Chapter 57- ICP

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    Increased Intracranial Pressure

    Monro-Kellie hypothesis:becauseof limited space in the skull, anincrease in any one skull componentbrain tissue, blood, or CSFnecessitates a change in the volume ofanother

    Compensation to maintain a normalICP of 10 to 0 mm !g is normally

    accomplished by shifting or displacingCSF"ith disease or in#ury, ICP may

    increaseIncreased ICP decreases cerebral

    perfusion, causes ischemia, cell death,

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    ICP and CPP

    &ormal ICP is 10 to 0 mm!g

    CCP $cerebral perfusion pressure% is

    closely linked to ICP

    CCP ' ()P $mean arterial pressure% * ICP

    &ormal CCP is +0 to 100

    ) CCP of less than 0 results in

    permanent neuralgic damage

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    -arly Signs of ICP.he earliest sign of increasingICP is a change in /C

    Slo2ing of speech and delayin response to verbal

    suggestions are other earlyindicators

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    3etecting -arly Indications ofIncreasing ICP

    3isorientation, restlessness, increasingagitation, increased respiratory e4ort $5ussmaulbreathing%, purposeless movements, andmental confusion

    Pupillary changes and impaired e6traocularmovements

    "eakness in one e6tremity or on one side of the

    body!eadache that is constant, increasing inintensity, and aggravated by movement orstraining

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    ther manifestations

    include7

    8ehavior changes

    Sei9ures

    &ausea and :omiting/ethargy

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    in ICP is a medical

    emergency.reatment should

    be initiatedimmediately

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    "ays to relieve an increase in ICP

    3ecrease Cerebral-dema

    (annitol

    Fluid ;estrictions )ssess 8P, skinturgor, mucousmembranes, urineoutput < osmolality

    I: Fluids prescribed* slo2 to moderaterate

    ral hygiene b=c ofdehydration

    (aintainingCerebral Perfusion

    3obutre6

    /evophed5eep head in amidline position

    )void e6treme hip

    >e6ion)void the :alsalvamaneuver

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    "ays to relieve an increase in ICP

    ;educing CSF andIntracranial 8lood

    :olume3rain CSF

    )septic techni?ueand assess for

    signs of infection!yperventilation *as a last resort

    Controlling Fever)ntipyretic medications

    !ypothermia blanket

    )void shivering in the

    patient;emoving all bedding overthe patient $e6cept for alight sheet%

    @iving cool sponge bathsand an electric fan to

    facilitate cooling

    (onitor temperaturefre?uently * monitorresponse to therapy and toprevent e6cess decrease in

    temperature and shivering

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    "ays to relieve an increase in ICP

    (aintaining6ygenation

    (aintain a patentair2ay

    3iscourage coughingand straining

    )uscultate lungs everyA hours

    (onitor )8@s andPulse o6ymetry

    ptimi9e hemoglobinsaturation

    ;educing (etabolic

    3emands!igh doses ofbarbiturates

    Paralytics

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    3ue to the use of paraly9ing agentspatient 2ill re?uire7

    Continuous cardiacmonitoring

    -ndotracheal intubation

    (echanical ventilation

    ICP monitoring

    )rterial pressure monitoring

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    (onitoring ICP

    :entriculostomy7

    )5) :entricular Catheter (onitoring 3evice

    Fine bore catheter is inserted into the nonB

    dominant hemisphere of the brainCatheter connected to a transducer that monitorsthe ICP and ;ecords dataBscillator scope

    )llo2s for ICP relief by allo2ing for CSF release

    thus relieving intercranial !.&Intraventricular (ed )dministration access

    )ir or contrast administration for :entriculography

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    Ventriculostomy with fber optictransducer-tipped device

    Complication of

    Ventriculostomy:

    Infection

    Meningitis

    Ventricular

    Collapse

    Occlusion of

    catheter device by

    brain or blood

    materials

    Problems with

    monitoring system

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    (onitoring ICP $continued%

    Subarachnoid Scre2 or 8olt7

    Scre2 or bolt is a hollo2 scre2 that isinserted through a hole drilled in the skull

    and through a hole cut in the dura materin to the subarachnoid space

    !ollo2 scre2 avoids complications from brainshifting

    3oesnt re?uire ventricular puncture

    Infection < clogging scre2 2ith brain mattera4ecting readings

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    Subarachnoid screw or bolt

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    (onitoring ICP $continued%

    -pidural Sensor7

    -pidural 3evice is placed through aburr hole drilled in the skull, #ust overthe epidural covering Esespneumatic pressure to signal analarm for pressure abnormalities

    -pidural lining is not perforated, thusless invasive < less infection

    Cannot relieve e6cess CSF

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    (onitoring ICP $continued%

    Fiber ptic Sensor

    Fiber ptic device can be insertedinto the ventricle, subarachnoid andsubdural space (iniB.ransducerconverts ICP readings into electronicdigital monitoring

    "hen inserted in to the ventricle canallo2 for CSF 2ithdra2al

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    .rending ICP :alues

    ICP "aves7

    ) "avesBCan last B0minutes 2ith amplitudesbet2een 0B100 mm!g

    8 "avesB0 seconds to minutes 2ithamplitudes up to 0mm!g

    C "aves * ccur up Gtimes a minute 2ithamplitudes up to mm!g

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    &e2 .rends in &euro (onitoring

    /ico6 Catheter

    ) in 1 2hite mattercatheter that measures ICP,.emperature, and endcapillary tissue o6ygen level

    @ives real time feed back ofICP management, guidingtherapy and o6ygenation oftissue at risk in thecerebrum

    .he temperature probe canbe replaced 2ith amicrodialysis probe

    Picture from I&.;-@) 2ebsite7 http7==222integraBiscom=P3Fs=lico6=&S+H0ICPH0CatheterH02H0I(C

    H08oltpdf

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    /ate (anifestations of Increased ICP

    Further deterioration of /C stuporto coma

    3ecreasing level of responsiveness accidity may occur

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    Decorticate Posturing

    Decerebrate Posturing

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    /ate (anifestations of Increased ICP cont

    )lterations in vital signs

    Increase in systolic blood pressure

    "idening of pulse pressure

    Slo2ing of the heart rate pulse may >uctuate

    rapidly from tachycardia to bradycardiaIncrease in temperature

    Cushings !riad7 bradycardia, hypertension,< bradypnea

    Immediate intervention re?uired to preventherniation of brain stem < occlusion of blood >o2

    Cessation of cerebral blood >o2 results in cerebralischemia, infarction, < brain death

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    /ate (anifestations of Increased ICPcont

    :isual changes pupillary changes re>ectingpressure on optic=oculomotor nerves

    Pupils decrease or increase in si9e or become une?ual

    /ack of con#ugate eye movement

    Papilledema

    Pro#ectile vomiting may occur 2ith increasedpressure on the re>e6 center in the medulla

    /oss of brain stem re>e6es, including pupillary,corneal, gag, < s2allo2ing re>e6es

    /oss of re>e6es is an ominous sign of approachingbrain death

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    /ate (anifestations of Increased ICPcont

    Classic J6edand dilated

    Kblo2n pupilL)bsence ofoculocephalicre>e6 or KdollseyeL

    Picture7 http7==imagesgooglecom=imgresMimgurl'http7==222o2lnetriceedu=Npsyc1=Images=3ilatedPupil#pg

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    /ate (anifestations of Increased ICPcont

    (a#or complication ofIncreased ICP B!ernation

    $1% !erniation of the

    cingulate gyrus under thefal6 cerebri $% Centraltranstentorial herniation$% Encal herniation of

    the temporal lobe intothe tentorial notch $R%Infratentorial herniationof the cerebral tonsils

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    /ate (anifestations of Increased ICPcont

    3iabetes insipidus is theresult of decreasedsecretion of antidiuretichormone $)3!%

    SI)3! is the result of

    increased secretion of)3!)ll information other than the /ico6 slide, and blo2n pupil slide is from 8runner < Suddarths .e6tbook of (edicalBSurgical

    &ursing, 11th edition http7==thepointeeditionl22com=pt=re=T+A0+A1+T+AG=bookcontent01GTGB11thO-ditionBRhtm#sessionid'[email protected]/VgV+m6@yvpykn;hhvP;:UG5pkpWsU..TA;tPFhy/XBTAG1TRX1A11TGTXA0T1XB1 Information compiled by Stephen Strom, (ichelle !arris, )ngela;eaves, Su9anne Finch, and )manda 5ing

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