAnti platelet agents /

Anti coagulants

Statins

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PROGNOSISISCHEMIC STROKE

Mortality rate in first 30 days is 8-12%

First 3 hrs after stroke - GOLDEN PERIOD

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PROGNOSIS INTRACEREBRAL HAEMORRHAGE

Mortality rate in first 30 days is almost 50%

Prognosis compared to ischemic type .

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Four PrinciplesSTROKE is a brain attack.

STROKE is an emergency.

STROKE is treatable.STROKE is preventable.

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