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REFERENCESECG
• previous CME talks
• Life In The Fast Lane
• Dr Smiths ECG blog
• Google Image Search
• Time on the Floor
THE NORMAL ECG
• Rhythm: <10% variation in R-R intervals
• Rate: 60 - 100bpm
• Axis -30 - -90
• P Waves
• 0.2-0.3mV
• 0.06 – 0.12s
• Upright in I, II, aVF, V2- V6
• Inverted in aVR
• Varies in III, aVLSinus origin
• PR Interval
• 0.12 – 0.2s
• Q Waves
• Small in I, II, aVL, V5, V6
• QRS Complex
• <0.12s
• ST Segment
• Isoelectric
• T Waves
• <2/3 height of preceding R wave
• 0.5mm in I, II, III
• <10mm in V1 – V6
• Same direction as preceding R wave
• U Waves
• <25% of T wave
• Same direction as T wave
• QTc
• <440ms in males
• <460ms in females
RATE
• Small square: 0.04sec
• Big square: 0.2sec
• If it looks fast
• 1500/small squares
• If it looks slow/normal
• 300/big squares
• If irregular, count complexes on rhythm strip x 6 for average
RHYTHM
• Regular
• Irregular
• Regularly Irregular
• Irregularly Irregular
• P-wave:
• Are they present?
• Are they regular?
• Is there a P for every QRS?
• Do the P-waves look similar?
• PR Interval
• Long? >0.12s
• Short? <0.06s
• Are the PR intervals Consistent?
• QRS
• Long? >0.12s
• Similar in appearance to each other?
• Atrial, AVNRT, Accessory Pathways, Junctional rhythms, Ventricular rhythmas
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? Normal Sinus Rhythm
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? 1st degree Heart Block
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? Atrial Fibrillation
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? Atrial Flutter 4:1 block
SUPRA VENTRICULAR TACHYCARDIASRHYTHM
• A tachydysrhythmia originating above the Bundle of His
• Sinus tachy, Atrial flutter, Atrial Fibrillation
• Atrio-Ventricular Re-Entry Tachycardia (AVRT)
• AV Nodal Re-Entry Tachycardia (AVNRT)
• Automatic Junctional Tachycardia
AVNRT
• Slow and fast pathway within the AV Node
• If a PAC arrives whilst a pathway is refractory, it can allow the other pathway to set up a re-entry pathway within the AV node
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? AVRT - Orthodromic
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? AVRT - Antidromic
VT VS SVT WITH ABERRANT CONDUCTIONRHYTHM
• VT is more likely when
• absence of typical RBBB/LBBB morphology
• Very broad complexes (>160ms)
• AV dissociation
• Fusion beats - when sinus and ventricular beat fuse to cause hybrid complex
• Entirely positive or entirely negative complexes
throughout V1-6
• Brugada’s - Distance from onset of QRS to nadir of S-wave is >100ms
• Josephson’s Sign - notching near nadir of S-wave
• RSR complexes w/ Left Rabbit Ear being taller. In contrast Right Rabbit Ear is taller in RBBB
RHYTHMRegular
?P waves? PR interval? - long, short, consistent?P for every QRS?
QRS? Ventricular Fibrillation
WHAT IS A LICHTENBERG FIGURE?RHYTHM QUIZ
11.
They are branching electric discharges that sometimes appear on the surface or in the interior of insulating
materials.
AXIS
• QRS Positive Deflection = axis toward that lead
• QRS Negative Deflection = axis away from the lead
• QRS Isoelectric = Axis perpendicular to that lead
DIFFERENTIALSAXIS
• RAD• RVH• RV Strain• Lateral STEMI• COPD• HyperK+• Na+ Blockade• WPW• Normal Paeds
ECG
• LAD• LVH• LBBB• Inf. MI• Ventricular
pacing• WPW
• Extreme Axis
• HyperK+• Ventricular
arrhythmias• Severe RVH
WHAT IS COMMOTIO CORDIS?AXIS QUIZ
7.
Commotio cordis (Latin, "agitation of the heart") is an often lethal disruption of heart rhythm that occurs as a result of a blow to the area directly over the
heart (the precordial region), at a critical time during the cycle of a heart beat causing cardiac arrest. It is a form of ventricular fibrillation (V-Fib), not
mechanical damage to the heart muscle or surrounding organs, and not the result of heart disease. The fatality rate is about 65% even with prompt CPR
and defibrillation, and more than 80% without.
THE P WAVE• P Wave
• Width <120ms
• Amplitude
• <2.5mm limb leads
• <1.5mm Precordial
• V1 biphasic
• aVF inverted
THE PR INTERVAL
• PR Interval
• 120-200ms
• Long - any heart block
• Short - WPW, pre-excitation
• Elevation/Depression - Pericarditis
NEGATIVE DEFLECTION BEFORE THE R WAVETHE Q WAVE
• They are pathological if:
• >40ms wide
• >2mm deep
• >0.25% QRS
• Present in leads V1-V3
• >2mm in leads III/avR can be a normal variant
NEGATIVE DEFLECTION BEFORE THE R WAVETHE Q WAVE
• They are pathological if:• >40ms wide• >2mm deep• >0.25% QRS• Present in leads V1-V3
• >2mm in leads III/avR can be a normal variant
FIRST POSITIVE DEFLECTION AFTER THE P-WAVETHE R WAVE
• Causes of a dominant R wave in V1• Normal in paediatric and young adults• RVH• RBBB• Posterior STEMI• WPW• Dextrocardia• HOCM• Dystrophy
• Myotonic• Duchennes
RVH
FIRST POSITIVE DEFLECTION AFTER THE P-WAVETHE R WAVE
• Dominant R wave in aVR• Poisoning - Na-Channel Blockers• Dextrocardia• Incorrect lead placement• Commonly in VT TCA poisoning
FIRST POSITIVE DEFLECTION AFTER THE P-WAVETHE R WAVE
• Poor R Wave Progression - R wave <4mm in V3• Prior anteroseptal MI• LVH• Inaccurate lead placement• Can be normal variant
THE QRS
• LBBB - depolarisation activated from RV via right bundle then to LV via left bundle
• RBBB - Right ventricular depolarisation is delayed and so depolarisation spreads from left to right
BUNDLE BRANCH BLOCK
LEFT VENTRICULAR HYPERTROPHYTHE QRS
• Muscle wall thickens
• Increased S wave in Right sided leads
• Increased R wave in Left sided leads
• Prolonged depolarisation
• Repolarisation abnormalities in lateral leads
• Causes
• HTN
• AR/AS
• MR
• HOCM
RIGHT VENTRICULAR HYPERTROPHYTHE QRS
• Right Axis
• Dominant R in V1 >7mm
• Dominant S in V6 >7mm
• RV Strain - ST depression in V1-4 and inferiorly
• Causes
• PE
• Chronic Lung Dx
• Pulm. HTN
• Mitral Stenosis
• Congenital Heart Dx
THE HEART OF A BLUE WHALE IS ABOUT AS BIG AS A…?
QRS QUIZ7.
A. Volkswagen BeetleB. Piano
C. Average PersonD. Bus
THE HEART OF A BLUE WHALE IS ABOUT AS BIG AS A…?
QRS QUIZ7.
A. Volkswagen BeetleB. Piano
C. Average PersonD. Bus
AN OCTOPUS HAS HOW MANY HEARTS?ST QUIZ
8.
3!!!Two brachial hearts on either side of the body that oxygenate blood by pumping it through the gills
One systemic heart pumps blood from gills to the rest of the body
START OF THE Q TO THE END OF TYOU QT
• Represents Ventricular de- and repolarization
• Inversely proportional to heart rate
• 440ms for males, 460 for females
• QT >500ms increases risk of TDP
VENTRICULAR REPOLARIZATIONT WAVES
• Can be inverted in V1 and aVR
• Flat, Biphasic, Inverted, Peaked, Tented
• Look for dynamic change
• Don’t miss Wellen’s Syndrome
• Hyperacute - early STEMI, prinzmetal Angina
• Inverted T - can be normal, MI, BBB, Hypertrophy, PE, HOCM
• Biphasic - Ischaemia, Hypokalaemia
WELLENS’ SYNDROMET WAVES
• Inverted or Biphasic T-waves in V2-3 (in patients presenting with ischaemic chest pain) highly specific for critical stenosis of LAD
Type A Type B
DE WINTERS - LAD OCCLUSIONT WAVES
• Peaked anterior T waves with ascending limb of T wave commencing below isoelectric line - tall, prominent, symmetric
• Upsloping ST depression in precordial leads
U WAVEOTHER WAVES
• Thought to be delayed depolarisation of Purkinje Fibers
• Prominent U waves: Bradycardia, Hypo-Ca/Mg, Hypothermia, LVH, HOCM, Digoxin, Class Ia, III antiarrhythmics
• Inverted U waves: CAD, HTN, Valvular HD, Congenital HD, Hyperthyroid
OSBORNE WAVEOTHER WAVES
• Positive deflection at J point
• Hypothermia, Normal Variant, HypercalcaemiaHypothermia Temp 32
WHAT IS A SHARKS SIXTH SENSE?BONUS POINTS: NAME THE ORGAN THAT DETECTS THIS
QUIZ TIME8.
They can sense electricity!
Ampullae of Lorenzini