Ophtha Case Report - Copy

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    C.R

    57/MMarried

    Patient Profile

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    3 yrs PTC, px noted gradual blurring of vision of

    the right eye

    (-) eye pain

    (-) redness

    (-) tearing

    (-) dicharge

    CONSULT

    History of Present Illness

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    Past Medical History

    HTN (Amlodipine, 2 yrs)

    Past Ocular History

    Unremarkable

    Family History

    Unremarkable

    History of Present Illness

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    SUDDEN GRADUAL TRANSIENT

    PAINLESS PAINFUL

    Ischemic Optic

    Neuropathy Retinal

    Artery/VeinOcclusion

    Acute Angle

    ClosureGlaucoma Corneal Ulcer Uveitis with

    Complications

    Cataract

    Error ofRefraction Open Angle

    Glaucoma DM

    Retinopathy Age-related

    MacularDegeneration

    Subacute

    AngleClosureGlaucoma

    Uveitis Optic

    Neuritis Uveitis

    Papilledema

    AmaurosisFugax

    Differential Diagnosis

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    Cataract57 y/oLeukocoria

    Error of Refraction Not improved with pinhole

    Open Angle Glaucoma Normal IOP

    DM Retinopathy No DM

    Age-related Macular Degeneration Central vision is not affected

    Gradual, Painless

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    Cataract Senile Mature, OD

    Assesment

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    Pathogenesis

    not completely understood.

    cataractous lensesprotein aggregates thatscatter light rays and reduce transparency.

    Factors:

    oxidative damage (from free radical reactions)

    ultraviolet light damage

    malnutrition

    NO medical treatment to retard or reversecataract formation.

    protective effect from dietary carotenoids (lutein)

    Cataract

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    Mature All of the lens protein is opaque Leukocoria

    Immature Some transparent protein Black opacity against a red orange background

    Intumescent Lens take up water

    Hypermature

    Cortical proteins have become liquidescapeshrunken lenswith wrinkled capsule

    Morgagnian Hypermature cataract in which lens nucleus floats freely in the

    capsular bag

    Cataract

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    Most common type

    Age-related

    Types

    Nuclear

    Cortical

    Subcapsular

    Senile Cataract

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    Nuclear (Hard) Cataract

    Hardening of the nucleus d/t compacting &

    sclerosing of the lens fiber in the center of the lensyellow-brown pigments

    Myopic shift in refraction

    Early onset of BOV (Far vision)

    Senile Cataract

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    Cortical (Soft) Cataract

    Hydration of the cortex with development of

    subcapsular vacuoles which disrupts regulararrangement of lens fiber

    Opacity begins at periphery (radial spoke-like

    opacities)

    Late onset of BOV

    Senile Cataract

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    Posterior Subcapsular Cataract

    cortex near the central posterior capsule.

    Posterior migration of lens epithelium

    Near vision is affected early

    Associated with DM, trauma, corticosteroid use

    (topical or systemic), inflammation, or exposure to

    ionizing radiation.

    Senile Cataract

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    Phacoemulsification

    Latest method

    Use of ultrasonic vibrator to

    disintegrate nucleus Sutureless

    Procedure:

    Capsulorrhexis

    Hydrodissection

    Sculpting of the Nucleus Cracking of the Nucleus

    Emulsification

    Removal of Cortex

    Insertion of IOL

    Management of Cataract