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Coronary Heart Disease (CHD). Leading cause of death in U.S. Narrowing coronary arteries Atherosclerosis. Angina Pectoris - Pathophysiology. Obstructed coronary artery Increased myocardial oxygen demand Lactic acid release Leads to pain Three types Stable Unstable - PowerPoint PPT Presentation
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Coronary Heart Disease (CHD)
• Leading cause of death in U.S.
• Narrowing coronary arteries– Atherosclerosis
Angina Pectoris - Pathophysiology
• Obstructed coronary artery• Increased myocardial oxygen demand• Lactic acid release• Leads to pain• Three types
– Stable– Unstable– Prinzmetal’s: is a syndrome typically consisting of angina
(cardiac chest pain) at rest that occurs in cycles. It is caused by vasospasm, a narrowing of the coronary arteries caused by contraction of the smooth muscle tissue in the vessel walls rather than directly by atherosclerosis
Angina Pectoris - Manifestations
• Chest pain
• Radiates
• Onset with exercise, etc.
• Relieved by rest, nitroglycerin (NTG)
• SOB, pallor, fear
Acute Coronary Syndrome
• Condition that includes:– Unstable angina– Acute myocardial ischemia with or without
muscle damage
• Associated with coronary artery stenosis and atherosclerotic plaque
Acute Myocardial Infarction (AMI)
• Pathophysiology– Occluded coronary artery stops blood flow
to part of cardiac muscle– Cellular death– Tissue necrosis– Description—heart area affected– Classification
AMI Manifestations
• Chest pain
• Radiates to shoulder, neck, jaw, arms
• Lasts longer than 15–20 minutes
• Not relieved with NTG
• Sense of impending doom
• SOB
• Diaphoresis
• Nausea and vomiting
AMI Manifestations (continued)
• Manifestations in women and elderly– May be atypical– Upper abdominal pain– No chest pain but other symptoms
AMI Complications
• Related to size and location of infarct
• Dysrhythmias
• Pump failure– Cardiogenic shock
• Pericarditis
Cardiac Dysrhythmias
• Pathophysiology– Due to altered formation of impulses or
altered conduction of the impulse through the heart
– Ectopic beats– Heart block– Reentry phenomenon– Classified to the site of impulse formation or
the site and degree of conduction block
Types of Cardiac Dysrhythmias (continued)
• PVCs
• Ventricular tachycardia
• Ventricular fibrillation
• AV conduction blocks– First degree– Second degree– Third degree
Types of Cardiac Dysrhythmias
• Supraventricular
• Sinus tachycardia
• Sinus bradycardia
• PAC
• Atrial flutter
• Atrial fibrillation
• Junctional
• Ventricular dysrhythmias
ECG Changes in Angina Pectoris vs. Myocardial
Infection
04/11/2009 15
Congestive Heart FailureCongestive Heart Failure
Dr Ibraheem Bashayreh, RN,PhD
04/11/2009 16
Heart failure
Normal heart function
04/11/2009 17
Congestive Heart FailureDefinition
Congestive Heart FailureDefinition
• Impaired cardiac pumping such that heart is unable to pump adequate amount of blood to meet metabolic needs
• Not a disease but a “syndrome”
• Associated with long-standing HTN and CAD
• Impaired cardiac pumping such that heart is unable to pump adequate amount of blood to meet metabolic needs
• Not a disease but a “syndrome”
• Associated with long-standing HTN and CAD
04/11/2009 18
Factors Affecting Cardiac Output
Cardiac OutputCO
Preload
Afterload Contractility
Heart RateStroke Volume
SV= X
SV: the volume of blood pumped from one ventricle of the heart with each beat
04/11/2009 19
Factors Affecting Cardiac Output
• Heart Rate– In general, the higher the heart rate, the lower
the cardiac• E.g. HR x Systolic Volume (SV) = CO
» 60/min x 80 ml = 4800 ml/min (4.8 L/min)
» 70/min x 80 ml = 5600 ml/min (5.6 L/min)
– But only up to a point. With excessively high heart rates, diastolic filling time begins to fall, thus causing stroke volume and thus CO to fall
04/11/2009 20
Heart Rate Stroke Volume Cardiac Output
60/min 80 ml 4.8 L/min
80/min 80/ml 6.4 L/min
100/min 80/ml 8.0 L/min
130/min 50/ml 6.5 L/min
150/min 40/ml 6.0 L/min
04/11/2009 21
Factors Affecting Cardiac Output
• Preload– The volume of blood/amount of fiber stretch
in the ventricles at the end of diastole (i.e., before the next contraction)
04/11/2009 22
Factors Affecting Cardiac Output
• Preload increases with:• Fluid volume increases
• Vasoconstriction (“squeezes” blood from vascular system into heart)
• Preload decreases with• Fluid volume losses
• Vasodilation (able to “hold” more blood, therefore less returning toheart)
04/11/2009 23
Factors Affecting Cardiac Output
• Starling’s Law – Describes the relationship between preload and cardiac
output– The greater the heart muscle fibers are stretched (b/c of
increases in volume), the greater their subsequent force of contraction – but only up to a point. Beyond that point, fibers get over-stretched and the force of contraction is reduced
• Excessive preload = excessive stretch → reduced contraction → reduced SV/CO
04/11/2009 24
Factors Affecting Cardiac Output
• Afterload – The resistance against which the ventricle must
pump. Excessive afterload = difficult to pump blood → reduced CO/SV
– Afterload increased with:• Hypertension• Vasoconstriction
– Afterload decreased with:• Vasodilation
04/11/2009 25
Factors Affecting Cardiac Output
• Contractility – Ability of the heart muscle to contract; relates
to the strength of contraction.
04/11/2009 26
Factors Affecting Cardiac Output
• Contractility decreased with:– infarcted tissue – no contractile strength– ischemic tissue – reduced contractile strength. – Electrolyte/acid-base imbalance– Negative inotropes (medications that decrease
contractility, such as beta blockers).
• Contractility increased with:– Sympathetic stimulation (effects of epinephrine)– Positive inotropes (medications that increase
contractility, such as digoxin, sympathomimmetics)
04/11/2009 27
Pathophysiology of CHF
• Pump fails → decreased stroke volume /CO. • Compensatory mechanisms kick in to increase CO
– SNS stimulation → release of epinephrine/nor-epinephrine
• Increase HR
• Increase contractility
• Peripheral vasoconstriction (increases afterload)
– Myocardial hypertrophy: walls of heart thicken to provide more muscle mass → stronger contractions
04/11/2009 28
Pathophysiology of CHF
– Hormonal response: ↓’d renal perfusion interpreted by juxtaglomerular apparatus as hypovolemia. Thus:• Kidneys release renin, which stimulates
conversion of antiotensin I → angiotensin II, which causes:
– Aldosterone release → Na retention and water retention (via ADH secretion)
– Peripheral vasoconstriction
04/11/2009 29
Pathophysiology of CHF
• Compensatory mechanisms may restore CO to near-normal.
• But, if excessive the compensatory mechanisms can worsen heart failure because . . .
04/11/2009 30
Pathophysiology of CHF
• Vasoconstriction: ↑’s the resistance against which heart has to pump (i.e., ↑’s afterload), and may therefore ↓ CO
• Na and water retention: ↑’s fluid volume, which ↑’s preload. If too much “stretch” (d/t too much fluid) → ↓ strength of contraction and ↓’s CO
• Excessive tachycardia → ↓’d diastolic filling time → ↓’d ventricular filling → ↓’d SV and CO
04/11/2009 31
Congestive Heart FailureRisk Factors
Congestive Heart FailureRisk Factors
• CAD• Age• HTN• Obesity• Cigarette smoking• Diabetes mellitus• High cholesterol• African descent
• CAD• Age• HTN• Obesity• Cigarette smoking• Diabetes mellitus• High cholesterol• African descent
04/11/2009 32
Heart failure
Underlying causes/risk factors• Ischemic heart disease (CAD)
• hypertension
• myocardial infarction (MI)
• valvular heart disease
• congenital heart disease
70%
• dilated cardiomyopathy
04/11/2009 33
Congestive Heart FailureTypes of Congestive Heart Failure
Congestive Heart FailureTypes of Congestive Heart Failure
• Left-sided failure
– Most common form
– Blood backs up through the left atrium into the pulmonary veins
• Pulmonary congestion and edema– Eventually leads to biventricular failure
• Left-sided failure
– Most common form
– Blood backs up through the left atrium into the pulmonary veins
• Pulmonary congestion and edema– Eventually leads to biventricular failure
04/11/2009 34
Congestive Heart FailureTypes of Congestive Heart Failure
Congestive Heart FailureTypes of Congestive Heart Failure
• Left-sided failure
– Most common cause: • HTN
• Cardiomyopathy
• Valvular disorders
• CAD (myocardial infarction)
• Left-sided failure
– Most common cause: • HTN
• Cardiomyopathy
• Valvular disorders
• CAD (myocardial infarction)
04/11/2009 35
Congestive Heart FailureTypes of Congestive Heart Failure
Congestive Heart FailureTypes of Congestive Heart Failure
• Right-sided failure– Results from diseased right ventricle– Blood backs up into right atrium and venous
circulation– Causes
• LVF• Cor pulmonale: failure of the right side of the heart brought on
by long-term high blood pressure in the pulmonary arteries and right
ventricle of the heart
• RV infarction
• Right-sided failure– Results from diseased right ventricle– Blood backs up into right atrium and venous
circulation– Causes
• LVF• Cor pulmonale: failure of the right side of the heart brought on
by long-term high blood pressure in the pulmonary arteries and right
ventricle of the heart
• RV infarction
04/11/2009 36
Congestive Heart FailureTypes of Congestive Heart Failure
Congestive Heart FailureTypes of Congestive Heart Failure
• Right-sided failure
– Venous congestion• Peripheral edema• Hepatomegaly• Splenomegaly• Jugular venous distension
• Right-sided failure
– Venous congestion• Peripheral edema• Hepatomegaly• Splenomegaly• Jugular venous distension
04/11/2009 37
Congestive Heart FailureTypes of Congestive Heart Failure
Congestive Heart FailureTypes of Congestive Heart Failure
• Right-sided failure
– Primary cause is left-sided failure
– Cor pulmonale
• RV dilation and hypertrophy caused by pulmonary pathology
• Right-sided failure
– Primary cause is left-sided failure
– Cor pulmonale
• RV dilation and hypertrophy caused by pulmonary pathology
04/11/2009 38
Acute Congestive Heart FailureClinical Manifestations
Acute Congestive Heart FailureClinical Manifestations
• Pulmonary edema (what will you hear?)
– Agitation
– Pale or cyanotic
– Cold, clammy skin
– Severe dyspnea
– Tachypnea
– Pink, frothy sputum
• Pulmonary edema (what will you hear?)
– Agitation
– Pale or cyanotic
– Cold, clammy skin
– Severe dyspnea
– Tachypnea
– Pink, frothy sputum
04/11/2009 39
Chronic Congestive Heart FailureClinical Manifestations
Chronic Congestive Heart FailureClinical Manifestations
• Fatigue
• Dyspnea
– Paroxysmal nocturnal dyspnea (PND)
• Tachycardia
• Edema – (lung, liver, abdomen, legs)
• Nocturia
• Fatigue
• Dyspnea
– Paroxysmal nocturnal dyspnea (PND)
• Tachycardia
• Edema – (lung, liver, abdomen, legs)
• Nocturia
04/11/2009 40
Chronic Congestive Heart FailureClinical Manifestations
Chronic Congestive Heart FailureClinical Manifestations
• Behavioral changes
– Restlessness, confusion, attention span• Chest pain (d/t CO and ↑ myocardial work)
• Weight changes (r/t fluid retention)
• Skin changes
– Dusky appearance
• Behavioral changes
– Restlessness, confusion, attention span• Chest pain (d/t CO and ↑ myocardial work)
• Weight changes (r/t fluid retention)
• Skin changes
– Dusky appearance
04/11/2009 41
Congestive Heart FailureClassification
Congestive Heart FailureClassification
• Based on the person’s tolerance to physical activity
– Class 1: No limitation of physical activity
– Class 2: Slight limitation
– Class 3: Marked limitation
– Class 4: Inability to carry on any physical activity without discomfort
• Based on the person’s tolerance to physical activity
– Class 1: No limitation of physical activity
– Class 2: Slight limitation
– Class 3: Marked limitation
– Class 4: Inability to carry on any physical activity without discomfort
04/11/2009 42
Congestive Heart FailureDiagnostic Studies
Congestive Heart FailureDiagnostic Studies
• Primary goal is to determine underlying cause
– Physical exam
– Chest x-ray
– ECG
– Hemodynamic assessment
• Primary goal is to determine underlying cause
– Physical exam
– Chest x-ray
– ECG
– Hemodynamic assessment
04/11/2009 43
Congestive Heart FailureDiagnostic Studies
Congestive Heart FailureDiagnostic Studies
• Primary goal is to determine underlying cause– Echocardiogram (Uses ultrasound to visualize
myocardial structures and movement, calculate EF)
– Cardiac catheterization
• Primary goal is to determine underlying cause– Echocardiogram (Uses ultrasound to visualize
myocardial structures and movement, calculate EF)
– Cardiac catheterization
04/11/2009 44
Acute Congestive Heart FailureNursing and Collaborative
Management
Acute Congestive Heart FailureNursing and Collaborative
Management
• Primary goal is to improve LV function by:
– Decreasing intravascular volume
– Decreasing venous return
– Decreasing afterload
– Improving gas exchange and oxygenation
– Improving cardiac function
– Reducing anxiety
• Primary goal is to improve LV function by:
– Decreasing intravascular volume
– Decreasing venous return
– Decreasing afterload
– Improving gas exchange and oxygenation
– Improving cardiac function
– Reducing anxiety
04/11/2009 45
Acute Congestive Heart FailureNursing and Collaborative
Management
Acute Congestive Heart FailureNursing and Collaborative
Management
• Decreasing intravascular volume
– Improves LV function by reducing venous return
– Loop diuretic: drug of choice
– Reduces preload
– High Fowler’s position
• Decreasing intravascular volume
– Improves LV function by reducing venous return
– Loop diuretic: drug of choice
– Reduces preload
– High Fowler’s position
04/11/2009 46
Acute Congestive Heart FailureNursing and Collaborative
Management
Acute Congestive Heart FailureNursing and Collaborative
Management
• Decreasing afterload
– Drug therapy: • vasodilation, Angiotensin-converting enzyme
(ACE) inhibitors
– Decreases pulmonary congestion
• Decreasing afterload
– Drug therapy: • vasodilation, Angiotensin-converting enzyme
(ACE) inhibitors
– Decreases pulmonary congestion
04/11/2009 47
Acute Congestive Heart FailureNursing and Collaborative
Management
Acute Congestive Heart FailureNursing and Collaborative
Management
• Improving cardiac function
– Positive inotropes
• Improving gas exchange and oxygenation– Administer oxygen, sometimes intubate and
ventilate
• Reducing anxiety
– Morphine
• Improving cardiac function
– Positive inotropes
• Improving gas exchange and oxygenation– Administer oxygen, sometimes intubate and
ventilate
• Reducing anxiety
– Morphine
04/11/2009 48
Chronic Congestive Heart FailureCollaborative Care
Chronic Congestive Heart FailureCollaborative Care
• Treat underlying cause
• Maximize CO
• Alleviate symptoms
• Treat underlying cause
• Maximize CO
• Alleviate symptoms
04/11/2009 49
Chronic Congestive Heart FailureCollaborative Care
Chronic Congestive Heart FailureCollaborative Care
• Oxygen treatment
• Rest
• Biventricular pacing
• Cardiac transplantation
• Oxygen treatment
• Rest
• Biventricular pacing
• Cardiac transplantation
04/11/2009 50
Chronic Congestive Heart FailureDrug Therapy
Chronic Congestive Heart FailureDrug Therapy
• ACE inhibitors
• Diuretics
• Inotropic drugs : drugs that influence the force of contraction of cardiac muscle
• Vasodilators-Adrenergic blockers
• ACE inhibitors
• Diuretics
• Inotropic drugs : drugs that influence the force of contraction of cardiac muscle
• Vasodilators-Adrenergic blockers
04/11/2009 51
Chronic Congestive Heart FailureNutritional Therapy
Chronic Congestive Heart FailureNutritional Therapy
• Fluid restrictions not commonly prescribed
• Sodium restriction
– 2 g sodium diet
• Daily weights
– Same time each day
– Wearing same type of clothing
• Fluid restrictions not commonly prescribed
• Sodium restriction
– 2 g sodium diet
• Daily weights
– Same time each day
– Wearing same type of clothing
04/11/2009 52
Chronic Congestive Heart FailureNursing ManagementNursing Assessment
Chronic Congestive Heart FailureNursing ManagementNursing Assessment
• Past health history
• Medications
• Functional health problems
• Cold, diaphoretic skin
• Past health history
• Medications
• Functional health problems
• Cold, diaphoretic skin
04/11/2009 53
Chronic Congestive Heart FailureNursing ManagementNursing Assessment
Chronic Congestive Heart FailureNursing ManagementNursing Assessment
• Tachypnea
• Tachycardia
• Crackles
• Abdominal distension
• Restlessness
• Tachypnea
• Tachycardia
• Crackles
• Abdominal distension
• Restlessness
04/11/2009 54
Chronic Congestive Heart FailureNursing Management
Nursing Diagnoses
Chronic Congestive Heart FailureNursing Management
Nursing Diagnoses
• Activity intolerance
• Excess fluid volume
• Disturbed sleep pattern
• Impaired gas exchange
• Anxiety
• Activity intolerance
• Excess fluid volume
• Disturbed sleep pattern
• Impaired gas exchange
• Anxiety
04/11/2009 55
Chronic Congestive Heart FailureNursing Management
Planning
Chronic Congestive Heart FailureNursing Management
Planning
• Overall goals: Peripheral edema Shortness of breath Exercise tolerance
– Drug compliance
– No complications
• Overall goals: Peripheral edema Shortness of breath Exercise tolerance
– Drug compliance
– No complications
04/11/2009 56
Chronic Congestive Heart FailureNursing Management
Nursing Implementation
Chronic Congestive Heart FailureNursing Management
Nursing Implementation
• Acute intervention
– Establishment of quality of life goals
– Symptom management
– Conservation of physical/emotional energy
– Support systems are essential
• Acute intervention
– Establishment of quality of life goals
– Symptom management
– Conservation of physical/emotional energy
– Support systems are essential
04/11/2009 57
What is Blood Pressure?
• The force of blood against the wall of the arteries.
• Systolic- as the heart beats
• Diastolic - as the heart relaxes
• Written as systolic over diastolic.
• Normal Blood pressure is less than 130 mm Hg systolic and less than 85 mm Hg diastolic.
04/11/2009 58
High Blood Pressure
• A consistent blood pressure of 140/90 mm Hg or higher is considered high blood pressure.
• It increases chance for heart disease, kidney disease, and for having a stroke.
• 1 out of 4 Americans have High Bp.
• Has no warning signs or symptoms.
04/11/2009 59
Why is High Blood Pressure Important?
• Makes the Heart work too hard.
• Makes the walls of arteries hard.
• Increases risk for heart disease and stroke.
• Can cause heart failure, kidney disease, and blindness.
04/11/2009 60
How Does It Effect the Body?The Brain
• High blood pressure is the most important risk factor for stroke.
• Can cause a break in a weakened blood vessel which then bleeds in the brain.
04/11/2009 61
The Heart
• High Blood Pressure is a major risk factor for heart attack.
• Is the number one risk factor for Congestive Heart Failure.
04/11/2009 62
The Kidneys
• Kidneys act as filters to rid the body of wastes.
• High blood pressure can narrow and thicken the blood vessels.
• Waste builds up in the blood, can result in kidney damage.
04/11/2009 63
The Eyes
• Can eventually cause blood vessels to break and bleed in the eye.
• Can result in blurred vision or even blindness.
04/11/2009 64
The Arteries
• Causes arteries to harden.
• This in turn causes the kidneys and heart to work harder.
• Contributes to a number of problems.
04/11/2009 65
What causes High Blood Pressure?
• Causes vary
• Narrowing of the arteries
• Greater than normal volume of blood
• Heart beating faster or more forcefully than it should
• Another medical problem
• The exact cause is not known.
04/11/2009 66
Who can develop High Blood Pressure?
• Anyone, but it is more common in:
• African Americans- get it earlier and more often then Caucasians.
• As we get older. 60% of Americans over 60 have hypertension.
• Overweight, family history
• High normal bp:135-139/85-89 mm Hg.
04/11/2009 67
Detection
• Dr.’s will diagnose a person with 2 or more readings of 140/90mm Hg or higher taken on more than one occasion.
• White-Coat Hypertension
• Measured using a spygmomameter.
04/11/2009 68
Tips for Having your blood pressure taken.
• Don’t drink coffee or smoke cigarettes for 30 minutes before.
• Before test sit for five minutes with back supported and feet flat on the ground. Test your arm on a table even with your heart.
• Wear short sleeves so your arm is exposed.
04/11/2009 69
Tips for having blood pressure taken.
• Go to the bathroom before test. A full bladder can affect bp reading.
• Get 2 readings and average the two of them.
• Ask the Dr. or nurse to tell you the result in numbers.
04/11/2009 70
Categories of High Blood Pressure
• Ages 18 Years and Older)
• Blood Pressure Level (mm Hg)
• Category Systolic Diastolic
• Optimal** < 120 < 80
• Normal < 130 < 85
• High Normal 130–139 85–89
04/11/2009 71
Categories of High Blood Pressure
High Blood Pressure
• Stage 1 140–159 /90–99
• Stage 2 160–179 /100–109
• Stage 3 180 /110
04/11/2009 72
Preventing Hypertension
Adopt a healthy lifestyle by:
• Following a healthy eating pattern.
• Maintaining a healthy weight.
• Being Physically Active.
• Limiting Alcohol.
• Quitting Smoking.
04/11/2009 73
DASH diet
• Dietary Approaches to Stop Hypertension.
• Was an 11 week trial.
• Differences from the food pyramid:
• an increase of 1 daily serving of veggies.
• and increase of 1-2 servings of fruit.
• inclusion of 4-5 servings of nuts,seeds, and beans.
04/11/2009 74
Tips for Reducing Sodium
• Buy fresh, plain frozen or canned “no added salt” veggies.
• Use fresh poultry, lean meat, and fish.
• Use herbs, spices, and salt-free seasonings at the table and while cooking.
• Choose convenience foods low in salt.
• Rinse canned foods to reduce sodium.
04/11/2009 75
Maintain Healthy Weight
• Blood pressure rises as weight rises.
• Obesity is also a risk factor for heart disease.
• Even a 10# weight loss can reduce blood pressure.
04/11/2009 76
Be Physically Active
• Helps lower blood pressure and lose/ maintain weight.
• 30 minutes of moderate level activity on most days of week. Can even break it up into 10 minute sessions.
• Use stairs instead of elevator, get off bus 2 stops early, Park your car at the far end of the lot and walk!
04/11/2009 77
Limit Alcohol Intake
Alcohol raises blood pressure and can harm liver, brain, and heart
04/11/2009 78
Quit Smoking
• Injures blood vessel walls
• Speeds up process of hardening of the arteries.
04/11/2009 79
Other Treatment
• If Lifestyle Modification is not working, blood pressure medication may be needed, there are several types:
• Diuretics-work on the kidney to remove access water and fluid from body to lower bp.
• Beta blockers-reduce impulses to the heart and blood vessels.
04/11/2009 80
Other Treatment
• ACE inhibitors- cause blood vessels to relax and blood to flow freely.
• Angiotensin antagonists- work the same as ACE inhibitors.
• Calcium Channel Blockers- causes the blood vessel to relax and widen.
• Alpha Blocker- blocks an impulse to the heart causing blood to flow more freely.
04/11/2009 81
Other Treatment
• Alpha-beta blockers- work the same as beta blockers, also slow the heart down.
• Nervous system inhibitors- slow nerve impulses to the heart.
• Vasodilators- cause blood vessel to widen, allowing blood to flow more freely.
04/11/2009 82
Conclusion
• Hypertension is a very controllable disease, with drastic consequences if left uncontrolled.