Chapter 074

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    Chapter 74

    Interventions for Clients with

    Renal Disorders

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    Polycystic Kidney Disease

    Inherited disorder in which fluid-filled

    cysts develop in the nephrons

    Etiology and Genetic Risk Autosomal dominant form

    50% chance of inheritance

    Autosomal recessive form

    25% chance of inheritance(Continued)

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    Polycystic Kidney Disease(Continued)

    Key features include: Abdominal or flank pain Hypertension

    Nocturia Increased abdominal girth Bloody or cloudy urine Kidney stones Constipation

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    Interventions/Complications

    Acute and chronic pain NSAIDs and ASA CAUTIOUSLY

    Lipid soluble antibiotics Bactrim, Septra, Cipro

    Constipation Fluid, fiber, exercise

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    Interventions/Complications

    Hypertension and renal failure Nursing interventions to promote self-management and

    understanding Fluid therapy

    Drink at least 2 liter of fluid per day

    Restrict sodium intake to control BP Fluid restriction is not necessary

    Drug therapy Antihypertensive agents diuretics

    Measure and record blood pressure Diet therapy

    Low sodium diet? Protein

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    Hydronephrosis,

    Hydroureter, and UrethralStricture Provide privacy for elimination. Conduct Cred maneuver as necessary.

    Apply double-voiding technique.

    Apply urinary catheter as appropriate.

    Monitor degree of bladder distention.(Continued)

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    Hydronephrosis,Hydroureter, and UrethralStricture(Continued) Catheterize for residual. Intermittently catheterize as appropriate.

    Follow infection protection measures.

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    Nephrostomy

    Client preparation

    Procedure

    Follow-up care including: Assess for

    amount of drainage.

    type of urinary damage expected.

    manifestations of infection. Monitor nephrostomy site for leaking urine.

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    Pyelonephritis

    Bacterial infection in the kidney (upper

    urinary tract)

    Key features include: Fever, chills, tachycardia, and tachypnea Flank, back, or loin pain

    Abdominal discomfort

    Turning, nausea and vomiting, urgency,frequency, nocturia

    General malaise or fatigue

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    Key Features of ChronicPyelonephritis Hypertension

    Inability to conserve sodium

    Decreased concentrating ability

    Tendency to develop hyperkalemia and

    acidosis

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    Acute Pain Interventions

    Pain management interventions

    Lithotripsy

    Percutaneous ultrasonic pyelolithotomy

    Diet therapy

    Drug therapy Antibiotics

    Urinary antiseptics

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    Surgical Management

    Preoperative care Antibiotics

    Client education

    Operative procedure: pyelolithotomy,

    nephrectomy, ureteral diversion, ureter

    reimplantaton

    Postoperative care for urologic surgery

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    Potential for Renal Failure

    Interventions include: Use of specific antibiotics

    Compliance with therapies and regular follow-

    up Blood pressure control

    Fluid therapy

    Diet therapy Other interventions

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    Renal Abscess

    A collection of fluid and cells caused by

    an inflammatory response to bacteria

    Manifestations: fever, flank pain, general

    malaise

    Drainage by surgical incision or needle

    aspiration

    Broad-spectrum antibiotics

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    Renal Tuberculosis

    Diagnosis

    Antitubercular therapy with rifampin,

    isoniazid, and pyrazinamide

    Complications renal failure, kidney

    stones, obstruction, and bacterial

    superinfection of the urinary tract

    Surgical excision possible

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    Acute Glomerulonephritis

    Assessment Onset approximately 10 days from time of

    infection.

    Streptococcal infection most common

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    Acute Glomerulonephritis

    Management of infection

    Prevention of complications Diuretics

    Sodium, water, potassium, and protein

    restrictions

    Dialysis, plasmapheresis

    Client education

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    ChronicGlomerulonephritis Develops over a period of 20 to 30 years

    or longer AssessmentEdema:

    presacral tissue

    Pedal

    Pretibial

    Adventitious breath

    sounds

    Uremic symptoms

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    Chronic Glomerulonephritis

    Interventions include: Slowing the progression of the disease and

    preventing complications

    Diet changes(Continued)

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    ChronicGlomerulonephritis(Continued) Fluid intake

    Drug therapy

    Dialysis, transplantation

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    Nephrotic Syndrome

    Condition of increased glomerular

    permeability that allows larger molecules

    to pass through the membrane into the

    urine and be removed from the blood Severe loss of protein into the urine

    (Continued)

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    Nephrotic Syndrome(Continued)

    Treatment involves: Immunosuppressive agents

    Angiotensin-converting enzyme inhibitors

    Heparin Diet changes

    If GFR is normal, dietary intake of complete proteins

    is needed

    Mild diuretics and sodium restriction

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    Nephrosclerosis

    Thickening in the nephron blood vessels,

    resulting in narrowing of the vessel lumen

    Occurs with all types of hypertension,

    atherosclerois, and diabetes mellitus Collaborative management: control high

    blood pressure and preserve renal

    function

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    Renovascular Disease

    Profoundly reduces blood flow to the kidneytissue

    Causes ischemia and atrophy of renal tissue Diagnosis

    Magnetic resonance angiography Renal vein renin levels Radionuclide imaging

    Interventions: drugs to control high bloodpressure and procedures to restore the renalblood supply

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    Diabetic Nephropathy

    Diabetic nephrophathy is a microvascular

    complication of either type 1 or type 2

    diabetes.

    First manifestation is persistentalbuminuria.

    Avoid nephrotoxic agents and

    dehydration. Assess need for insulin.

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    Cysts and Benign Tumors

    Thorough evaluation for cancer is needed.

    Cyst can fill with fluid and cause local

    tissue damage as it enlarges.

    Many cysts cause no symptoms.

    Cysts are a structural birth defect that

    occur in fetal life.

    Simple renal cysts are drained by

    percutaneous aspiration.

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    Renal Cell Carcinoma

    Paraneoplastic syndromes include

    anemia, erythrocytosis, hypercalcemia,

    liver dysfunction, hormonal effects,

    increased sedimentation rate, andhypertension.

    PTH produced by tumor cells can cause

    hypercalcemia (Continued)

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    Renal Cell Carcinoma(Continued)

    Nonsurgical management includes: Radiofrequency ablation, although effect is

    not known

    Chemotherapy: limited effect Biological response modifiers and tumor

    necrosis factor: lengthen survival time

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    Surgical Management

    Preoperative care

    Operative procedure

    Postoperative care:

    Monitoring Assess for hemorrhage and adrenal insufficiency

    Altered LOC

    Hypotension

    Decreased urine output

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    Surgical Management

    Post-op Care Cont. pain management

    Opioid analgesics

    prevention of complications

    Antibiotics Steroids

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    Renal Trauma

    Minor injuries such as contusions, small

    lacerations

    Major injuries such as lacerations to the

    cortex, medulla, or branches of the renalartery

    Pedicle injuries are lacerations or breaks

    in the renal artery or renal vein.

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    Renal Trauma

    Collaborative management

    Nonsurgical management: drug therapy

    and fluid therapy Dopamine

    Clotting

    Surgical management: nephrectomy or

    partial nephrectomy Bench surgery