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BSPD Specialists’ meeting, Leeds, 14 th March 2003 Molar-Incisor Hypoplasia Prevalence and Histology. Karin Weerheijm Dept of Cariology, Endodontology, Pedodontology Academic Centre for Dentistry (ACTA), Amsterdam. [email protected] Proposed definition = Hypomineralisation of systemic origin of 1-4 permanent first molars, frequently associated with affected incisors. (c.f. Weerheijm KL, et al. Caries Res 2001; 35: 390-391). Observations: Some teeth affected by MIH exhibit significant sub- surface porosity. This results in breakdown of the enamel. Primary teeth are not affected. Presentation may be asymmetrical. Affected teeth are sensitive, even if the enamel has not broken away.

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BSPD Specialists’ meeting, Leeds, 14 th March 2003

Molar-Incisor Hypoplasia Prevalence and Histology.

Karin Weerheijm

Dept of Cariology, Endodontology, Pedodontology Academic Centre for Dentistry

(ACTA), Amsterdam.

[email protected]

Proposed definition = Hypomineralisation of systemic origin of 1-4 permanent first

molars, frequently associated with affected incisors.

(c.f. Weerheijm KL, et al. Caries Res 2001; 35: 390-391).

Observations:

Some teeth affected by MIH exhibit significant sub-surface porosity. This results

in breakdown of the enamel.

Primary teeth are not affected.

Presentation may be asymmetrical.

Affected teeth are sensitive, even if the enamel has not broken away.

The cause of the increased sensitivity is not yet known.

The prevalence is equal in the upper and lower jaws

(c.f. Weerheijm KL, et al. J Dent Child 2001; 68: 259-262).

It is not known if there is a racial difference.

It is not known if the tips of the permanent canines are affected.

Affected enamel exhibits increased carbon content, and decreased calcium and

phosphorus concentrations (Jälevik, et al. Arch Oral Biol 2001; 46: 239-247).

In fluorosis, enamel is caries resistant. In MIH, affected teeth are more caries

susceptible, and post-eruptive breakdown occurs. The condition may be

misdiagnosed as hypoplasia.

Problems associated with MIH:

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Caries development

Post-eruptive enamel breakdown is unpredictable

Anaesthesia

Prevalence of MIH: Ranges 4 – 25% (prevalence data are limited).

Aetiology of MIH:

Multifactorial

Ameloblasts are affected at the late secretory or early maturation stage.

Some ameloblasts are irreversibly damaged, resulting in yellow-brown opacities,

and more porosities.

Where ameloblasts have recovered, white opacities result.

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Cosmetic management of anterior teeth with MIH

Stephen Fayle, Consultant in Paediatric Dentistry, Leeds Dental Institute.

[email protected]

Treatment options for opacities:

Microabrasion Better suited to diffuse opacities

Defects in MIH are usually full-thickness

Carbamide peroxide bleaching Lightens the whole tooth, making the

opacity less obvious, but doesn’t affect

the opacity.

Direct and indirect veneers

Aesthetic composites

Esthetex (Dentsply)

Cosmodent real enamel system (v.expensive)

Polishing systems

Enhance (Dentsply)

Soflex (3M)

Prisma gloss

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Problems and restorative options for first permanent molars with MIH

Stephen Fayle

Analgesia can be a problem in MIH.

The phenomenon of tachyphylaxis has been observed (LA has taken effect, but

once it wears off, subsequent analgesia is difficult to achieve).

Special techniques of analgesia may be required:

Intra-ligamental

Maxillary molar block (c.f. Adatia 1966) – after-pain may be a problem due to

decreased blood supply caused by vasoconstriction

Palatal analgesia is required

Computerised analgesia (“the wand”) may prove useful (c.f. Allen et al. Paed

Dent 2002; 24: 315-320. Gibson et al. Paed Dent 2000; 22: 458-462. Ram et al.

Int J Paed Dent 2002: 12: 80-89).

Desensitising agents:

Fluoride varnish – According to Stephen Fayle, Colgate Fluorogard Gel-Kam

works well in MIH (0.4% SnF).

Desensitising toothpastes

Bonding agents (hydrophilic recommended). In a study by Stephen Fayle, “Prime

& Bond” didn’t work.

Fissure sealants – glass ionomer based FSs may prove useful in MIH.