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© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect for lasting results. Presented By - John Apsley, MD(E), DC In alliance with: The International College of Colloidal Therapeutics (ICCT TM ) & School of Constitutional & Eclectic Medicine TM

© 2011 by John W. Apsley, II, MD(E), DC Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

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Page 1: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Cancer No More: Setting up the Four Pillars

approach at home to induce The Regeneration Effect for lasting results.

Presented By - John Apsley, MD(E), DC

In alliance with: The International College of Colloidal Therapeutics (ICCT TM) &

School of Constitutional & Eclectic MedicineTM

Page 2: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

What Causes Cancer (How Does Cancer Arise)? There are always key reasons why people get cancer (it's all about the milieu or "turf"):  The first and foremost is from deficient oxygen saturation and/or defective utilization (which contributes to

hyperacidity & inflammation). The second reason is from a special kind of dehydration unique to cells, which translates into chaotic

water "structure" (which contributes to hyperacidity & inflammation). The third is a colloidal mineral complex deficiency or dysregulation in your body cells, especially potassium,

magnesium, manganese, zinc, copper, iodine and rubidium (which also contributes greatly to hyperacidity). The fourth is unsuspected low thyroid function (which greatly contributes to inflammation). The fifth is poor digestive function (i.e., "death begins in the colon" - lower hydrochloric acid output, lower enzyme

output, lower sodium bicarb output, and protein malabsorption, which progressively contributes to systemic hyperacidity & inflammation).

The sixth is a congested liver, usually from low thyroid function, low fiber diets and disturbed intestinal flora (low populations of good probiotics like acidophilus), AND from exposures to toxic substances from the environment that greatly impacts the liver (which also contributes to inflammation).

The seventh is a deficient lifestyle plagued with: lack of sufficient raw food factors, lack of proper exercise, addictive behaviors and, maintaining occupations that harm ourselves, those around us and/or our planet in general.

The eighth is exposures to harmful mental conditioning such as:• negative or unproductive attitudes towards oneself, one's family, and the Universe in general; • lack of discipline;  • deficiency of loving, calming and charitable practices; and possibly • unhealthy compulsive or abusive behaviors.

Page 3: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

* See: http://davelafferty.com/

Page 4: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

* See: http://davelafferty.com/

Page 5: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

* See: http://davelafferty.com/

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Page 6: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

The turf of Cancer is one of cellular depravity and regional chaos. In a very real biological sense, it is cellular mindlessness. Specifically, the cell to cell communications are all nonsensical, garbled, and muted. This leads to bizarre adaptation of the cells into forms which function the best they can in this chaotic turf while starving for energy.  Thus, their internal programs, deprived of outside references to indicate otherwise, kick into a "survival at all costs" mode and to hell with protocol! There can be no finer example of the low-ground or low-gain milieu than the mindless, chaotic turf of cancer.

Just like a placenta in tow with its embryo finding themselves in an 'in utero' barren wasteland, the cells spare no effort to improvise, and in the process go rogue. 

Cope FW. Pathology of structured water and associated cations in cells (the tissue damage syndrome) and its medical treatment. Physiol Chem Phys 1977;9(6):547-53.

Page 7: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

* See: http://davelafferty.com/

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Page 8: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

In review - the first engine acts as a rogue placenta emulating the beast Cerebrus. This member of the cancer quartetresembles what most oncologists have thought to be tumor cells. However, its unrecognized structure and function was really to feed a rogue embryo. This rogue embryo is comprised of rogue cells called a stroma, which behaves as if a lost soul because it is not supposed to be there. Both co-evolved in order to execute the only option left open to them to survive an extremely rough & rugged turf - the wrath of Hell. Very little actual waste is produced that cannot be recycled to grow and support more cancer cells, but only cancer cells and not normal healthy cells. Twenty-four/seven, emergency rationing plus extreme efficiency rules the day, which gains a significant competitive advantage and means to survive over adjacent normal cells.

Martinez-Outschoorn UE, et al. The autophagic tumor stroma model of cancer or “battery-operated tumor growth:” A simple solution to the autophagy paradox, Cell Cycle, 2010 Nov 1; 9:21, 4297-306. © 2010 Landes Bioscience.

Page 9: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

* See: http://davelafferty.com/

Page 10: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

The rogue placenta is vulnerable to blowing up if autophagy becomes short-circuited by way of overload. We term this lethal autophagy. The capacity for catastrophic autophagy falls under the control of innate mechanisms within the body whose purpose is to re-establish control over rogue recycling of cell debris. Under strategies utilized by Eclectic Oncology, all-natural tools are employed which may excite this innate wisdom of the body to overload such rogue cell recycling, with little harm to normal healthy cells.   The rogue embryo is also vulnerable to blowing up, in this case by re-establishing apoptosis under the cell's autopilot control which suffers from amnesia. Under strategies utilized by Eclectic Oncology, normal healthy apoptosis may become fully reawakened, thereby shutting down this rogue embryo.

Akar U, et al. Silencing of Bcl-2 expression by small interfering RNA induces autophagic cell death in MCF-7 breast cancer cells. Autophagy 2008 Jul 1;4(5):669-79.

Serini S, Piccioni E, Merendino N, Calviello G. Dietary polyunsaturated fatty acids as inducers of apoptosis: implications for cancer. Apoptosis. 2009 Feb;14(2):135-52.

Page 11: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

* See: http://davelafferty.com/

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© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

High titers of pro-inflammatory extremes play tag team in the milieu of cancer by bringing about hostile weather conditions intended to trigger deadly cellular crashes. As the crashes inevitably multiply, the wreckage is strewn far and wide, across the turf in all directions. Many explosions follow, launching flaming wreckage airborne. Eerily as this race for life & death continues, Molotov cocktails now rain down all over the speedway. The turf is now perfectly reminiscent of what would have been the scene encircling Pearl Harbor on December 7th, 1941 (right after the first salvo of explosions were underway thrusting giant plumes of thick ash & soot skyward). As cancer wages its battle over life, death & more turf, with all the injured being tossed into concentration camps of the Holocaust, the light turns into darkness as if Heaven and Hell swapped places.

Flavin, DF. Clinical-Patient Studies: A lipoxygenase inhibitor in breast cancer brain metastases. Journal of Neuro-Oncology. 2007 Mar;82(1):91-3.

Page 13: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Eclectic Oncology does not seek to treat the disease, but rather the patient by using methods to restore the patient’s constitution. Eclectic Oncology starts at the ground floor level with techniques that: Reconstitute the patient’s cells, then Optimizes cell to cell communications, then Initiates the process of tissue and organ regeneration, and Empowers Mind-over-Body excellence, which Culminates into revitalizing the patient’s Regeneration Effect within.

Page 14: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Conventional Oncology’s Strategies are Threefold, with a Plan “B”

Surgery

Chemotherapy

Radiation Therapy

Immunotherapy when patient goes into crises

Page 15: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Five Cancers Will Account for Approximately 62% of All Cancer Deaths in the U.S.

Lung Cancer

Colon Cancer

Breast Cancer

Prostate Cancer

Gynecological Cancers (Ovary, Uterus, etc…)

CA Cancer J Clin 2004;54:8029. In: Faguet GB. The War On Cancer: An Anatomy of Failure, A Blue Print for the Future. Springer, Dordrecht, The Netherlands, 2005; p. 9.

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© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Survival Gains made against these top five deadly cancers have nothing to do with

Conventional Cancer Treatments. The biggest reason why gains in survival are reported is due to

“Lead-Time Bias,” which is simply that more cancers are being detected now in their earlier stages, and thus the patient would naturally live longer with or without conventional treatments.

Lung cancer has slightly declined mostly due to folks giving up the tobacco habit, and not due to conventional cancer treatments.

Other minor reasons why folks with these cancers are living longer have to do with improved hygiene and safer foods, and not to conventional cancer treatments.

Surveillance, Epidemiology and End Results (SEER) Program of the National Cancer Institute (NCI). In: : Faguet GB. The War On Cancer: An Anatomy of Failure, A Blue Print for the Future. Springer, Dordrecht, The Netherlands, 2005; pp. 15-17.

Page 17: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

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Iatrogenic death (Physician Induced Death) from Conventional Cancer Treatments.

Up until now, conventional oncology has had a difficult time solving the riddle as to precisely what is & what is not the cancerous state, especially in regard to its turf (milieu). Thus, conventional treatment development has consistently ignored critical extracellular and intracellular milieu factors to the great detriment of the patient's own immune system.

Indeed, conventional oncology's strategy of "cut, burn and poison" frequently is devastating to the patient's remaining healthy tissues and milieu. In fact, fatalities from conventional treatment (and not from the cancer) approaches 40%! And of these iatrogenic deaths, approximately one-half will occur within the first 30 days of conventional treatment because they are so caustic to essential tissues and organs.

Webb SD, Sherratt JA, Fish RG. Mathematical modelling of tumour acidity: regulation of intracellular pH. J Theor Biol. 1999 Jan 21;196(2):237-50. See: www.gilbertling.orgMartinez-Outschoorn UE, et al. Oxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cells. Cell Cycle. 2010 Aug 15;9(16):3256-76.See: NCEPOD-SACT Report.pdf

Page 18: © 2011 by John W. Apsley, II, MD(E), DC  Cancer No More: Setting up the Four Pillars approach at home to induce The Regeneration Effect

© 2011 by John W. Apsley, II, MD(E), DC www.doctorapsley.com

Iatrogenic Death From Chemotherapy Chemotherapy is most effective for childhood cancers such as leukemia and lymphoma.

But the after affects of conventional treatment can be devastating. For example, according to the New England Journal of Medicine, conventional treatments for childhood Hodgkin's disease are 1,800% more likely later to develop secondary malignant tumors. Girls are at great risk too. 35% of young girls treated for Hodgkin's disease with conventional methods should expect to develop breast cancer on or before they reach 40 years of age. This astounding figure is 7,500% times greater than the risk of developing breast cancer for women in general.

The risk for developing leukemia increases alarmingly at the four year post-treatment mark and continues to increase in risk until the 14th year. Beyond that, the increased likelihood of developing secondary solid tumors remains high (approaches 30% of all patients treated conventionally as a child for Hodgkin's disease), at the 30 year post-treatment mark.42 Between these horrific figures, it gives us all pause to ask if there were not a better way of going about this?

Bhatia S, et al. Breast cancer and other second neoplasms after childhood Hodgkin's disease. N Engl J Med. 1996 Mar 21;334(12):745-51.

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Effectiveness of Conventional Chemotherapy for all other cancersChemotherapy Failure Rate   Morgan G, Wardy R, Bartonz M. Overview: The Contribution of Cytotoxic Chemotherapy to 5-year Survival in Adult Malignancies. Clinical Oncology, 2004;16: 549-60. *Department of Radiation Oncology, Northern Sydney Cancer Centre, Royal North Shore Hospital, Sydney, NSW; Department of Medical Oncology, St Vincent’s Hospital, Sydney, NSW; Collaboration for Cancer Outcomes Research and Evaluation, Liverpool Health Service, Sydney, NSW, Australia  

Abstract Aims: The debate on the funding and availability of cytotoxic drugs raises questions about the contribution of curative or adjuvant cytotoxic chemotherapy to survival in adult cancer patients. Materials and methods: We undertook a literature search for randomised clinical trials reporting a 5-year survival benefit attributable solely to cytotoxic chemotherapy in adult malignancies. The total number of newly diagnosed cancer patients for 22 major adult malignancies was determined from cancer registry data in Australia and from the Surveillance Epidemiology and End Results data in the USA for 1998. For each malignancy, the absolute number to benefit was the product of (a) the total number of persons with that malignancy; (b) the proportion or subgroup(s) of that malignancy showing a benefit; and (c) the percentage increase in 5-year survival due solely to cytotoxic chemotherapy. The overall contribution was the sum total of the absolute numbers showing a 5-year survival benefit expressed as a percentage of the total number for the 22 malignancies. Results: The overall contribution of curative and adjuvant cytotoxic chemotherapy to 5-year survival in adults was estimated to be 2.3% in Australia and 2.1% in the USA. Conclusion: As the 5-year relative survival rate for cancer in Australia is now over 60%, it is clear that cytotoxic chemotherapy only makes a minor contribution to cancer survival. To justify the continued funding and availability of drugs used in cytotoxic chemotherapy, a rigorous evaluation of the cost-effectiveness and impact on quality of life is urgently required. doi:10.1016/j.clon.2004.06.007

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Lethal Iatrogenic Diseases from Radiation Therapy Women over fifty-five years of age dramatically increase their risk-to-benefits when

electing for radiation therapy in terms of reducing their chances of a local recurrence and then face increased risks of radiation-induced cardiovascular complications, as well as other cancers of the breast, esophagus, lung, and stomach and leukemia .

NCI’s SEER data, researchers showed a 1,600% increased risk of angiosarcoma of the breast and chest wall following irradiation for primary breast cancer.

Women younger than forty-five receiving higher radiation exposures for postlumpectomy radiotherapy (as compared to post-mastectomy radiation) had a 150% increase in subsequent contralateral breast cancers. This was especially true for younger women reporting they have a significant family history of breast cancer .

See: http://www.breastcancerfund.org/assets/pdfs/publications/state-of-the-evidence-2010.pdf p. 64.Veronesi U, Luini A, Del Vecchio M, et al. Radiotherapy after breast-preserving surgery in women with localized cancer of the breast. New England J Med, 1993;328:1587-91.EBCTCG: Early Breast Cancer Trialists’ Collaborative Group. Favorable and unfavorable effects on long-term survival of radiotherapy for early breast cancer: An overview of the randomized trials. Lancet, 2000;355:1757-1770.Mellemkjaer L, Friis S, Olsen JH, et al. Risk of second cancer among women with breast cancer. Int J Cancer, 2006;118:2285-92. Roychoudhuri R, Evans H, Robinson D, et al. Radiation-induced malignancies following radiotherapy for breast cancer. Br J Cancer, 2004;91:868-72.Huang J, Mackillop WJ. Increased risk of soft tissue sarcoma after radiotherapy in women with breast carcinoma. 2001;Cancer, 92:532-6.Hooning M, Aleman B, Hauptmann M, et al. Roles of radiotherapy and chemotherapy in the development of contralateral breast cancer. J Clin Oncol, 2007;10.1200/JCO.2007.16.0192.

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Lethal Iatrogenic Injuries from Conventional Immunotherapy Due to the toxicity of chemotherapy and radiation therapy,

oftentimes desperate methods are called in (at great expense and pain) in an attempt to restore immune function before the patient dies from opportunistic infectious disease. Commonly, this requires that the patient undergoes bone marrow transplantation (BMT). However, too frequently and unacceptably BMT brings about rapid death as well.

Specifically, such fatalities from BMT account for up to 26% of the overall number of patients dying from the treatments themselves (and not the cancer).

Gratwohl A, et al. Cause of death after allogeneic haematopoietic stem cell transplantation (HSCT) in early leukaemias: an EBMT analysis of lethal infectious complications and changes over calendar time. Bone Marrow Transplant. 2005 Nov;36(9):757-69.

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Conventional Survival Statistics“…between 1950 and 2000 twice as many Americans died of cancers with increasing rather than with decreasing mortality rates…To address these issues, we will examine survival gains for the five most lethal cancers in the United States (Lung, Colorectal, Breast, Prostate, and Pancreas) that account for 57% of all cancer deaths in 2000. Five-year survival gains between 1950-54 and 1992-99 ranged from 1.5 -fold improvement for breast cancer to 4.4-fold in 5-year survival in pancreatic cancer. However, a 4.4-fold improvement in 5-year survival in pancreatic cancer is meaningless as only approximately 4% of patients reached that landmark and the average survival for this cancer remains unchanged (3-4 months) since 1950. Likewise, lung cancer patients on the average live 7-9 months from diagnosis today despite a reported 2.5-fold improvement in 5-year survival since 1950-54. Moreover, while improvements in 5-year survival are frequently presented to the public and to policymakers as evidence of success in the War On Cancer they should not be… Improvements in supportive medical care and better screening and diagnostic tools…enable detection of more cases in curable and non-curable early stages of the disease. Because their cancer was diagnosed earlier in its course, these patients will survive longer (called lead-time bias) than individuals with more advanced diseases diagnosed in the past independent of treatment.”

Faguet GB. The War On Cancer: An Anatomy of Failure, A Blue Print for the Future. Springer, Dordrecht, The Netherlands, 2005; pp. 14-16.

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Eclectic Oncology, above all else, protects and reconstitutes the patient’s internal turf.The no-spin truth is, cancer can never be 'cured' by further mucking up its turf. Mucking up cancer's turf simply facilitates the growth of more cancer with

a vengeance. In essence as far as the turf (milieu) to cancer is concerned, it self-sustains itself in such a way as to blur any clear cut barriers between normal cellular biology, cellular pathology and microbe pathology. The pro-inflammatory agents plus the highly toxic free-radical peroxynitrite facilitate this self-sustaining muck. Put another way, cancer cells often act as though they were chameleons in such degenerated milieus, as do many microbes.

This feat of both human cells and microbes acting as chameleons is known as pleomorphism, meaning cells of many shapes which love to "morph" into ever changing stages of differentiation.

Cope FW. Pathology of structured water and associated cations in cells (the tissue damage syndrome) and its medical treatment. Physiol Chem Phys 1977;9(6):547-53. Gerson M. The cure of advanced cancer by diet therapy: a summary of 30 years of clinical experience. Physiol Chem Phys 1978;10(5):449-64.Ling GN. Life at the Cell and Below - Cell Level: A Hidden History of a Fundamental Revolution in Biology. Pacific Press, NY. 2001; especially pages 26-7.. Flavin, DF. Clinical-Patient Studies: A lipoxygenase inhibitor in breast cancer brain metastases. Journal of Neuro-Oncology. 2007 Mar;82(1):91-3.

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Eclectic Oncology has a better track record, and causes no Iatrogenic death, especially for Advanced Cancers (Stages III to IV). Analysis of 57% of cases seen at The Bio-Medical Center shows advanced cancer

cases achieving a 11.4% success rate for 5-year survival.

Analysis of Stage III Melanoma patients seen at The Gerson Hospital (CHIPSA) achieved 82% success rate for 5-year survival.

Analysis of cases seen by Dr. William Donald Kelly, who complied 100% to his protocols, shows advanced Pancreatic cancer cases achieving a Median Survival of 9 years.

Of 160 advanced cancer patients following Dr. Kelly’s protocols with confirmed pathology reports, 50 were selected for analysis covering 25 kinds of cancer. Most had been treated first with conventional treatments which had failed. Average survival among all 50 patients complying with Dr. Kelly’s protocols was 10.4 years.

For Stage IIIb cancers and below, providing there is no bone cancer present and arterial delivery of sodium bicarbonate is properly administered directly into the tumor(s), upwards of 90% survival exceeding 5 years is readily obtainable.

Richardson MA, et al. Assessment of outcomes at alternative medicine cancer clinics: a feasiblity study. J Altern Complement Med. 2001 Feb;7(10:19-32.Hildenbrand GL, et al. Five-year survival rates of melanoma patients treated by diet therapy after the manner of Gerson: a retrospective review. Altern Ther Health Med. 1995 Sep;1(4):29-37.Gonzalez NJ. One Man Alone: An Investigation of Nutrition, Cancer and William Donald Kelly. New Springs Press, 2010, NY, NY; pp. 54-6.Simoncini T. Cancer is a Fungus: A Revolution in Tumor Therapy. Edizioni Lampis. Casale Marittimo, Italy, 2005, pp.163-205.

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Eclectic Oncology seeks first and foremost to strengthen the patient's constitutional state with The Regeneration Effect. This gleans an unprecedented advantage over the cancerous state. For example, inducing unscheduled fibroblast repair while insuring for homeostasis and epigenetic integrity fosters tissue regeneration which punitively impacts the formation of cancer or its continued survival. Then, by coercing the tumor cells to readily assimilate foods impregnated to choke & suffocate both the invasive placenta as well as the rogue embryo, it gleans its second major advantage over the cancerous state. The coercing strategy would necessarily involve treatment protocols sensitive to chronobiology.  Thirdly, Eclectic Oncology selects dietary items and nutraceuticals that restore immunity in a properly paced sequential manner over sufficient time to regenerate tissues (12 to 24 months in all).  Fourthly, all inflammatory pathways are quenched at not only their source, but also at their locations by upregulating targeted detox elimination channel(s). This finishing touch enables the patient's own immune system to complete the job. Finally, Mind-over-Body techniques are employed to prevent recurrence.

Carrel A. Tissue Culture and Cell Physiology. Physiol Rev 1924;4:1-17.Bryant S. Regeneration and pattern formation - an interview with Susan Bryant. Interviewed by Richardson MK, Cheng-Ming C. Int J Dev Biol. 2009;53(5-6):827-33.Eismann EA, Lush E, Sephton SE. Circadian effects in cancer-relevant psychoneuroendocrine and immune pathways. Psychoneuroendocrinology. 2010 Aug;35(7):963-76. Cope FW. Pathology of structured water and associated cations in cells (the tissue damage syndrome) and its medical treatment. Physiol Chem Phys 1977;9(6):547-53. Flavin, DF. Clinical-Patient Studies: A lipoxygenase inhibitor in breast cancer brain metastases. Journal of Neuro-Oncology. 2007 Mar;82(1):91-3. See: http://www.authorstream.com/Presentation/aSGuest9984-135251-postive-mental-attitude-critical-fo-optimal-health-spiritual-inspirational-ppt-powerpoint/

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The Regeneration Effect is the underlying determinant or authority over our self-healing capacity.

The Regeneration Effect is the driving force that can repair perfectly at near quantum speeds as is known to occur in healthy 25 year olds.

All disease and aging may be defined as the ability or inability to maintain this driving force.

For example, a healthy 25 year old is known to undergo over 100,000 genetic mutations or alterations daily in each of their 50 to 75 trillion human cells.

That equals approximately 7,500,000,000,000,000,000 mutations or alterations daily, just in the main gene pool alone.

Yet, each and every 24 hours, all of these mutations or alterations are perfectly repaired.

This rate of repair equals 868,055,555,555,556 corrections each and every second 24/7!

What is The Regeneration Effect?

© 2010 by John W. Apsley, II, MD(E), DC – www.doctorapsley.com

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* See: http://davelafferty.com/

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Under strategies utilized by Eclectic Oncology, all-natural tools are employed which may excite this innate wisdom of the body to induce lethal autophagy in such rogue placental cells, with little harm to normal healthy cells. 

Next, strategies utilized by Eclectic Oncology induce normal healthy apoptosis to become fully reawakened, thereby shutting down the rogue embryo.

Eclectic Oncology then goes one step further helping to insure these two engines never operate again. This is done in many cases by turning up cancer's very own system of necrosis using all-natural means. Typically, necrosis only occurs at the outside layers of quickly growing tumors, and does nothing to harm the most dangerous inner tumor layers. However, there are strategies that "induce" and/or "amplify" necrosis (called necroptosis) throughout the tumor site.

Nguyen TM, et al. Endostatin induces autophagy in endothelial cells by modulating Beclin 1 and beta-catenin levels. Cell Mol Med. 2009 Sep;13(9B):3687-98. Tomasin R, Cintra Gomes-Marcondes MC. Oral administration of Aloe vera and honey reduces walker tumour growth by decreasing cell proliferation and increasing apoptosis in tumour tissue. Phytother Res. 2010 Sep 13.Galluzzil L, Kroemer G. Necroptosis: A specialized pathway of programmed necrosis. Cell, 2008 Dec 26; 135:1161-3.

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Those highly skilled in the art & science of treating cancer by Eclectic Oncology do not have difficulty dissolving tumors. The keys to proper tumor resolution are: To keep the process at the perfect pace, and… To prevent cancer from ever returning.

This is accomplished via the 12 Step Protocols each with a duration of one month, with each Step divided into two equal parts “A” & “B,”

Then enhanced by part “C” designed to first initiate, then gradually optimize The Regeneration Effect within.

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Nature has provided solutions for cancer utilized by Eclectic Oncology that are effectively divided into three groups. Group “A” induce Apoptosis in animal and human rogue

tissues, such as Green Tea

Group “B” induce Lethal Autophagy in animal and human rogue tissues, such as Hops

Groups “C” is internal, and maintains healthy Necroptosis. This effort is led by sodium bicarbonate, pancreatic enzymes and mega-Vitamin C + Vitamin K.

Meeran SM, et al. A Novel Prodrug of Epigallocatechin-3-gallate: Differential Epigenetic hTERT Repression in Human Breast Cancer Cells. Cancer Prev Res (Phila). 2011 Mar 16. [Epub ahead of print]Delmulle L, et al. Treatment of PC-3 and DU145 prostate cancer cells by prenylflavonoids from hop (Humulus lupulus L.) induces a caspase-independent form of cell death. Phytother Res. 2008 Feb;22(2):197-203.Gonzalez NJ, Isaacs LL. Trophoblast and the Origins of Cancer: One solution to the medical enigma of our time. New Spring Press (2009). ISBN-13: 978-0982196502.Gilloteaux J, et al. Cell damage and death by autoschizis in human bladder (RT4) carcinoma cells resulting from treatment with ascorbate and menadione. Ultrastruct Pathol. 2010 May;34(3):140-60.

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