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Ncp-Ineffective Tissue Perfusion(Aortic Stenosis)
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Name: Mr. Heartbreaker Age: 52 years old Ward: Andrew Hall 1Chief Complaint: abdominal pain Diagnosis: Aortic Stenosis, Cardiomegaly, CHF III, Acute Kidney Injury secondary to Ischemic Nephropathy with Complicated UTI
CUES NURSING DIAGNOSIS
SCIENTIFIC RATIONALE
EXPECTED OUTCOME
NURSING INTERVENTIONS
RATIONALE EVALUATION
SUBJECTIVE:“Nadudu-as daw it akon im-im yakan nira ngan it akon kulo nagbuburusag gihap” as verbalized by the patient.
“Danay tigda la nalipong nak ulo labi na kun nabuhat ako” as verbalized by the patient.
“Nagkukuri gihap ako pagginhawa” as verbalized by the patient
OBJECTIVES:
V/S taken as follows: T= 36.1 ˚C RR= 31cpm HR= 60bpm BP= 140/70 mm
Hg Decrease
hemoglobin count (115g/dL)N=142-175g/dL
Pallor Pale nail beds Pale lips Shortness in
breath Chest pain
Ineffective tissue perfusion related to
vasoconstriction secondary to
redistribution of cardiac output to vital
organs
Vasoconstriction mediated by the sympathetic nervous system is largely responsible for the redistribution of cardiac output which serves as an important compensatory mechanism when flow is reduced. This redistribution is most marked when a patient with heart failure exercises or when an additional burdened is imposed, such as fever or anemia, but as heart failure advances, redistribution occurs even in the basal state. Blood flow is redistributed so that the delivery of oxygen to vital organs, such as the brain and myocardium, is maintained at normal or near normal levels, while flow to less critical areas, such as cutaneous and muscular beds and splanchnic viscera which results to ineffective perfusion to these areas.
SHORT TERM:
After 6 hours of nursing intervention, the patient will be able to maintain hemodynamic stability as evidenced by: Decrease blood
pressure and warm skin
Minimize report of chest pain
Normal capillary refill
Skin, palpebral conjunctiva, nasal mucosa, lips, oral mucosa and gums of natural pale, pink color
Absence or alleviation of lightheadedness
LONG TERM:
After 1 week of nursing intervention, the patient will be able to demonstrate increase tissue perfusion as evidenced by: No signs of pallor Able to perform
ADL’s without difficulty and dyspnea.
1. Assess general condition of the patient.
2. Assess vital signs.
3. Assess skin for coolness, pallor.
4. Provide a warm environment.
5. Perform capillary refill test.
6. Monitor urine output.
7. Encourage quiet, restful atmosphere.
8. Encourage active range of motion but provide rest periods.
9. Elevate bed at night.
COLLABORATIVE10. Administer
medications as prescribed:
Isoket (isosorbide
1. To assess the current status of client’s condition.
2. To obtain baseline data.
3. May indicate hypoxemia.
4. A warm environment promotes vasodilation, which decreases preload and tissue perfusion.
5. To check the adequacy of blood flow or circulation.
6. Decreased perfusion to the kidneys may result in oliguria.
7. Conserves energy and lowers tissue O2 demand.
8. Range of motion helps decrease venous pooling and promotes tissue perfusion.
9. To increase gravitational blood flow.
>Promotes peripheral
Goals partially met
After nursing interventions, the client had: Decreased blood
pressure with warm skin
Minimized report of chest pain
Capillary refill normal
Normal skin,palpebral conjunctiva, nasal mucosa, lips, oral mucosa and gum color
Absence of lightheadedness
Absence of pallor
Was able to perform some of his ADL’s like self care with less difficulty
Patient’s hematologic result slightly increased.
Delayed capillary refill (4 seconds)N-
Body malaise and activity intolerance
Fatigue Abnormal ABG
result (pH=7.530 N=7.350-7.450; pCO2=19.3mmHg N=35-45; pO2=113mmHg N=80-100)
Additional Diagnosis: Acute kidney injury secondary to ischemic nephropathy secondary to CHF, complicated UTI
Final diagnosis: severe aortic stenosis, cardiomegaly, CHF III ascites
Focus on Pathophysiology page 476 by Barbara L. Bullock and Reet L. Henze
Patient’s hematologic laboratory results will be normal or at least increase in value
dinitrate) 20mg 1 tab OD
Simvastatin(antihyperlipidemic) 20mg 1 tab OD
Bisoprolol (antihypertensive) 5mg ½ tab BID
11. Monitor ABG.
12. Oxygen administration as prescribed.
vasodilation and reduces preload and afterload, decreasing myocardial oxygen consumption and increasing cardiac output. Also dilates coronary arteries, increasing blood flow and improving collateral circulation
>Inhibits the enzyme that catalyzes the first step in the cholesterol synthesis pathway, resulting in decrease in serum cholesterol, and serum LDL’s.
>Decreases the excitability of the heart, thus decreasing the cardiac output and oxygen consumption, decreasing the release of rennin from the kidney, and lowering the BP.
11. Identifies hypoxemia, effectiveness or need for therapy.
12. To promote adequate tissue perfusion.