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    AMC CLINICAL EXAM RECALL BRISBANE FEBRUARY 2008

    AMC CLINICAL EXAM RECALL

    BRISBANE FEBRUARY 2008

    1. A 17 year-old girl with a known peanut allergy has had moderate episode yesterday.

    TASK; advise her of the management.This is a common station. Advise for the potential dangers of the peanut allergy, possibly

    associated allergies to other nuts, Epipen carrying instructions with explanation of

    contents and their use. Bracelet, referral to allergologist, advice for meticulous checking

    of any food for descriptive labels of traces of nuts, leaflet. The chance for the allergy to

    burn out over the years is small.

    No questions from the role-player or the examiner.

    Peanut Induced Anaphylaxis

    2. A young man with nausea, vomiting, and abdominal pain which started out as central

    but which moved to the right iliac fossa. There is guarding in the right iliac fossa onpalpation.

    TASK; explain the possible likely diagnosis, give differential diagnoses. Management.

    The patient was told that the likely diagnosis is appendicitis. I explained additional

    examination/tests I would do to prove this, namely Rovsing, Psoas, obturator symptom.

    The differential diagnoses I mentioned were renal colic, mesenteric adenitis, Crohns

    disease. After that the conversation was about the forthcoming operation, why it was

    needed, possible complications, and the regime for care after the operation. This refers to

    case no. 140 in the Australian Medical Council Handbook of Clinical Assessment.

    Initial Management Of Acute Appendicitis

    3. Case no 113 in the Australian Medical Council Handbook of Clinical Assessment.

    Sepsis (Paediatrics)

    4. Case no. 79 in the Australian Medical Council Handbook of Clinical Assessment.

    Here I nearly missed assessing the blood group for rhesus negative. The examiner

    prompted me, asking whether I had anything else to add, and I remembered and was

    saved.Threatened abortion (O&G)

    5. A young woman unsuccessfully trying to conceive has been seen by your colleague

    yesterday and now is in your consulting room with the investigation results, which are

    borderline anaemia, thyroid function normal, follicle-stimulating hormone low, and

    normal electrolytes. Her body mass index is 14.4; BP normal, without postural drop.

    TASK; history, explanation of results, and management.

    History- no problems at home, work or socially. She has been this skinny since her teensand claims to feel very comfortable. Not tired, has enough energy. Has had no periods

    for several years. I missed asking about illicit drug use and hyperthyroidism symptoms.

    I explained to her the connection between low BMI and failure to conceive, and the

    hypothalamus, pituitary and ovary interrelationships. I explained osteoporosis following

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    anovulation, and referred her for a bone density test. She was keen to start to eat so as to

    conceive, and so was sent to a dietician, and to the community mental health service.

    The examiner said nothing, but looked extremely grim.

    Anorexia Nervosa, Diagnosis

    6. A woman with a previous caesarian section, who is now at 28 wks gestation, has been

    having vaginal bleeding, and has come to the emergency dept. She is rhesus positive.

    TASK; focused history, examination findings from the examiner, management.

    In the history I find that she has had an uncomplicated pregnancy till this moment. She

    does not abuse drugs and has undergone no recent trauma except possibly that she has

    had sex the previous night. Her morphology scan at 18 weeks showed the placenta to be

    in a low position. Presently, she is feeling well. She does not feel dizzy when she stands

    up..

    On examination, she is haemodynamically stable, with BP 120/80 and HR 70 or so. Theabdomen is not tender. She is not in pain. The foetal HR was 140BPM.

    I explained that this is probably a case of placenta praevia, and that I would urgently call

    the obstetrics registrar for an ultrasound examination and CTG to discover the reason for

    the bleeding, and to make sure of the babys well-being. I admitted her because the

    bleeding may resume. I will NOT examine her vaginally (this would have been a critical

    error; because it may cause bleeding).

    The role-player asked me about the mode of delivery. I answered, If the case is placenta

    praevia, delivery will only be via caesarian section.

    The young Indian examiner was hostile from the beginning, making angry exclamations

    when I mentioned wanting to check the vital signs in the first instance, and after that,

    continued to look angrily at me for whole duration of the station. Perhaps somebody had

    angered him before I arrived. In any case I will be surprised if I fail such an easy station.

    Well, it happened that I did in fact fail this station.

    It was because she looked so well and her vital signs were so stable that I didnt order a

    blood cross-match for a possible transfusion while waiting for the obstetrician to arrive,

    and this might have been the critical error.

    Vaginal Bleeding At 26 Weeks Gestation (O&G) (Failed)

    7. A 2 year-old child with limping and pain in one leg.

    TASK; history, examination results from examiner, differential diagnoses, diagnosis,

    management.

    History; the pain began last night. There is no history of trauma, of easy bruising. The

    childs delivery and developmental history to this moment have been normal. The father

    has not noticed the child to be hot. He had a respiratory tract infection, possibly flu,

    some ten days ago.

    The examination findings from the examiner were; general appearance; normal, restrictedand painful internal rotation and abduction in the hip. The child is not febrile.

    I explained that this is probably a case of irritable hip, with this typical history of flu 10

    days ago, and restricted hip movement. The differential diagnoses are avascular necrosis

    (Pertes Disease), septic arthritis, and perhaps, though unlikely, developmental dysplasia

    of the hip.

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    I would order C-reactive protein, erythrocyte sedimentation rate and full blood count and

    X-ray of the hip, and I would refer the patient to the orthopaediatrician. Meanwhile, the

    child will need at least a weeks rest.

    30 seconds before the bell, the father turned to me and asked provocatively, Are you

    sure that the problem is in the hip? I felt somewhat panicked, and asked the examiner

    for the examination findings in the groin, knee and ankle. With a guarded smile, the

    examiner answered, Normal. Then I replied that yes, I was certain that the problem

    was in the hip.

    Transient Synovitis Of Hip Irritable Hip (Paediatrics)

    8. Case no. 119 in the Australian Medical Council Handbook of Clinical Assessment.

    A man wants to discuss his wifes condition.

    Patient confidentiality

    9. Full page of text impossible to read in 2 minutes, describing investigation done on a manwith abnormal liver function test. He has a pacemaker fitted.

    TASK; history, investigations from the examiner, management.

    When I entered, I asked for permission to read for a little longer, and after 30 seconds

    started to take the history. The patient had never used any drugs that could damage the

    liver, or any alcohol. On questioning about his ancestry, he kept replying Australian!

    He reported no skin-colour change. Hepatitis serology tests were negative, as was

    monospot test. On the list of investigations given, there were normal Alkaline

    Phosphotase (no cholestasis), but increased Alanine Aminotransferase (hepatocellular

    damage). I asked for a serum iron study, and the examiner supplied an additional listwith significantly increased ferritin and transferrin saturation. After that, it was easy.

    This refers to case no. 69 in the Australian Medical Council Handbook of Clinical

    Assessment.

    The management; once weekly venesection 500ml for at least 2 years, then every 2 to 3

    months. screen 1st degree relatives for haemochromatosis.

    Abnormal Liver Function Tests

    10. Case no. 97 in the Australian Medical Council Handbook of Clinical Assessment.

    A pregnant woman of 28 weeks gestation, with a positive Glucose Challenge Test result.

    The scenario given was nearly the same as the above-mentioned Handbook case, with

    just the one difference that the blood-sugar levels after 2 hours were not so high, but

    rather were only 8-point-something.

    After I began to take the history, the examiner stopped me to tell me that he was unable

    to hear me. So I made my first mistake; instead of raising my voice, I moved my chair

    closer to his. This violated his personal space and ruined his preferred arrangement of the

    room. And then, after a time, asking about investigations, I said urinalysis, and he said

    something like what?. I mistakenly thought that he had not heard me and repeated

    URINALYSIS!, loudly. He looked insulted, and answered me with quiet venom

    What.For.?. of course this was a test for protein, but the damage had been done.After that, I covered the case according to the book, except for neglecting to ask for foetal

    lie and presenting part, and I didnt advise the mother to test her blood-sugar levels 4

    times daily, which was a critical error. (I thought this was needlessly cruel, given that the

    glucose levels were not so excessive).

    Abnormal glucose challenge test (O&G) (Failed)

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    11. A 3 year-old child has had a problem breathing through his/her nose (mostly mouth-

    breathes), night-snoring, and frequent upper-respiratory tract infections.

    TASK; history, examination, investigations from examiner, diagnosis, management.

    This has been happening for the last several months. There are no sleep apnoea episodes.

    Otherwise the childs development has been normal, has had all the proper vaccinations

    and is thriving. The childs father suffers from allergies.. The mother hasnt noticed any

    hearing problem in the child. The child does not take any medications.

    On examination, there were difficulties with nose-breathing, with bilateral nasal secretion

    (not malodorous) and constriction. Otherwise the throat was given to be normal (via

    pharyngoscopy), as were the chest (via auscultation), and the ears (otoscopy).

    The examiner was surprised by my attempts to view enlarged adenoids through the nose

    or via the indirect epipharyngeal route; it seems that this is not done in this country.

    Following my past experience, I explained to the mother what I thought was the likeliest

    diagnosis, of adenoiditis, and that I would like to refer the patient to an ENT specialist for

    eventual surgery.

    I perceived that neither the role-player nor the examiner were satisfied with what I had

    said. The role-player started to ask me about other possible management, and especially

    about anti-biotics, and in an attempt to salvage some success I answered that we could

    use an intranasal saline rinse, and then eventually antibiotics. But I forgot to ask for

    about pharyngeal post-nasal drip, or for a nasopharyngeal swab, or about enlarged

    cervical lymph nodes.

    I felt that something had gone wrong. I mentioned that an allergy could be playing a rolehere, given the family history, and offered to arrange a skin-prick test. At this point, 20

    seconds before the end of the station, the answer given by the examiner was not

    available.I think it was a mistake to send the child for prospective surgery without trying

    less invasive treatments first. I wonder whether perhaps they wanted me to speak mostly

    about perennial rhinitis, and about strategies for dust-mite reduction etc. I forgot to

    advise against smoking in the house as well.

    See p 243 in the Royal Childrens Hospital Paediatric Handbook

    Rhinitis (Paediatrics) (Failed)

    12. Case no. 83 in the Australian Medical Council Handbook of Clinical Assessment.

    A woman with chronic schizophrenia recently started a new medication, and gained 15

    kg in 3 months. The role player was extremely convincing, and appeared to be authentic.

    I asked her PASS questions (Psychosis, Anhedonia, Substance Abuse, Suicide). She had

    no extrapyramidal side-effect symptoms, nor anticholinesterase symptoms. She had no

    decreased tolerance to cold. The name of the drug she has started 3 months ago was

    unknown to me (probably deliberately), and so I asked is this Olanzapine?. Examiner;

    Yes. I ordered a blood sugar test, thyroid function test, a lipid profile. I forgot to ask

    for a serum prolactin test.

    My management was to explain this common side-effect of the drug to the patient, and togive her the option to start diet and exercise (referral to dietician), or to consider a change

    of medication, urging her though, to take into consideration the probable side-effects of

    any eventual new drug. I told her that a change of medication could be carried out only

    by a psychiatrist, with gradually withdrawal from the old and introduction of the new.

    The examiner seemed want to hear something else about my management. at almost the

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    last possible moment, he said, The psychiatrist could see her not earlier than in a weeks

    time. Shes taking 20 mg of Olanzapine!. I thought for 5 seconds and said, I cantchange her treatment myself (20 mg of Olanzapine, it turns out, is a normal dose, so this

    seems to have been a case of the examiner attempting to provoke me into making an

    error). But still I am not sure whether or not a GP is empowered to prescribe or change

    antipsychotics. Please check this.

    Weight Gain Side Effect Of Antipsychotic Drug

    13. Case 59 in the Australian Medical Council Handbook of Clinical Assessment.

    The examiner was the lovely Dr Marshal, from the AMC examination video. You dont

    have to memorise the signs and tests for brain-death; the list is inside the room. The role-

    player only wants you to explain the logic of the tests.

    Diagnosis Of Brain Death

    14. A 50-something-year-old man with a history of 2 weeks of headache.

    TASK; take history, a real focused examination, diagnosis, management.

    I questioned the patient about the nature of his pain; severity, duration, constancy,

    nausea, vomiting, blurred or double vision, history of similar symptoms, and about any

    history of drug abuse or trauma. I asked whether there had been any recent fever.

    In the examination, I checked for neck stiffness, facial and pupillary asymmetry, and

    visual abnormalities. The pain was definitely in the temporal area, but pulsation of the

    temporal artery was present. I mentioned ophthalmoscopy and the examiner asked what I

    was checking for.

    For the management of temporal arteritis, see case no. 93 in the Australian MedicalCouncil Handbook of Clinical Assessment.

    Headache

    15. A middle-aged woman has had a black-out yesterday.

    TASK; history, examination findings from examiner, differential diagnoses, diagnosis,

    management.

    The patient has a history of good health, but yesterday, while playing tennis, she had

    fallen suddenly unconscious. She had no visual disturbances prior to the episode and was

    not disoriented after it. She does not use drugs and her BP is usually normal. She

    doesnt feel any numbness in her limbs and has no headache (I thought I knew what this

    case would turn out to be, but tried to hide this fact from the examiner, and so I didnt

    even ask about cardiovascular symptoms at this point).

    After I offered to examine her, the examiner pointedly asked me, Do you want to finish

    your history?. I asked about shortness of breath during exertion, palpitations,

    orthopnoea, swelling of the ankles.

    The examination revealed a normal cranial nerve, no carotid bruit, and no displaced apexbeat. There was no thrill, but there was a grade 3 systolic murmur over the aortic valve

    propagating to the carotid artery. I asked about the Valsalva manoeuvre to exclude

    hypertrophic obstructive cardiomyopathy.

    I diagnosed aortic stenosis.

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    The examiner asked me about differential diagnoses for syncope, and I mentioned

    Transient ischaemic attack, hypoglycaemia, epilepsy, orthostatic hypotension, vasovagal

    syncope, and hypotension-causing medications. I forgot to mention arrhythmia.

    In the end, the examiner told me, You forgot, taking your history, something connected

    with aortic stenosis. What did I miss?? The answer came to mind after the exam;

    ANGINA. But I would not have expected to fail for this kind of omission.

    This refers to case no. 36 in the Australian Medical Council Handbook of Clinical

    Assessment.

    Syncope (Failed)

    16. A middle-aged man is concerned about the possibility of his contracting prostate cancer.

    TASK; assess the risk of prostate cancer for this particular patient. Inform him about the

    prostatic screening strategy in Australia.

    Upon my entering the room, the examiner asked me whether I had understood the test,

    and although I answered in the affirmative, he stood beside me and read to me the entire

    stem. Probably he was upset with the levels of comprehension of previous candidates.

    The role-players father had had prostate cancer of >70 years of age onset. The role-

    player had not had any radioscopy and neither had he worked with toxins. I asked him

    carefully about bladder-outlet obstruction and haematuria, neither of which he had, but I

    neglected to ask about back-pain. The examiner wanted to hear specifically the triad of

    screening tests;

    i/ Digital Rectal Examination

    ii/ prostatic-Specific Antigeniii/ Abdominal Ultrasonography

    The required content is on pages 77 and 1102 in the latest edition of Murtagh.

    Most decisive in this case was that at the end of the station, the role-player asked me to

    describe the advantages and disadvantages of prostate cancer screening.

    My comments were that the benefits are as for any screening; to discover problems early.

    The disadvantage is inaccuracy; there are a lot of false negatives and false positives. And

    the patient is subjected to high levels of anxiety that can not necessarily be relieved by

    favourable results, and may be needlessly stressed by an unfavourable one.

    Carcinoma of the prostate

    CONCLUSION

    More than half of the stations were from the Australian Medical Council

    Handbook of Clinical Assessment.

    The strong tendency in past exams for examiners to behave as observers remains

    stable.

    These particular examiners -and I have heard this about the Brisbane examiners-

    do not appear to feel obliged to provide any positive feedback after you finish.Most of them maintain stony faces, and this could seem very discouraging,

    especially if they have met you at the beginning of the station smilingly.

    There were two relatively young examiners, and they both failed me for to my

    mind- insufficient reason (see the vaginal bleeding and syncope cases).

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