HTN & CRF

Embed Size (px)

Citation preview

  • 8/3/2019 HTN & CRF

    1/15

    DIABETIC NEPHROPATHY

    INTRODUCTIONHypertension, is the medical term for high blood pressure. Hypertension, on its own, is

    the second most common cause of end-stage renal failure, next to diabetes. It is common

    for all types of chronic kidney disease to eventually cause hypertension (approx. 80% of

    chronic kidney disease patients develop hypertension at some point). The reason for this

    is two-fold. With reduced kidney function, there may be a certain amount of clinical and

    sub-clinical fluid retention in the body, due to poor elimination of fluids and poor control

    of sodium. Perhaps more importantly, the kidneys are a major component in the body's

    regulation of blood pressure. As such, the kidneys have their own ability to raise blood

    pressure via release of a hormone called renin. Release of renin triggers a cascade of

    events all over the body which eventually cause constriction of the blood vessels

    (vasoconstriction). This cascade of events is called the renin-angiotensin-system, or RAS

    for short. When the kidneys sense that the glomeruli (the actual filters in the kidneys) are

    not getting the blood perfusion that they need (this means good, adequate blood flow

    within the glomeruli), they cause release of more renin, and blood pressure is eventually

    raised throughout the body. Since chronic kidney disease does affect blood perfusion

    within the glomeruli, chronic kidney disease is almost always accompanied by

    hypertension to some extent, even if there is no fluid retention. Some high blood pressure

    medications work by inhibiting the renin-angiotensin-system specifically, and these are

    therefore most effective in the context of chronic kidney disease.

    Causes, incidence, and risk factors

    Chronic kidney disease (CKD) slowly gets worse over time. In the early stages, there

    may be no symptoms. The loss of function usually takes months or years to occur. It may

    be so slow that symptoms do not occur until kidney function is less than one-tenth of

    normal.

    The final stage of chronic kidney disease is called end-stage renal disease (ESRD). The

    kidneys no longer function and the patient needs dialysis or a kidney transplant.

    http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000500/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003005/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000500/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003005/
  • 8/3/2019 HTN & CRF

    2/15

    Chronic kidney disease and ESRD affect more than 2 out of every 1,000 people in the

    United States.

    Diabetes and high blood pressure are the two most common causes and account for most

    cases.

    Many other diseases and conditions can damage the kidneys, including:

    Problems with the arteries leading to or inside the kidneys

    Birth defects of the kidneys (such as polycystic kidney disease)

    Some pain medications and other drugs

    Certain toxic chemicals

    Autoimmune disorder

    Injury or trauma

    Glomerulonephritis

    Kidney stones and infection

    Reflux nephropathy (in which the kidneys are damaged by the

    backward flow of urine into the kidneys)

    Other kidney diseases

    Chronic kidney disease leads to a buildup of fluid and waste products in the body. This

    condition affects most body systems and functions, including red blood cell production,

    blood pressure control, and vitamin D and bone health.

    Symptoms

    The early symptoms of chronic kidney disease often occur with other illnesses, as well.

    These symptoms may be the only signs of kidney disease until the condition is more

    advanced.

    Symptoms may include:

    General ill feeling and fatigue

    http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001214/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000468/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000502/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000484/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000458/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000459/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003089/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003088/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001214/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000468/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000502/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000484/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000458/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000459/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003089/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003088/
  • 8/3/2019 HTN & CRF

    3/15

    Generalized itching (pruritus) and dry skin

    Headaches

    Weight loss without trying to lose weight

    Appetite loss

    Nausea

    Other symptoms that may develop, especially when kidney function has worsened:

    Abnormally dark or light skin

    Bone pain

    Brain and nervous system symptoms

    Drowsiness and confusion

    Problems concentrating or thinking

    Numbness in the hands, feet, or other areas

    Muscle twitching or cramps

    Breath odor

    Easy bruising,bleeding, or blood in the stool

    Excessive thirst

    Frequent hiccups

    Menstrual periods stop (amenorrhea)

    Sleep problems, such as insomnia, restless leg syndrome,

    and obstructive sleep apnea

    Swelling of the feet and hands (edema)

    Vomiting, typically in the morning

    http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003217/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003024/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003107/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003117/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003242/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003208/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003205/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003206/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003296/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003058/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003235/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000045/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003085/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003149/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A002106/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000807/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000811/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003103/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003217/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003024/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003107/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003117/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003242/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003208/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003205/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003206/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003296/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003058/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003235/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000045/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003085/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003149/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A002106/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000807/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000811/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003103/
  • 8/3/2019 HTN & CRF

    4/15

    Treatment

    Controlling blood pressure is the key to delaying further kidney damage.

    Angiotensin-converting enzyme (ACE) inhibitors or angiotensin

    receptor blockers (ARBs) are used most often.

    The goal is to keep blood pressure at or below 130/80 mmHg

    Other tips for protecting the kdineys and preventing heart disease and stroke:

    Smoking restriction.

    Consumption of meals that are low in saturated fat and cholesterol

    Regular exercise

    Drugs to lower your cholesterol, if necessary.

    Keeping blood sugar under control.

    Other treatments may include:

    Special medicines called phosphate binders, to help prevent

    phosphorous levels from becoming too high

    Treatment for anemia, such as extra iron in the diet, iron pills, special

    shots of a medicine called erythropoietin, and blood transfusions

    Extra calcium and vitamin D (always talk to your doctor before taking)

    Fluid restriction.

    Restriction of salt,potassium, phosphorous, and other electrolytes.

    When loss of kidney function becomes more severe, dialysis or kidney transplants

    become the potential options

    Dialysis depends on different factors, including lab test results, severity

    of symptoms, and readiness.

    http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0000089/http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0000089/
  • 8/3/2019 HTN & CRF

    5/15

    Expectations (prognosis)

    Many people are not diagnosed with chronic kidney disease until they have lost much of

    their kidney function.There is no cure for chronic kidney disease. Untreated, it usually

    progresses to end-stage renal disease. Lifelong treatment may control the symptoms of

    chronic kidney disease.

    Complications

    Anemia

    Bleeding from the stomach or intestines

    Bone, joint, and muscle pain

    Changes in blood sugar

    Damage to nerves of the legs and arms (peripheral neuropathy)

    Dementia

    Fluid buildup around the lungs (pleural effusion)

    Heart and blood vessel complications

    Congestive heart failure

    Coronary artery disease

    High blood pressure

    Pericarditis

    Stroke

    High phosphorous levels

    Highpotassium levels

    Hyperparathyroidism

    Increased risk of infections

    http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000500/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000560/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000593/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000739/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000086/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000158/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A007115/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000182/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000726/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001179/http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0000089/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001215/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000500/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000560/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000593/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000739/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000086/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000158/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A007115/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000182/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000726/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001179/http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0000089/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001215/
  • 8/3/2019 HTN & CRF

    6/15

    Liver damage or failure

    Malnutrition

    Miscarriages and infertility

    Seizures

    Weakening of the bones and increased risk of fractures

    Prevention

    Treating the condition that is causing the problem may help prevent or delay chronic

    kidney disease. People who have diabetes should control their blood sugar andblood

    pressure levels and should not smoke

    http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000404/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001488/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001191/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003200/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003398/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003398/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A000404/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001488/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A001191/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003200/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003398/http://www.ncbi.nlm.nih.gov/pubmedhealth/n/pmh_adam/A003398/
  • 8/3/2019 HTN & CRF

    7/15

    CASE SCENARIO

    Ab a 22 year old college student presented to the emergency room with headaches and

    shortness of breath. On examination he had a blood pressure of 200/120 mmHg.Ab reports that over the past year his weight has increased about 5 kilos although his diet

    has remained unchanged. He attributed this weight gain to decrease exercise and a busy

    class schedule.

    Past medical history

    Ab has had no recent viral illness sore throat or occurred respiratory infection. He has no

    family history of renal disease. He is not taking any medications and has no drug or food

    allergies.

    Social history:He shares a dormitory room with a fellow student who is in good health.

    He does not consume alcohol and dose not smoke.

    Review of systems

    General: fatigue, weakness, shortness of breath

    Vital signs: temperature, pulse rate are normal except for blood pressure as recorded.

    Anthropometry:

    Height: 0 5 ft 9

    Current weight: 77.3 kgs

    Usual weight: 70.5 kgs

    Extremities: peripheral edema on both legs, abdomen no hepatomegaly, general well

    developed male

    Biochemical investigation

    Biochemical parameters Actual value Reference range

    Chloride 111meq/L

    Heamoglobin 8.3g/dl 13 16 g/dlBUN 30 mg/dl 8.0-23.0 mg/dl

    Creatinine 2 mg/dl 0.8-1.6 mg/dl

    Sodium 158 mmol/l 134-145 mmol/l

    Potassium 6 mmol/l 3.5-5.2 mmol/l

  • 8/3/2019 HTN & CRF

    8/15

    Urine output 200 ml/day

    NUTRITION CARE PLAN

    PRINCIPLE/NUTRITION CARE GOALSDiabetic Nephropathy can be managed by adopting the following principles:

    To achieve:

    Regular meal timings.

    Incorporation of fiber rich food over refined food and unsaturated fats over

    saturated fats

    Low salt/low sodium and potassium intake

    Fluid restriction

    Different ways in which the client can abide by the low sodium and low

    potassium intake.

    Concept and importance of leeching and the foods that have to be subjected to

    leeching.

    A change in lifestyle

    PATIENT PROFILEName: AB

    Age: 22 years

    Gender: Male

    Lifestyle: Sedentary physical activity

    Socio economic status: Middle Income Group

    Food Habit : Not specified

    ANTHROPOMETRY

    Height: 5 ft 9

    Current weight: 77.3 kg

    Usual weight: 70.5 kg

    BMI: 23.8

  • 8/3/2019 HTN & CRF

    9/15

    Interpretation: The calculated Body Mass Index for Mr. AB was found to be 23.8.

    This explains that the BMI lied in the normal range (i.e between 18 -24.9)

    which indicates that the weight of Mr. AB was under control and the diet

    planned should emphasize on diabetes management in combination with

    sodium, potassium and fluid restriction.

    BIOCHEMICAL ASSESSMENT

    Biochemical parameters Actual value Reference range

    Chloride 111meq/L 95 105 meq/L

    Heamoglobin 8.3g/dl 13 16 g/dl

    BUN 30 mg/dl 8.0-23.0 mg/dl

    Creatinine 2 mg/dl 0.8-1.6 mg/dl

    Sodium 158 mmol/l 134-145 mmol/l

    Potassium 6 mmol/l 3.5-5.2 mmol/l

    Urine output 200 ml/day 800 2000 ml/day

    Interpretation:

    The elevated levels of BUN in blood indicate renal failure.

    The level of Creatinine is also elevated indicating possible malfunction or

    failure of the kidneys.

    Sodium values are elevated, elevated sodium values increase hypertension

    which in turn worsens the functioning of kidney.

    Decrreased urine output indicated oliguria due to kidney malfunction

    Shows higher concentration of sodium, potassium and chloride /electroytes

    due to kidneys inability to excrete them.

  • 8/3/2019 HTN & CRF

    10/15

    NUTRIENT RECOMMENDATIONSNUTRIENT RDA PRINCIPLE OF

    MODIFICATION

    MODIFIED RDA

    Energy 2320 kcal 35 kca/kg IBW 2537.5kcal

    Protein 60g 1.2g /kg IBW 87 g

    Fat 25 g 25g 25 g

    DISTRIBUTION OF ENERGY

    NUTRIENT PERCENTAGE IN KCAL IN GRAMS

    Carbohydrates 60 % - 70 % 1522.50 -1776.25 380.6 444.06

    Protein 12% 304.50 76.12

    Fat 10 % -15 % 253.75 380.62 28.19 - 42.29

    EXCHANGE PLAN

    Food Group Exchange Amount

    (g)

    Energy

    (kcal)

    Protein

    (g)

    Fat

    (g)

    Cereals 10 300 1000 30.0 8.0

    Pulses 5 150 500 30.0 2.8

    Milk 3 300 210 9.0 9.0

    Roots and

    Tubers

    2 200 160 2.6 -

    Green Leafy

    Vegetables

    1 100 46 3.6 0.4

    Other

    vegetables

    2 200 56 3.4 0.4

    Fruits 2 200 80 - -

    Sugar 3 15 60 - -

    Fats and oils 4 20 340 - 20TOTAL 2452.0 78.6 40.6

    SKELETAL MENU

    Early Morning (6:30 a.m) 1 glass milk

    Breakfast (8:30 a.m) 3 gobi paranthas with curds and 1 apple

  • 8/3/2019 HTN & CRF

    11/15

    Mid Morning (11:00 a.m) 1 aloo mehti sandwich

    Lunch (2:30 p.m) 1 bowl of rajma salad, 1 bowl of steamed rice with mixed pulse curry

    With ladies finger sabji

    Evening (5:00 p.m) 1 bowl of upma and 1 bowl of papaya

    Pre Dinner (6:30 p.m) 1 bowl of cucumber salad

    Dinner (8:00 p.m) 1 bowl of amaranth khichdi

    Bed Time(10:30 p.m) 1 glass of skimmed milk

    DETAILED CALCULATIONS

    Meal/

    Time

    Menu Ing Ex Amt

    (g)

    E

    (kcal)

    P

    (g)

    F

    (g)

    Na

    (mg)

    K

    (mg)

    Phos.

    (mg)

    Early

    Morning

    (6:30 a.m)

    Milk 1 100 67 3.2 4.1 73 140 90

    Gobi

    paranthhas

    Wheat flour 3 90 306.9 10.

    8

    1.5 18 283.5 319.5

    Cauliflower 1 100 30 2.6 0.4 53 138 57

    Onion 25 12.5 0.3 0.02 1.0 31.7 12.5

    Curds Curds 50 30 1.5 2.0 16 65 46.5

    Apple Apple 1 100 59 0.2 0.5 28 75 14.0

    Mid

    Morning

    (11:00 am)

    Aloo mehti

    sandwich

    Bread 2 60 73.5 2.3 0.21 8.3 39 -

    Potato 50 48.5 0.8 0.05 5.5 123 20

    Mehti 50 24.5 2.2 0.45 38 15 25.5

    Lunch

    (2:30 p.m)

    Rajmah

    salad

    *rajmah 1 30 103.8 6.6 0.39 - - 93

    Onion 25 12.5 0.3 0.02 1.0 31.7 12.5

    Tomato 25 5.0 0.2 0.05 3.2 36.5 10.0

    Rice Rice 3 90 310.5 6.1 0.45 - - 144

    Soy bean

    curry

    *soybean 2 60 219.0 26 11.1 - - 280

    Tomato 25 12.5 0.3 0.02 1.0 31.7 12.5

    Ladies

    finger

    sabji

    Ladiesfinger 50 5.0 0.2 0.05 3.2 36.5 10.0

    Onion 25 12.5 0.3 0.02 1.0 31.7 12.5

    Tomato 25 5.0 0.2 0.05 3.2 36.5 10.0

    Curds Curds 50 30 1.5 2.0 16 65 46.5

    sugar 1 5 19.9 - - - - 0.05

    Evening

    (5-00 pm)

    Upma Semolina 2 60 104.4 3.1 0.24 6.3 24.9 30.6

    Onion 25 12.5 0.3 0.02 1.0 31.7 12.5

    Papaya Papaya 1 100 32 0.6 0.1 6 69 13

    Dinner Cumber Cumber 1 100 45 2.6 0.03 16 35 41.2

    Amaranth Rice 2 60 200.5 4.1 0.25 - - 94

  • 8/3/2019 HTN & CRF

    12/15

    (8.00 pm) Khichidi 2

    Green gram

    dal

    2 60 369 54 10.4 - - 320

    Amaranth 50 24 0.3 0.8 - - 31

    Bed time

    (10.00 pm)

    Milk Milk 1 100 67 3.2 4.1 73 140 90Sugar 1 5 19.9 - - - - 0.05

    Total Oil 4 20 340 - 20 - - -

    Total 2478.6 79.3 31.6 276.7 1236.4 1259.2

    * leach before use

    EVALUATION :

    NUTRIENT PRESCRIBED

    VALUES

    CALCULATED

    VALUES

    Energy (kcal) (E) 2537.5 2478.6

    Protein (g) (P) 87 79.3Fat (g) (F) 25 31.6

    Potassium (mg) (K) 2000 (2g) 1236.4 = 1.23g

    Sodium (mg) (Na) 2000 (2g) 276.70

    Phosphate 0.8 g 1.2g 1.25 g

    EVALUATION OF THE DIET

    A Nutrition Care Plan was formulated for Mr. AB, a 22 year old student who is

    hypertensive and has been diagnosed with renal failure.

    A diet was planned which was formulated keeping in mind the principles of hypertension

    management in combination with sodium, potassium and fluid restriction.

    Potassium, sodium and fluid restriction was crucial in order to reduce the load on the

    kidneys and to prevent further complications like water retention etc.

    The diet focused on timely, regular and portion controlled meals that were rich in fiber in

    order to manage diabetes.

    Mostly vegetables with moderate amounts of potassium were carefully chosen during the

    construction of the diet. Group III vegetables (K > 200 mg) that were used were leeched

    pre use.

    Pulses used for the preparation of rajmah salad, dal and khichdi were also leeched before

    cooking in order to drain out the excess potassium.

  • 8/3/2019 HTN & CRF

    13/15

    A very low sodium diet was planned consisting of 276.70 mg of sodium excluding salt.

    Only 1 tsp of salt was permitted and could be achieved by using salt in sachet form

    during meals.

    The fluid intake in the form of coffee and milk was given an allowance of 3 exchanges i.e

    300ml in order to achieve adequate consumption of proteins and the water intake was

    kept low keeping in mind fluid restriction as adviced by the physician.

    Minimum amounts of fats in the form of oil were used for seasoning purpose. Saturated

    fats were discouraged which is crucial for hypertension management.

    COUNSELLING

    Establish a good rapport with the client

    Assess the clients nutritional status through anthropometric measurements,

    biochemical investigations, clinical signs and symptoms and diet therapy.

    Plan a diet to serve diabetes management along with sodium, potassium and fluid

    restriction

    Educate the client

    about the disease condition

    About the consequences of the disease.

    About hidden salt

    To read food labels.

    Dos

    Have regular and timely meals

    Have portion controlled meals

    Have small and frequent meals

    Choose foods rich in fiber over refined food

    Do not add extra salt at the table.

    Cook the food without salt and use only 4-5 g per day, i.e. 1 tsp per day).

    Do not consume any raw vegetables, except cucumber.

    Reduce the consumption of oil, salt & coconut used in cooking

  • 8/3/2019 HTN & CRF

    14/15

    LEACHING:

    Leaching is very important to remove excess potassium that is present in

    pulses/legumes/lentils and vegetables.

    Boil excess water & soak dal & vegetables in it for 1 hour, after that discard the

    water & then cook the dal and vegetables.

    FOODS TO BE AVOIDED:

    CEREALS: Ragi, Maida, bajra, barley

    PULSES: Consume in moderation

    VEGS: potatoes, beetroot, green leafy vegetables,

    Soya bean, mushrooms.

    FRUITS: Avoid all fruits & fruit juices & tender coconut water.

    DAIRY PRODUCTS: Condensed milk, cheese, cream, butter, ghee.

    MEAT: Liver, kidney, sausages.

    MISCELLANEOUS FOODS: Sugar, honey, jaggery, soups, chocolates, bourn vita,

    horlicks, pickles, papads,

    Sauces, instant coffee.BAKERY FOODS: buns, puffs, cakes, pastries. .

    FRIED FOODS: chips, mixtures, vada, bonda, Fried meats.

    DRINKS: Tender coconut water, coca-cola, Pepsi, etc.

    NUTS: All nuts like coconut, groundnut, and cashew nut

    SPICES: All spices like chilly powder, pepper, garam masala & garlic to be used in

    moderation.

    FOODS ALLOWED:

    CEREALS: Rice, wheat, jowar, maize, rava (moderation)

    PULSES: Urad dal, thur dal, green gram, sprouted grams after leaching.

    NON-VEG: egg whites, chicken, fish- 2 medium pieces (thrice a week.)

  • 8/3/2019 HTN & CRF

    15/15

    VEGS: Cucumber, pumpkin, cabbage, beans, carrot, onion, bottlegourd, ridgegourd,

    snake gourd, capsicum, raddish, khnol-khol, ladiesfinger.

    FRUITS: Apple/Guava/Pear/Papaya: once a week(100g)

    MILK: milk, buttermilk, curd in moderation

    DRINKS: Tea/ coffee in moderation.