Allergic Contact Dermatitis Picture

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Allergic contact dermatitis


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Allergic contact dermatitis

a) What is Hives?Hives, or the medical term 'Urticaria' is a common

allergic reaction on the skin. It is characterized by

raised, red skin welts that are more than 5mm in

diameter. Hives are extremely itchy, and often have a

pale border surrounding the red area. The urticarial

rash can be isolated or many, sometimes they join

together and form a large area of raised, red rash.

b) Statistics on HivesUrticaria is a common disease - it affects up to 25%

of the general population.

c) Risk Factors for HivesThere is no specific predisposing factors for urticaria. Geographical regions do not have any influence on cases of

urticaria. The number of cases are similar internationally.Acute urticaria happens equally in men and women; but

chronic urticaria seemed to be more common in women. Urticaria happens in all ages, but chronic urticaria is more

common in the 40-50s.

d) Progression of HivesUrticaria is divided into 2 forms according to the duration of disease:

acute: lasting


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chronic: lasting >6 weeksAcute urticaria is more common than chronic one. More than two-thirds of the cases of

urticaria are acute. The rashes of acute urticaria are characterised by a rapid onset and rapid resolution within

several hours. A trigger can occasionally be identified.As for chronic urticaria, it account for approximately 30% of the

cases. Clinically both acute and chronic forms are indistinguishable. However resolution time in chronic urticaria is

longer - sometimes up to 36 hours before resolution. The following are the recognised causes or associations of

urticaria:

autoimmune diseases: thyroid diseases

drugs: antibiotics (penicillins, etc), sulfur containing medications, progesterone (oral contraceptive pills, hormone

replacement therapy), local anaesthetic agents, opiates, etc.- physical contact: animal saliva, plant products, resins,

raw fish, vegetables, latex, metals (nickel)

stinging insects: bees, wasps, hornets

aeroallergens: allergens that travels in the air, like pollens, grass, weeds.

foods: milk, egg, peanuts, nuts, soy, wheat

infections: acute bacterial and viral infections such as Coxsackie A/B, hepatitis A/B/C, parasitic infections

physical agents: pressure, cold, heat (in exercise, sweating, hot showers causing cholinergic urticaria)

e) How is Hives Diagnosed?The diagnosis of urticaria is based on the signs and symptoms of the patient, and the history of exposure to

allergens. Therefore it does not rely on laboratory testing.

f) Prognosis of HivesUrticaria causes significant distress in terms of the symptoms produced, however prognosis is excellent as it is not

life-threatening. Acute urticaria is usually self-limited with resolution occurring within 24 hours. Chronic urticaria lasts

for more than 6 weeks, hence it can cause significant distress to the patient. However, urticaria does not results in

any long term complications.

g) How is Hives Treated?There is no cure for urticaria, the treatment aims at symptom control only. Regardless of the cause of urticaria, the

treatment options are generally successful in controlling the symptoms.Depending on the severity of urticaria, the

following treatment options can either be given alone or with combination:

allergen avoidance: prevention and avoidance of known triggers are the mainstay of treatment. This include limiting

outdoor exposure, shutting windows, avoiding the drugs that are identified, etc.
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antihistamines: this class of drugs directly blocks the action of histamine, the major substance responsible for

urticaria. It is effective in reducing symptoms such as itchiness (this is the most distressing symptoms for patients

with urticaria). Antihistamines include fexofenadine, loratadine and cetirizine.

steroids: steroids has limited use in most patients with acute urticaria. In individuals who have persistent or

recurrent attacks of acute urticaria (or associated with angioedema), they may respond to steroid therapy. Steroids

should only be used after a trial of maximal dose of antihistamines.Treatment should include treating the underlying

diseases if found any. This will be discussed in separate topics.

II. ANGIOEDEMA

A. What is angioedema?Angioedema is a skin reaction similar to hives orurticaria. It is most often characterised by an abrupt

and short-lived swelling of the skin and mucous membranes. All parts of the body may be affected but

swelling most often occurs around the eyes and lips. In severe cases the internal lining of the upperrespiratory tract and intestines may also be affected.

Angioedema

Image provided by Dr Janjua
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B. What's the difference between angioedema and urticaria?Angioedema and urticaria are very similar in many ways and can co-exist and overlap. Urticaria occurs

more commonly and is less severe than angioedema as it only affects the skin layers whereasangioedema affects the tissues beneath the skin (subcutaneous tissue). The main differences between

urticaria and angioedema are shown in the following table.

Feature Angioedema Urticaria

Tissues

involved

Subcutaneous and submucosal surfaces (beneath

the dermis)

Epidermis (outer layer of skin) and dermis

(inner layer of skin)

Organs

affected

Skin and mucosa, particularly the eyelids and lips Skin only

Duration Transitory (usually lasts between 24-48 hours) Transitory (usually lasts < 24 hours)

Physical signs Red or skin coloured swellings occurring below

the surface of the skin

Red patches and weals on the surface of skin

Symptoms May or may not be itchy. Often accompanied by

pain and tenderness.

Usually associated with an itch. Pain and

tenderness uncommon.


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F12 gene on chromosome 12, encoding coagulation factor XII.

Type 1 and II result in low levels of circulating C1 inhibitor; this is

normal in Type III

Occurs in 1 in 50,000 males and females; Type III more severe in

women.

Decreased C1 inhibitor activity leads to excessive kallikrein, which in

turn produces bradykinin, a potent vasodilator

Acquired C1 inhibitor

deficiency Acquired during life rather than inherited

May be due to B-cell lymphoma or antibodies against C1 inhibitor

Whatever the cause of angioedema, the actual mechanism behind the swelling is the same in allcases. Small blood vessels in the subcutaneous and/or submucosal tissues leak watery liquid through

their walls and cause swelling. This same mechanism occurs in urticaria but just closer to the skinsurface.

D. What are the clinical features of angioedema?Symptoms and signs of angioedema may vary slightly between the different types of angioedema butin general some or all of the following occur.

Marked swelling, usually around the eyes and mouth

Throat, tongue, hands, feet and/or genitals may be affected too

Skin may appear normal, i.e. no hives or other rash

Swellings may or may not be itchy

Swellings may be painful, tender or burning

In severe angioedema swelling of the throat and/or tongue may make it difficult to breath

Swelling of the lining of the intestinal tracts may cause gastrointestinal pain and cramps

Some features specific to the different types of angioedema are listed below.


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Angioedema type Clinical features

Acute allergic Almost always occurs with urticaria

Angioedema and urticaria both usually occur within 1-2 hours of exposure to

allergen (exception is ACE inhibitor induced angioedema that usually occurs within

the first week of treatment but can occur weeks to months later)

Reactions are usually self-limiting and subside within 1-3 days

Reactions will recur with repetitive exposures or exposure to cross-reactive

substances

Non-allergic drug

reaction ACE inhibitor induced angioedema occurs without urticaria

Idiopathic/chronic

Similar to acute allergic but angioedema keeps on recurring and often no known

cause is found

Hereditary

Patients often experience no symptoms until they reach puberty

Swellings can occur without any provocation or induced by precipitating factors,

including local trauma, vigorous exercise, emotional stress, alcohol, and hormonal

factors (menstruation, pregnancy, oestrogen)

Some patients may get a transitory prodromal non-itchy rash, headache, visual

disturbance or anxiety

Face, hands, arms, legs, genitals, digestive tract and airway may be affected;

swellings spread slowly and may last for 3-4 days

Abdominal cramps, nausea, vomiting, difficulty breathing and rarely urinary

retention from swelling of internal tracts

Urticaria (wealing) does not occur


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E. How is the diagnosis made?A detailed medical history is invaluable in diagnosing angioedema.

Keep a diary of exposure to possible irritants

Tell your doctor about all medications you take, including over-the-counter (OTC) drugs and

herbal remedies (even if taking them irregularly)

Any family history of skin rash, allergies

Skin prick testing may be performed to try and identify any allergens. If hereditary angioedema issuspected, blood tests to check for levels and function of specific complement blood proteins may bedone.

F. What is the treatment for angioedema?Treatment of angioedema depends on the severity of the condition. In cases where the respiratory

tract is involved the first priority is to secure the airway. Patients may need emergency hospital careand require intubation (placement of a tube in the throat to keep the airway open).

Patients with mild acute angioedema can usually be treated the same way as those with an allergicreaction (seeurticaria). In many cases the swelling is self-limiting and resolves spontaneously after afew hours or days. In more severe cases where there is persistent swelling, itchiness or pain thefollowing medications may be used.

Subcutaneous adrenaline (epinephrine)

Oral or intravenous corticosteroids

Oral or injectedantihistamines

Chronic angioedema with chronic autoimmune or chronic idiopathic urticaria is often difficult to treatand response to medication variable. The following treatment steps are recommended. Each step isadded to the previous one if inadequate response is obtained.

Step 1: non-sedating antihistamines, e.g. cetirizine

Step 2: sedating antihistamines, e.g. diphenhydramine

Step 3: a) oral corticosteroids, e.g. prednisone

b) immunosuppressants e.g.ciclosporin ormethotrexate

The goal of treatment for chronic angioedema is to reduce itch, swelling, tenderness and pain to atolerable level to maintain function (e.g. work, school, sleep).
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Acute episodes of hereditary angioedema do not respond to adrenaline, antihistamine andcorticosteroids. Fortunately most acute episodes of Type I and II hereditary angioedema are non life-threatening.

The mainstay of emergency medical treatment is intravenous C1 inhibitor concentrate (a blood

product).

If this is unavailable, fresh frozen plasma can be infused, but this occasionally exacerbates the

angioedema.

New medications inhibit bradykinin, e.g. icatibant, or kallikrein, e.g. ecallantide. Ecallantide

has been reported to causeanaphylaxisin some cases.

The chance of an attack can be reduced with the following medications:

C1 inhibitor concentrate infused an hour before a surgical procedure

Anabolic steroids (stanazolol, oxandrolone and danazol) to increase circulating levels of normal

functional C1 inhibitor. These have 'male-like' hormonal activity, so may cause weight gain,

menstrual irregularities and virilism.

Tranexamic acid has been used in pre-pubertal children and may be effective in Type III

herediary angioedema.
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G.What is Atopic Dermatitis?

Atopic dermatitis is a persistent and chronicinflammatory condition. It wanes and relapses on a continual basis and manifests with symptoms

of dry, flaky, red, itchy skin. It goes by many other names, including infantile eczema and

flexural eczema.

Unlike most dermatitis conditions, atopic dermatitis is not circumstantial and subject to various

environmental stimuli. It may be made to outbreak, however, due to almost anything. The

number of circumstantial stimuli that can spark an outbreak of atopic dermatitis are many, andthe hallmark of the ailment. The comedy stereotype of the nerdy kid whose none-acne skin

condition will flare up at the drop of a hat originates from atopic dermatitis.


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Atopic dermatitis is frequently identified during infancy. Despite being a chronic condition, it is

not serious on its own, and most simply deal with it the way they would a minor allergy, bysimply avoiding the offending circumstances and influences that might provoke an outbreak.

H. Who gets Atopic Dermatitis?Atopic dermatitis is a very common ailment. Some one in nine individuals suffer from atopic

dermatitis to varying degrees of severity. Some will be in a constant state of inflammation, and

others will have only very rare outbreaks in response to strong reactions.

Atopic dermatitis is almost always found in childhood. It is generally present from birth, with its

discovery occurring upon its first breakout. This usually occurs during infancy, but it may rarely

lurk undiscovered until adolescence, or even puberty, when raging and tumultuous hormones candrag it out of hiding. From here, it will usually persist.

Individuals of Caucasian descent are almost always more likely to suffer from atopic dermatitis.While the condition does not discriminate and is not contagious, Caucasian individuals have skin

that is much more susceptible to such damage and difficulty. Thus, atopic dermatitis is more

likely to present in Caucasians. This is not because atopic dermatitis is more likely to present inCaucasians in and of itself, but rather because Caucasians are more susceptible to the symptoms

themselves.

There is a very clear family link in cases of atopic dermatitis. Parents with sensitive skin are far

more likely to give birth to children with sensitive skin. It is entirely possible for a child to dodge

this genetic bullet, but statistically speaking they will likely suffer the same trouble as theirparents. If both parents suffer from atopic dermatitis, this becomes even more likely.

Contributing factors are likely to be similar as well, much like certain allergies. Parents who

have exceptional difficulty with sunlight or particular dietary issues will be more likely to have

children who suffer from the same.

I. What causes Atopic Dermatitis?The causes of atopic dermatitis are not clear. Generally, an increase in the various chemicalreceptors that can make skin more or less sensitive to various external stimuli are involved, but

this is not universally true. Because it is poorly understood, there is no true cure. It is likely thatthere is not one simple natal cause, but rather a number of elements coinciding to make the skin

more sensitive.

It should be elucidated that one cannot catch atopic dermatitis. It may not present as early in all

as it does in most, but it cannot be contracted later in life. It is entirely possible to developvarious allergies later in life with similar results, this is a very different process. Nothing can

cause atopic dermatitis after birth.


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Stimuli that can cause outbreaks of atopic dermatitis are nearly endless, however. The stereotype

of a nerdy individual with skin that will break out at the drop of the hat is both originated fromatopic dermatitis and, further, not very far off from reality.

Heat is a major offender, especially coupled with sun exposure. The skin may be extremely

sensitive to dehydration and will almost immediately dry, flake and peel, itching and swellingbadly. The result may be very much like a sunburn, symptomatically, but the underlying cause

and the actual true effect is different. The permanent damage that a sunburn may entail will notbe a relevant factor.

Dietary influences frequently play a role. Any number of foods may be too oily for the skin tohandle, and this may subsequently cause a breakout.

Hormonal influences, as is frequently true with dermatological conditions, are likely to play arole. Female hormone cycles can be a major offender in this, as can the various hormonal

overhauls that an individual suffers during puberty. Any disturbance, from those overt changes

that may occur during natural biological processes to those that may occur when one switchesfrom one contraceptive to another to the very subtle changes that may occur due to the hormone

content of various dairy and meat products may trigger an outbreak of atopic dermatitis.

J. What does Atopic Dermatitis cause?Atopic dermatitis can cause a number of uncomfortable symptoms. Frequently, these do not

extend beyond simple irritation and inflammation. Itching is very common. Flaking and redness

of the skin is almost guaranteed. Beyond this, the symptoms of atopic dermatitis and the ultimateresult of an atopic dermatitis outbreak is primarily dependent upon the cause of the outbreak.

More severe outbreaks of atopic dermatitis may result in minor scarring or scaling of the skin leftafter the outbreak subsides. Scarring will almost invariably heal after no more than a week or

two. Scaling can be easily resolved with the application of proper lotions or moisturizers. These

are almost purely cosmetic. Any true complications from either of these would have to be wildly

atypical and likely result from the trigger condition.

Inflamed skin is frequently vulnerable to damage. Between the dryness and flaking that makes itvulnerable and the itching that inclines individuals to scratch at it, it is relatively easy to make

inflamed skin become damaged skin. This damaged skin subsequently may become infected by

bacteria, which can lead to something far more severe than the atopic dermatitis itself.

The most profound effects of atopic dermatitis tend to be mental. Individuals suffering from

atopic dermatitis frequently have confidence issues due to the alterations to their appearance that

they have no control over. Atopic dermatitis can manifest almost anywhere on the body(although not necessarily everywhere at once), which makes it a sharp contrast to acne, which

frequently afflicts a limited area. In children, atopic dermatitis may have social implications, aschildren may ostracize others based on such things. The tendency of affected skin to dry andflake away only compounds this. Later in life, the social implications are generally diminished


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by the increased maturity level of the affecteds peers, but the psychological implications and

discouragement may persist.

K.How serious is Atopic Dermatitis?

Atopic dermatitis is not serious from a medical standpoint. It has no

mortality rate to speak of, and no notable co-morbid conditions that need be worried about

should atopic dermatitis present in an individual. Most of the worst symptoms are cosmetic and

do not last long. There are, however, circumstantial occurrences that can render atopic dermatitisa more serious condition to be reckoned with.

Atopic dermatitis is most serious when it results in infectious complications as a result of

mechanical excoriation of inflamed skin. That is to say, if an individual picks at the dried,

swollen and itchy skin of an atopic dermatitis outbreak, they may leave it vulnerable to bacterialinfection. This infection will nearly always be more severe than the initial dermatitis damage if it

should occur. However, this is relatively easy to prevent by keeping skin that has broken out

insulated against damage with proper clothing and moisturization.

While atopic dermatitis is chronic and cannot truly be cured, its relatively benign nature means it

is rarely more than a nuisance to be dealt with on a regular basis. Thus, it isnt a true risk, butsimply a factor to be considered when relevant.

L. What does Atopic Dermatitis treatment look like?Atopic dermatitis treatment is almost purely symptomatic. Atopic dermatitis is chronic and prone

to relapse. It cannot be treated in such a way as to cure it. However, treatment options do existto mollify or nullify atopic dermatitis symptoms as they present or to prevent their coming to the

fore to begin with.

Moisturizer is a major boon with respect to the primary itchiness and dryness that can afflictanyone suffering from an atopic dermatitis outbreak. While proper hydration through water

consumption is nearly always more effective than lotions or moisturizers with respect to keepingthe skin healthy with enough water content, lotions and moisturizers can alleviate the itching and

burning that may be experienced during an outbreak. The act of rubbing lotion into the skin can

help clear away dead flakes of skin without letting them be scratched away, and the lotion itselfcan further insulate the skin against damage and irritation. The moisture content will also help to

salve the irritation somewhat.


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Outbreaks can also be prevented by keeping triggering factors at bay. Individuals who suffer

from atopic dermatitis due to sunlight should wear sunblock or SPF-rated moisturizers and keeptheir skin covered or otherwise protected from UV radiation. Individuals who suffer from diet-

triggered atopic dermatitis should avoid the foods that are likely to provoke an outbreak.

Individuals who suffer from an allergic reaction should compensate accordingly, avoiding the

plants or animals that provoke their reaction to the best of their ability and washing their skinthoroughly after any accidental, unavoidable contact.

It is recommended that afflicted individuals keep a journal of triggered outbreaks to help

determine what affects them most strongly. This can help to weed out various incidental factors

from circumstantial factors and let the afflicted avoid atopic dermatitis-triggering conditions inthe future.

Many, of course, will not feel any need to pursue such measures. Some individuals suffering

from atopic dermatitis have cases so mild that they simply dont care about the minorsymptomsthat may occur. This is especially true for individuals who suffer from atopic dermatitis that

primarily triggers due to sunlight; any outbreaks will simply be written off as sunburn, andextended sun exposure will be avoided just as anyone with or without atopic dermatitis. Manyothers who technically suffer from atopic dermatitis will simply not be aware of their symptoms.

Thus, these treatment measures will vary widely.

In any case, unless very severe complications arise from a related bacterial infection (which is in

and of itself fairly rare), it is unlikely that any serious medical intervention will be required.

Unless ones case of atopic dermatitis is severe enough to interfere with day-to-day life (mostcommon in young children), medical treatment will likely not need to be considered. Atopic

dermatitis is, for this, almost completely harmless and benign, and a nuisance at worst.

M.How do I know if I have Atopic Dermatitis?Atopic dermatitis is identified by its persistent and consistent nature. Much like an allergy, one

will not simply react to a situation once and not again. An allergy can be noted when one

repeatedly has an issue with a substance or circumstance and must adjust their behavior to avoidit. Atopic dermatitis is similar; having a rash once, due to poison ivy, sunburn, sleeping all night

under an electric blanket or some other single factor does not denote atopic dermatitis. Rash-

sensitive skin from childhood that may break out under nearly any circumstance, be it sunexposure, dietary intake or even cloth rubbing uncomfortably.

Atopic dermatitis is almost always identified in infancy, when an individual is most vulnerable.

It is rare for one to make it to adolescence without their condition having revealed itself, butfeasible, especially with respect to chemical sensitivities such as caffeine, or stress triggers that

may not come into play until a child has begun working under higher pressure in school.

If a persistent and consistent rashing condition seems to be troubling you, you may be suffering

from atopic dermatitis. Self-diagnosis is not a reliable method or replacement for a medical

professionals input, however. While there is no effective cure, there are a number of things


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that can be done to prevent outbreaks and alleviate outbreaks that do occur. It is thus a good idea

to speak to a medical professional for consultation and advice, the sooner the better.

N. What is Sun Poisoning Rash?

Picture 1Sun Poisoning Rash

Sourcetandurust

Sun Poisoning Rashes are skin rashes that develop as an allergic reaction of the body in response

to prolonged exposure to the sun. Anyone can suffer from Sun Poisoning Rashes. However, fair-

skinned people are more susceptible to this condition as their skin lack the necessary

pigmentation to combat the harmful UV rays of the sun.
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O. Sun Poisoning Rash SymptomsKnow about some of the major symptoms of Sun Poisoning Rashes :

1) Skin RashesSkin rashes are the most visible symptom of this condition. Sun Allergy Rashes arise as a bodilyreaction against exposure to the sun rays. It is often a case of tanning gone wrong.

Sun Poisoning Rashes appear to view as small, red patches on the skin. These are usually itchy,

painful and elevated. The rashes may appear within a few minutes of exposure of skin to sun or itmay arise a good few hours afterwards.

The condition is usually a mild one and marked only by the appearance of skin rashes. However,there can be other associated symptoms in severe cases. These are

2) HeadacheSkin rashes can also be accompanied with headache. The person may experience an acute pain inthe temple that can impair activities and make it very discomforting for the person.

3) NauseaThe person can also experience nauseating symptoms along with headache. There is a theory thatnausea arises from acute pain in the head.

4) DizzinessThe affected person also suffers from dizziness. There may be a mild or severe reeling sensation

in the sufferer.

5) FeverA sufferer may also experience increased body temperatures due to the condition. Fever is

accompanied with chills.

P. Sun Poisoning Rash CausesRead on to know about some of the major causes of Sun Poisoning Rash :

1) UV RaysAs aforementioned, prolonged exposure of the skin to the harmful UV rays of the sun result in

Sun Poisoning of the skin.


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2) Absence of PigmentationAs aforesaid, fair-skinned people are at more risk of having Sun Poisoning Rashes as their skin

do not have the pigmentation necessary to act as a protective barrier between the epidermis andthe UV rays of the sun.

3) DrugsCertain medicines increase the vulnerability of the skin to UV rays. Components like

Salicylanilide and Hexachlorophene, found in industrial cleaner and antibacterial soaps

respectively, Tetracycline Antibiotics, birth control pills, tranquilizers and blood-pressure

reducing drugs increase the sensitivity of the skin. These drugs can give rise to burns and rashesin the skin even without any exposure to the sun.

Other potentially risky drugs are Thisazide Diruretics and antibiotics. Certain chemicals incosmetic products like soaps, perfumes and lipsticks also invite skin rashes similar to Sun

Poisoning Rashes.

Q. Sun Poisoning Rash DiagnosisThe condition is mainly diagnosed by physical observation. An experienced healthcareprofessional looks at the rashes with naked eye and diagnoses the condition.

R. Sun Poisoning Rash TreatmentFor rashes arising out of Sun Poisoning Treatment consists of reducing the excess temperature of

the body that gives rise to rashes. This is done by making the body cool and hydrated by naturalmethods and lifestyle modification. Know about the common methods used in treatment for SunPoisoning.

1) ShowersTaking cool water showers after every few hours will help reduce the body temperature andprevent rise of skin rashes.

2) Avoid Strong Soaps

Do not use hard soaps on the skin while taking bath. This may lead to allergy in the rash spotsand further complicate the situation.

3) Treat the Rashes GentlyGently rub the affected spots with a soft towel to dry them. Never scratch the itchy rashes with

your bare finer nails. This may chafe the region. Use the soft end of a cloth or towel to scratch

the spots.


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4) Sponge the SpotsSoak a cotton ball with a small amount of cold water or milk. Press it over the rashes. Soak it

again and continue the process. Go on doing this for 15-20 minutes to draw out heat from thebody.

5) Increase fluid IntakeDrink plenty of fluids. This will keep the body hydrated and reduce prevalence of rashes.

6) DrugsItching and inflammatory symptoms are usually controlled with anti-itching medicines. Though

analgesics and over-the-counter anti-itching and anti-inflammatory oral drugs and topicalointments are available, it is better if you consult your doctor prior to using any of these. Make

your doctor know if you suffer from any other disease like diabetes before he/she prescribes

drugs.

S.

Sun Poisoning Rash Natural TreatmentSome of the most important Sun Poisoning Rash natural remedies include :

1) Baking soda SolutionMix a little baking soda to a small amount of water to make a paste. Apply this over the rashes toget relief from painful symptoms.

2) Calamine LotionItchiness is a major complain for sufferers of Sun Poisoning Rashes. Calamine lotion is found tobe quite effective in reducing itchiness in such type of skin rashes.

3) Antioxidant FoodsSufferers must incorporate into their diet foods that are rich in antioxidants such as berries,broccoli, beans, tomatoes, spinach, whole grains, carrots and soy. Green tea is also a rich source

of antioxidants.

T. Sun Poisoning Rash HealingPeople suffering from sun blisters are generally seen to recover within a few hours or within a

week. Some people may take a little longer. But the condition usually resolves in a couple of

weeks. Following effective Sun Poisoning Rash cure can make the rashes go away much earlier.


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U.

Sun Poisoning Rash PicturesCurious to know what does Sun Poisoning Rash look like? Here are some useful Sun Poisoning

Pictures. These pictures of skin poisoning rash show people who have suffered from rash aftersun exposure. If you are suffering from rashes, these Sun Poisoning Rash photos can help you

decide whether you are really suffering from the condition.

Picture 2Sun Poisoning Rash Picture

Sourceparaorkut

Picture 3Sun Poisoning Rash PhotoSource - typepad

If you are suffering from sun blisters, it is advisable that you use the natural sun rash treatments

at home. If the condition shows no improvement in a day or two, you should get professional

medical assistance. This will help you make a faster recovery from this non-threatening butdiscomforting condition.

Our skin is a good barometer of how healthy the body is. A frequent recurrent infection of

the skin may indicate that theimmunitysystem is weak.Skinhas its own interference withlifestyle in colour and texture. Our skin controls and regulates the water content of thebody. It protects body as a covering and works as defence force against infection. It is

helpful in activating vitamin D when it is exposed to sunlight. If skin is damaged watercontent will be lost. Infection will spread resulting in renal shut down and collapse.

Whenskinor body is confronted with any other foreign substance, our immune system

counteracts to withstand any ill effects from that substance. Antigen-antibody reaction willdevelop to inhibit all the ill effects. If this is excessive and it creates disease like condition
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then it is called Allergy. So the heightened reaction of the body to external agent is called

Allergy. Antigens developed from this reaction attach to blood cells and form mast cells.

When this gets contact with allergen, it secretes histamine which is primary chemicalconductor and responsible for allergic reactions. When the body has been exposed to theelements known as allergen, which has been previously exposed,allergiesform on the

body. Allergies are different types asallergic rhinitis, sinusitis, asthma and allergic skin

disorder. All allergies are serious and very uncomfortable. Allergies can be due to anyforeign material natural or artificial.

If any foreign substance enters into the nose, wesneeze. If this occurs continuously as allergy then it is

called as allergicRhinitis. Likewise, if there is any

irritation in the skin, body reacts as red itchy swellinginitially, which in due course with subsequent exposureleads to allergic skin disorder.

Skin allergy usually subsides by it self, and denotesabout the bodies sensitivity. If this is excess it becomes

allergic skin disorder.

Skin allergies or Allergic Skin Disorders are a serious and uncomfortable type. Any red,bumpy, scaly itchy, swollen skin can signify an allergicskin condition. This is the result ofimmune reaction against an irritant. From an overall healthcare the problem of allergy scare

is dismissed as minor annoyances without due consideration for diagnosis and treatment.The majority sufferers are females leading to dismissal more readily than if the symptomswere experienced by men. Allergy in some form occurs in all individuals.

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Documents

Allergic Contact Dermatitis Picture