Upload
heronbayanin15
View
83
Download
0
Embed Size (px)
DESCRIPTION
Pathophysiology of Urinary Retention
Citation preview
Stagnation of urine
Increase hydrostatic pressure against the bladder wall
Urea Splitting Micororganisms
Hypertrophy of detrusor muscle
Hypersensitivity to afferent stimuli
Formation of trabeculae
Formation of diverticula
Increase in Ammonia
Alkalinity (Increasing pH of urine)
Decrease bladder contraction
Renointestinal reflexes and shared nerve
supply between the ureters and intestine
Calcium salts gets to urethra
Builds up and blocks the opening in the
urethra
Increase in residual urine volume
Overdistention of the urinary bladder
Overflow IncontinenceFrequency in Urination
Urgency
Nausea, Vomiting and Abdominal Distention
Feeling of Incomplete Voiding
Dysuria, Anuria
Vaginal Delivery Anticholinergics, Antispasmodics
Anesthesia
Large size of the baby
Pelvic trauma or injury during delivery
Injury to S2-S4Damage Stretch Receptor
Blocks action of Acetycholine
Inhibits Bladder Contractility
Blocks pain signals
Impaired Nerve Functioning
Decrease intrabladder pressure
Inhibits Micturition reflex
Loss of sensation of bladder during filling
Relaxation of detrusor muscle
Decrease intravesicular pressure
Residual Volume up to 2,000 ml
Overdistention of the bladder
Bladder reaches breakthrough point
Small amounts of urine dribble
Overflow Incontinence
Prostatic enlargement Constipation
Gland presses against the urethra
Bladder wall becomes thicker and irritable
Bladder contracts even when it contains small
amounts of urine
Frequent urination
Bladder weakens and loses its ability to empty
itself
Increase residual urine volume
Decrease elasticity of bladder
Feeling of Incomplete Voiding Overflow Incontinence
Hard stool in the rectum
Pushes against the bladder Pushes against the urethra
Narrowing of urethra
Decrease amount of urine release
Decrease force of urinary stream
Dysuria, Anuria