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8/18/2019 Vision Experiment
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8/18/2019 Vision Experiment
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VISIONSosa, Diane
Visual Field
Visual Acuity Train Nystagmus
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PERIMETRY
a visual feldtest o eacheye
To diagnoseblindness in
specifc portiono the retina
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P!"#T!$ %&A!T
'FT $ !"(&T $
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VISUALFIELD
Visual area seen bythe eye at a giveninstant)
NASA' F"'D *FV"S"*N
Area seen on thenasal side
'AT!A' F"'D *FV"S"*N
Area seen on thelateral side
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The normalextent of eld ofv!on
+-nasally)
.-superiorly
/-ineriorly )
0-
temporally
/
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FA%T*!S T&AT '"#"TS V"S1A' F"'D
• Superiorly2 – !oo o the *rbit,
Forehead, and
(labella)• #edially2
– Nose
• "neriorly2 – %hee3 bones
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V"S1A' F"'D DF%TS
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%auses2
optic atrophy
indirect opticneuropathy
acute optic neuritistraumatic avulsiono optic nerve)
%haracterised by2complete blindness in
a4ected eye 5ith losso both direct onipsilateral 6concensual lightre7e8 on
contralateral side)Near re7e8 ispreserved)
g) !ight opticnerve
involvement
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Le!on! thro'(h)roxmal )art ofo)t* nerve %
ipsilateral
blindness) contralateralhemianopia
abolition o directlight re7e8 on
a4ected side 6concensual lightre7e8 oncontralateral side)
near re7e8 intact)
g) !t optic nerve
"nvolvement in
Pro8imal part
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causes2 suprasellar aneurysm
tumors o pituitarygland
craniopharyngioma
suprasellarmeningioma 6 glioma o9rd ventricle)
third ventriculardilatation due to
obstructivehydrocephalus)
chronic chiasmalarachnoiditis)
$hara*ter!ed -.%
:itemporalhemianopia
:itemporalhemianopic
paralysis o pupillary
re7e8) ;usually lead topartial descending optic
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/#Lateral *ha!malle!on! %
causes2
•
Distension o 9rd
ventricle causingpressure on each sideo optic chiasma
•Atheroma o carotids6 posterior
communicating artery)
%haracterised by
•:inasal hemianopia
•:inasal hemianopic
parallysis opupillary re7e8;usually lead to partialdescending opticatrophy<
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tra*t %
%auses2
Syphilitic meningitis=gumma)
Tuberculosis
Tumors o opticthalamus
Aneurysm o superiorcerebellar or posteriorcerebral arteries)
%haracterised by 2•"ncongruoushomonymoushemianopia 5ith %='hemianopic pupillaryreaction; 5ernic3e>s
reaction<
• These lesions usuallylead to partialdescending opticatrophy 6 may be
associated 5ith %=' 9rd
nerve paralysis 6
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1#Le!on! oflateral
(en*'late-od. %
leads tohomonymoushemianopia 5ithsparing opupillary re7e8es6 may end inpartial opticatrophy)
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2#Le!on! of o)t*radaton! %
%auses2
Vascular occlusionPrimary 6 secondarytumors
Trauma
%haracterised by 2
%*#P'T&*#*N$#*1S&#"AN*P"A; sometimes sparing macula<
T*TA' *PT"%!AD"AT"*N
"NV*'V#NT
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'S"*NS *FPA!"TA' '*:
;involvingsuperior fbres
o opticradiations<
"NF!"*!?1AD!ANT"%&#"AN*P"A;
P" *N T&F'**!<
'S"*NS *F T#P*!A'
'*: ;involving
inerior fbres ooptic radiations<
S1P!"*!?1AD!ANT"%
&#"AN*P"A; P"
*N T&!**F<
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3#Le!on! of
v!'al*ortex %
pupillary lightre7e8 is normal
6 optic atrophydoes not occurollo5ing visualcorte8 lesions)
%ongruoushomonymous
hemianopia;sparing macula<
*cclusion oposterior cerebral
artery supplyinanterior part ooccipiatl corte8
%ongruoushomonymous
macular deect
&ead in@ury=gunshot in@ury
leading to lesionso tip o occipital
corte8
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P&$S"*'*("%A' :'"ND SP*T
%orresponding to optic nerve head
B. deg temporal to point o f8ation
Span C . deg horiontal
EE / deg vertical
T5o thirds belo5 the horiontalmeridian
B
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V"S1A' A%1"T$
A measure o spatial resolution o theeye
An estimation o its ability todiscriminate bet5een t5o points
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!S1'TS
R(htE.e
LeftE.e
Githout'ens
9=B =B
Gith'ens
=9 =.
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F*VA %NT!A'"S
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V"S1A' PAT&GA$
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V"S1A' PAT&GA$
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SN''N>S %&A!T
• Principle2 D"STANTV"S1A' A%1"T$
• the ratio o one>svisual acuity tothat o a person5ith normal visualacuity
"V V"S1A' A%1"T$
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"V) V"S1A' A%1"T$
EEEEH
the distancethat thepatient 5asmade to standrom the chart
the distanceassigned to thelast ro5 they5ere able toread
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RETINAL OR TRAIN
NYSTA4MUSJ*PT*K"NT"% N$STA(#1SLEhappens 5hen repetitive stimuli traverse the feld ovision
Eallo5s the eye to ollo5 ob@ects in motion 5hen headremains stationary
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!etinal ;train nystagmus<
E JDancing eyesL
E *scillatory or rhythmic movementso the eye in 5hich there is a ast anda slo5 phase)
E "t is named according to the directiono the ast phase, because it is moreeasil observed)
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!etinal ;train nystagmus<
O)to5net* Re6ex ,O7R#
- "s a second mechanism by 5hich thenervous system stabilie the visualscene on the retina
E Activated by movement o the visualscene)
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!etinal ;train nystagmus<
Sa**ade!
- To be able to move the eye 5ith respect to
the 5orld
- So that ob@ects o importance can be ocusedonto the ovea
E Scrutinied 5ith this highEresolution part othe retina)
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!etinal ;train nystagmus<
Smooth P'r!'t
E This system allo5s us to 3eep itstable on the ovea despite itscontinued motion)
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!etinal ;train nystagmus<
N.!ta(m'!
E these re7e8es 5ill initially counterErotate the eyes in an attempt tomaintain a stable image on the retina)
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!etinal ;train nystagmus<
Ver(en*e
E these re7e8es 5ill initiallycounterrotate the eyes in an attempt tomaintain a stable image on the retina)
E allo5s a target to be maintained onboth ovea during eye movement
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T&ANK $*1