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NEUROPHYSIOLOGIC AND BEHAVIORAL MEASURES OF PHONETIC
PERCEPTION IN ADULT SECOND LANGUAGE
SPEAKERS OF SPANISH
by
Jaden Hellewell
A thesis submitted to the faculty of
Brigham Young University
in partial fulfillment of the requirements for the degree of
Master of Science
Department Communication Disorders
Brigham Young University
April 2007
BRIGHAM YOUNG UNIVERSITY
GRADUATE COMMITTEE APPROVAL
of a thesis submitted by
Jaden Hellewell
This thesis has been read by each member of the following graduate committee and by majority vote has been found to be satisfactory. Date David L. McPherson, Chair Date Shawn Nissen Date Christopher Dromey
BRIGHAM YOUNG UNIVERSITY
As chair of the candidate’s graduate committee, I have read the thesis of Jaden Hellewell in its final form and have found that (1) its format, citations, and bibliographical style are consistent and acceptable and fulfill university and department style requirements; (2) its illustrative materials including figures, tables, and charts are in place; and (3) the final manuscript is satisfactory to the graduate committee and is ready for submission to the university library. Date David L. McPherson Chair, Graduate Committee Accepted for the Department Ron W. Channell Graduate Coordinator Accepted for the College K. Richard Young Dean, David O. McKay School of Education
ABSTRACT
NEUROPHYSIOLOGIC AND BEHAVIORAL MEASURES OF PHONETIC
PERCEPTION IN ADULT SECOND LANGUAGE
SPEAKERS OF SPANISH
Jaden Hellewell
Department of Communication Disorders
Master of Science
Infants 6-8 months old are able to identify nearly every speech sound contrast on
which they have been tested, regardless of whether that contrast represents an across-
category (two different phonemes) phonetic change in their native language or not. A
child’s ability to identify non-native consonant contrasts greatly diminishes by 11-12
months of age. The present study examined speech sound discrimination in three groups
of listeners. Adult second language (L2) listeners of Spanish were compared with native
English listeners with no knowledge of Spanish and native Mexican listeners to
determine if they would show the ability to discriminate phonetic distinctions that are
present in Spanish and not English. Behavioral and Electrophysiological measures were
obtained in response to a continuum of bilabial consonant-vowel (CV) stimuli that
differed in voice onset time (VOT) from -50 to 30 ms VOT (/ba/ to /pa/). The -50 ms
VOT stimulus was paired with each of the others to form stimulus pairs with varying
degrees of acoustic difference. Behavioral data showed that L2 listeners of Spanish
perceived a transition from /ba/ to /pa/ later than native Spanish listeners but earlier than
English only listeners. Electrophysiological data using the mismatch negativity revealed
that the both L2 Spanish and native Spanish listeners perceived a distinction between
stimuli in the stimulus pairs 20 ms earlier than English only listeners. These results
support the theory that underlying neural-sensory representations of speech may be
altered by linguistic experiences, and that the loss of non-native speech sound
discrimination abilities in infancy and the regaining of these abilities in adulthood is not
due merely to attentional bias.
ACKNOWLEDGMENTS
Many hours of work went into this project and not all of them were mine. I want
to thank my thesis committee chair Dr. David McPherson for his dedicated help and
teaching. I would also like to thank the other members of my committee Dr. Shawn
Nissen and Dr. Christopher Dromey. Thank you to my many participants many others
who gave me advice and help throughout the process. I am especially grateful to my
loving wife, Kira, for her constant encouragement and unwavering support.
vii
TABLE OF CONTENTS
Page
List of Tables ..................................................................................................................... ix
List of Figures ..................................................................................................................... x
List of Appendixes ............................................................................................................ xii
Literature Review................................................................................................................ 1
Developmental Speech-Sound Perception .............................................................. 1
The Mismatch Negativity ....................................................................................... 4
The Mismatch Negativity and Speech-Sound Discrimination ................................ 7
The Mismatch Negativity and Second Language Learning .................................... 8
Method .............................................................................................................................. 11
Participants ............................................................................................................ 11
Stimuli ................................................................................................................... 11
Procedures for Behavioral Experiment ................................................................. 14
Procedures for Electrophysiological Experiment ................................................. 14
Data Analysis ........................................................................................................ 15
Results ............................................................................................................................... 16
Behavioral Results ................................................................................................ 16
MMN Results ........................................................................................................ 16
Scalp Distribution Within Groups ........................................................................ 21
Scalp Distribution Across Groups ........................................................................ 49
Statistical Analysis ................................................................................................ 54
Discussion ......................................................................................................................... 55
References ......................................................................................................................... 59
viii
Appendixes ....................................................................................................................... 64
ix
LIST OF TABLES
Table Page
1. Descriptive Statistics for Average Peak Latencies (Plat) of the MMN ........................ 19
2. Descriptive Statistics for Average Peak Amplitudes (Pamp) of the MMN .................. 20
3. Descriptive Statistics for RMS Amplitudes of the MMN at the CPZ Electrode .......... 22
x
LIST OF FIGURES
Figure Page
1. Waveforms representing the eight stimuli used in the experiment ............................. 13
2. Mean identification functions for the /bɑ/-/pɑ/ continuum for each group are
represented in Figures 2A-2C. Figure D shows all three groups................................17
3. Graphs representing presence or absence of MMN in each group at each stimulus . 18
4. Scalp distribution map for group 1 -40 ms VOT......................................................... 23
5. Scalp distribution map for group 1 -30 ms VOT .........................................................24 6. Scalp distribution map for group 1 -20 ms VOT......................................................... 25
7. Scalp distribution map for group 1 -10 ms VOT .........................................................27 8. Scalp distribution map for group 1 0 ms VOT …........................................................ 28 9. Scalp distribution map for group 1 10 ms VOT .......................................................... 29 10. Scalp distribution map for group 1 20 ms VOT ......................................................... 30
11. Scalp distribution map for group 1 30 ms VOT .......................................................... 31 12. Scalp distribution map for group 2 -40 ms VOT .........................................................33 13. Scalp distribution map for group 2 -30 ms VOT .........................................................34 14. Scalp distribution map for group 2 -20 ms VOT ........................................................ 35
15. Scalp distribution map for group 2 -10 ms VOT .........................................................36 16. Scalp distribution map for group 2 0 ms VOT …........................................................ 38 17. Scalp distribution map for group 2 10 ms VOT .......................................................... 39 18. Scalp distribution map for group 2 20 ms VOT .......................................................... 40 19. Scalp distribution map for group 2 30 ms VOT .......................................................... 38
20. Scalp distribution map for group 3 -40 ms VOT .........................................................42
xi
21. Scalp distribution map for group 3 -30 ms VOT .........................................................43 22. Scalp distribution map for group 3 -20 ms VOT .........................................................45 23. Scalp distribution map for group 3 -10 ms VOT .........................................................46 24. Scalp distribution map for group 3 0 ms VOT ............................................................ 47 25. Scalp distribution map for group 3 10 ms VOT .......................................................... 48 26. Scalp distribution map for group 3 20 ms VOT .......................................................... 50 27. Scalp distribution map for group 3 30 ms VOT .......................................................... 51
xii
LIST OF APPENDIXES
Appendix Page
A. Informed Consent Document ...................................................................................... 64
B. Words Used for Stimulus Recordings ......................................................................... 66
C. List of Sentences Read by Speaker ............................................................................. 67
D. Tables of RMS Amplitudes......................................................................................... 69
1
Literature Review
Little is known about the neurological processes involved in speech sound
perception and second language (L2) learning. A few recent studies have begun to
explore this topic directly, and have dealt mainly with the perception of non-native
vowels (Nenonen, Shestakova, Huotilainen, & Naatanen, 2005; Shestakova, Huotilainen,
Ceponiene, & Cheour, 2003; Winkler & Kujala et al., 1999). The exploration of this topic
began with studies devoted to developmental language learning and speech sound
perception.
Developmental Speech-Sound Perception
A significant body of research has shown that infants 6-8 months old are able to
identify nearly every speech sound contrast on which they have been tested, regardless of
whether that contrast represents an across-category (AC, two different phonemes)
phonetic change in their native language or not. Infants can identify contrasts that form a
within-category (WC, same phoneme) phonetic change in their native language and an
AC phonetic change in another language, to which the infant has not been exposed. The
research has also shown that a child’s ability to identify non-native consonant contrasts
greatly diminishes by 11-12 months (Werker, 1991). Research by Best, McRoberts, and
Sithole (1988) comparing Zulu click contrasts in English speaking adults and infants
provided a caveat to this theory. They concluded that if the nonnative contrasts are not
likely to be assimilated into any native phonemic category (such as a Hindi retroflex /ɖɑ/
being perceived by English speakers as dental /dɑ/), the ability to discriminate the sounds
will continue into adulthood because perception will focus on purely auditory or phonetic
properties.
2
A study by Werker, Gilbert, Humphrey, and Tees (1981) used 6-8 month old
infants, native Hindi speakers, and native English speakers to test consonant contrasts
that are present in Hindi, but not in English. The first Hindi consonant contrast involved a
change in place of articulation, a voiceless dental stop verses voiceless retroflex stop. The
second involved a contrast between a voiceless aspirated stop and a breathy voiced dental
stop, in which a change in voice onset time (VOT) is the main distinction. The study
found that the 6 month old infants were able to discriminate both Hindi sound pairs along
with the native Hindi speaking adults. The native English speaking adults, however, were
not able to distinguish the place of articulation contrast, and only some were able to
distinguish the VOT contrast.
The early age at which children lose this discrimination ability was shown by
Werker and Lalonde (1988), who tested discrimination abilities in native Hindi speaking
adults, native English speaking adults, 6-8 month old English-learning infants, and 11-13
month old English-learning infants. The stimuli used were two phoneme pairs with
varying VOT. The first represented a phonetic change common to both languages, and
the second represented a change only in Hindi. The researchers found that the younger
infants were able to discriminate both the contrast common to both languages and the
contrast found only in Hindi, while the older infants and the native English speakers were
only able to discriminate the common contrast.
Further research has helped describe the development of vowel discrimination.
Polka and Werker (1994) tested the discrimination of two German vowel contrasts in
English-learning infants of 6-8 months and 10-12 months of age. This study showed, as
expected, that the 6-8 month old infants were able to distinguish the German vowels with
3
more accuracy than the 10-12 month old infants. The difference between the two groups
was not as significant as expected, however. The researchers then tested another, younger
group of infants (four months old) on the same stimuli. The four month old infants
showed improved discrimination beyond that of the 6-8 month old group. The researchers
concluded that there is a developmental shift from a general phoneme recognition pattern
to language-specific phoneme recognition pattern within the first year of life, as shown
by previous consonant discrimination studies, but they also concluded that this change
appears to begin earlier for vowels than it does for consonants. Additionally, research by
Kuhl, Williams, Lacerda, Stevens, and Lindblom (1992) used the “perceptual magnet
affect” to show that at six months of age English and Swedish speaking infants’ vowel
perception was already influenced by the language to which they were exposed. Both
groups of infants showed a stronger magnet effect for their own native language
prototype vowel than for the other languages prototype vowel.
Evidence from these studies suggest the inability of adults and children to
distinguish nonnative speech sounds. Because of the ability that adults and children have
to relearn how to discriminate nonnative contrasts when learning a new language, the
prevailing theory has been that this loss of speech sound discrimination is due to
attentional bias rather than neural-sensory loss (Pisoni, Lively, & Logan, 1994; Werker,
1994). More recent studies (Dehaene-Lambertz, 1997; Sharma & Dorman, 1999) using
electrical brain responses, specifically the auditory evoked potential (AEP), have
investigated the underlying neural-sensory representations of speech to determine if they
are actually altered by linguistic experiences.
4
The Mismatch Negativity
The mismatch negativity (MMN) is commonly used in AEP studies of speech
sound discrimination. The MMN is a negative component of the AEP elicited by a
physically deviant stimulus following a series of homogeneous, or standard, stimuli, and
generally occurs between 100 and 200 ms after change onset (Naatanen, Gaillard, &
Mantysalo, 1978). The MMN is specific to auditory stimuli (Nyman et al., 1990) and is
currently the only valid objective measure of central auditory processing accuracy in the
brain. The MMN is best recorded when the subject’s attention is directed away from the
auditory stimulation (Naatanen & Escera, 2000) because processes underlying the MMN
are automatic and pre-attentive (Aaltonen, Niemi, Nyrke, & Tuhkanen, 1987). In other
words, the process takes place before conscious attention is paid to the auditory
stimulation.
Repeated exposure to a given auditory stimulus creates a memory trace in the
brain which accurately represents the stimulus features. The memory trace can be created
after only a few repetitions. When the deviant stimulus is presented, it is compared with
the previously formed memory trace. If a mismatch is present in the comparison of the
deviant stimulus and the memory trace, the MMN is elicited (Cheour et al., 1998;
Naatanen & Escera, 2000; Naatanen, Jiang, Lavikainen, Reinikainen, & Paavilainen,
1993; Naatanen, Schroger, Karakas, Tervaniemi, & Paavilainen, 1993). The MMN is
elicited, not only by changes in the acoustic signal itself, but also by changes to the silent
intervals between stimuli, or the inter-stimulus interval (Naatanen & Jiang et al., 1993).
Long term neural learning effects can be found with continued presentation of unfamiliar
stimuli; however, long term adaptive changes require attention and effort (Naatanen and
5
Schroger et al., 1993).
The MMN tolerates some variation in the standard stimuli. Winkler et al. (1990)
demonstrated that slight changes in frequency and intensity in the standard stimuli affect
the amplitude (μV) and area (μV × ms) of the MMN response but do not eliminate it. The
amplitude and area decrease with increased variation of the standard stimuli (Winkler et
al., 1990). The amplitude of the MMN also varies as a function of the magnitude of
change between the deviant stimulus and the standard stimuli. As the difference between
the standard and the deviant stimuli increases, the amplitude of the MMN also increases
(Jaramillo, Paavilainen, & Naatanen, 2000; Savela et al., 2003).
The MMN has been elicited by a range of stimulus variations including changes if
phonetic structure such as frequency, intensity, spatial locus of origin, rise time, and
duration (Naatanen, 1992). The MMN has also been elicited by changes in natural and
synthetic speech sounds (Aaltonen et al., 1987; Savela et al., 2003; Sharma, Kraus,
McGee, Carrell, & Nicol, 1993; Winkler & Lehtokoski et al., 1999). There is some
disagreement as to whether the MMN reflects processing of only the acoustic aspects of
speech or whether it also reflects phonetic processing into categories. Sharma et al.
(1993) compared the MMN elicited by two stimulus pairs across the /dɑ/ - /ɖɑ/
continuum. They found that a WC pair, where both phonemes were perceived as /dɑ/,
and an AC pair, where one phoneme was perceived as /dɑ/ and the other as /ɖɑ/, both
elicited equal MMN responses. The MMN did not differ in latency (onset, peak and
offset), amplitude, or area and the acoustic difference (starting F2 and F3 frequencies)
between the two pairs of stimuli were equivalent. The authors therefore concluded that
the MMN reflects the processing of only acoustic aspects of speech. Another study by
6
Savela et al. (2003) used Finnish subjects to compare native vowels with non-native
vowels (a Finnish vowel was paired with a Komi vowel) in an electrophysiological
experiment, measuring the MMN, and a behavioral experiment, measuring the reaction
time for identification of the deviant vowel. They used three stimulus pairs with varying
amounts of acoustic difference between them. They found that in one stimulus pair, /e-ε/,
the reaction time was longer when the Komi vowel /ε/ was the standard stimulus than
when the Finnish vowel /e/ was the standard stimulus. The same pattern was not found
with regard to the MMN. They concluded that the phonemic status of the standard
stimulus only plays a role at the attentive level, not the pre-attentive level.
Winkler and Lehtokoski et al. (1999) found that the pre-attentive change-
detection process generating the MMN utilizes both auditory and phonetic
representations. They used Hungarian and Finnish speaking subjects, and tested them
with two vowel stimulus pairs. One pair represented an AC change in Hungarian and a
WC change in Finnish, while the other pair represented an AC change in Finnish and a
WC change in Hungarian. The MMN data that they collected showed a larger amplitude
in AC contrasts than in WC contrasts in both language groups. Based on this evidence,
the researchers concluded that there is both auditory and phonetic involvement in
generation of the MMN.
The MMN can be used to delineate the stages of auditory information processing
in the same way as behavioral responses. However, MMN experiments provide more
accurate and specific information about the emergence of auditory stimulation
representations than do behavioral experiments. The pre-attentive nature of the MMN
protects it from contamination by voluntary processes. Although the MMN reflects pre-
7
attentive auditory perception, the absence of the MMN does not rule out behavioral
discrimination. For this reason, it is imperative that sound discrimination experiments
done using the MMN are accompanied by behavioral experiments (Naatanen & Winkler,
1999).
The Mismatch Negativity and Speech-Sound Discrimination
Many experiments have used the MMN to study speech sound discrimination in
children and adults because of the accuracy and specificity of the information obtained by
using the MMN. A study by Dehaene-Lambertz (1997), one of the earliest to use MMN
data to study speech sound discrimination, used changes in place and manner of
articulation to elicit the MMN; the stimuli were similar to those used by Werker and
Lalonde (1988). The stimulus set represented two different phonemes for the French
subjects (/bɑ/ and /dɑ/) while native Hindi speakers distinguished three phonemes (/bɑ/,
dental /dɑ/, and retroflex /ɖɑ/). Behavioral data for French subjects corresponded with
MMN results which showed an ability to discriminate the native contrasting sounds, but
not the nonnative contrast. The MMN was significantly stronger in the French subjects
when the stimulus represented an AC phonetic change in French, than when it
represented an AC phonetic change in Hindi and a WC phonetic change in French. The
authors concluded that the subjects’ inability to discriminate non-native phonetic
contrasts does not seem related to attention bias, but rather to a loss of auditory
discrimination abilities.
Other studies have contrasted different types of phonetic boundaries.
Discrimination of VOT changes in a /dɑ/-/tɑ/ continuum in English (/dɑ/ and /tɑ/ are
primarily distinguished by VOT) measured using behavioral and electrophysiological
8
methods, showed that the MMN evoked by an AC change were much stronger than those
evoked by a WC change for native English speakers. The behavioral identification of the
VOT changes coincided with the MMN data (Sharma & Dorman, 1999). A later study
contrasted English and Hindi listeners’ perception of VOT changes across a /bɑ/-/pɑ/
continuum. They found that when the stimuli changes presented were phonetically
significant to native Hindi listeners but not native English listeners, a significantly larger
MMN was evoked in the Hindi listeners. Again, a behavioral experiment showed
perceptual boundaries coincided with the boundaries revealed by the MMN. All of the
syllables presented were pre-voiced, therefore, English listeners perceived only /bɑ/,
while Hindi speakers perceived differences between /bɑ/ and /pɑ/ depending on the
amount of pre-voicing (Sharma & Dorman, 2000).
The Mismatch Negativity and Second Language Learning
Electrophysiological data have also supplied valuable information about brain
function when an L2 is learned after the first year of life; which may be after the ability to
distinguish nonnative sounds has been lost. Winkler and Kujala et al. (1999) used the
MMN to measure electrophysiological responses to native and nonnative vowel contrasts
in native Hungarian speakers who are fluent L2 Finnish speakers. The researchers
contrasted a group of native Hungarians fluent in Finnish with a group of native
Hungarians with no exposure to Finnish and a native Finnish group. A vowel contrast
that is found only in Finnish was compared to a vowel contrast that is present in both
languages. The MMN was elicited in both the native Finnish and fluent Hungarian
subjects when presented with the Finnish-only vowel contrast, but it was not elicited in
the Hungarians with no knowledge of Finnish. The MMN was elicited in all three subject
9
groups when the vowel contrast common to both languages was presented. These results
coincided with perceptual abilities of the three groups in the behavioral portion of the
study. When the Finnish-only vowels were presented, the Finns identified them with 90%
accuracy and the Finnish-fluent Hungarians identified them with 80% accuracy, while the
Finnish-naïve Hungarians identified the vowels at a chance level. The fact that the MMN
was present in the fluent Hungarians as well as the native Finns, and not the naïve
Hungarians shows evidence of L2 learning at a neural-sensory level.
The ability to perceive nonnative vowel contrasts, as shown by
electrophysiological data, was also demonstrated by Finnish children (3 to 6 years old)
learning French. A group of Finnish children enrolled in a French school was compared
to a group of Finnish children enrolled in Finnish-only schools and daycares. They were
presented with contrasting French vowels that do not correspond closely to Finnish
vowels. The MMN elicited in the French-learning group was significantly larger than
MMN elicited in the Finnish-only group after only 3 to 4 months of enrollment in the
French schools. This study again shows evidence of L2 learning at a neural-sensory level.
Overall, however, the body of research relating to AEPs and L2 learning is still relatively
small (Shestakova et al., 2003).
In several of the above studies the Hindi language has been compared to English
because of the differences in VOT between English and Hindi stops. Sharma and Dorman
(2000), for example, found that Hindi listeners identified bilabial plosive phonemes with
VOTs of 0 and -20 ms as /p/, and bilabial phonemes with VOTs of -50 and -90 ms as /b/.
The English listeners, however, perceived all four phonemes as /b/. Research findings
have indicated that English listeners do not consistently perceive /p/ until the VOT
10
reaches 25 ms or more (Lisker & Abramson, 1970). A comparison between English and
Spanish shows a similar phenomenon. While in English /b/ is produced with a VOT of
approximately 0 ms and /p/ is produced with a VOT of 50 ms or more, in Spanish /b/ is
pre-voiced with a VOT of less than -50 ms and /p/ is produced with a VOT of
approximately 0 ms (Lisker & Abramson, 1964). Based on this VOT data it is expected
that English listeners would perceive Spanish /b/ and /p/ only as /b/. Voice onset times for
bilabial stops have been shown to differ somewhat in different Spanish dialects.
However, all the Spanish dialects tested show similar differences from English VOTs
(Rosner, Lopez-Bascuas, Garcia-Albea, & Fahey, 2000).
The current study addresses the issue of the MMN in L2 learners as it relates to
VOT. More evidence is needed to know if speech-sound discrimination abilities that are
lost after infancy can be relearned in adulthood at a neural-sensory level as shown by the
MMN, particularly in consonant sound pairs. As stated above, the review by Pisoni,
Lively, and Logan (1994) points out that people have the ability to relearn nonnative
phonetic contrasts; therefore, it is also expected that as a native English speaker learns
Spanish, the speaker will learn to distinguish the Spanish phonemes of /b/ and /p/
according to common Spanish VOTs. The current study was designed to test this
hypothesis behaviorally and electrophysiologically, to find out whether behavioral
responses and the MMN evoked by changes in VOT in L2 adult speakers of Spanish
resemble the MMN of native Spanish speakers when the stimuli represent an AC change
in Spanish, but a WC change in English.
11
Method
Participants
Thirty adult listeners between the ages of 18 and 35 took part in this study.
Participants were divided into 3 groups. The first group of participants consisted of 10
native English listeners with no knowledge of Spanish. The second group of participants
consisted of 10 native Mexican listeners who spent at least the first 17 years of their life
in their native country. The third group of participants consisted of 10 native English
listeners who have learned Spanish as a second language in adulthood and had recently
(within 18 months of return) spent 20-24 months in Mexico. All participants
demonstrated normal hearing with pure-tone thresholds of ≤ 15 dB HL at 250, 500, 1000,
2000, and 4000 Hz bilaterally (American National Standards Institute [ANSI], 1996), and
displayed normal, type A, tympanograms (Martin & Clark, 2005). No participant had a
recent (within 60 days) respiratory infection or reported history of neuropsychiatric
disorders or head trauma.
Stimuli
A female native Mexican speaker who lived in Mexico until at least age 17 served
as talker for the recording of the stimuli. The speaker was recorded while saying the list
of Spanish sentences found in Appendix C. Each sentence contained a Spanish bisyllabic
word beginning with /bɑ/ or /pɑ/. Each sentence was used five times and the order was
randomized. The list of words is found in Appendix B. The sentences were recorded
online to a computer in a sound isolated chamber. A low-impedance dynamic
microphone (DPA 4011) and an analog/digital converter (Apogee Mini-me) were used to
record the participant’s productions. The microphone was affixed to a microphone stand
12
and placed approximately 6 inches from the speaker’s lips during recording. The speech
tokens were sampled at 96 kHz and low-pass filtered at 22.05 kHz with 24 bit
quantization.
The experimental stimuli consisted of a continuum of 9 stimulus tokens (T1-T9)
differing in terms of voice onset time (-50 ms to +30 ms VOT). Each syllable differed by
10 ms. The initial token in the stimulus continuum (T1 or -50 ms VOT) is commonly
perceived by both English and Spanish listeners as /bɑ/ while the terminal token (+30 ms
VOT) is often perceived as /pɑ/. The experimental stimuli were created using Adobe
Audition (2003). The initial /bɑ/ from the word “banda” was chosen because the pre-
voicing segment was regular and represented average Spanish VOTs. The initial /bɑ/ was
extracted from the word and the final 3 ms of the vowel were ramped to eliminate noise
from the segment. The pre-voicing segment of the /b/, which was originally 80 ms long,
was cut to 50 ms for the first sample. The pre-voicing was cut to 40 ms for the second
sample, 30 ms for the third, 20 ms for the fourth, 10 ms for the fifth, and the pre-voicing
was cut completely for the sixth. To create the positive VOT syllables, the /p/ burst from
the word “panda” was cut at a zero crossing of the waveform and added in the place of
the 50 ms pre-voicing segment of the first token, which was also cut at zero crossings.
This was done to achieve a naturalistic burst while keeping a consistent subsequent vowel
nucleus for all continuum tokens. The original /p/ burst was 10 ms long. Small portions
of that burst were copied and added in again to create the +20 and +30 ms VOT syllables.
The vowel portions of the burst syllables were also ramped during the final 3 ms so that
all samples had the exact same vowel. Waveforms for the 9 stimuli used are found in
Figure 1.
13
Figure 1. Waveforms representing the nine stimuli used in the experiment.
14
Procedures for Behavioral Experiment
Following the tympanograms and hearing screenings, the participants were seated
in a comfortable recliner in a sound-isolated chamber. The stimuli were presented
through insert phones with an intensity of 70 dB HL. The participants used a push button
controller with buttons labeled “ba” and “pa”. They were instructed to push the button
corresponding to the syllable they heard. The participants were presented with two trials
of 180 syllables. Each syllable was presented 20 times per trial. No training was given
prior to the start of the experiment.
Procedures for Electrophysiological Experiment
The participants were again seated in a comfortable recliner in a sound-isolated
chamber. The stimuli were presented through insert phones with an intensity of 70 dB
HL. The participants brought a book of their choosing and were instructed to stay alert
and read while the sounds were presented to them. They were told to ignore the stimuli.
The MMN was obtained by presenting a “common” or “standard” stimulus in contrast to
a second “deviant” stimulus with a stepped variation from the first stimulus token. The -
50 ms VOT /bɑ/ token was used as the standard and was paired with each of the other 8
tokens to form the stimulus pairs. Each paired comparison was presented with 480 trials.
The common syllable occurred 399 times (83%), and the deviant syllable occurred 81
times (17%). The participants were fitted with a NeuroScan 32-electrode cap. Each
electrode was filled with ECI Electro-gel to reduce impedance to 10 kOhms or less. A
NeuroScan computer using Scan 4.2 software was used to collect and analyze the AEP
waveforms. Raw electrical potentials were bandpassed from 0.05 to 70 Hz.
15
Data Analysis
The AEP waveforms obtained for each participant were averaged for both the
standard and the deviant conditions and analyzed. Grand averages were also computed
across subjects in the standard and deviant conditions and analyzed. Since the MMN is a
potential elicited by the deviant stimulus in a deviant paradigm, amplitude, area, and
latency of the MMN were measured from a difference wave computed by subtracting the
deviant response from the standard response. Difference waveforms were computed on
each set of standard and deviant condition waveforms for each participant, as well as for
the grand average waveforms.
The latency of the MMN was measured at the peak (point of maximum
negativity) as well as at the onset (beginning) and the offset (end) points of the MMN
component of the difference waveform. The MMN peak was defined as the prominent
negative peak within the latency range of 100-300 ms at the Fz recording site. The wave
duration was the time from onset to offset.
The magnitude of the MMN at Fz was obtained by measuring the amplitude of the
difference waveform from the baseline to the peak amplitude of the MMN. In addition,
the RMS amplitude of the MMN was determined by taking the average RMS amplitude
within the time window of the MMN.
16
Results
Behavioral Results
For the behavioral experiment, the number of responses for /bɑ/ and /pɑ/ were
totaled in each group. The line graphs in Figure 2 describe how often each response was
chosen according to the stimulus presented. The graph in the upper left represents group
1, the English only group. Group 2, in the bottom left is the native Spanish group, and the
L2 Spanish group, group 3, is in the upper right. The graph in the bottom right shows all
three groups. The perceptual crossover from /bɑ/ to /pɑ/ occurred earliest with the native
Spanish group, as was expected. Perceptual crossover was at about 0 ms VOT. The
crossover for the English only listeners occurred about 10 ms later. The L2 Spanish group
crossed over in between the two.
A one-way ANOVA for VOT by group showed significant differences, F(2, 27) =
5.689, p < 0.009. A post hoc t-Test showed significant differences between group 1 and
group 2, t(18) = 2.877, p < 0.010 and between group 1 and group 3, t(18) = 2.946, p<
0.009; however, failed to reach significance between group 2 and group 3, t(18) = 0.004,
p < 0.997.
MMN Results
Descriptive statistics. The graphs in Figure 3 below show how often a MMN was
present and absent at each of the stimuli in each of the three groups. Figure 3A represents
the English only group, group1; Figure 3B represents the native Spanish group, group2;
and Figure 3C represents the L2 Spanish group, group 3.
The average peak latencies of the MMN for each of the stimuli in all 3 groups are
represented in Table 1. The average peak amplitudes are represented in Table 2. The
17
Figure 2. Mean identification functions for the /bɑ/-/pɑ/ continuum for each group are
represented in Figures 2A-2C. Figure 2D shows all three groups.
18
Figure 3. Graphs representing presence or absence of MMN in each group at each
stimulus.
19
Table 1
Descriptive Statistics for Average Peak Latencies (Plat) of the MMN
Group Stimulus VOT Plat Mean Plat SD Plat Min Plat Max
1 -40 ms 212.60 34.56 158.00 248.00
1 -30 ms 203.50 56.48 129.00 264.00
1 -20 ms 190.25 21.06 174.00 219.00
1 -10 ms 183.83 34.89 137.00 217.00
1 0 ms 196.11 51.74 132.00 272.00
1 10 ms 188.50 38.11 136.00 232.00
1 20 ms 208.57 53.16 155.00 279.00
1 30 ms 195.25 46.88 128.00 258.00
2 -40 ms 180.50 46.75 122.00 255.00
2 -30 ms 178.60 38.06 124.00 224.00
2 -20 ms 188.00 47.07 128.00 257.00
2 -10 ms 179.38 45.46 124.00 241.00
2 0 ms 204.80 57.52 125.00 269.00
2 10 ms 173.80 24.44 147.00 196.00
2 20 ms 169.00 27.96 142.00 207.00
2 30 ms 196.13 45.98 123.00 255.00
3 -40 ms 203.00 44.05 141.00 252.00
3 -30 ms 207.75 55.27 139.00 253.00
3 -20 ms 174.83 44.46 125.00 238.00
3 -10 ms 195.67 42.28 158.00 256.00
3 0 ms 177.63 29.71 137.00 217.00
3 10 ms 145.43 25.84 117.00 180.00
3 20 ms 159.50 38.02 104.00 200.00
3 30 ms 222.83 66.57 121.00 296.00
20
Table 2
Descriptive Statistics for Average Peak Amplitudes (Pamp) of the MMN
Group Stimulus VOT Pamp Mean Pamp SD Pamp Min Pamp Max
1 -40 ms -2.66 1.31 -4.50 -1.12
1 -30 ms -2.26 0.38 -2.65 -1.86
1 -20 ms -2.39 1.61 -5.00 -0.91
1 -10 ms -2.55 1.24 -4.50 -1.15
1 0 ms -2.45 1.09 -4.74 -0.75
1 10 ms -3.12 1.20 -5.78 -1.83
1 20 ms -2.30 0.85 -3.71 -1.32
1 30 ms -1.62 1.89 -2.98 2.80
2 -40 ms -2.39 0.75 -3.22 -1.34
2 -30 ms -2.86 0.50 -3.71 -2.37
2 -20 ms -1.93 0.48 -2.69 -1.47
2 -10 ms -2.41 0.72 -3.63 -1.49
2 0 ms -2.34 1.25 -3.91 -0.56
2 10 ms -1.61 0.86 -2.52 -0.49
2 20 ms -2.50 0.32 -2.71 -2.02
2 30 ms -2.32 0.73 -3.76 -1.52
3 -40 ms -2.66 1.47 -5.02 -0.99
3 -30 ms -1.62 0.39 -2.00 -1.19
3 -20 ms -3.05 2.27 -7.16 -1.14
3 -10 ms -2.19 1.26 -3.48 0.13
3 0 ms -2.83 2.03 -5.90 -0.42
3 10 ms -2.74 1.41 -5.43 -1.51
3 20 ms -2.17 1.49 -4.94 -0.41
3 30 ms -2.48 1.37 -4.13 -0.64
21
RMS amplitudes for each of the stimuli in each group at the CPZ electrode are found in
Table 3. The RMS amplitudes for all of the electrodes are found in Appendix D.
Scalp Distribution Within Groups
The grand averages for each group and stimulus were analyzed using scalp
distributions. Each figure represents the difference between the -50 VOT scalp
distribution and the VOT as stated on the individual figure. Each figure contains 25
individual maps representing an 8 ms time frame as labeled below each map.
Group 1 -40 ms VOT (Figure 4). The distribution shows some early negativity in
the left hemisphere at 90-97 ms. Some occipital negativity occurs first at 114 ms and then
again between 186 and 225 ms. The strongest occipital negativity occurs between 258
and 265 ms but is still relatively small. There are no large negativities in this distribution.
Group 1 -30 ms VOT (Figure 5). The first signs of negativity in this distribution
occur in the occipital region at 106 to 113 ms although they are very slight negativities.
Small occipital negativities also occur at 170 to 177 ms, and from 218 to 249 ms. These
negativities are again very small. The most prominent negativities occur between 258 and
289 ms in the frontal and temporal regions, showing strongest on the right side.
Group 1 -20 ms VOT (Figure 6). This distribution again shows some early
negative processing in the frontal and temporal regions from 90 to 105 ms. There is
scattered negativity throughout the distribution occurring maximally at 154 to 161 ms in
the left temporal region. As with earlier distributions, all negativities occur in only small
portions. There is also some positive processing shown first, at 138 to 145 ms, and again
from 242 to 265 ms, occurring maximally form 258 to 265 ms.
22
Table 3
Descriptive Statistics for RMS Amplitudes of the MMN at the CPZ Electrode
Group Stimulus VOT Mean SD Min Max
1 -40 ms -0.99 0.63 -1.55 -0.11
1 -30 ms -1.10 0.45 -1.68 -0.67
1 -20 ms -0.62 0.38 -0.93 0.01
1 -10 ms -1.02 1.00 -2.09 0.69
1 0 ms -0.90 0.49 -1.50 0.02
1 10 ms -1.49 1.09 -4.12 -0.78
1 20 ms -0.98 0.82 -2.55 0.17
1 30 ms -0.94 0.72 -1.64 0.46
2 -40 ms -1.10 0.79 -2.01 -0.03
2 -30 ms -1.00 0.34 -1.42 -0.59
2 -20 ms -0.94 0.44 -1.54 -0.33
2 -10 ms -1.18 0.51 -2.01 -0.36
2 0 ms -0.97 0.88 -1.87 0.42
2 10 ms -0.46 0.59 -0.99 0.53
2 20 ms -1.50 0.36 -1.98 -1.17
2 30 ms -1.28 0.63 -2.67 -0.74
3 -40 ms -0.87 0.53 -1.49 0.04
3 -30 ms 0.13 0.98 -1.17 0.99
3 -20 ms -1.61 1.54 -4.48 -0.38
3 -10 ms -0.73 1.01 -1.99 0.91
3 0 ms -1.33 1.52 -4.68 0.29
3 10 ms -1.47 0.92 -3.02 -0.52
3 20 ms -0.93 1.16 -2.73 0.51
3 30 ms -0.99 1.55 -2.93 1.59
23
Figure 4. Scalp distribution map for group 1 -40 ms VOT.
24
Figure 5. Scalp distribution map for group 1 -30 ms VOT.
25
Figure 6. Scalp distribution map for group 1 -20 ms VOT.
26
Group 1 -10 ms VOT (Figure 7). The first prominent negativities show up in this
distribution. First, frontally from 106 to 121 ms. Negativity recurs generally from 130 to
145 ms, strongest first in the right frontal and temporal regions then moving to the right.
Another prominent negativity occurs at 186 to 193 ms appearing mainly in the right
central and temporal areas. The strongest negativity occurs occipitally from 266 to
289 ms.
Group 1 0 ms VOT (Figure 8). Negativity shows up occipitally from 106 to 121
ms and again from 138 to 145 ms. A large negativity occurs generally from 218 to 241
ms with the largest negativity occurring from 226 to 233 ms. Although the negativity is
generalized to all areas during this time, it its strongest in the occipital area. Some smaller
occipital negativities persist during the last 40 ms of the distribution.
Group 1 10 ms VOT (Figure 9). This distribution is filled with large negativities.
The most prominent of these occur from 130 to 145 ms in the entire right hemisphere,
and again from 154 to 161ms and from 194 to 201 ms. There is, again, a very large
negativity from 226 to 233 ms, this time in the left hemisphere. Smaller negativities
persist from 258 to 289 ms.
Group 1 20 ms VOT (Figure 10). In this distribution prominent early negativity
occurs in the left temporal and occipital areas from 90 to 105 ms. Although some
negativity continues during the majority of the distribution, the largest and strongest
negativities occur frontally from 154 to 193 ms. The most prominent of these is from 154
to 161 ms.
Group 1 30 ms VOT (Figure 11). There is some early negative and positive
processing during this distribution, but there is nothing prominent until about 186 ms.
27
Figure 7. Scalp distribution map for group 1 -10 ms VOT.
28
Figure 8. Scalp distribution map for group 1 0 ms VOT.
29
Figure 9. Scalp distribution map for group 1 10 ms VOT.
30
Figure 10. Scalp distribution map for group 1 20 ms VOT.
31
Figure 11. Scalp distribution map for group 1 30 ms VOT.
32
Large negativities occur from 210 to 233 ms and again from 250 to 257 ms, always
appearing strongest in the occipital areas.
Group 2 -40 ms VOT (Figure 12). The first distribution of group 2 contains many
small, scattered negativities throughout the distribution. The largest of these occur
frontally from 210 to 217 ms and from 242 to 257 ms. None of the negativities are very
large or prominent.
Group 2 -30 ms VOT (Figure 13). At -30 ms VOT negativities are again scattered
but occur slightly stronger and with less frequency than in the previous distribution.
There is some large left hemisphere negativity from 98 to 105 ms. From 106 to 193 ms
there is some frontal and occipital negativity. The most prominent of these occurs from
122 to 129 ms where the negativity is focused occipitally although it is also present in the
frontal and temporal areas.
Group 2 -20 ms VOT (Figure 14). The early portions of the -20 ms VOT
distribution show only small negativities. Prominent negativity appears at from 178 to
201 ms, showing strongest from 186 to 193 ms in the occipital and central areas. A
prominent left hemisphere negativity occurs from 226 to 241 ms with minor scattered
negativity after that.
Group 2 -10 ms VOT (Figure 15). Some strong negativity in the left frontal areas
occurs from 122 to 137 ms in the -10 ms VOT distribution. Large general negativity
occurs from 210 to 226 ms. Although the negativity is spread to all areas of the brain, the
strongest negativity is present in the left frontal quadrant. It is particularly strong from
218 to 225 ms.
33
Figure 12. Scalp distribution map for group 2 -40 ms VOT.
34
Figure 13. Scalp distribution map for group 2 -30 ms VOT.
35
Figure 14. Scalp distribution map for group 2 -20 ms VOT.
36
Figure 15. Scalp distribution map for group 2 -10 ms VOT.
37
Group 2 0 ms VOT (Figure 16). This distribution contains only light scattered
negativity throughout. The strongest of these occurs in the left hemisphere from 146 to
153 ms and in the frontal area from 282 to 289 ms.
Group 2 10 ms VOT (Figure 17). Small negativity is again scattered throughout
the distribution. A large negativity fills the central area from 138 to 145 ms with some
right hemisphere negativity lingering from 146 to 153 ms. Some large but relatively weak
negativity also occurs in the right hemisphere from 178 to 193 ms.
Group 2 20 ms VOT (Figure 18). Prominent negativity occurs throughout the left
and occipital areas from 122 to 129 ms during this distribution. From 130 to 137 ms
negativity is still strong in the left central and occipital areas, but has shifted from the left
frontal to right frontal area. The only other prominent negativity occurs frontally from
202 to 209 ms.
Group 2 30 ms VOT (Figure 19). The 30 ms VOT distribution is full of large
negativities beginning in the right central areas from 122 to 129 ms and from 138 to 146
ms. Large right central negativity occurs from 178 to 194 ms. There is another shift back
to the left from 218 ms to 234 ms. The most prominent negativity occurs from 242 to 257
ms where the negativity is spread throughout the brain. The negativity remains but
gradually diminishes for the remainder of the distribution.
Group 3 -40 ms VOT (Figure 20). There is some early processing in the frontal
areas from 98 to 122 ms. Prominent negativity is present in most of the front half of the
distribution from 210 to 249 ms. The strongest negativity is in the frontal portion.
Group 3 -30 ms VOT (Figure 21). This distribution again shows early frontal
negativity from 98 to 122 ms. Prominent negativity does not show up again until 250 ms
38
Figure 16. Scalp distribution map for group 2 0 ms VOT.
39
Figure 17. Scalp distribution map for group 2 10 ms VOT.
40
Figure 18. Scalp distribution map for group 2 20 ms VOT.
41
Figure 19. Scalp distribution map for group 2 30 ms VOT.
42
Figure 20. Scalp distribution map for group 3 -40 ms VOT.
43
Figure 21. Scalp distribution map for group 3 -30 ms VOT.
44
and lasts through 289 ms. The negativity is focused mostly in the right hemisphere with
some minor negativity on the left edge. Small negativity on the left edge is present
throughout most of the distribution.
Group 3 -20 ms VOT (Figure 22). Strong negativity is present centrally from 90
to 130 ms. From 138 to 145 ms the processing has shifted mainly to the right hemisphere.
The strongest negativity shows up from 90 to 105 ms and is focused near the frontal
areas. Strong frontal negativity is again present from 226 to 265 ms.
Group 3 -10 ms VOT (Figure 23). Strong general negativity shows up from 138 to
177 ms. The largest and most prominent negativity is present from 146 to 169 ms. During
this time the strongest negativity is focused toward the occipital region. Smaller
negativities are scattered throughout the distribution.
Group 3 0 ms VOT (Figure 24). As with most samples from this group, strong
negativity is present in the early processing from 90 to 113 ms. In this sample the focus is
near the occipital region. The negativity fades but shows up in the frontal portion from
130 to 153 ms. Other, smaller patches of negative processing are present near the
occipital region from 178 to 209 ms, frontally from 242 to 249 ms, and in a small patch in
the right hemisphere from 258 to 289 ms.
Group 3 10 ms VOT (Figure 25). Strong general negativity dominates the
distribution from 90 to 146 ms. Early on the negativity is strongest in the frontal and left
portions of the distribution and fades overall from 106 to 113 ms. The negativity comes
on strong again covering the entire map except the edges from 122 to 145 ms. Other
prominent negativities appear centrally and right temporally from 218 to 225 ms, and in
the right hemisphere from 274 to 289 ms.
45
Figure 22. Scalp distribution map for group 3 -20 ms VOT.
46
Figure 23. Scalp distribution map for group 3 -10 ms VOT.
47
Figure 24. Scalp distribution map for group 3 0 ms VOT.
48
Figure 25. Scalp distribution map for group 3 10 ms VOT.
49
Group 3 20 ms VOT (Figure 26). Strong negativity is limited from 122 ms to 129
ms where the entire map is dominated. Prior to that, from 106 to 121 ms negative
processing is found in the occipital and left temporal portions. Some occipital and right
temporal processing appears later on from 234 to 257 ms. See figure 26 below.
Group 3 30 ms VOT (Figure 27). Prominent negative processing is present from
106 to 121 ms focused mainly in the left hemisphere. Strong negativity shows up
frontally from 162 to 178 ms and again from 258 to 289 ms. Negativity dominates the
entire map from 274 to 281 ms and focuses in the frontal and left areas of the map.
Scalp Distribution Across Groups
The grand average scalp distributions were again analyzed using the brain maps.
This time the maps were analyzed across groups for each of the 8 stimuli.
-40 ms VOT. No prominent negativity shows up in either group 1 or group 2 with
the -40 ms VOT stimuli. Although group 2 shows slightly more negativity in the frontal
areas from 170 to 265 ms, the difference is minor. Group 3, however, shows prominent
frontal negativity from 210 to 249 ms.
-30 ms VOT. For the -30 ms VOT stimulus the most prominent processing is
again found in group 3, although the processing is very late, from 250 to 289 ms. Group 1
also shows some late processing but is not nearly as strong. Group 2 lacks the late
processing but shows some minor negativities in the intermediate times, particularly from
178 to 193 ms. Neither group 1 or group 3 show any prominent processing during this
time. Some early processing is found in groups 2 and 3, group 3 showing the stronger
negativities from 98 to 129 ms.
50
Figure 26. Scalp distribution map for group 3 20 ms VOT.
51
Figure 27. Scalp distribution map for group 3 30 ms VOT.
52
-20 ms VOT. At -20 ms VOT group 1 again shows the least amount of prominent
processing with only early perceptual processing showing up from 90 to 106 ms. Group 3
not only shows very strong early perceptual processing but also displays strong
processing from 122 to 145 ms. Very strong frontal negativity is also found from 226 to
257 ms. Group 2 lacks the early negativity but shows some strong negativity centrally
and occipitally from 186 to 201 ms. Some prominent negativity is also found overlapping
with that of group 3 from 226 to 241 ms although it is not as strong and is focused almost
completely in the left hemisphere.
-10 ms VOT. At -10 ms VOT group 1 finally shows some negative processing that
could represent a MMN. Frontal processing is present from 106 to 145 ms. Occipital
processing appears very late from 266 to 289 ms. Groups 2 and 3 still show more
prominent processing. Group 2 shows strong left hemisphere negativity from 122 to
137 ms and very strong negativity, again focused mainly on the left, from 210 to 233 ms.
Group 3 shows very strong negativity earlier than does group 2. The strongest negativity
is from 138 to 177 ms.
0 ms VOT. The differences found at 0 ms VOT are unexpected as group 2 shows
almost no negativity. Only very small negativities from 138 to 146 ms seem to have any
significance. Group 1 shows very strong negativity focused mainly in the occipital
portion from 218 to 226 ms. Group 3 again shows the most processing with strong early
perceptual processing from 90 to 113 ms and more strong frontal negativity from 130 to
153 ms. Smaller occipital and right central processing is found throughout the rest of the
distribution, particularly from 194 to 217 ms.
53
10 ms VOT. Strong negativities are present in all three groups at 10 ms VOT. The
strong negativities are limited in group 2 showing up only from 138 to 145 ms. Limited
smaller negativities are also found from 146 to 153 ms and from 178 to 193 ms. Groups 1
and 3 display extensive prominent negative processing. While the negativity in group 3 is
clustered together lasting from 90 to 161 ms and fading after that, group 1 has strong
negative processing spread throughout the distribution. The strongest negativities are
from 130 to 145 ms, from 154 to 161 ms, from 194 to 201 ms, from 226 to 233 ms, and
from 266 to 289 ms.
20 ms VOT. At 20 ms VOT all 3 groups again show some prominent processing,
but less than at 10 ms. Group 1 displays strong frontal processing from 146 to 193 ms.
Group 2 again shows less, but strong negativity is present from 122 to 137 ms. Group 3
displays very strong processing only from 122 to 129 ms, with some prominent
processing on before and after. There is also some strong occipital negativity from 234 to
257 ms.
30 ms VOT. All three groups show scattered but strong negative processing at 30
ms VOT. Group 1 lacks negativity in the early stages but shows strong processing from
186 to 201 ms, from 210 to 233 ms, and from 250 to 257 ms. Group 2 displays small
spots of strong negativity from 122 to 129 ms and from 138 to 153 ms. More wide spread
negativity is found from 170 to 201 ms and from 210 to 273 ms. The focus of the
negativity shifts significantly as time passes. Group 3 shows strong negative processing
that is not quite as sporadic as in group 2, but more spread out than in group 1. Early
processing is found from 106 to 121 ms. Very strong processing then occurs from 162 to
185 ms. The very late stages from 258 to 289 ms also show some strong processing.
54
Statistical Analysis
A multivariate analysis of variance for the independent variables of groups and
stimuli failed to show any significant F ratios at p ≤ .05.
55
Discussion
The present study examined /bɑ/-/pɑ/ speech sound discrimination in 3 different
groups of listeners using an array of stimuli on a VOT continuum from -50 ms to 30 ms.
The -50 ms stimulus was used as the standard and all other stimuli were compared to it as
the deviants in an odd-ball paradigm to elicit the MMN. At some point in that continuum
the difference between the standard and the deviant stimuli represented a WC change for
native English listeners and an AC change for native Spanish listeners. The group of
interest was those who had learned Spanish as a second language. It was of interest to
know if they were able to make the sound discrimination that native Spanish listeners
make when the sound pair represents a WC change for native English listeners and
whether or not the MMN shows this discrimination.
Statistical analysis of the behavioral data showed a significant difference between
native Spanish listeners and English-only listeners regarding when they perceive /pɑ/
instead of /bɑ/. There was also a significant difference between L2 Spanish listeners and
English-only listeners, but not between native Spanish listeners and L2 Spanish listeners.
This indicates that the L2 Spanish listeners’ ability to distinguish Spanish speech sounds
was similar to that of the native Spanish listeners, and it was better than the English only
listeners.
Although statistical analysis of the groups and stimuli at the CPZ electrode was
not significant, the difference in strong negativities shown on the distribution maps
support the theory explored in previous studies that underlying neural-sensory
representations of speech may be altered by linguistic experiences (Dehaene-Lambertz,
1997). Analysis using groupings of electrodes or looking at all electrodes may have
56
shown significant differences and future studies should address different methods of
analysis. The differences between the three groups found in the distribution maps did not
occur at the same place as those found in the behavioral data. Scalp distribution maps
showed strong negativity within the time frame of the MMN at -20 and -10 ms VOT in
both the native Spanish and L2 Spanish listeners, while English only listeners did not
show strong negativity within the time frame of the MMN until 0 ms VOT. The anomaly
is that at 0 ms VOT, where behavioral data showed that native Spanish listeners had
begun to perceive /pɑ/ about 40% of the time and English-only listeners were at about
10%, very little negativity appeared in the native Spanish listeners’ maps. At 10 ms VOT
all three groups showed strong negativities in the time frame of the MMN but the native
Spanish listeners showed the weakest negativities. The reason for these weak negativities
at 0 ms in native Spanish listeners is unclear. One possibility is that the behavioral task is
more sensitive than the MMN to VOT.
Both the behavioral and the electrophysiological data showed differences between
the groups, but the differences did not correspond. The behavioral data showed a
separation between 0 and 20 ms VOT, where native Spanish listeners perceived a
difference between /bɑ/ and /pɑ/ slightly earlier than did English only listeners, and L2
Spanish listeners were in between. The electrophysiological data showed a distinction
between -20 and 0 ms VOT where native Spanish and L2 Spanish listeners perceived a
difference between /bɑ/ and /pɑ/ 20 ms earlier than did English only listeners. Previous
studies using the MMN to test L2 learning have not reported any differences between
behavioral and electrophysiological results (Nenonen et al., 2005; Shestakova et al.,
2003; Winkler & Kujala et al., 1999). Studies such as that by Sharma and Dorman (2000)
57
that explored the MMN in cross-language phonetic perception without exploring an L2
similarly did not show a discrepancy between behavioral and electrophysiological data.
The major difference between the current study and these studies is that none of them
used a large continuum of stimulus tokens. They used only one or two stimulus pairs and
any significant difference between behavioral and MMN data might have contradicted
the findings of their study. Although a difference existed in the current study between
behavioral and MMN data, both sets of data support the theory that L2 language learning
may alter underlying neural-sensory representations of speech. More research must be
done in order to determine if this discrepancy is an anomaly to this study or if similar
patterns could be found in other languages or with other phoneme comparisons.
Overall, the findings from the study support the work by Winkler & Kujala et al.
(1999). Their study using Hungarian-only listeners, Hungarians fluent in Finnish, and
native Finnish listeners, similarly found that when tested on Finnish vowel contrasts not
found in Hungarian, an MMN response was not elicited in the Hungarian-only listeners.
The MMN was elicited in the other two groups, however. The researchers concluded that
L2 listeners developed cortical memory representations for the phoneme system of the
new language enabling them to process these phonemes pre-attentively as do native
listeners. The current study similarly concludes that the loss of non-native speech sound
discrimination abilities that seems to occur after 6-8 months of age and the regaining of
these abilities in adulthood after an L2 is learned is apparently not due merely to
attentional bias. Research by Shestakova et al. (2003) was conducted using Finnish
children learning French as an L2, who had only been exposed to the new language for
12-16 weeks. Despite the limited exposure, they too found that L2 learners had increased
58
MMN magnitudes over Finnish-only children when presented with French vowel
contrasts.
Future studies should continue to explore the effect of L2 learning on neural-
sensory representations of speech. Non-native consonant contrasts other than the /bɑ/-
/pɑ/ contrast, and other language comparisons besides English-Spanish should be done.
Such studies could help to explain whether there are differences between L2 learning of
vowels and L2 learning of consonants. Up until now most L2 studies using the MMN
have focused on vowels and have shown that the neural-sensory representations of speech
are affected in L2 learners (Shestakova et al., 2003; Winkler & Kujala et al., 1999). The
current study tested consonants that differed in VOT and demonstrated the same thing,
but there are aspects of consonant distinction, other than VOT, such as place of
articulation that could be explored to shed more light on how the brain learns a new
language.
59
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APPENDIXES
Appendix A
Informed Consent Document
CONSENT TO ACT AS A HUMAN RESEARCH SUBJECT Speech Sound Discrimination in L2 Spanish Listeners David L. McPherson, Ph.D. Department of Audiology and Speech Language Pathology Brigham Young University (801) 422-6458 Name of Participant: _____________________________ Date of Birth: _____________ Purpose of Study: This investigation is designed to study how the brain processes nonnative speech sounds in second language learners. Participation in this study will help scientists better understand how second languages are learned in adulthood. Procedures: I have been asked to participate in a research study being conducted by Dr. David L. McPherson and/or such assistants as may be selected by him. The study will be conducted in room 109 and 111 of the John Taylor Building on the Brigham Young University campus. I will be asked questions but do not have to answer any questions that I do not want to answer. Participation in this study, including orientation and testing, requires two sessions, equaling about 2 hours, total. These two sessions will be scheduled on separate days, but it is possible for the two sessions to be combined into one visit. The first session will be for a hearing screening and a question and answer session, which should last approximately 20 minutes. The second session will be for the actual data collection, which should last about 1 ½ hours. I have been advised that these times are an estimate, and it may take more or less time, depending on how easy it is to set up the equipment. I may ask for a break at any time during testing. I will be given a standard hearing test screening. My ears will be looked into with a scope, my eardrums will be tested to see how they move, and my hearing will be tested. Surface electrodes (metal discs about the size of a dime) will be used to record electrical activity of the brain. These discs will be applied to the surface of the skin with a gel and are easily removed with water. Blunt needles will be used as a part of this study to help apply the electrode gel, but they will never be used to puncture the skin. Electrical activity of my brain will be recorded, but no electrical impulses/shocks will be applied to the brain. This is similar to what is known as an “EEG” or brain wave test. These procedures show actual measurements of normal, continuous, electrical activity in the brain. Some sounds will be presented though earphones. I will be asked to push one button when the sounds are the same and another when they are different. These clinical procedures are routine, similar to those used to test hearing, although some techniques of
65
analysis are experimental. Risks: There may be some local skin irritation resulting from the electrode discs. This will be treated in the usual manner by removing the discs and exposing the area to air, which results in alleviation of the irritation. Another possible, but unlikely, discomfort would be if the scalp received an abrasion when the blunt needle is used to place the electrode gel. The electrodes would be removed immediately, and any gel on the injured site would be removed. If this were to occur I would be given the option to discontinue my participation in the study. There are no other known risks with this procedure. Benefits: Possible benefits from participating in this study will be the assessment of my hearing. I will be notified of any irregularities in my ears (structures) and/or hearing abilities found during the routine hearing tests. If irregularities are discovered, I may be advised to have a professional examine my ears/hearing, or I may be advised of possible treatments (if any). These procedures will benefit me by providing (possible) early treatment. I also understand there may be no direct benefit to me. However, the information obtained will help people further understand how the brain processes auditory information and about differences in these processes between native speakers of a language and second language learners. Confidentiality: Participation in this study is voluntary and I have a right to refuse to participate or withdraw at any time, without penalty. All information obtained from testing is strictly confidential and is protected under the laws governing privacy. No information specifically pertaining to me, other than reporting of test results without identifying information may be released without my signature. All identifying references will be removed and replaced by control numbers which will identify any disclosed or published data. Data collected in this study will be stored in a secured area accessible only to personnel associated with the study. Other Considerations: There are no charges incurred by me for participation in this study. There is no treatment or intervention involved in this study although I may be counseled to seek such treatment or intervention. I understand that for any reason I may withdraw from the study at any time without penalty. The procedures listed above have been explained to me by: __________________ in a satisfactory manner and my questions relating to such risks and procedures have been answered. If I have any questions about the research I may ask any of the investigators or contact Dr. David L. McPherson, Audiology and Speech-Language Pathology, 129 TLRB, Provo, Utah 84602-8633; email: [email protected]; phone: 801-422-6458. If I have any questions as to my rights as a participant in this research project I may contact Renea Beckstrand, Chair of Institutional Review Board, email: [email protected]; phone: 801-422-3873. I consent to participate in the above explained study. __________________________________ ________________ Signature of Participant Date
66
Appendix B Words Used for Stimulus Recordings
/pɑ/ words
Paca (bale, pack)
Pase (pass, come in)
Pala (shovel)
Paño (cloth)
Panda (Panda, group)
Pasta (paste)
/bɑ/ words
Baca (roof, rack)
Base (base)
Bala (bullet)
Baño (bathroom)
Banda (band, ribbon)
Basta (enough)
67
Appendix C
List of Sentences Read by Speaker
Diga la palabra paca otra vez.
Diga la palabra pala otra vez.
Diga la palabra basta otra vez.
Diga la palabra banda otra vez.
Diga la palabra pala otra vez.
Diga la palabra baño otra vez.
Diga la palabra bala otra vez.
Diga la palabra base otra vez.
Diga la palabra paño otra vez.
Diga la palabra panda otra vez.
Diga la palabra baca otra vez.
Diga la palabra pasta otra vez.
Diga la palabra paca otra vez.
Diga la palabra pala otra vez.
Diga la palabra basta otra vez.
Diga la palabra banda otra vez.
Diga la palabra pala otra vez.
Diga la palabra baño otra vez.
Diga la palabra bala otra vez.
Diga la palabra base otra vez.
Diga la palabra paño otra vez.
Diga la palabra panda otra vez.
Diga la palabra baca otra vez.
Diga la palabra pasta otra vez.
Diga la palabra paca otra vez.
Diga la palabra pala otra vez.
Diga la palabra basta otra vez.
Diga la palabra banda otra vez.
Diga la palabra pala otra vez.
Diga la palabra baño otra vez.
Diga la palabra bala otra vez.
Diga la palabra base otra vez.
Diga la palabra paño otra vez.
Diga la palabra panda otra vez.
Diga la palabra baca otra vez.
Diga la palabra pasta otra vez.
Diga la palabra paca otra vez.
Diga la palabra pala otra vez.
Diga la palabra basta otra vez.
Diga la palabra banda otra vez.
Diga la palabra pala otra vez.
Diga la palabra baño otra vez.
68
Diga la palabra bala otra vez.
Diga la palabra base otra vez.
Diga la palabra paño otra vez.
Diga la palabra panda otra vez.
Diga la palabra baca otra vez.
Diga la palabra pasta otra vez.
Diga la palabra paca otra vez.
Diga la palabra pala otra vez.
Diga la palabra basta otra vez.
Diga la palabra banda otra vez.
Diga la palabra pala otra vez.
Diga la palabra baño otra vez.
Diga la palabra bala otra vez.
Diga la palabra base otra vez.
Diga la palabra paño otra vez.
Diga la palabra panda otra vez.
Diga la palabra baca otra vez.
Diga la palabra pasta otra vez.
69
Appendix D
Tables of RMS Amplitudes
Table D1
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of -40 ms VOT
Location Mean SD Min Max
O2 0.02 0.35 -0.31 0.60
O1 0.47 0.96 -0.36 2.13
OZ 0.04 0.35 -0.47 0.44
PZ -0.73 0.52 -1.36 0.04
P4 -0.57 0.69 -1.42 0.37
CP4 -0.73 0.88 -1.96 0.31
P8 -0.42 0.55 -1.06 0.22
C4 -1.03 1.02 -2.52 0.23
TP8 -0.43 0.56 -1.05 0.30
T8 -0.60 0.61 -1.62 -0.17
P7 0.15 0.35 -0.25 0.45
P3 -0.21 0.53 -0.99 0.49
CP3 -0.41 0.55 -0.98 0.51
CPZ -0.99 0.63 -1.55 -0.11
CZ -1.09 0.80 -1.74 0.26
FC4 -1.13 1.19 -3.05 0.21
FT8 -0.78 0.94 -2.44 -0.18
TP7 -0.06 0.47 -0.61 0.44
C3 -0.60 0.63 -1.05 0.45
FCZ -1.33 0.89 -2.27 0.14
FZ -1.41 1.19 -3.25 0.05
F4 -1.06 1.27 -3.24 0.09
F8 -0.86 1.07 -2.74 -0.18
T7 -0.37 0.56 -0.90 0.54
FT7 -0.44 0.74 -1.28 0.56
FC3 -1.13 0.70 -1.73 0.00
F3 -1.06 0.88 -2.26 0.12
70
FP2 -0.58 1.34 -2.88 0.48
F7 -0.66 0.79 -1.63 0.53
FP1 -0.67 1.07 -2.38 0.57
71
Table D2
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of -30 ms VOT
Location Mean SD Min Max
O2 -0.10 0.65 -0.83 0.70
O1 -0.13 0.31 -0.34 0.32
OZ -0.31 0.53 -0.96 0.34
PZ -0.98 0.89 -1.85 0.25
P4 -0.93 0.48 -1.47 -0.33
CP4 -1.24 0.41 -1.85 -0.97
P8 -0.46 0.29 -0.85 -0.16
C4 -1.19 0.38 -1.62 -0.82
TP8 -0.67 0.21 -0.87 -0.39
T8 -0.84 0.19 -1.11 -0.67
P7 0.06 0.78 -0.54 1.21
P3 -0.25 1.04 -1.22 1.18
CP3 -0.63 1.11 -1.77 0.74
CPZ -1.10 0.45 -1.68 -0.67
CZ -1.07 0.37 -1.60 -0.79
FC4 -1.05 0.44 -1.64 -0.60
FT8 -0.73 0.38 -1.15 -0.23
TP7 -0.03 0.91 -0.94 1.22
C3 -0.63 1.18 -1.96 0.80
FCZ -0.80 0.24 -1.00 -0.46
FZ -0.55 0.23 -0.81 -0.31
F4 -0.99 0.63 -1.56 -0.13
F8 -0.82 0.48 -1.40 -0.23
T7 -0.04 0.94 -0.86 1.26
FT7 0.08 0.89 -0.84 1.29
FC3 -0.28 0.63 -0.86 0.50
F3 -0.03 0.34 -0.42 0.39
FP2 -0.73 0.79 -1.48 0.17
F7 0.06 0.89 -0.83 1.29
FP1 -0.31 0.09 -0.41 -0.23
72
Table D3
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of -20 ms VOT
Location Mean SD Min Max
O2 -0.56 0.66 -1.48 0.21
O1 -0.22 0.55 -0.93 0.34
OZ 0.05 0.56 -0.59 0.70
PZ -0.57 1.08 -1.43 1.31
P4 -1.24 1.32 -3.48 -0.05
CP4 -0.93 0.52 -1.71 -0.29
P8 -0.46 0.68 -0.99 0.53
C4 -0.54 0.32 -0.92 -0.22
TP8 -0.35 0.58 -0.93 0.45
T8 -0.29 0.51 -0.92 0.16
P7 0.16 0.83 -0.68 1.44
P3 -0.27 0.68 -1.04 0.41
CP3 -0.21 0.53 -0.85 0.43
CPZ -0.62 0.38 -0.93 0.01
CZ -0.55 0.43 -1.11 -0.02
FC4 -0.23 0.54 -0.87 0.58
FT8 0.08 0.57 -0.80 0.79
TP7 0.25 0.75 -0.57 1.46
C3 -0.22 0.47 -0.83 0.49
FCZ -0.67 0.74 -1.31 0.45
FZ -0.30 0.81 -1.32 0.66
F4 -0.07 0.80 -1.12 1.02
F8 -0.01 0.74 -1.20 0.79
T7 0.02 0.43 -0.73 0.31
FT7 0.04 0.45 -0.75 0.38
FC3 -0.19 0.54 -0.78 0.69
F3 -0.05 0.63 -0.81 0.66
FP2 -0.14 0.79 -1.32 0.58
F7 -0.02 0.42 -0.75 0.31
FP1 -0.01 0.66 -0.88 0.58
73
Table D4
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of -10 ms VOT
Location Mean SD Min Max
O2 -0.11 0.54 -0.96 0.37
O1 0.17 0.70 -1.12 0.99
OZ 0.06 0.72 -1.19 1.00
PZ -0.88 0.81 -1.99 0.37
P4 -0.58 0.78 -1.30 0.93
CP4 -0.70 0.96 -1.36 1.18
P8 -0.35 0.30 -0.66 0.11
C4 -0.53 1.17 -1.51 1.65
TP8 -0.40 0.21 -0.61 -0.12
T8 -0.33 0.58 -1.25 0.43
P7 0.02 1.02 -1.47 1.69
P3 -0.38 1.02 -1.99 0.98
CP3 -0.51 1.15 -1.89 1.44
CPZ -1.02 1.00 -2.09 0.69
CZ -0.87 0.99 -1.88 0.85
FC4 -0.14 1.21 -1.39 1.96
FT8 -0.24 0.83 -1.60 0.73
TP7 0.11 0.87 -0.99 1.66
C3 -0.55 0.81 -1.58 0.72
FCZ -0.61 1.04 -1.64 1.14
FZ -0.30 0.73 -1.31 0.64
F4 -0.13 0.81 -1.22 0.81
F8 -0.14 0.82 -1.60 0.67
T7 0.03 0.61 -0.79 1.09
FT7 0.06 0.50 -0.26 1.07
FC3 -0.45 0.47 -0.90 0.36
F3 -0.42 0.28 -0.88 -0.12
FP2 -0.09 1.02 -1.64 1.39
F7 0.02 0.52 -0.33 1.06
FP1 -0.58 0.51 -1.45 0.13
74
Table D5
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of 0 ms VOT
Location Mean SD Min Max
O2 -0.84 0.61 -1.76 0.06
O1 -0.61 0.48 -1.48 0.29
OZ -0.78 0.57 -1.48 0.31
PZ -1.02 0.51 -1.62 -0.11
P4 -1.02 0.99 -2.62 0.61
CP4 -0.86 0.83 -1.61 0.91
P8 -0.53 0.85 -2.42 0.91
C4 -0.54 0.87 -1.62 1.39
TP8 -0.42 0.94 -2.23 1.41
T8 -0.37 0.87 -1.59 1.56
P7 -0.02 0.53 -0.93 1.00
P3 -0.58 0.50 -1.48 0.25
CP3 -0.47 0.56 -1.15 0.90
CPZ -0.90 0.49 -1.50 0.02
CZ -0.71 0.91 -1.89 1.15
FC4 -0.44 1.21 -1.90 1.72
FT8 -0.48 1.03 -2.74 1.17
TP7 0.09 0.51 -0.87 1.01
C3 -0.43 0.70 -1.35 1.38
FCZ -0.55 1.17 -1.97 1.51
FZ -0.38 1.17 -1.53 1.73
F4 -0.36 1.11 -1.77 1.51
F8 -0.43 1.16 -2.89 1.34
T7 0.16 0.48 -0.52 1.19
FT7 0.03 0.62 -0.73 1.22
FC3 -0.46 0.89 -1.63 1.73
F3 -0.23 0.88 -1.00 1.89
FP2 -0.37 1.05 -2.22 1.10
F7 0.07 0.77 -1.21 1.21
FP1 -0.14 0.97 -1.85 1.74
75
Table D6
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of 10 ms VOT
Location Mean SD Min Max
O2 -0.58 0.75 -1.96 0.40
O1 -0.62 0.40 -1.30 -0.08
OZ -0.36 0.58 -1.59 0.34
PZ -1.28 0.71 -2.79 -0.69
P4 -1.06 0.62 -2.14 -0.22
CP4 -0.96 0.75 -2.46 -0.17
P8 -0.49 0.54 -1.56 -0.03
C4 -0.79 0.73 -2.06 -0.03
TP8 -0.19 0.30 -0.51 0.36
T8 0.05 0.50 -0.62 0.72
P7 -0.29 0.42 -1.31 -0.02
P3 -0.97 0.77 -1.96 0.01
CP3 -1.17 0.79 -2.58 -0.26
CPZ -1.49 1.09 -4.12 -0.78
CZ -1.46 1.24 -4.28 -0.37
FC4 -0.74 0.99 -2.56 0.59
FT8 0.15 0.60 -0.58 0.98
TP7 -0.31 0.30 -1.01 -0.03
C3 -1.13 0.83 -2.59 0.03
FCZ -1.33 1.31 -4.29 -0.27
FZ -0.91 1.29 -3.46 0.60
F4 -0.46 0.99 -2.01 1.02
F8 0.15 0.67 -0.72 1.09
T7 -0.29 0.32 -0.99 -0.01
FT7 -0.36 0.34 -0.98 0.01
FC3 -0.87 1.08 -2.90 0.76
F3 -0.77 0.87 -2.38 0.17
FP2 -0.17 0.78 -1.30 0.87
F7 -0.39 0.35 -1.01 0.01
FP1 -0.57 0.61 -1.56 0.32
76
Table D7
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of 20 ms VOT
Location Mean SD Min Max
O2 -0.25 0.73 -1.36 1.01
O1 -0.03 0.98 -0.95 1.84
OZ -0.08 0.80 -1.00 1.35
PZ -0.88 0.60 -1.70 -0.03
P4 -0.47 0.79 -1.80 0.52
CP4 -0.56 1.21 -2.75 0.93
P8 -0.25 0.62 -1.10 0.48
C4 -0.79 1.25 -3.20 0.90
TP8 -0.18 0.78 -1.32 0.78
T8 -0.51 0.81 -1.89 0.38
P7 0.18 0.72 -0.98 1.29
P3 -0.33 0.62 -1.26 0.67
CP3 -0.57 0.66 -1.69 0.04
CPZ -0.98 0.82 -2.55 0.17
CZ -1.18 0.98 -3.32 -0.43
FC4 -1.06 1.16 -3.60 -0.23
FT8 -0.54 0.68 -1.55 0.46
TP7 0.08 0.77 -1.00 1.26
C3 -0.77 0.86 -2.52 -0.13
FCZ -1.36 1.10 -3.76 -0.68
FZ -1.31 1.23 -3.96 -0.48
F4 -1.21 1.07 -3.17 -0.18
F8 -0.52 0.97 -2.45 0.45
T7 -0.22 0.64 -1.17 0.55
FT7 -0.29 0.73 -1.56 0.57
FC3 -0.95 1.32 -3.75 0.18
F3 -0.95 1.18 -3.50 -0.15
FP2 -0.76 0.92 -2.03 0.16
F7 -0.41 0.76 -1.55 0.57
FP1 -0.55 0.66 -1.56 0.35
77
Table D8
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 1 with Stimulus of 30 ms VOT
Location Mean SD Min Max
O2 -0.09 0.53 -0.94 0.53
O1 -0.31 0.71 -1.17 0.92
OZ -0.21 0.54 -0.81 0.65
PZ -0.77 0.73 -1.63 0.66
P4 -0.61 0.80 -1.56 1.01
CP4 -0.63 0.82 -1.21 1.07
P8 -0.08 0.50 -0.84 0.54
C4 -0.61 0.88 -1.57 1.13
TP8 -0.03 0.53 -0.76 0.66
T8 0.11 0.45 -0.38 0.78
P7 -0.43 0.48 -1.21 0.24
P3 -0.77 0.78 -1.77 0.85
CP3 -0.92 0.79 -2.05 0.60
CPZ -0.94 0.72 -1.64 0.46
CZ -0.91 0.71 -1.86 0.22
FC4 -0.70 0.89 -1.61 0.80
FT8 0.00 0.57 -0.72 1.24
TP7 -0.51 0.37 -0.99 0.15
C3 -0.87 0.68 -1.82 0.31
FCZ -0.96 0.94 -2.19 0.33
FZ -1.01 0.93 -2.17 0.15
F4 -0.82 1.00 -2.27 0.54
F8 0.00 0.64 -0.87 1.27
T7 -0.47 0.48 -1.26 0.10
FT7 -0.56 0.68 -1.85 0.10
FC3 -1.07 0.82 -2.41 -0.10
F3 -0.92 0.98 -2.40 0.67
FP2 -0.44 0.68 -1.61 0.82
F7 -0.47 0.74 -1.81 0.58
FP1 -0.52 0.63 -1.51 0.64
78
Table D9
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of -40 ms VOT
Location Mean SD Min Max
O2 -0.26 0.91 -1.47 0.74
O1 -0.61 0.65 -1.57 0.28
OZ -0.49 0.53 -1.29 0.07
PZ -0.74 0.94 -2.20 0.28
P4 -0.67 0.93 -1.86 0.16
CP4 -0.71 0.98 -2.01 0.27
P8 -0.41 0.90 -1.74 0.44
C4 -0.95 1.00 -2.52 -0.02
TP8 -0.42 0.65 -1.28 0.25
T8 -0.34 0.78 -1.49 0.71
P7 -0.50 0.44 -1.04 0.23
P3 -0.84 0.62 -1.61 -0.11
CP3 -1.18 0.53 -1.63 -0.37
CPZ -1.10 0.79 -2.01 -0.03
CZ -1.28 0.67 -2.13 -0.33
FC4 -0.82 0.82 -2.31 0.04
FT8 -0.22 0.85 -1.50 0.93
TP7 -0.49 0.48 -1.02 0.25
C3 -1.08 0.52 -1.60 -0.45
FCZ -1.22 0.76 -2.13 -0.03
FZ -1.16 0.65 -1.95 -0.01
F4 -0.67 0.92 -1.87 0.28
F8 -0.35 0.75 -1.52 0.58
T7 -0.55 0.53 -1.25 0.26
FT7 -0.39 0.42 -0.94 0.10
FC3 -1.12 0.53 -1.73 -0.34
F3 -0.83 0.60 -1.46 0.01
FP2 -0.37 0.99 -1.51 1.00
F7 -0.37 0.52 -1.11 0.24
FP1 -0.55 0.89 -1.71 0.57
79
Table D10
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of -30 ms VOT
Location Mean SD Min Max
O2 -0.58 0.74 -1.62 0.40
O1 -0.06 0.56 -0.76 0.78
OZ -0.33 0.90 -1.57 0.81
PZ -0.66 0.65 -1.27 0.11
P4 -0.66 0.90 -1.50 0.48
CP4 -0.69 0.68 -1.19 0.43
P8 -0.43 0.48 -0.88 0.25
C4 -0.63 0.49 -1.00 0.08
TP8 -0.32 0.44 -0.88 0.14
T8 -0.29 0.58 -0.93 0.46
P7 -0.26 0.36 -0.61 0.20
P3 -0.94 0.45 -1.52 -0.45
CP3 -1.02 0.66 -2.12 -0.51
CPZ -1.00 0.34 -1.42 -0.59
CZ -0.73 0.38 -1.27 -0.30
FC4 -0.81 0.16 -1.02 -0.58
FT8 -0.29 0.68 -1.21 0.28
TP7 -0.27 0.51 -0.86 0.31
C3 -0.72 0.51 -1.46 -0.04
FCZ -0.52 0.48 -0.97 0.26
FZ -0.39 0.47 -0.74 0.37
F4 -0.41 0.53 -1.06 0.37
F8 -0.15 0.70 -1.17 0.62
T7 -0.17 0.64 -0.87 0.49
FT7 -0.15 0.85 -1.15 0.74
FC3 -0.47 0.65 -1.25 0.41
F3 -0.39 0.86 -1.50 0.60
FP2 -0.14 0.67 -1.15 0.47
F7 -0.01 0.82 -0.99 0.83
FP1 -0.24 0.79 -1.52 0.52
80
Table D11
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of -20 ms VOT
Location Mean SD Min Max
O2 -0.65 0.52 -1.44 0.04
O1 -0.38 0.80 -1.22 0.57
OZ -0.59 0.71 -1.61 0.11
PZ -0.63 0.11 -0.74 -0.45
P4 -0.61 0.34 -1.13 -0.28
CP4 -0.79 0.59 -1.48 0.12
P8 -0.54 0.50 -1.11 0.22
C4 -0.67 0.83 -1.34 0.67
TP8 -0.69 0.60 -1.42 0.18
T8 -0.44 0.94 -1.23 0.77
P7 -0.32 0.67 -1.18 0.22
P3 -0.83 0.79 -2.12 0.00
CP3 -0.73 0.63 -1.83 -0.27
CPZ -0.94 0.44 -1.54 -0.33
CZ -0.56 0.77 -1.07 0.70
FC4 -0.58 1.03 -1.53 0.90
FT8 -0.41 1.01 -1.34 0.78
TP7 -0.23 0.76 -1.12 0.48
C3 -0.70 0.54 -1.63 -0.23
FCZ -0.07 1.25 -1.53 1.60
FZ -0.41 0.89 -1.16 0.98
F4 -0.48 1.10 -1.75 0.94
F8 -0.22 1.01 -1.37 0.82
T7 -0.32 0.64 -1.26 0.28
FT7 -0.20 0.80 -1.31 0.83
FC3 -0.51 0.66 -1.40 0.44
F3 -0.40 0.69 -1.22 0.65
FP2 -0.16 1.07 -1.40 1.06
F7 -0.03 0.56 -0.54 0.83
FP1 -0.01 0.65 -0.44 1.13
81
Table D12
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of -10 ms VOT
Location Mean SD Min Max
O2 -0.42 0.60 -1.18 0.66
O1 -0.32 0.67 -0.95 1.03
OZ -0.26 0.72 -0.97 1.15
PZ -0.88 0.43 -1.48 -0.10
P4 -0.70 0.32 -1.25 -0.32
CP4 -1.14 0.76 -2.82 -0.27
P8 -0.65 0.42 -1.16 0.15
C4 -1.07 0.53 -2.14 -0.57
TP8 -0.64 0.80 -2.11 0.59
T8 -0.56 1.01 -2.14 0.98
P7 -0.52 0.55 -1.27 0.19
P3 -0.77 0.63 -1.56 0.21
CP3 -1.04 0.68 -2.10 0.04
CPZ -1.18 0.51 -2.01 -0.36
CZ -1.26 0.76 -2.66 -0.33
FC4 -1.09 0.55 -1.93 -0.39
FT8 -0.76 1.18 -2.88 0.58
TP7 -0.53 0.56 -1.42 0.24
C3 -1.18 0.84 -2.82 0.01
FCZ -1.37 0.55 -2.25 -0.59
FZ -1.24 0.71 -2.24 -0.31
F4 -1.08 0.76 -2.66 -0.20
F8 -0.65 1.14 -3.14 0.42
T7 -0.61 0.55 -1.36 0.48
FT7 -0.69 0.44 -1.33 0.07
FC3 -1.13 0.66 -1.75 0.09
F3 -0.95 0.65 -2.25 -0.25
FP2 -0.89 1.26 -3.69 0.46
F7 -0.73 0.54 -1.37 0.06
FP1 -0.84 0.99 -3.05 0.23
82
Table D13
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of 0 ms VOT
Location Mean SD Min Max
O2 -0.11 0.50 -1.01 0.19
O1 -0.08 1.08 -0.83 1.81
OZ -0.09 0.84 -0.67 1.37
PZ -0.55 1.00 -1.40 0.97
P4 -0.41 0.47 -0.82 0.31
CP4 -0.77 0.60 -1.35 -0.02
P8 -0.15 0.71 -0.81 1.03
C4 -0.87 0.87 -1.65 0.53
TP8 -0.26 0.46 -0.75 0.42
T8 -0.35 0.56 -1.00 0.44
P7 -0.20 0.76 -0.74 1.11
P3 -0.55 0.88 -1.35 0.86
CP3 -0.75 0.81 -1.36 0.64
CPZ -0.97 0.88 -1.87 0.42
CZ -1.21 0.85 -1.89 0.26
FC4 -1.14 0.75 -1.90 0.08
FT8 -0.70 0.24 -0.93 -0.35
TP7 -0.42 0.81 -0.98 0.98
C3 -1.01 0.62 -1.45 0.01
FCZ -1.24 0.88 -1.97 0.23
FZ -0.91 0.75 -1.53 0.33
F4 -1.02 0.52 -1.47 -0.17
F8 -0.77 0.25 -1.15 -0.51
T7 -0.39 0.49 -1.00 0.30
FT7 -0.17 0.63 -0.72 0.71
FC3 -0.93 0.94 -1.63 0.60
F3 -0.74 0.70 -1.33 0.46
FP2 -0.65 0.50 -1.24 0.09
F7 0.05 0.91 -0.71 1.52
FP1 -0.13 1.00 -0.90 1.45
83
Table D14
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of 10 ms VOT
Location Mean SD Min Max
O2 -0.35 0.42 -1.03 0.05
O1 -0.56 0.39 -1.01 -0.16
OZ -0.77 0.75 -2.00 -0.20
PZ -0.54 0.39 -0.92 -0.03
P4 -0.59 0.76 -1.45 0.31
CP4 -0.57 0.86 -1.38 0.36
P8 -0.32 0.70 -0.99 0.45
C4 -0.43 0.97 -1.49 0.74
TP8 -0.24 0.78 -0.91 0.67
T8 -0.25 0.87 -1.15 0.79
P7 0.01 0.53 -0.40 0.90
P3 -0.26 0.29 -0.60 -0.01
CP3 -0.10 0.44 -0.60 0.36
CPZ -0.46 0.59 -0.99 0.53
CZ -0.35 0.73 -0.82 0.86
FC4 -0.56 1.04 -1.56 0.94
FT8 -0.24 0.87 -0.87 1.10
TP7 0.14 0.43 -0.32 0.75
C3 -0.04 0.58 -0.56 0.88
FCZ -0.23 0.85 -0.84 1.17
FZ -0.22 0.84 -1.30 1.03
F4 -0.42 1.07 -1.54 1.22
F8 -0.29 0.93 -1.22 1.10
T7 0.21 0.60 -0.39 1.19
FT7 0.20 0.48 -0.37 0.77
FC3 -0.19 0.61 -0.49 0.91
F3 -0.16 0.56 -0.79 0.74
FP2 -0.20 1.03 -1.49 1.36
F7 0.26 0.26 -0.01 0.60
FP1 0.07 0.72 -0.96 1.01
84
Table D15
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of 20 ms VOT
Location Mean SD Min Max
O2 -0.52 0.24 -0.84 -0.27
O1 -0.73 0.43 -1.16 -0.35
OZ -0.92 0.47 -1.44 -0.38
PZ -1.41 0.35 -1.75 -1.05
P4 -0.65 0.80 -1.11 0.54
CP4 -0.83 0.75 -1.43 0.24
P8 -0.14 1.08 -1.07 1.41
C4 -0.74 0.83 -1.62 0.34
TP8 0.11 0.89 -0.58 1.41
T8 -0.05 0.87 -0.87 1.18
P7 -0.55 0.42 -1.08 -0.14
P3 -1.34 0.37 -1.70 -0.93
CP3 -1.19 0.37 -1.64 -0.87
CPZ -1.50 0.36 -1.98 -1.17
CZ -1.51 0.82 -2.18 -0.45
FC4 -1.20 1.06 -1.75 0.38
FT8 0.11 0.85 -0.72 1.21
TP7 -0.33 0.66 -0.85 0.62
C3 -1.08 0.33 -1.47 -0.77
FCZ -1.50 0.79 -2.13 -0.43
FZ -1.26 1.11 -2.24 0.29
F4 -0.90 1.15 -1.64 0.82
F8 -0.04 0.84 -0.70 1.20
T7 -0.24 0.59 -0.66 0.62
FT7 -0.30 0.73 -0.96 0.73
FC3 -1.11 0.67 -1.60 -0.19
F3 -0.83 0.75 -1.65 0.07
FP2 -0.77 0.80 -1.53 0.33
F7 -0.39 0.49 -0.94 0.09
FP1 -0.39 0.61 -1.03 0.41
85
Table D16
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 2 with Stimulus of 30 ms VOT
Location Mean SD Min Max
O2 -0.47 0.82 -1.93 0.69
O1 -0.79 0.91 -2.23 0.70
OZ -0.78 0.96 -2.73 0.68
PZ -1.09 0.54 -2.02 -0.38
P4 -1.14 0.91 -2.38 0.18
CP4 -1.17 0.63 -1.98 -0.40
P8 -0.65 0.75 -1.54 0.43
C4 -1.03 0.55 -1.94 -0.45
TP8 -0.54 0.46 -1.11 0.22
T8 -0.50 0.55 -1.26 0.37
P7 -0.47 0.45 -1.12 0.14
P3 -1.20 1.21 -3.99 -0.25
CP3 -0.89 0.45 -1.66 -0.37
CPZ -1.28 0.63 -2.67 -0.74
CZ -1.15 0.50 -2.27 -0.67
FC4 -0.95 0.62 -1.72 0.06
FT8 -0.61 0.81 -1.84 0.38
TP7 -0.40 0.42 -1.36 -0.03
C3 -0.72 0.42 -1.43 0.03
FCZ -0.94 0.55 -1.83 -0.33
FZ -0.65 0.52 -1.30 0.05
F4 -0.59 0.89 -1.58 0.78
F8 -0.64 1.05 -2.46 0.40
T7 -0.32 0.57 -1.33 0.27
FT7 -0.18 0.51 -1.14 0.35
FC3 -0.53 0.42 -1.12 0.14
F3 -0.29 0.42 -1.06 0.37
FP2 -0.41 0.65 -1.54 0.12
F7 -0.11 0.51 -0.82 0.58
FP1 -0.14 0.56 -1.03 0.93
86
Table D17
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of -40 ms VOT
Location Mean SD Min Max
O2 0.12 0.77 -0.99 1.14
O1 0.05 0.23 -0.30 0.37
OZ -0.04 0.35 -0.39 0.59
PZ -0.56 0.28 -0.85 -0.20
P4 -0.45 0.65 -1.59 0.42
CP4 -0.48 0.67 -1.27 0.60
P8 -0.30 0.65 -1.44 0.46
C4 -0.89 0.68 -1.63 -0.08
TP8 -0.38 0.56 -1.42 0.05
T8 -0.34 0.74 -1.51 0.51
P7 -0.15 0.47 -0.81 0.42
P3 -0.16 0.38 -0.58 0.50
CP3 -0.53 0.37 -1.12 -0.07
CPZ -0.87 0.53 -1.49 0.04
CZ -0.87 0.80 -2.17 -0.06
FC4 -0.81 1.48 -3.71 0.16
FT8 -0.41 0.64 -1.50 0.12
TP7 -0.14 0.45 -0.80 0.42
C3 -0.60 0.51 -1.51 0.00
FCZ -1.17 1.37 -3.84 0.11
FZ -0.93 1.99 -4.89 0.35
F4 -0.84 1.92 -4.69 0.32
F8 -0.27 0.75 -1.52 0.35
T7 -0.21 0.62 -1.19 0.68
FT7 -0.39 0.91 -1.72 0.72
FC3 -0.91 1.42 -3.65 0.34
F3 -0.71 1.48 -3.59 0.34
FP2 -0.62 1.58 -3.74 0.40
F7 -0.30 0.81 -1.27 0.69
FP1 -0.27 1.44 -3.15 0.72
87
Table D18
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of -30 ms VOT
Location Mean SD Min Max
O2 -0.08 0.77 -1.08 0.76
O1 -0.27 0.85 -1.10 0.78
OZ 0.06 0.81 -0.85 1.09
PZ 0.15 0.87 -0.66 1.33
P4 0.31 0.73 -0.58 1.21
CP4 0.02 0.70 -0.63 0.67
P8 0.01 0.71 -1.02 0.49
C4 -0.03 0.65 -0.82 0.76
TP8 -0.01 0.62 -0.92 0.45
T8 -0.30 0.61 -1.06 0.21
P7 -0.40 0.48 -1.09 -0.08
P3 -0.33 1.13 -1.97 0.47
CP3 -0.21 0.87 -1.48 0.46
CPZ 0.13 0.98 -1.17 0.99
CZ -0.28 1.03 -1.77 0.58
FC4 -0.42 0.99 -1.61 0.58
FT8 -0.32 0.40 -0.78 0.07
TP7 -0.25 0.63 -1.09 0.31
C3 -0.35 1.10 -1.81 0.67
FCZ -0.57 1.18 -1.66 0.49
FZ -0.43 1.22 -1.67 0.72
F4 -0.27 0.51 -0.84 0.20
F8 -0.44 0.41 -0.88 0.10
T7 -0.72 1.08 -1.64 0.47
FT7 -0.59 1.15 -1.66 0.46
FC3 -0.46 1.42 -1.96 0.92
F3 -0.48 1.00 -1.57 0.38
FP2 -0.04 0.25 -0.42 0.11
F7 -0.70 1.10 -1.68 0.57
FP1 -0.39 0.55 -1.02 0.33
88
Table D19
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of -20 ms VOT
Location Mean SD Min Max
O2 -0.31 0.66 -1.12 0.75
O1 -0.23 0.68 -1.16 0.67
OZ -0.54 0.66 -1.13 0.67
PZ -1.41 1.10 -3.44 -0.35
P4 -1.11 1.38 -3.15 0.87
CP4 -1.21 1.63 -4.32 0.49
P8 -0.92 0.79 -2.22 -0.09
C4 -1.12 1.75 -4.59 0.06
TP8 -0.68 0.90 -2.22 0.42
T8 -0.36 0.78 -1.65 0.46
P7 -0.39 0.52 -0.98 0.24
P3 -0.98 0.73 -2.22 -0.06
CP3 -0.98 1.06 -2.90 -0.14
CPZ -1.61 1.54 -4.48 -0.38
CZ -1.30 1.75 -4.61 0.04
FC4 -0.97 1.51 -3.97 0.12
FT8 -0.36 0.70 -1.67 0.31
TP7 -0.31 0.54 -1.00 0.22
C3 -0.90 1.14 -2.92 -0.04
FCZ -1.08 1.80 -4.49 0.86
FZ -0.70 1.12 -2.90 0.26
F4 -0.62 1.06 -2.35 0.49
F8 -0.51 0.68 -1.66 0.28
T7 -0.30 0.59 -1.37 0.34
FT7 -0.18 0.74 -1.62 0.35
FC3 -0.76 1.01 -2.47 0.31
F3 -0.23 0.49 -1.14 0.23
FP2 0.14 0.55 -0.52 1.08
F7 -0.13 0.89 -1.75 0.68
FP1 0.32 0.34 -0.10 0.72
89
Table D20
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of -10 ms
Location Mean SD Min Max
O2 -0.20 0.73 -0.89 1.09
O1 -0.54 0.60 -1.34 0.25
OZ -0.63 0.80 -2.08 0.00
PZ -0.70 1.20 -1.93 1.43
P4 -0.40 0.97 -1.52 1.25
CP4 -0.54 0.48 -1.15 -0.04
P8 0.13 0.30 -0.25 0.47
C4 -0.19 0.64 -1.17 0.72
TP8 0.28 0.60 -0.47 1.12
T8 0.23 0.59 -0.48 1.01
P7 -0.48 0.36 -1.02 -0.07
P3 -0.75 0.78 -1.64 0.72
CP3 -0.74 0.56 -1.32 0.22
CPZ -0.73 1.01 -1.99 0.91
CZ -1.03 1.20 -2.99 0.31
FC4 -0.39 0.61 -1.04 0.37
FT8 0.45 0.69 -0.48 1.36
TP7 -0.41 0.42 -1.03 0.16
C3 -0.81 0.44 -1.30 -0.19
FCZ -0.72 0.73 -1.48 0.25
FZ -0.49 0.99 -1.46 1.07
F4 -0.09 0.57 -0.62 0.75
F8 0.45 0.61 -0.50 1.37
T7 -0.38 0.59 -1.15 0.41
FT7 -0.35 0.45 -1.01 0.04
FC3 -0.82 0.57 -1.38 0.15
F3 -0.35 0.90 -1.17 0.87
FP2 -0.12 0.41 -0.55 0.42
F7 -0.33 0.72 -1.04 0.72
FP1 -0.22 0.85 -0.93 1.38
90
Table D21
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of 0 ms VOT
Location Mean SD Min Max
O2 -0.34 0.51 -1.35 0.09
O1 -0.27 0.42 -0.83 0.16
OZ -0.34 0.48 -1.23 0.23
PZ -1.21 1.54 -4.52 0.31
P4 -0.67 0.85 -2.52 0.08
CP4 -0.89 1.14 -3.50 0.08
P8 -0.40 0.67 -1.88 0.22
C4 -0.87 1.35 -4.00 0.13
TP8 -0.15 0.78 -1.87 0.76
T8 -0.26 0.71 -1.81 0.36
P7 -0.10 0.43 -0.72 0.42
P3 -0.69 0.67 -2.06 -0.04
CP3 -0.65 0.67 -1.67 0.25
CPZ -1.33 1.52 -4.68 0.29
CZ -1.08 1.48 -4.33 0.27
FC4 -0.84 1.32 -3.79 0.31
FT8 -0.13 0.76 -1.81 0.46
TP7 -0.13 0.44 -0.73 0.42
C3 -0.54 0.66 -1.68 0.26
FCZ -0.88 1.68 -4.58 0.46
FZ -0.52 1.23 -3.18 0.87
F4 -0.48 1.00 -2.58 0.91
F8 -0.18 0.78 -1.76 0.80
T7 -0.18 0.28 -0.57 0.18
FT7 -0.09 0.36 -0.63 0.34
FC3 -0.55 0.95 -1.95 0.45
F3 -0.23 0.73 -1.41 0.92
FP2 0.04 0.92 -0.88 1.67
F7 -0.06 0.35 -0.63 0.39
FP1 -0.13 0.76 -1.17 1.08
91
Table D22
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of 10 ms
Location Mean SD Min Max
O2 -0.22 0.38 -0.64 0.33
O1 -0.13 0.64 -0.76 0.87
OZ -0.35 0.63 -1.09 0.54
PZ -1.12 0.92 -2.28 0.38
P4 -0.79 0.54 -1.85 -0.20
CP4 -0.91 0.61 -1.87 0.20
P8 -0.24 0.51 -0.62 0.66
C4 -1.15 0.42 -1.79 -0.55
TP8 -0.42 0.20 -0.67 -0.06
T8 -0.39 0.30 -0.80 -0.07
P7 -0.28 0.48 -0.89 0.55
P3 -0.64 0.93 -1.86 0.69
CP3 -1.02 0.60 -2.10 -0.24
CPZ -1.47 0.92 -3.02 -0.52
CZ -1.43 0.82 -2.60 -0.34
FC4 -1.16 0.66 -2.02 -0.18
FT8 -0.53 0.54 -1.35 0.09
TP7 -0.27 0.48 -0.87 0.55
C3 -1.09 0.64 -1.94 -0.35
FCZ -1.27 0.92 -2.29 0.23
FZ -1.26 0.99 -2.57 0.42
F4 -1.33 1.27 -3.81 0.05
F8 -0.40 0.37 -0.83 0.11
T7 -0.54 0.09 -0.63 -0.41
FT7 -0.47 0.81 -1.69 0.92
FC3 -1.07 0.99 -2.36 0.82
F3 -1.05 1.23 -2.31 0.81
FP2 -0.34 0.82 -1.65 0.84
F7 -0.77 0.63 -2.10 -0.17
FP1 -0.90 1.13 -2.50 0.55
92
Table D23
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of 20 ms VOT
Location Mean SD Min Max
O2 -0.52 1.09 -2.93 0.46
O1 -0.50 0.81 -1.86 0.36
OZ -0.52 1.06 -2.67 0.63
PZ -1.07 0.97 -2.71 0.08
P4 -0.92 0.98 -2.99 0.01
CP4 -0.84 1.08 -2.37 0.63
P8 -0.56 0.65 -1.59 0.45
C4 -0.51 1.08 -2.29 0.90
TP8 -0.53 0.55 -1.34 0.19
T8 -0.25 0.84 -1.35 0.60
P7 -0.44 0.36 -1.08 0.03
P3 -0.70 0.77 -2.11 0.41
CP3 -0.53 0.66 -1.63 0.25
CPZ -0.93 1.16 -2.73 0.51
CZ -0.45 1.00 -1.62 0.80
FC4 -0.17 1.01 -1.80 0.93
FT8 -0.14 0.69 -1.38 0.88
TP7 -0.36 0.46 -1.05 0.35
C3 -0.28 0.75 -1.38 0.56
FCZ -0.13 0.95 -1.30 1.06
FZ 0.06 0.92 -1.03 1.46
F4 -0.02 0.94 -1.50 1.52
F8 -0.10 0.76 -1.37 1.18
T7 0.09 0.71 -0.45 1.73
FT7 0.19 0.56 -0.37 1.44
FC3 -0.10 0.88 -1.55 0.85
F3 0.30 0.80 -0.71 1.60
FP2 -0.06 0.99 -1.33 1.80
F7 0.26 0.63 -0.36 1.68
FP1 0.29 1.07 -0.61 2.30
93
Table D24
Descriptive Statistics for RMS Amplitudes of the Mismatch Negativity for Group 3 with Stimulus of 30 ms VOT
Location Mean SD Min Max
O2 -0.58 0.86 -1.88 0.60
O1 -0.62 1.07 -2.49 0.45
OZ -0.59 1.03 -2.52 0.25
PZ -1.13 1.63 -3.13 1.50
P4 -0.70 1.35 -2.15 1.50
CP4 -0.94 1.12 -2.16 0.92
P8 -0.63 1.07 -2.38 0.25
C4 -0.83 1.10 -1.73 1.15
TP8 -0.56 0.84 -2.12 0.35
T8 -0.61 0.81 -1.96 0.37
P7 -0.59 1.40 -2.19 1.68
P3 -0.78 1.68 -2.50 2.13
CP3 -0.77 1.65 -2.41 2.25
CPZ -0.99 1.55 -2.93 1.59
CZ -1.11 1.28 -2.87 0.88
FC4 -0.80 0.89 -1.61 0.37
FT8 -0.75 0.95 -2.00 0.46
TP7 -0.53 1.31 -1.67 1.71
C3 -0.87 1.23 -2.05 1.22
FCZ -1.18 1.12 -2.43 0.42
FZ -0.80 0.92 -2.10 0.34
F4 -1.11 0.87 -2.27 -0.07
F8 -0.80 0.93 -2.02 0.42
T7 -0.45 1.03 -1.49 1.09
FT7 -0.31 0.80 -1.27 0.64
FC3 -0.78 1.05 -1.97 0.89
F3 -0.73 0.94 -1.68 0.24
FP2 -0.61 0.95 -1.69 0.54
F7 -0.36 0.87 -1.39 0.69
FP1 -0.53 1.04 -1.63 0.94