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Eyes and Ears
Special Senses
Eyes
Review the function and structure of the eye.
A & P - External Eye
Lacrimal Apparatus
6 Extraocular Muscles
A & P - Internal Eye
Internal StructureOuter Layer
transparent cornea covers the iris sclera - protects and site for attachment of extraocular muscles
Middle Layer pupil - SNS =dilates. PNS= constricts, vitreous humor maintains shape,
Inner Layer retina- extension of optic nerve, receives light impulses to be transmitted to
occipital lobe Optic disc: vessels converge at center, yellow-orange in colour, round or oval,
physiologic cup within the disc (smaller lighter circle). Retinal vessels: paired artery &vein Fovea centralis -Macula: slightly darker pigment at center has the sharpest and
keenest vision due to cones and rods.
Visual Pathways
EYE
Health History
Subjective information
Health History
AgeInfant/Child
Peripheral vision intact in newborn Most neonates (80%) are born farsighted (gradually
decreases after 7 to 8 years) Macula (area of keenest vision) begins development by
4 months and is mature by 8 months. Decreased eye cordination at birth, eye movement
sometime sluggish to eyes
Health HistoryAge
Middle agePresbyopia(difficulty with near vision)Hypertensive RetinopathyVisual acuity diminished gradually after 50 yrs
(continues)
Health History
Age (cont’d) Elderly
Develop cataracts (lens opacity), glaucoma (increased ocular pressure) and macular degeneration (loss of central vision)Entropion, ectropionExternal eye changes: wrinkling and droopingDry eyes- Lacrimal glands involute: decreased
tear production, dryness and burning
(continues)
Health History
Gender- Female - dry eyesRace- Glaucoma (African Americans), melanona of
eye (Caucasians)Common chief complaints
Changes in visual acuity Pain Drainage Itching Dryness
(continues)
Health History
Characteristics of chief complaints Location Quality Associated manifestations Aggravating and alleviating factors Setting Timing
Past Health History
Medical: eye-specificSurgical: eye-specificMedicationsAllergiesInjuries and accidentsSpecial needsChildhood illnesses
(continues)
Past Health History
FamilySocial
Work environment
Health maintenance activities Diet Use of safety devices Health check-ups
Assessment of the EyeEquipment
Ophthalmoscope Penlight Vision charts Vision occluder
General approach Lighting Environment
Preparation for Physical Exam of the Eye
Position the client sitting up with the head at your eye level
Use orderly approach moving from the extraocular structures to the intraocular structure(1-6 p 348)
Visual Acuity
Assessment of Cranial nerve IIDistance vision - Use Snellen Chart N= 20/20
Myopia (nearsightedness) Amblyopia (loss of visual acuity due to uncorrected
strabismus= crossed eyed or DM,alcoholism, uremia)
Near vision - use Rosebaum or snellen card N = reading is possible at 14 inches until late 30-40s Hyperopia (farsightedness)
Color vision N = can id primary colors on snellen chart
Visual Fields
Confrontation technique
Assess all fields N= pt covers 1 eye,
use own visual field as control, can see stimulus at 90°temporally, 60°nasally, 50°superiority, 70°inferiority
Visual Fields
Types of defects (p352 image)
Hemianopsia Circumferential
blindness Unilateral blindness
External Eye and Lacrimal Apparatus
Eyelids- inspect N = symmetrical eyelids, can raise eyelids
symmetrically (CN 3) Abnormal findings
Ptosis (drooping of lid) Exophthalmos (protrusion of eyes out of orbit) Entropion(turning inward or inversion of lower lid) ectropion (outward turning of lower eyelid) hordeolum (acute localized inflammation- internal =
conjunctival side of lid, & external =sty)
External Eye and Lacrimal Apparatus
Lacrimal apparatus- inspect, palpate N = no enlgmt, no swelling or no redness, no
exudate and minimal tearing. No dischrg from punctum apon palpation.
Abnormal findings dacryoadenitis (acute inflammation of lacrimal gland) dacryocystitis - obstruction of lacrimal duct dt
inflammation
Extraocular Muscle Function CN III, IV, VI
Corneal light reflex (Hirschberg test)Cover/uncover testCardinal fields of gaze
Extraocular Muscle Function CN III, IV, VI
Corneal light reflex (Hirschberg test)
N= light reflex seen symmetrically in center of each eye Abnormal findings
due extraocular muscle weakness
• extropia (outward turning of eye
• esotropia (inward turning or eye)
Extraocular Muscle Function CN III, IV, VI
Cover/uncover test N= eyes are aligned,
no movement of either eye
Abnormal findings phoria =latent
misalignment of eye exists
Extraocular Muscle Function CN III, IV, VI
6 Cardinal fields of gaze N=both eyes move smoothly
and symmetrically in 6 fields of gaze & converge on the object as it converges on then nose
note : nystagmus= involuntary movement
Abnormal findings deviations from N
Anterior Segment StructuresConjunctiva- inspect
N= transparent, sm bld vessels, white Abnormal findings
conjunctiva, edema, lesions, foreign bodies,
Sclera -inspect N= white , sm bld vessels Abnormal findings
jaundice, blue (osteogenesis imperfecta-thinning of sclera)
Cornea- inspect with penlight N= corneal surface is moist, shiny Abnormal findings
presence of discharge, cloudiness, opacities, irregularities
Anterior Chamber
Iris Inspect the iris for color, nodules, vascularity
N=even color or mosaic, smooth no vascularity Shine light obliquely through the anterior chamber
from lateral side towards nasal chamber N=the entire iris will be illuminated Abnormal findings
hyphema (bleeding into iris dt trama)
Anterior Chamber
Pupil (CN III) darken room & note size and shape of pupil,
move penlight from side to front of eye, observe pupillary reaction
N=PERRLA N= direct light flex (pupil constrict with light) N= Consensual light reflex (move penlight in
front of one eye and observe other eye for pupillary constriction)
N = Accommodation (pupils constrict as converge onto closer object)
Abnormal findings anisocoria - sm diff in pupil size oculomotor nerve damage- a fixed and
dilated pupil is seen (see pg 363)
Anterior Chamber
Lens shine penlight directly into
pupil , note color of lens
N= transparent in color
Abnormal findings cataract- cloudiness or
opacity in the lens
Posterior Segment Structures
Assessment techniques
use of ophthalmoscope
Posterior Segment Structures
Retinal structures instruct pt to look at distant object use ophthalmoscope, shine into each eye
N= Red Reflex present (pupil appears red through ophthalmoscope)
N= observe intact optic disc (on nasal side of retina by following any retina vessel centrally)
Abnormal findings absent red reflex - dt cataract
Posterior Segment Structures
Macula move ophthalmoscope
towards ear (temporal lobe) and observe for black circle around fovea.
N= macula is darker, avascular area with a pinpoint reflective center known as the fovea centralis
Gerontological Variations
Changes in visual acuityPresbyopiaCataractsMacular degenerationGlaucoma
Review of Normal Findings
Visual acuity 20/20Near vision acuity at 14 inchesAble to identify all six colorsVisual fields intact
(continues)
Review of Normal Findings
Eyelids symmetrical; no drooping, infections, or tumors
No enlargement, swelling, or redness of the lacrimal apparatus
Light reflex is symmetrical in the center of each cornea
(continues)
Review of Normal Findings
Eyes aligned on cover/uncover testExtraocular eye movements intact in all
six fieldsBulbar conjunctiva is transparentPalpebral conjunctiva is pink and moist
(continues)
Review of Normal Findings
Sclera are white, without exudate, lesions, or foreign bodies
Cornea is moist, shiny, without discharge, cloudiness, or opacities
Entire iris is illuminatedColor of iris is evenly distributed
(continues)
Review of Normal Findings
Pupils are deep, black, round, and of equal diameter
Pupil size is 2–6 mmLens is transparentRed reflex is presentOptic disc is pinkish orangeMacula is darker, avascular
Ears
Review the function and structure of the Ear
(continues)
Anatomy and Physiology of the Ear
Three sections External ear Middle ear Inner ear
External ear Auricle or pinna
External, Middle, Inner Ear Structure
External Ear Structure
Middle Sar Structures
Air filled cavity Tympanic membrane Ossicles ( 3 tiny bones - malleus,
incus, stapes)
2 muscles involved in movement of ossicles- tensor tympani- pulls inward, stapedius - pulls outwards
Eustachian tube - connected to nasopharynx by the auditory canal (relieves air pressure within the middle cavity) see next slide
Inner Ear Structures
Controls hearing and equilibrium/balance closed fluid-filled system of
interconnecting tubes called the Labyrinth cochlea (snail shape structure containing
perilymph & endolymph which vibrate and stimulate vestibulocochlear nerve CNVIII)
semicircular canals (provide balance and equilibrium for the body)
vestibule (btwn cochlea & semicircular canals)
Frequency range of 20–20,000 Hz Decibel range 0–140
Pathways of Hearing
1. Air Conduction (AC): most efficient.
AC>BC
2. Bone Conduction (BC)
3. See OH
Ears
Health History
Subjective Data
Ears-Subjective Data**Note the following**
EaracheInfectionsDischarge (otorrhea)Hearing lossEnvironmental noise TinnitusVertigo Self care behaviours
What further information would you gather if the client is an infant and children?
Ear infections (how many, 1st one?)
Parent = 1. Does the child seem to have hearing loss?
2. Does the child put objects in the ears?
Ears – Physical Examination
Objective Data
Equipment
OtoscopeTuning fork
(continues)
Assessment of the Ear
Consists of three parts Auditory screening(CN VIII) Inspection and palpation of external ear Otoscopic assessment
Auditory Screening
Voice-whisper test instruct pt to occlude 1 ear with finger stand 2 feet behind the other ear and whisper ask pt to repeat whispered words
N= able to repeat words whispered at a distance of 2 feet
(continues)
Auditory Screening- Tuning fork tests
Weber test N= able to hear sound
equally in both ears
Abnormality Determines whether
hearing loss is conductive or sensorineural
Auditory Screening- Tuning fork tests
Rinne test Normal finding: air
conduction > bone conduction
Abnormality Determines whether
hearing loss is conductive or sensorineural
Hearing Loss
1. Central deafness: occurs with pathologic conditions above the junction of the acoustic nerve and the brain stem. E.G. brain tumor, vascular changes which deprive the inner ear of blood supply, CVA.
2. Conduction deafness: mechanical dysfunction of the external or middle ear. Partial loss (must increase amplitude). E.G. impacted cerumen, foreign bodies, perforated tympanic membrane, pus in middle ear.
3. Sensorineural deafness: pathology of the inner ear, CN VIII or auditory areas of the cerebral cortex. E.G. presbycusis (gradual nerve deterioration), ototoxic drugs (affect the hair cells in the cochlea)
External Ear
Inspection & Palpate Note position, size, color, and shape
N= flesh color, top of ear = to outer canthus of eye, cerumen is moist & does not obscure the tympanic membrane, no foreign bodies, redness, drainage, deformities, nodules, or lesions
Abnormal findings Pale, red, cyanotic Small-size or large-size ears Purulent drainage Clear or bloody drainage Hematoma behind ear over mastoid Pain or tenderness on palpation
(continues)
Otoscopic Assessment Inspect both external ear canal using
otoscope N= No redness, swelling, tenderness, lesions, drainage,
foreign bodies Tympanic membrane is pearly gray with well-defined landmarks Light reflex present at 5 o’clock in right ear and 7 o’clock in left
ear Tympanic membrane moves when patient blows against
resistance
Abnormal findings Chalky patches on tympanic membrane Severe pain Redness, swelling, narrowing, pain Drainage Hard, dry, very dark yellow cerumen Reddened tympanic membrane
Ear Abnormalities
Acute Otitis Media tympanic membrane is red
with decreased motility,and possible bulging due inflammation of middle ear
Chronic & Acute Otitis Externa redness, swelling, narrowing
and pain of external ear, drainage present due to inflammation of external ear
Risk Factors for Otitis Media
Less than 2 years of age Frequent upper respiratory infections Cold weather Male gender Caucasians, Native Americans, Alaska natives Family history Smoky environment Bottle fed Down syndrome
Ear Abnormalities
Tympanic Membrane Perforation due to untreated ear
infection secondary to increasing pressure or trama to the ear canal.
Equilibrium Abnormalities
Labyrinth becomes inflammed and sends the wrong information to the brain.
Which develops into what we call…..
Vertigo: staggering gait, strong spinning, whirling sensation.
Developmental Considerations
Infants/Children Rubella in 1st trimester can damage the organ of
Corti and impair hearing Eustachian tube is shorter and wider, position is
more horizontal than the adult’s Greater risk for ear infection External auditory canal is shorter and sloped is
opposite to the adult’s
Developmental Considerations
Aging Adult Cilia becomes coarse and stiff Cerumen is dryer and impaction is a common
reversible cause of hearing loss. Presbycusis occurs with aging “50s” (nerve
degeneration in the inner ear or auditory nerve) “70s” takes longer to process sensory input and
to respond to it.
Transcultural Considerations
Otitis Media (OM) incidence and severity increased in Native Americans, Alaskan and Canadian Inuits & Hispanics.
Also increased in premature infants and those with Down Syndrome, and bottle fed babies in supine position.
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