Upload
sachiko-yosores
View
1.221
Download
6
Embed Size (px)
DESCRIPTION
NCM106
Citation preview
1. Discuss different Acute Biologic Crisis conditions together with the roles and responsibilities of the nurse in the care of the following.
•Cardiac failure - Acute Myocardial infarction• Acute pulmonary failure • Acute renal failure • Stroke • Increased Intracranial pressure• Metabolic emergencies – e.g. DKA/HHNK• Massive Bleeding • Extensive surgeries• Extensive Burns • Emerging illnesses (SARS, Avian Flu)• Multiple injuries
2. Use critical thinking in the management of these cases3. Familiarize with the different treatment modalities and equipments used
Acute Biologic CrisisCondition that may result to patient
mortality if left unattended in a brief period of time.
Condition that warrants immediate attention for the reversal of disease process and prevention of further morbidity and mortality.
1. Coronary Artery Disease & Acute Coronary Syndromes
Most Common cause of cardiovascular disability and death.
It refers to a spectrum of illnesses that range from the least life threatening to the most life threatening acute coronary syndrome(AMI/ Heart attack).
Coronary Artery Disease & Acute Coronary Syndromes
Incomplete occlusion of the coronary arteries lead to Angina (ischemia)
Complete occlusion of the coronary arteries lead to Myocardial Infarction
The heart will pump harder to meet the O2 demand leading to Congestive Heart Failure.
Non Modifiable Risk Factors of CAD/ ACSAgeGenderRaceHeredity
Modifiable Risk Factors of CAD/ ACSStress
Diet
ExerciseCigarette SmokingAlcohol
Hypertension
norepinephrine
tachycardia
vasoconstriction
Na, cholesterol & fat CVD
Circulation, maintains vascular tone& enhances release of chemical activators that prevent blood clotting
Vasoconstriction & spasm of arteries.
Myocardial demand
20 ml = vasodilation 30 ml = vasoconstriction
As a result of Systemic vascular resistance
Modifiable Risk Factors of CAD/ ACS
HyperlipidimiaDiabetes MellitusObesityPersonality Type or
Behavioral FactorsContraceptive Pills
Accumulation of fatty plaques
Glucose cannot be transported into the cells due to insulin insufficiency or Increases resistance to insulin
Increase cardiac workload
Type A – competitive, impatient, aggressive has been correlated to CAD
Cardiovascular AssessmentChest PainMost commonDue to Ischemia or MIPrecipitated by stress or can be relieved by
Nitroglycerin (NTG)In MI, it is more intense, unrelated to
activities and can’t be relieved by NTGIf it occurs during breathing, suspect
respiratory problems
Rough diagram of pain zones in myocardial infarction (dark red = most typical area, light red = other possible areas, view of the chest).
Cardiovascular AssessmentDyspneasubjective feeling (inability to get enough
air).Dyspnea on exertion is due to increased O2
myocardial demand.Orthopnea is related to blood pooling in the
pulmonary bed; suspect Pulmonary EdemaAny sudden or acute dyspnea may be a sign
of Pulmonary Embolism
Tightness of Chest
Cardiovascular AssessmentCough/sputumMucoid and foamy sputum can be a sign of
CHFPink-tinged frothy appearance may signal
Pulmonary Edema.Whitish, viral infectionChange in color other than the above
mentioned may signify bacterial infection.
Cardiovascular AssessmentCyanosisBluish discoloration of the skin and
mucous membraneSat O2 is below 90%
FatigueMay be due to Anemias or related to
decreased Cardiac Output
Cardiovascular AssessmentPalpitationsAwareness of rapid or irregular heart beatAutonomic Nervous System and Adrenal
Glands response (stress)
SyncopeTransient loss of consciousnessDue to decreased cerebral tissue perfusion
Cardiovascular AssessmentEdemaDue to: Increased Hydrostatic Pressure
(HP)Decreased Colloidal Oncotic
Pressure (COP)Obstructed Lymphatic or
Vascular System Related to Inflammatory reaction
Types of EdemaBilateral edema
= CHF or Renal FailureUnilateral edema
= Vascular or Lymphatic obstruction
Non-pitting edema= Inflammatory
Pitting edema= HP and
COP derangement
Cardiovascular AssessmentSkinColor, temperature, hair growth,
nails, capillary refillspooning of fingers /clubbing of
fingers
Clubbing of Fingers
Cardiovascular AssessmentHeart rate – 60-100Rhythm – regular or irregularBruits and Thrills – murmurlike; vascular
in origin
- palpate a thrill, auscultate a bruitBlood PressureJugular venous pressure
Cardiovascular AssessmentCardiac rate and rhythmTachycardia = ↑ 100 beats/minuteBradycardia = ↓ 60 beats/minuteArrhythmias = irregular rate and
rhythm
Cardiovascular AssessmentMurmurs- turbulence of blood flow; if positive
watch out for FVE; normal until 1 year oldPericardial Friction Rub -“squeaking sound”;
suspect pericardial effusion if this is heardMuffled Heart Sound - if positive rule out
Cardiac Tamponade and other similar problems like Effusion
Laboratory & Diagnostic TestComplete Blood Count- RBC suggest tissue
oxygenation.
Elevated WBC may indicate infectious heart disease and MI.
Erythrocyte Sedimentation Rate (ESR)- Its is elevated in infectious heart disorder or MI.
Normal range: Males: 15-20mm/hr
Females: 20-30 mm/hr
Laboratory & Diagnostic TestBlood Coagulation Test:
1.Prothrombin Time (PT, Pro Time)- It measures time required for clotting to occur. Used to evaluate effectiveness of COUMADIN. Normal range 11-16 secs.
2.Partial Thromboplastin Time (PTT)- Best screening test for disorders of coagulation. Used to determine the effectiveness of HEPARIN. Normal Range: 60-70 secs.
Laboratory & Diagnostic TestBlood Urea Nitrogen (BUN)- Indicator of
renal function
Normal Range: 10-20mg/dl (5-25mg/dl is also accepted).
Blood Lipids:
1.Serum Cholesterol: 150-200mg/dl
2.Serum Triglycerides: 140-200mg/dl.
Laboratory & Diagnostic TestSerum Enzymes Studies
1.Aspatate Aminotransferase(AST)- Elevated level indicates tissue necrosis. Normal Range: 7-40mu/ml
2.CK-MB- Elevated 4-6hrs from the onset of infarction; peaks 24-36 hrs. returns to normal 4-7 days.
Normal Range: males: 50-325mu/ml; Females: 50-250mu/ml
Laboratory & Diagnostic TestSerum Enzymes Studies
3. Lactic Dehydogenase (LDL)- Onset: 12hrs; Peak: 48hrs; returns to normal: 10-14 days
4. Hydroxybuterate Dehydroxynase (HBD)- it is valuable in detecting silent MI because it is elevated for a long period of time.
Onset: 10-12hrs; Peaks: 48-72hrs; Returns to Normal 12-13 days
Laboratory & Diagnostic TestSerum Enzymes Studies
5. Troponin- Most specific lab test to detect MI. Troponin has 3 compartments: I,C, &T .
Troponin I persist for 4-7 days.
Angina Myocardial Infarction
Chest Pain- tightness & heaviness
Severe crushing, stabbing chest pain
Relieved quickly:3-15min by rest or sublingual nitrogen.
Not relieve by rest and medication
Initiated by physical exertion or stress
Pain last longer >20min
Radiation may or may not be present
May or may not have radiation of pain
Frequently associated with shortness of breath
Laboratory & Diagnostic TestSerum Electrolytes/ Blood Chemistry:
1.Sodium (Na)
2.Potassium (K)
3.Calcium (Ca)
4.Magnessium (Mg)
5.Glucose
6.Glycosylated Hemoglobin (Hemoglobin A1c)
Laboratory & Diagnostic TestECG/ EKG- ST segment elevation and T
wave inversion
Diagnostic TestRadiologic Findings
Chest X-RayNormalCardiomegalySigns of CHF
Diagnostic TestHemodynamic Monitoring
Swan-Ganz CatheterizationRight side of the heartPulmonary artery pressurePulmonary artery occlusive pressureRight atrial pressureCardiac output
Swan-Ganz Catheterization
Diagnostic TestCoronary Angiogram
allows to visualize narrowings or obstructions
therapeutic measures can follow immediately.
Goal:Pain reliefReduction of myocardial oxygen consumption
Prevention and treatment of complications
InterventionAdmit to the CCU/ ICUActivity
Day 1: bed rest, if stableDay 2-3: bed rest, but patient
may be allowed to sit on a chair for 15-20 minutesEarly mobilization is
recommended for uncomplicated AMI
InterventionMonitoring Vital Signs
First 6 hours- q30-60 minutesNext 24 hours- q 2 hoursThereafter q 4 hours
DietNPO: 1st 24 hoursIf stable low salt, low cholesterol
diet
InterventionIV Fluids
D5W to KVOIf unable to take food/fluid per orem1000ml/8 hours
K supplement
InterventionPain Medication
Morphine SO4 (2-5mg/IV dose)Potent analgesicPeripheral venous vasodilationPulmonary venous distentionInferior wall MI: may increase vagal
discharge
TranquilizresTo decrease anxietyDiazepam (5-10 mg per IV/orem)
LaxativeTo prevent straining during defecation
Lactulose (HS)
Drugs to Limit Infarct SizeBeta Blockers
Hyperdynamic states, HPN w/o evidence of heart failure
Reduce myocardial oxygen consumption by decreasing: BP. Heart Rate, Myocardial Contractility and calcium output.
Ex: Propranolol, Metoprolol, Atenolol
Nursing Consideration:
1.Assess Pulse Rate before administration; withhold if bradycardia is present.
2.Administer with food, may cause GI upset.
3.Do not administer with asthma it causes Bronchoconstriction.
4.Do not give to patient with DM, it causes hypoglycemia.
5.Antidote for Beta Blocker poisoning is Glucagon
NitratesAct by augmenting perfusion at the border
of ischemic zone.Generalized vasodilationReducing myocardial O2 demand
Lowering preloadLowering afterload
Ex: IV Nitroglycerine, Sublingual Niotroglycerine, Oral/Transdermal Nitroglycerine
Nursing Considerations:
1.Only a maximum of 3 doses at 5 min. interval.
2.Offer sips of water before giving it sublingually.
3.Store the medication in a cool, dry place; use dark /amber container.
4.If side effects is noticed do not discontinue the drug this is usual in the first few doses of medication.
5.Rotate skin sites for nitro patch.
ACE inhibitors reduce mortality rates after MI. Administer ACE inhibitors as soon as
possible ACE inhibitors have the greatest benefit in
patients with ventricular dysfunction. Continue ACE inhibitors indefinitely after
MI. Angiotensin-receptor blockers may be used
as an alternative adverse effects, such as a persistent cough,
Aspirin and/or antiplatelet therapy
Continue aspirin indefinitelyClopidogrel may be used as an alternative only if resistance or allergy to aspirin.
Nursing Considerations:
1.Assess for signs and symptoms of Bleeding.
2.Avoid straining at stool to avoid rectal bleeding.
3.It should be given with food.
4.Observe for toxicity- Tinnitus (ringing of ears).
5.May cause Bronchoconstriction- Observe for wheezing.
Heparin
1.Assess for S/S of Bleeding.
2.Keep Protamine Sulfate available.
3.If used SQ. do not aspirate to prevent hematoma formation.
4.Monitor for PTT or APTT
5.Used for a maximum of 2 weeks.
Coumadin (Warfarin Sodium)
1.Assess for bleeding
2.Keep Vitamin K available.
3.Monitor for Prothrombin Time
4.Do not give together with aspirin to prevent bleeding.
5.Minimize green leafy vegetables in the diet.
thombolytic therapyThe effectiveness:
highest in the first 2 hoursAfter 12 hours, the risk associated with thrombolytic
therapy outweighs any benefitcontraindicated
unstable angina and NSTEMIand for the treatment of individuals with evidence of
cardiogenic shockstreptokinase, urokinase, and alteplase (recombinant
tissue plasminogen activator, rtPA), reteplase, tenecteplase
Surgical Care
Percutaneous Transluminal Coronary Angioplasty-treatment of choice
PCI provides greater coronary patency lower risk of bleedingand instant knowledge about the extent
of the underlying disease.A specially designed balloon – tipped
catheter is inserted uder flouroscopic guidance and advance to the site of the obstruction.
Intravascular StentingBiologic Stent is produced through
coagulation of collagen, ellastin and other tissues in the vessel wall by laser, photocoagulation or radio frequency.
It is done to prevent restenosis after Percutaneous Transluminal Coronary Angioplasty.
Emergent or urgent coronary artery graft bypass surgery (CABG) is indicatedangioplasty fails Severe narrowing of 1
or more coronary artery.
Commonly used: Saphenous vein and internal mamary artery.
ComplicationsInflammationMechanicalElectrical abnormalities
Cardiac RehabilitationA process which a person restored to
health and maintains optimal physiologic, psychosocial and recreational functions.
Begins with the moment a client is admitted to the hospital for emergency care, it continues for months and even years after the client is discharged from the health care facility.
Goals of Rehabilitation:
1.To live as full, vital and productive life as possible.
2.Remain within the limits of the heart’s ability to respond to activity and stress.
Activities: Exercise may gradually
implemented from the hospital onwards.
Exercise session is terminated if any one of the following occurs: cyanosis, cold sweats, faintness, extreme fatigue, severe dyspnea, pallor, chest pain, PR more than 100/ min., dysrhythmias greater than 160/95mmHg.
Teaching and CounselingSelf management education guide.
Control hypertension with continued medical supervision.
DietWeight reduction programProgressive exerciseStress management techniquesResumption of sexual activity
after 4-6 weeks from discharge, if appropriate.
Teaching guide on resumption of sexual activities:
Assume less fatiguing position.The non- MI partner take the active roleTake nitroglycerine before sexual activityIf dyspnea, chest pain or palpitations
occur, moderation should be observed; if symptom persist stop sexual activity.
Develop other means of sexual expression.
ACUTE RENAL FAILURE Rapid onset of oliguria (<400 ml /day) , with severe rise in BUN & creatinine (Azotemia – accumulation of nitrogen in blood )
Acute renal failure is classified as pre renal, intra renal or post renal. All conditions that lead to pre renal failure impair blood flow to the kidneys (renal perfusion), resulting in a decreased glomerular filtration rate and increased tubular resorption of sodium and water. Intra renal failure results from damage to the
Onset – 1-3 days with ^ BUN and creatinine and possible decreased UOP
Oliguric – UOP < 400/d, ^BUN,Crest, Phos, K, may last up to 14 d
Diuretic – UOP ^ to as much as 4000 mL/d but no waste products, at end of this stage may begin to see improvement
Recovery – things go back to normal or may remain insufficient and become chronic
Complications ARF
Hyperkalemia – most dangerous complication, may lead to cardiac arrest if rise in K+ is too fast
Nursing Care ARF
Daily WeightCVP monitoringDiuretic as prescribedLow protein, K,Na &
high carbohydrate diet
Nursing Care ARFEmergency
management of Hyperkalemia : insulin & dextrose Kayexalate enema
Chronic Renal failure
Chronic irreversible progressive reduction of functioning renal tissue
Common causes CRFDiabetic nephropathyHypertensive nephropathyGlomerulonephritisChronic pyelonephritis
Stages CRF1. Reduced Renal Reserve high
BUN no clinical symptoms yet
2. Renal insufficiency- mild Azotemia – impaired urine concentration , nocturia
Stages CRF3. Renal failure – Severe
azotemia, acidosis,concentrated urine, severe anemia & electrolyte imbalances
CRF systemic SSHyper K, Hypernatremia, Hypocalcemia
AnemiaAnorexia, nausea & vomiting
CRF systemic SSAmmoniacal breathImmunosuppressionHTN, CHFPulmonary edemaSevere pruritusPeripheral neuropathyUremic amaurosis
Nursing Care ESRDLow Protein, Low Na dietPrepare client for peritoneal / hemodialysis
Monitor Anemia
Nursing Care ESRDAdminister epoietin alpha (Epogen), diuretics, antihypertensives as prescribed
Kidney transplant
Peritoneal DialysisPeritoneal Dialysis
Peritoneal DialysisPeritoneal Dialysis
HemodialysisHemodialysis
HEMODIALYSIS: Is the diffusion of dissolved particles from the blood into the dialysate bath of the hemodialysis machine across the semipermeable membrane of the dialyzer.
Hemodialysis requires vascular access:
Subclavian vein/ Femoral vein (temporary)
Arteriovenous fistula, arteriovenous shunt,/ arteriovenous graft
( Permanent)
HemodialysisHemodialysis
Hemodialysis
Nursing Management: Assess the integrity of the hemodialysis access site
Monitor VSAssess client for fluid overload
Nursing Management:
Weigh the client before and after the dialysis treatment ( to determine fluid loss)
Hold meds that can be dialyzed off
Monitor for SS of Shock & Disequilibrium syndrome
Complication: Disequilibrium Syndrome – is the rapid change in composition of extracellular fluid where the solutes of the blood are removed from the blood faster than that of the CSF, causing osmotic movement of fluid into the CSF causing cerebral edema.
Nursing Management: Disequilibrium syndrome:
Assess for Nausea & vomitingAssess for headacheRestlessness, agitation & or confusion
Watch out for seizures
Nursing Management: Disequilibrium syndrome:
Notify physician if SS of disequlibrium syndrome occurs
Reduce environmental stimuliDialyze the patient at a shorter period
and at a slower rate
Kidney Transplant
Cell destruction of the layers of the skin and resultant depletion of fluid and electrolytes
Types of BurnsThermal : exposure to flameChemical: exposure to strong acids or alkali
Electrical: Caused by electrical strong electrical current results in internal tissue injury
Burn Depth:
Superficial thickness burn (1st degree)- mild to severe erythema of skin, blanches with pressure – heals in 3-7 days
Partial thickness burn(2nd degree) – large blisters; painful heals 2-3 weeks
Burn Depth:
Full thickness burns (3rd degree) – white yellow deep red to black (eschar) disruption of blood flow, no pain; scarring and wound contractures will develop. Grafting is required; healing takes weeks to months
Burn Depth:
Deep full thickness burn (4th degree) – Involves injury to muscle and bone= appears black(eschars) – hard and inelastic healing takes weeks to months; grafts are required
Nursing Diagnosis
Decreased Cardiac output Related to Fluid shifts
Rule Of 9
Head and neck 9%Anterior trunk 18% ( chest-9 abdomen-9)
Posterior trunk-18%
Rule Of 9Arms 9% each (forearms only or upper arms only 4.5%)
Legs – 18% each Perineum-1%
Rule of 9
PARKLAND (BAXTER) FORMULA FOR FLUID REPLACEMENT
4ml Lactated Ringer’s sol x Kg body mass x total percentage of body surface burned
PARKLAND (BAXTER)
•1st 8 hours = ½ of total 24 hour fluid replacement
•next 8 hours = ¼ of total
•last 8 hours= ¼ of total
A man Suffered from a 3rd degree burn involving the head and neck, front of the torso (chest & abdomen), and whole left arm. Weight is 50 kg
Calculate the: TBSA burned
24 hour fluid replacement in ml1st 8 hours fluid replacement
2nd 8 hour remaining 8 hour
TBSA:
Head & neck= 9%
front of torso = 18% Whole left arm = 9%
TBSA burned 36%
24 hour replacement: Parkland Baxter formula
4mlX 50 kgs x (TBSA)36%
= 7200 ml
1st 8 hours :7200 ml
2
= 3600 ml = 1st 8 hours
2nd 8 hours & remaining 8 hours respectively :
3600 ml 2
= 1800 ml = 2nd 8 hours= 1800 ml = last 8 hours
MANAGEMENT OF BURNS:Administer fluids as prescribedMaintain a high calorie, high
protein dietMonitor intake and outputMonitor for infections of burn site
Burn Medications:Nitrofurazone ( Furacin) – broad spectrum antibiotic ointment or cream – used when bacterial resistance to other drugs is a problem : apply 1/16 inch thick film directly to burn
Burn Medications:Mafenide ( Sulfamylon) – water
soluble cream bacteriostatic gr + - bacteria- apply 1/16 inch directly to burn – notify physician if hyperventilation occurs as this drug may ppt. metabolic acidosis.
Burn Medications:Silver Sulfadiazene
( Silvadene) – cream Broad spectrum to gr+ - ; does not cause metabolic acidosis – keep burn covered at all times with Sulfadiazine – (1/16 inch thick);
Monitor CBC – causes leukopenia
Burn Medications:Silver Nitrate – Antiseptic solution against gr-, dressings are applied to the burn and then kept moist with Silver nitrate ; used on extensive burns that may precipitate fluid and electrolyte imbalance.
DKA( Diabetic Ketoacidosis) / HHNS
( Hyperglycemic
Hyperosmolar Nonketotic Syndrome)
DKA- Is a life threatening complication of DM type 1 = develops because of severe insulin deficiency
MANIFESTATATIONS
1. Hyperglycemia
2. Dehydration
3. Electrolyte loss and acidosis
CAUSE; Missed insulin dose, or infection
HHNS- SIMILAR TO dka WITH EXTTREME hyperglycemia except that in HHNS there is no acidosis. This is for DM type 2
ASSESSMENT: Blood glucose – 300 – 800 mg/dl
Low bicarbonate & low pH
Dehydration
ASSESSMENT: Mental status changesNeurological deficitsSeizures
NURSING INTERVENTION:Administer Insulin IV push 5-10 units
1st then IV infusion
NURSING INTERVENTION:Restore Fluids ( administer fluids as
prescribed)Treat dehydration w/ rapid infusion of NSS or .45% saline
when blood glucose reaches 250-300 mg/dl D5NS, or D5 .45%Saline is used
NURSING INTERVENTION:Always use infusion pump for IV
insulinMonitor serum potassium ( initially
as a result of acidosis Hyperkalemia is present upon admin of insulin K+ level drops)
NURSING INTERVENTION:Monitor LOC= too rapid decrease in
blood glucose may cause cerebral edema
THYROID CRISIS – (THROID STORM/ Thyrotoxicosis)- Acute life threatening condition that occurs in a client with uncontrollable hyperthyroidism – maybe a result of manipulation of thyroid gland during surgery(release of thyroid hormones to bloodstream)
THYROID CRISIS – (THROID STORM/ Thyrotoxicosis)-
Causes: Undiagnosed , untreated hyperthyroidism, infection, trauma
Medical management:Antithyroid medications; beta blockers; glucocorticoids & iodides are given before surgery to prevent thyroid crisis
Medical management:Antithyroid meds: Iodide, Propylthiouracil, Methimazole
Iodides/ Iodine = Reduce the vascularity of the thyroid gland before thyroidectomy,
Medical management:Iodides= used in the treatment of
thyroid storm because it enables the storage of TH in the thyroid gland.
However it is given only for 10-14 days Because eventually it looses its effect on the thyroid gland.
NURSING INTERVENTION:ASSESSMENT : elevated Temp
( high fever); tachycardia; agitation; tremors
Maintain a patent airway
Administer antithyroid meds as prescribed ( sodium iodide solution)
Monitor VS
MULTI ORGAN DYSFUNCTION SYNDROME
(MODS)
SEPSIS, DEAD TISSUE, PNEUMONITIS, PANCREATITIS
RESPIRATORY FAILURE
INTUBATION (maybe stable for 7-14 days)
MALFUNCTION of GI
SEEDING OF BACTERIA FR. GI TO OTHER ORGANS
HYPERMETABOLIC STATE
HYPERMETABOLIC STATE (hyperglycemia, hyperlactacidemia, ulceration in GI- seeding of bacteria from GI to other organs)(skin breakdown, loss of muscle mass, delayed healing of surgical wounds)(mortality rate 60%)
LIVER FAILURE (jaundice)
RENAL FAILURE(mortality rate 90-100%)
Criteria for Diagnosis of MODS
Cardiovascular Failure presence of 1 or more of the ff:
<54 bpmSystolic < 60 mm HgVtach/ V fibpH < 7.24
Respiratory FailureRR < 5/min RR> 49/min
Renal Failure presence of 1 or more of the ff:Output < 479 ml/24 hr or < 159 ml/ 8 hrBUN > 100mg/dlCrea > 3.5mg/dl
Hematologic Failure presence of 1 or more of the ff:
WBC < 1000 uLPlatelets < 20,000HCT < 20%
Hepatic failure presence of both of the FF:
Bilirubin > 6 mg %PT > 4 sec over control in absence
of anticoagulation (normal PT – 11-12sec)
Neurologic Failure
GCS < 6 in absence of sedation
Medical Management: Control of infection w/ antibiotics
( common MRSA & Vancomycin resistant
Aggressive pulmonary care mech vent & O2 (intubation)
Enteral (NGT) feeding
Nursing Management
Limited : effective client & family coping
The only way to keep your health is to eat what you don't want, drink what you don't like, and do what you'd rather not.
Mark Twain